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1 e time course of pleasure, across stimuli, is well-fit by a model with one free parameter: pleasure amplitude.
2 coupled an optimality-based plant nitrogen (N) acquisition model with a microbe-focused soil organic matter (SOM) model.
3 Further, we couple the calibrated platelet aggregation model with a tissue-factor/contact pathway coagulation cascad
4 our novel profilin1 mutant mouse line may provide a new ALS model with the opportunity to gain unique perspectives into m
6 cial isolation has been established as a widely used animal model with translational relevance for neurodevelopmental psy
7 We base our method on an arbitrary-order Markov chain model with community structure, and develop a nonparametric B
9 ta were analyzed using the validated two-tissue compartment model with arterial plasma input function with total volume o
11 ter the beginning of hypercapnia and a 1-tissue-compartment model with flow-dependent extraction correction.
14 By combining a multispecies Gompertz model with a Bayesian state-space framework, we quantify comm
16 llow the energy-momentum dependence calculated by a Hubbard model with suitable finite-range interactions.
17 rt GFP into the rigid, ligand-binding head of the integrin, model with Rosetta the orientation of GFP and its transition
18 cope with uncertainty, we extended a reinforcement learning model with a belief state about the perceptually ambiguous st
19 gion-specific means were compared by using a general linear model with false discovery rate control for multiple comparis
20 On the basis of comparison of this MDFF model with an earlier-derived crystallographic model for the
22 ngth of combining QTL mapping and RNA-Seq data from a mouse model with association studies in human case-control samples
23 We used whole-genome bisulfite sequencing in a mouse model with nonrandom XCI to examine allele-specific DNA methy
24 docytes in the treatment of proteinuria, we created a mouse model with podocyte-specific deletion of the glucocorticoid r
25 , in megakaryocytes (MKs) and platelets, we created a mouse model with Vps34 deletion in the MK/platelet lineage (Pf4-Cre
26 role in disease pathogenesis, we generated a knock-in mouse model with NB disruption mediated by 2 point mutations (C62A/
27 Here, the authors generate a novel mouse model with titratable expression of DUX4, and show that it re
28 Our studies utilized the well-characterized Pax9(-/-) mouse model with a consistent cleft palate phenotype to test small-
29 als in mammalian tibial nerve axons using an in vitro mouse model with a dextran-conjugated fluorescent calcium indicator
30 in the native membranes, and is described by a multisquare model with Schiff base deprotonation at the lumirhodopsin I i
32 Mathematically integrating the mutation model with background selection model gives a more complete d
33 ASSO (wgLASSO) algorithm to integrate a data-driven network model with prior biological knowledge (i.e., protein-protein
34 We assess an improved trait-driven carbon optimality model with in situLL data for 105 species in two Panamanian f
37 canines with open (n=3) and closed chest (n=5) and in a pig model with features of human heart failure and preserved ejec
39 ION: We developed and validated a novel clinical prediction model with good discriminative performance to identify patien
40 and resuscitation-induced microvascular leakage using a rat model with intravital microscopic imaging.
42 sensitive ex vivo detection of endogenous CO in a realistic model with facile, inexpensive synthesis, and displays many a
43 We constructed a mixed-effects linear regression model with the individual physician as the random effect in t
44 ing the output of a well-established computational saliency model with the activation of neurons in the primate superior
45 Hence, we develop a flexible Bayesian variable selection model with efficient computational techniques for such integr
47 indings were used for the creation of a multistate survival model with 3 states (entry, EEG risk, and seizure).
48 However, above a local noise level of 20%, the model with nonspecific plasticity outperforms the standard, s
50 ameters of breast cancer were calculated by using the Tofts model with T1 values before and after B1 correction.
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