コーパス検索結果 (1語後でソート)
通し番号をクリックするとPubMedの該当ページを表示します
1 genus Nannomys, are susceptible to ecotropic Moloney and Friend mouse leukemia viruses (MLVs) but not
6 ch repair of retroviral double-stranded DNA, Moloney leukemia virus (MLV)-based vectors with a mutati
8 d others reported that ectopic expression of Moloney leukemia virus 10 (MOV10) protein strongly inhib
10 Dnmt3b during de novo methylation of murine Moloney leukemia virus provirus DNA in virus-infected ES
11 T is different from the basic loop of either Moloney leukemia virus RNase H or Escherichia coli RNase
14 ls also stimulated infection by amphotrophic Moloney leukemia virus, herpes simplex virus, and viruse
18 ble with LacZ pseudotypes having AKV Env and Moloney MLV (MoMLV) Gag proteins, further indicating tha
19 unctional genes, we inoculated neonates with Moloney MLV (MoMLV) or amphotropic MLV (A-MLV) and scree
22 nge at position 8 (H8R) in the SPHQ motif of Moloney MLV blocks infection by arresting virus-cell fus
23 ne change at position 84 (D84K) of ecotropic Moloney MLV Env abolishes virus binding and infection.
24 iphasic response that differentiated between Moloney MLV Env and VSV G at low concentrations, at whic
25 gave comparable dose-dependent inhibition of Moloney MLV Env and VSV G pseudotypes, suggesting that t
27 s with different affinities for CBF into the Moloney MLV genome, and determined the effects of these
30 ification of large quantities of recombinant Moloney MLV NC protein, and have studied its interaction
34 fection of NIH 3T3 cells, endocytosis brings Moloney MLV to early lysosomes, where the virus encounte
36 ection could be enhanced by preincubation of Moloney MLV with cathepsin B, consistent with SU cleavag
37 x2 mice with Moloney murine leukaemia virus (Moloney MLV) also led to a dramatic acceleration of tumo
38 e site in the Moloney murine leukemia virus (Moloney MLV) enhancer was previously shown to be an impo
39 The neurovirulent retroviruses FrCasE and Moloney MLV-ts1 cause noninflammatory spongiform neurode
43 the latent period of disease onset, in that Moloney MLVs with high-affinity CBF binding sites induce
45 recombinant generation in the animal during Moloney MuLV (M-MuLV) infection is unknown, and the exac
46 on has been unclear, since gPr80gag-negative Moloney MuLV (M-MuLV) mutants are replication competent
47 ominantly with a single group of proviruses, Moloney MuLV (M-MuLV) recombines with at least two disti
52 ed protein for a glycine residue (gly-491 in Moloney MuLV Env) that is otherwise conserved in all of
53 t interact with the reverse transcriptase of Moloney MuLV resulted in the identification of eRF1, the
54 previously generated between the ecotropic (Moloney-MuLV) and amphotropic (4070A) SU and TM proteins
55 Neonatal infection of CD2-Runx2 mice with Moloney murine leukaemia virus (Moloney MLV) also led to
56 iring within the Psi-RNA packaging signal of Moloney murine leukaemia virus (MoMuLV) expose conserved
58 show that, unlike classical chaperones, the Moloney murine leukaemia virus NC uses a unique mechanis
59 ar reporter genes were delivered into ECs by Moloney murine leukaemia virus or human immunodeficiency
63 e greater than those generated by a standard Moloney murine leukemia retroviral vector, and they were
65 n adult floxed mice by a vector based on the Moloney murine leukemia retrovirus, expressing Cre recom
67 dimerization domain in genomes isolated from Moloney murine leukemia virions using a quantitative and
68 nal structure of the double hairpin from the Moloney murine leukemia virus ([Psi(CD)](2), 132 nt, 42.
70 mbinant avian myeloblastosis virus (AMV) and Moloney murine leukemia virus (M-MLV) reverse transcript
72 substrates and the reverse transcriptases of Moloney murine leukemia virus (M-MuLV) and human immunod
73 t structural studies have suggested that the Moloney murine leukemia virus (M-MuLV) CA protein may as
74 ly stained, membrane-bound, histidine-tagged Moloney murine leukemia virus (M-MuLV) capsid protein (h
75 s shown that gPr80gag facilitates release of Moloney murine leukemia virus (M-MuLV) from cells along
78 wo-end integration reaction catalyzed by the Moloney murine leukemia virus (M-MuLV) integrase (IN) wa
81 nces within terminal inverted repeats of the Moloney murine leukemia virus (M-MuLV) LTR were synthesi
82 system that mimics the assembly of immature Moloney murine leukemia virus (M-MuLV) particles to exam
83 were generated within the 3' terminus of the Moloney murine leukemia virus (M-MuLV) pol gene encoding
85 an immunodeficiency virus type 1 (HIV-1) and Moloney murine leukemia virus (M-MuLV) were shown to req
86 as constructed by replacing the U3 region of Moloney murine leukemia virus (M-MuLV) with homologous s
87 n immunodeficiency virus, type 1 (HIV-1) and Moloney murine leukemia virus (M-MuLV), we determined th
88 -1 (human immunodeficiency virus type 1) and Moloney murine leukemia virus (M-MuLV), we evaluated how
94 cently discovered that the NC protein of the Moloney murine leukemia virus (MLV) can bind with high a
96 nalyze more than 40 derivatives of ecotropic Moloney murine leukemia virus (MLV) envelope, containing
97 ion of psi (i.e., leader) sequences from the Moloney murine leukemia virus (MLV) genome into the 3' u
98 racterized the structure and function of the Moloney murine leukemia virus (MLV) IN protein in viral
99 identified as a common integration site for Moloney murine leukemia virus (MLV) in rat thymic lympho
101 ansportin 3 protein under conditions whereby Moloney murine leukemia virus (MLV) integrase failed to
103 linkages, we digested deproteinized RNA from Moloney murine leukemia virus (MLV) particles with RNase
105 n HIV-1-based lentiviral vector to that by a Moloney murine leukemia virus (MLV) retroviral vector, u
106 ups of synthetic RNA-DNA hybrids with either Moloney murine leukemia virus (MLV) RT or human immunode
108 mmaretroviruses, we engineered a fluorescent Moloney murine leukemia virus (MLV) system consisting of
109 purvalanol A, and methoxy-roscovitine, block Moloney murine leukemia virus (MLV) transcription events
110 mo- and heterodimerization were examined for Moloney murine leukemia virus (MLV) using nondenaturing
111 f human hematopoietic stem cells (HSCs) with Moloney murine leukemia virus (MLV) vectors have been an
112 vity, while infection by the gammaretrovirus Moloney murine leukemia virus (MLV) was unaffected.
113 escribe the properties of the Gag protein of Moloney murine leukemia virus (MLV), a gammaretrovirus.
114 n, with functionally homologous regions from Moloney murine leukemia virus (MLV), a murine retrovirus
115 ins from four different retroviruses: HIV-1, Moloney murine leukemia virus (MLV), Rous sarcoma virus
116 file that may be safer than that of standard Moloney murine leukemia virus (MLV)-derived retroviral v
120 retroviral promoters and enhancers from the Moloney Murine Leukemia Virus (MMLV) and the Myeloprolif
123 ed to play a similar preintegrative role for Moloney murine leukemia virus (MMLV) in addition to HIV-
124 ies of the 3' end processing site within the Moloney murine leukemia virus (MMLV) LTR d(TCTTTCATT), a
125 (RT) fidelity, we measured the error rate of Moloney murine leukemia virus (MMLV) RT in the presence
126 s in the pE vectors have been taken from the Moloney murine leukemia virus (MMLV) vector pMFG, which
127 ocytes are refractory to gene transfer using Moloney murine leukemia virus (MMLV)-based retroviral ve
128 ansplantation model, the authors evaluated a Moloney murine leukemia virus (MMLV)-based vector encodi
129 In this report, we made use of a chimeric Moloney murine leukemia virus (MMLV)-HIV-1 Gag in which
130 n JSRV and the unrelated murine retroviruses Moloney murine leukemia virus (MMuLV) and mouse mammary
131 etherin markedly inhibits the replication of Moloney murine leukemia virus (Mo-MLV) and is required f
135 be transgenic mice expressing fusions of the Moloney murine leukemia virus (Mo-MuLV) Gag protein to s
138 nuclear localization signals (NLSs) into the Moloney murine leukemia virus (Mo-MuLV) integrase (IN) p
139 B1/DNMT3A/DNMT3L complex to newly integrated Moloney murine leukemia virus (Mo-MuLV) proviral DNA.
143 tion of a novel domain in the Gag protein of Moloney murine leukemia virus (MoLV) that is important f
145 es, whereas the prototypical gammaretrovirus Moloney murine leukemia virus (MoMLV) favors strong enha
147 1), feline immunodeficiency virus (FIV), and Moloney murine leukemia virus (MoMLV) integrases were st
148 Retroviral reverse transcriptase (RT) of Moloney murine leukemia virus (MoMLV) is expressed in th
149 n factor (BAF) blocks the autointegration of Moloney murine leukemia virus (MoMLV) PICs in vitro.
152 polymerase, but not the RNase H function of Moloney Murine Leukemia Virus (MoMLV) RT and also inhibi
153 ays which show that tetherin does not affect Moloney murine leukemia virus (MoMLV) spread, and only m
154 he HIV-1 vector was also more efficient than Moloney murine leukemia virus (MoMLV) vectors for transd
155 nd particle ultrastructure highly similar to Moloney murine leukemia virus (MoMLV), another gammaretr
156 xamine the early events in the life cycle of Moloney murine leukemia virus (MoMLV), we analyzed the i
157 lope protein (Env) can be used to pseudotype Moloney murine leukemia virus (MoMLV)-based retrovirus v
165 Recent studies with small fragments of the Moloney murine leukemia virus (MoMuLV) genome suggested
168 ropathogenic temperature-sensitive mutant of Moloney murine leukemia virus (MoMuLV-ts1), results in m
170 rmation from scarce, femtomole quantities of Moloney murine leukemia virus (MuLV) RNA inside authenti
175 onine-specific proviral integration site for Moloney murine leukemia virus (PIM) kinases PIM1 and PIM
176 We also determined whether the wild type Moloney murine leukemia virus (wt-MoMuLV) and one of its
178 e kinase PIM1 (Proviral Integration site for Moloney murine leukemia virus 1) has emerged as a key re
179 oplasmic domain derived from the amphotropic Moloney murine leukemia virus 4070A GP, revealed about 1
180 n cells expressing envelope protein (Env) of Moloney murine leukemia virus and target cells were stud
181 able to gene delivery by wild-type ecotropic Moloney murine leukemia virus and vesicular stomatitis v
182 l helper virus, pAM3-IRES-Zeo, that contains Moloney murine leukemia virus as a helper virus and a pi
183 ssive cis-acting DNA sequences identified in Moloney murine leukemia virus but remains sensitive to t
184 e full length, positive-strand genome of the Moloney Murine Leukemia Virus contains a "core encapsida
185 sults also suggest that the leader region of Moloney murine leukemia virus contains inhibitory/regula
187 erived MLE-15 cells infected with a chimeric Moloney murine leukemia virus driven by the JSRV enhance
190 l receptor-binding sequence of the ecotropic Moloney murine leukemia virus envelope glycoprotein with
191 we have studied how the protomeric units of Moloney murine leukemia virus envelope protein (Env) are
192 e we report that tryptophan 142 in ecotropic Moloney murine leukemia virus envelope protein is essent
196 by eight footprints representing regions of Moloney murine leukemia virus gag, some previously uncha
197 dful" was tested by examining the ability of Moloney murine leukemia virus genomes lengthened by 4, 8
198 coated with the parental wild-type ecotropic Moloney murine leukemia virus glycoprotein through the e
199 al analyses of the p12 Gag phosphoprotein of Moloney murine leukemia virus have demonstrated its part
200 nuclear magnetic resonance structure of the Moloney murine leukemia virus IN (M-MLV) C-terminal doma
202 To study the function of the HHCC domain of Moloney murine leukemia virus IN, the first N-terminal 1
203 receptor-5-positive (Lgr5(+)) and B lymphoma moloney murine leukemia virus insertion region homolog-1
205 rabidopsis (Arabidopsis thaliana) B lymphoma Moloney murine leukemia virus insertion region1 homolog
207 e examine the role of Bmi-1 (B-cell-specific Moloney murine leukemia virus integration site 1) as a r
210 igh expression of proviral insertion site of Moloney murine leukemia virus kinases (Pim-1, -2, and -3
211 ng hybrid duplex substrates derived from the Moloney murine leukemia virus long terminal repeat, we i
212 internal grp78 promoter, in contrast to the Moloney murine leukemia virus long terminal repeat, wher
213 xpression in vivo from vectors driven by the Moloney murine leukemia virus long-terminal repeat (LTR)
214 repeat (LTR) was found to be higher than the Moloney murine leukemia virus LTRs in both LNCaP and WPM
218 irus type 1 nucleocapsid protein and for the moloney murine leukemia virus nucleocapsid zinc knuckle
219 tated sequences both within and flanking the Moloney murine leukemia virus polypurine tract (PPT) and
223 n which the host, the N-terminal fragment of Moloney murine leukemia virus reverse transcriptase (MML
224 Inhibitor resistance of several commercial Moloney murine leukemia virus reverse transcriptase (MML
225 an effort to increase the thermostability of Moloney Murine Leukemia Virus reverse transcriptase (MML
226 A resolution of an N-terminal fragment from Moloney murine leukemia virus reverse transcriptase comp
229 ed on the ability of the RNase H activity of Moloney murine leukemia virus reverse transcriptase to c
230 , including D114A and R116A substitutions in Moloney murine leukemia virus reverse transcriptase, are
231 conserved residues in the fingers domain of Moloney murine leukemia virus reverse transcriptase, res
232 t includes the fingers and palm domains from Moloney murine leukemia virus reverse transcriptase.
233 n of human immunodeficiency virus type 1 and Moloney murine leukemia virus reverse transcriptases wit
234 ng model hybrid substrates and the HIV-1 and Moloney murine leukemia virus reverse transcriptases, we
235 to complete a single round of intracellular Moloney murine leukemia virus reverse transcription appr
238 ddition, PARP-1-deficient MEFs infected with Moloney murine leukemia virus showed no decrease in viru
239 he generation of retroviral vectors based on Moloney murine leukemia virus that specifically transduc
240 ic HTLV-1 with a replaced envelope gene from Moloney murine leukemia virus to allow HTLV-1 to fuse wi
241 substitutions were introduced into the NC of Moloney murine leukemia virus to examine further its rol
243 s on the neurovirulent viruses FrCas(NC) and Moloney murine leukemia virus ts1 indicate that the nasc
249 1 (fti-1) (feline leukemia virus) and Ahi-1 (Moloney murine leukemia virus) shows that these display
251 ces between XMRV and the intensively studied Moloney murine leukemia virus, architectures of the regu
252 mate lentiviruses and distinct from those of Moloney murine leukemia virus, avian sarcoma leukosis vi
253 retrovirus, MoFe2, or with the parent virus, Moloney murine leukemia virus, caused significant reduct
255 ation, followed by reverse transcription via Moloney murine leukemia virus, degradation of chromosoma
257 elops in mice infected with ts1, a mutant of Moloney murine leukemia virus, resembles human AIDS.
258 eviously, we have shown that, in the case of Moloney murine leukemia virus, the U3 region of the LTR
260 pendent kinase proviral integration site for Moloney murine leukemia virus-1 (PIM-1), which in turn r
261 evere combined immunodeficiency (SCID-X1), a Moloney murine leukemia virus-based gamma-retrovirus vec
262 atal mice were injected intravenously with a Moloney murine leukemia virus-based retroviral vector (R
264 in transduction efficiency by both HIV- and Moloney murine leukemia virus-based retroviral vectors.
267 HIV-1 replication than cells transduced with Moloney murine leukemia virus-based vectors expressing T
268 te dehydrogenase (hG6PD) gene transferred by Moloney murine leukemia virus-based vectors into murine
270 deletion of sequences in the 3' U3 region of Moloney murine leukemia virus-derived retroviral vectors
280 study, we tested replication properties for Moloney murine leukemia viruses with targeted mutations
282 oduce an in vitro methylated or unmethylated Moloney murine leukemia-based provirus in MEL cells.
284 The fundamental dimerization unit for the Moloney murine sarcoma gamma retrovirus spans a 170-nucl
285 examination of the in vitro dimerization of Moloney murine sarcoma virus (MuSV) RNA in the context o
286 iously, we proposed that dimerization of the Moloney murine sarcoma virus genomic RNAs relies upon th
287 dimerization active sequence (MiDAS) for the Moloney murine sarcoma virus in the final dimer state us
289 , a 2954-base-pair (bp) laboratory-generated Moloney murine sarcoma virus, induced subcutaneous tumor
291 ory activity toward proviral integrations of Moloney (PIM) virus 3 kinase but not ZIPK, had no effect
292 , expressed by tumor cells induced by Friend/Moloney/Rauscher (FMR) MuLV, is not immunogenic, even wh
293 Current strategies for genetic therapy using Moloney retroviruses require ex vivo manipulation of hem
294 e delivery of scFv4 into SU-CCS-1 cells by a Moloney sarcoma retroviral vector (SRalpha-Fv4) signific
295 ts as a chimeric transcription factor on the Moloney sarcoma virus long terminal repeat and a synthet
296 e beta1-subunit (Na,K-beta) in highly motile Moloney sarcoma virus-transformed Madin-Darby canine kid
300 nificant shift in disease specificity of the Moloney virus from T-cell leukemia to erythroleukemia.
WebLSDに未収録の専門用語(用法)は "新規対訳" から投稿できます。