ライフサイエンスコーパス: molecular layer

1 a network of inhibitory interneurons in the molecular layer.

2 ayer between the top metal electrode and the molecular layer.

3 at least one recurrent mossy fiber into the molecular layer.

4 Cs extended to the outer edge of the dentate molecular layer.

5 in the Purkinje cells and other cells of the molecular layer.

6 the metal atoms penetrating or damaging the molecular layer.

7 e cell and Purkinje cell layers, but not the molecular layer.

8 cted following electrical stimulation in the molecular layer.

9 mossy fibers) in the granule cell layer and molecular layer.

10 hat terminate in the outer two-thirds of the molecular layer.

11 lized foci on the superficial surface of the molecular layer.

12 er, terminates in the inner one-third of the molecular layer.

13 directly affected by orbital energies of the molecular layer.

14 ure granule cells and formed synapses in the molecular layer.

15 By 48 h the dendrites first appeared in the molecular layer.

16 le cell layer but only Kv4.3 subunits in the molecular layer.

17 ausing their dendrites to extend through the molecular layer.

18 d branching dendrites that extended into the molecular layer.

19 hibitory HIPPs distribute axon fibers in the molecular layer.

20 rkinje cells are misplaced in the cerebellar molecular layer.

21 they had developing synapses on them in the molecular layer.

22 ociated with decreased NeuN densities in the molecular layer.

23 layer preceding that of GABA neurons in the molecular layer.

24 f the electron energy after it traverses the molecular layer.

25 thin middle-to-outer portions of the dentate molecular layer.

26 in the granule cell layer and Veli-3 in the molecular layer.

27 the sulcus do not innervate the superficial molecular layer.

28 n the PCL and the extracellular space of the molecular layer.

29 e number of migrating granule neurons in the molecular layer.

30 local synapses and vasculature in the inner molecular layer.

31 ell dendrites invading a given volume in the molecular layer.

32 pines in different dendritic segments in the molecular layer.

33 in revealed immunostaining of the cerebellar molecular layer.

34 elrhodopsin or electrical stimulation in the molecular layer.

35 fibers and boutons extended into the dentate molecular layer.

36 ctrode was directly evaporated on top of the molecular layer.

37 eep layers and parallel fiber input in their molecular layers.

38 und along the direction perpendicular to the molecular layers.

39 e within a single molecular layer or between molecular layers.

40 ns permit the growth of an integer number of molecular layers.

41 terfaces between metallic thin films and C60 molecular layers.

42 ound along dendrites in the granule cell and molecular layers.

43 In the hilus and molecular layer, 4% of VAChT-ir terminals contacted dend

44 ell nanoparticles with two components in the molecular layer, a framework molecule to form the shell,

45 At photon energies where the molecular layer absorbs, a secondary phenomenon is opera

46 genitors migrate aberrantly to the hilus and molecular layer after prolonged seizures and differentia

47 , involving fiber sprouting into the dentate molecular layer and a parallel fiber retraction from the

48 spinous terminals in the dentate gyrus inner molecular layer and CA1 stratum radiatum and in (ii) pos

49 ive loss of synaptophysin in the hippocampal molecular layer and frontal neocortex of a transgenic mo

50 mma-7 is enriched in Purkinje neurons in the molecular layer and glomerular synapses in the granule c

51 the hippocampal CA1 region and dentate inner molecular layer and hilus.

52 CD200/CD200R interaction seem to include the molecular layer and pial surface in neonates and blood v

53 other types of neurons sprout into the inner molecular layer and synapse with granule cell dendrites

54 including reductions in the thickness of the molecular layer and the internal granule layer, as well

55 -Mott limit, and the interaction between the molecular layer and the substrate acts to influence the

56 ing from partial charge transfer between the molecular layer and the substrate.

57 rmed dense axonal arborizations in the inner molecular layer and throughout the hilus.

58 s on the affected side were increased in the molecular layer and upper cortex (+244 to +18%), decreas

59 e cortical shrinkage, atrophy of hippocampal molecular layers and corpus callosum, and enlargement of

60 cell dendrites (which are restricted to the molecular layer) and on interneuron dendrites in the hil

61 icantly larger dendritic arborization in the molecular layer, and a more triangular cell body than gr

62 nd dendrites, sprout axon collaterals in the molecular layer, and form new synapses with granule cell

63 pyramidale neuropil and dentate gyrus inner molecular layer, and increases in the strata oriens and

64 ice also had larger somata, more axon in the molecular layer, and longer dendrites than controls.

65 ry glomeruli, hippocampal CA3 and cerebellar molecular layer, and moderate binding in the cerebral co

66 fibres extending to the outer portion of the molecular layer, and more climbing fibre-Purkinje cell s

67 actory glomeruli, CA3-hilus area, cerebellar molecular layer, and pars reticulata of the substantia n

68 tiple potassium sites within initially dense molecular layers, and thus interact with the PAH anion p

69 l connectivity of the GABAergic cells in the molecular layer are not well understood.

70 Such molecular layers are at the quantum limit of device mini

71 Molecular layers are prepared by allowing sessile drops

72 Analysis of the molecular layer as a function of coverage reveals an unp

73 Granule cell axons enter the molecular layer as parallel fibers without bifurcating.

74 f the Ca(2+) indicator Fluo-4 throughout the molecular layer as well as the granule cells.

75 luation of thickness changes in the adsorbed molecular layers as a result of chaperone binding to rec

76 l layer potentiated responses throughout the molecular layer, as well as in the CA3 region.

77 This study confirms that thinning of the molecular layer associated with disease pathogenesis is

78 Films are constructed one molecular layer at a time by using the layer-by-layer, d

79 sistive switching are strongly affected by a molecular layer at the graphene/BaTiO3 interface.

80 Molecular layering at the lipid-nanotube interface is re

81 radial responses did not activate measurable molecular layer beams in transverse slices.

82 In the molecular layer, both isoforms were at highest levels wi

83 rials that consist of one or a few atomic or molecular layers, bottom-up assembly of 2D crystalline m

84 and large central cells are confined to the molecular layer but are not planar.

85 rth, with a primary dendrite pointing to the molecular layer, but at this stage, with several neurite

86 in the inner and outer zones of the dentate molecular layer, but it was reduced in the dentate hilus

87 nt path potentiated responses throughout the molecular layer, but left responses in the CA3 region un

88 dendrites of Golgi cells are confined to the molecular layer, but their axon arbors are either confin

89 ions studied (dentate gyrus granule cell and molecular layers, CA2/3-hilus, and CA1), and across the

90 inous GABAergic synapse numbers in the outer molecular layer changed most.

91 ices were restricted to sublayers within the molecular layer, conducting slowly away from the stimulu

92 irection of rectification is reversed if the molecular layer consists of naphtalene diimides having l

93 ils, based mainly on solid-state NMR data, a molecular layer consists of two beta-sheets (residues 12

94 colloidal synthesis, in which self-assembled molecular layers control the alignment between materials

95 MeHg-induced increase in fluorescence in the molecular layer correlated with that of increased sEPSC

96 The molecular-layer deposition of a flexible coating onto Si

97 ells that were ectopically positioned in the molecular layer displayed a more rapid functional matura

98 Distances shorter than four molecular layers displayed solid behavior with interlaye

99 h marked uniaxial characteristics within the molecular layer due to the presence of one-dimensional p

100 nd an enlargement of the CF territory in the molecular layer during development.

101 Shear fluidization of a film of five to six molecular layers during an individual slip event should

102 asing lattice resistance to shear sliding of molecular layers during plastic deformation.

103 Molecular layer ectopia, clusters of misplaced cells in

104 lacunosum-moleculare of CA1 and the dentate molecular layer elicited GABAergic inhibitory responses

105 In cerebellar molecular layer, expression of BDNF correlated significa

106 s increased dark Purkinje cells (PC), a thin molecular layer, fewer synapses, a loss of dendritic spi

107 eased Kv1.2, Kv1.3, and Kv1.6 in the dentate molecular layer following chronic Ro25,6981 treatment.

108 l mossy fiber terminals of the dentate inner molecular layer formed frequent asymmetrical synapses wi

109 Although previous studies focused on the molecular layer, here, we shift attention onto the mossy

110 ochemical analyses on confined electroactive molecular layers, herein exemplified with electroactive

111 cribe the prevalence and cytoarchitecture of molecular-layer heterotopia in C57BL/6J mice and related

112 particular, we found that the prevalence of molecular-layer heterotopia vaired according to the sex

113 nt protein were abnormally positioned in the molecular layer, hilus, and granule cell layer.

114 naptic terminals in both the outer and inner molecular layers; however, no significant changes were s

115 The inner molecular layer (IML) displayed a corresponding volumetr

116 ic reorganization of the dentate gyrus inner molecular layer (IML) is a pathophysiological process th

117 row time window and stack their axons in the molecular layer in chronological order from deep to supe

118 noreactivity was more prominent in the inner molecular layer in prenatally choline-deficient rats com

119 tput (I/O) relationship in the dentate gyrus molecular layer in response to lateral perforant path st

120 rains of electrical stimuli delivered to the molecular layer in vitro, release adenosine via a proces

121 eficits, as measured by the thickness of the molecular layer, in all mice in which Klhl1 was deleted

122 limited to fibers located in the granule and molecular layer, in an orientation consistent with granu

123 The structures and energy levels of the molecular layers included donor molecules with relativel

124 ion of parallel fibers, migration across the molecular layer, incorporation into the internal granule

125 Gene-ontology analysis across different molecular layers indicates that although chromatin recon

126 ted staining was detected at the inner third molecular layer indicating glutamatergic sprouting.

127 immature granule cell neurons in the dentate molecular layer induced by ECS.

128 ity into a beam-like response, implying that molecular layer inhibition does not prevent beam-like re

129 Therefore, molecular layer inhibition is compartmentalized into zeb

130 Molecular layer inhibitory interneurons generate on-beam

131 a low-affinity antagonist at parallel fiber-molecular layer interneuron (PF-MLI) synapses.

132 Furthermore, CFs systematically targeted molecular layer interneuron cell bodies, especially at t

133 idence indicates electrical coupling between molecular layer interneurons (basket and stellate cells)

134 increased by release of endogenous GABA from molecular layer interneurons (MLIs) and spillover activa

135 A receptors (NMDARs) expressed by cerebellar molecular layer interneurons (MLIs) are not activated by

136 munication between climbing fibers (CFs) and molecular layer interneurons (MLIs) in the cerebellum is

137 Inhibition from molecular layer interneurons (MLIs) is thought to play a

138 known that, during movement, the activity of molecular layer interneurons (MLIs) of the cerebellar co

139 aired recordings from synaptically connected molecular layer interneurons (MLIs) of the rat cerebellu

140 Molecular layer interneurons (MLIs) provide powerful inh

141 elieved to receive GABAergic inhibition from molecular layer interneurons (MLIs).

142 (TARPs) on presynaptic AMPARs in cerebellar molecular layer interneurons (MLIs).

143 Molecular layer interneurons (MLIs, stellate and basket

144 GABA release from molecular layer interneurons activates parallel fiber GA

145 cal discoveries concerning the importance of molecular layer interneurons and their plasticity, the s

146 Because they receive input from many CFs, molecular layer interneurons are well positioned to dete

147 Molecular layer interneurons exhibit bidirectional firin

148 or (NMDAR) expression on axons of cerebellar molecular layer interneurons have produced conflicting r

149 Here we perform cell-attached recording of molecular layer interneurons in cerebellar slices from r

150 By recording from multiple molecular layer interneurons in the cerebellar cortex, w

151 racellular recordings from granule cells and molecular layer interneurons in vivo, selective pharmaco

152 ortex has led to suggestions that cerebellar molecular layer interneurons laterally inhibit Purkinje

153 ive currents obtained under voltage clamp in molecular layer interneurons of juvenile rats or mice re

154 simple synapses between parallel fibers and molecular layer interneurons of rat cerebellar slices.

155 A(A) antagonists and reflect the activity of molecular layer interneurons on their targets.

156 Feed-forward inhibition from molecular layer interneurons onto granule cells (GCs) in

157 inoid release from PCs and GABA release from molecular layer interneurons provide the primary means o

158 ithin the 480 mum lateral distance examined, molecular layer interneurons reduced the strength of gra

159 we examined how feedforward inhibition from molecular layer interneurons regulates adaptation of the

160 upting this balance by selectively silencing molecular layer interneurons results in unidirectional f

161 pal slices and identified a subpopulation of molecular layer interneurons that expressed immunocytoch

162 ion from granule cells to Purkinje cells and molecular layer interneurons will be strengthened during

163 okes inward GABAergic currents in cerebellar molecular layer interneurons with rise times of 2 ms, co

164 made up of Purkinje cells, climbing fibers, molecular layer interneurons, and cerebellar efferent ce

165 nd adults, PCs make synapses onto other PCs, molecular layer interneurons, and Lugaro cells, but not

166 nctional connectivity between granule cells, molecular layer interneurons, and Purkinje cells in rats

167 ical synapses and gap junctions with various molecular layer interneurons, including other NGFCs.

168 h prior studies of spillover transmission to molecular layer interneurons, these results reveal that

169 xcitatory granule cells (GCs) and inhibitory molecular layer interneurons-in processing of whisking s

170 parallel fiber synaptic input to downstream molecular layer interneurons.

171 lity at synapses between parallel fibers and molecular layer interneurons.

172 rites of Purkinje cells, efferent cells, and molecular layer interneurons.

173 targets, the Purkinje cells, Golgi cells and molecular layer interneurons.

174 The molecular layers investigated included donor molecules w

175 When the molecular layer is based on a molecule with a high HOMO-

176 In the cerebellum, the molecular layer is regarded as the main NVC determinant.

177 Discrete molecular layer lesions demonstrate that PFs contribute

178 characterized by progressive thinning of the molecular layer, loss of Purkinje cells and increasing m

179 The partly charged end-groups of the grafted molecular layer may act as a top gate.

180 In the molecular layer, mGluR2/3 labeling slightly declined in

181 hat NGFCs form an integral part of the local molecular layer microcircuitry generating feed-forward i

182 ges in TrkB protein expression in cerebellar molecular layer mirrored those for BDNF during the first

183 reas dentate gyrus granule cells (DGGCs) and molecular layer (ML) interneurons predominantly express

184 e from the outer to the inner regions of the molecular layer (ML) of the DG.

185 ) including a granule cell layer (GCL) and a molecular layer (ML) that continuously crosses adjacent

186 ward migration of GABA interneurons into the molecular layer (ML) were arrested, disrupting layer and

187 al cells (BGs), astrocytes of the cerebellar molecular layer (ML), express various receptors that can

188 fibers (pfs) and apical GoC dendrites in the molecular layer (ML).

189 lar colocalization numbers were found in the molecular layer of cerebellar sections.

190 piny branchlets of the Purkinje cells in the molecular layer of cerebellum and in the olfactory bulb.

191 naptic terminals of GABAergic neurons in the molecular layer of cerebellum.

192 hinal cortex, CA1/subiculum border and outer molecular layer of dentate) were initially affected, wit

193 tative DGCs also were found in the hilus and molecular layer of epileptic human dentate gyrus.

194 e coating is a covalently tethered, flexible molecular layer of perfluorocarbon, which holds a thin l

195 ining of GIRK2 was detected in the lacunosum molecular layer of the CA3 area of the hippocampus.

196 The molecular layer of the cerebellar cortex is populated by

197 alterations to the general morphology of the molecular layer of the cerebellar cortex that are the re

198 ressed in punctate structures throughout the molecular layer of the cerebellar cortex.

199 pic neurons and progenitors collected in the molecular layer of the cerebellum and adjacent tectum.

200 rpendicular deposits of prion protein in the molecular layer of the cerebellum.

201 layers I and II of the cerebral cortex, and molecular layer of the cerebellum.

202 in developing GABAergic interneurons of the molecular layer of the cerebellum.

203 VGLUT1 was highly expressed in VCNm and the molecular layer of the DCN, whereas VGLUT2 was expressed

204 ted a decrease in spine density in the outer molecular layer of the dentate gyrus (DG) beginning as e

205 hippocampus proper, and especially the inner molecular layer of the dentate gyrus (DGiml).

206 of compact beta-amyloid plaques in the outer molecular layer of the dentate gyrus and Layers II and I

207 ll decreases in synapse density in the outer molecular layer of the dentate gyrus at both 6-9 and 15-

208 ults indicate that MOPP cells located in the molecular layer of the dentate gyrus contribute to feed-

209 nificantly increased synaptic density in the molecular layer of the dentate gyrus in Tg2576 mice.

210 ifically, spines of new neurons in the outer molecular layer of the dentate gyrus were more readily i

211 it labeling at perisynaptic locations in the molecular layer of the dentate gyrus where these subunit

212 which innervates the outer two-thirds of the molecular layer of the dentate gyrus, was one of the fir

213 and amyloid deposits are often found in the molecular layer of the dentate gyrus, which is the termi

214 s. radiatum, CA2 and CA3 s. pyramidale, and molecular layer of the DG, was increased by 34.0%, 8.9%,

215 sicular glutamate transporter 1 in the outer molecular layer of the hippocampal dentate gyrus on the

216 n the activated dendritic lamina, the middle molecular layer of the hippocampal dentate gyrus.

217 density of GABAergic synapse markers in the molecular layer of the hippocampus of TS mice.

218 and ectopic neurons in the dentate hilus and molecular layer of the hippocampus.

219 extracellular space (ECS) of the anisotropic molecular layer of the isolated turtle cerebellum.

220 erminalis, but they remained confined to the molecular layer of the MEApd from E14 through P1.

221 evealed a number of immature features in the molecular layer of the mutant cerebellar cortex, includi

222 in in dendritic spines down to 40 mum in the molecular layer of the visual cortex of an anesthetized

223 Similar beams were observed in the molecular layer of thick transverse slices but not sagit

224 A single molecular layer of titanium diselenide (TiSe2) is a prom

225 3 GPa are found using Raman spectrometry for molecular layers of 1-nm in thickness.

226 intracellular distribution of Fabp7 mRNA in molecular layers of hippocampus.

227 successive hopping diffusion into successive molecular layers of molecular spintronics devices.

228 scopy reveals that these ribbons, only a few molecular layers of protein thin, rival the mechanical p

229 ilins, are found at synapses and localize to molecular layers of the adult mouse hippocampus and acce

230 f synapses per neuron in the outer and inner molecular layers of the dentate gyrus parallel those see

231 of Y and Zr exist only within the first few molecular layers of the sample.

232 synapse densities in the dentate gyrus outer molecular layer (OML) correlated with DNMS recognition a

233 ume of the somatostatin-immunopositive outer molecular layer (OML), innervated by projections from th

234 ce intensity as well as the thickness of the molecular layer on the observed photocurrent.

235 cterization of structure and interactions of molecular layers on an optical waveguide surface for a w

236 and disilenes may be available for building molecular layers on C(001).

237 mperature and then regenerated with the same molecular layer or a different one by "renewing" the int

238 epending on whether they are within a single molecular layer or between molecular layers.

239 the number of GABAergic interneurons in the molecular layer or hilus.

240 It is organized in ridges of ganglionic and molecular layers, oriented perpendicular to the granular

241 numbers of excitatory synapses in the inner molecular layer per granule cell could be calculated.

242 ge of 2,280 asymmetric synapses in the inner molecular layer per granule cell, which was reduced to 6

243 numbers of excitatory synapses in the inner molecular layer per hippocampus in pilocarpine-treated c

244 modified rabies virus, we demonstrated that molecular layer perforant pathway (MOPP) cells innervate

245 g that CB1R downregulation in the cerebellar molecular layer plays a key role in THC-induced cerebell

246 her density of dendritic spines in the inner molecular layer postlesion accompanied by an increase in

247 e signals conveyed by parallel fibers in the molecular layer predict the patterns of sensory input to

248 iod of 4.5 A for distances greater than four molecular layers, revealing liquid behavior.

249 ture the electrochemical transformation of a molecular layer self-assembled on a tapered gold microel

250 zed excitatory neuronal class in the dentate molecular layer, semilunar granule cell (SGC), has been

251 ranule cells but had axon collaterals in the molecular layer, significantly larger dendritic arboriza

252 e they require a Burgers vector spanning two molecular-layer spacings, distorting the crystal lattice

253 In the rat cerebellar molecular layer, spillover of glutamate between parallel

254 Molecular layer stimulation of the intact cerebellum evo

255 ons reliably phase-lock to regular trains of molecular layer stimulation.

256 an energy loss that is strongly dependent on molecular layer structure and thickness.

257 ity, and allow examination of the effects of molecular-layer structure, thickness and contact work fu

258 of IPSCs evoked by direct stimulation in the molecular layer, suggesting that DGCs are susceptible to

259 issural/associational afferents in the inner molecular layer, suggesting that mRNA localization is in

260 cell formations extending into the hilus and molecular layer, suggesting that seizures alter migrator

261 In the cerebellar molecular layer, synaptic input reduces [Ca](o) by up to

262 l layer that contains all somata and (2) the molecular layer that contains the dendritic forest.

263 s (SGCs), glutamatergic neurons in the inner molecular layer that generate long-duration plateau pote

264 These results reveal UNG as a new molecular layer that shapes the specificity of AID-induc

265 f local field potential power in the dentate molecular layer that were accompanied by transient incre

266 ome shell proteins, which self-assemble into molecular layers that most likely constitute the facets

267 thickness of a material approaches a single molecular layer, the large surface energy can cause piez

268 ness of approximately 8 nm consisting of 7-8 molecular layers, the deposition of FgAalpha251 was term

269 ith the transverse-oriented, slow-conducting molecular layer, thereby permitting complex temporal pro

270 xponential dependence of junction current on molecular layer thickness (d) similar to that observed f

271 isease progression as assessed by histology (molecular layer thickness and an overall severity score)

272 deletions) and an intermediate reduction in molecular layer thickness in mice with reduced levels of

273 id not result in full recovery of cerebellar molecular layer thickness or prevent Bergmann glia degen

274 unctions, the energy loss is linear with the molecular layer thickness, with a slope of 0.31 eV/nm.

275 type, but did not prevent BG process loss or molecular layer thinning, while excision of ataxin-7 fro

276 d on electronically addressing a molecule or molecular layer through the formation of a metallic cont

277 d by direct electron-beam evaporation on the molecular layers through masks defined by electron-beam

278 ethod of forming 'soft' metallic contacts on molecular layers through surface-diffusion-mediated depo

279 m a horizontal bipolar shape situated in the molecular layer, through a transitional triangular and t

280 deposited remotely and then diffuse onto the molecular layer, thus eliminating the problems of penetr

281 The grafting of molecular layers to carbon-based materials provides a wa

282 labeling of the delta subunit in the dentate molecular layer was decreased by 4 d after status epilep

283 napses in the inner one-third of the dentate molecular layer was Glu-CB1 -RS, 53.19% (glutamatergic t

284 dendritic segments in different areas of the molecular layer were differentially regulated.

285 leptic rats excitatory synapses in the inner molecular layer were larger than in controls.

286 Those in the middle molecular layer were more likely to be influenced by the

287 of HEGC apical dendrites that penetrated the molecular layer were significantly reduced compared with

288 hereas granule cells and interneurons in the molecular layer were strongly immunopositive for beta bu

289 rection of rectification was reversed if the molecular layers were also reversed.

290 ut Ag present, and the spectra of oligomeric molecular layers were completely consistent with those o

291 The structure and orientation of the molecular layers were probed with ATR-FT-IR and Raman sp

292 When the energy levels of the two molecular layers were similar, the device had electronic

293 xclusively in the ganglionic layer below the molecular layer where parallel fibers terminate.

294 al dendrites extended into the middle of the molecular layer where they ramified.

295 e cell dendrites in the inner portion of the molecular layer, whereas parallel fibres form synapses o

296 mitochondrial staining, particularly in the molecular layer, which was independent of stress-induced

297 om the interaction of excitons in an organic molecular layer with surface plasmons in a metallic film

298 n of diazonium reagents enables formation of molecular layers with sufficient integrity for use in mo

299 f deep stellate cells are distributed in the molecular layer, with fine axon arbors in the ganglionic

300 ns of the outer and middle one-thirds of the molecular layer, with frequent axonal projections across