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1 sion or of cognitive therapy compared with a monoamine oxidase inhibitor.
2 es and aromatic amino acid decarboxylase and monoamine oxidase inhibitors.
3 at such patients responded preferentially to monoamine oxidase inhibitors.
4 st and present clinical trials of tricyclic, monoamine oxidase inhibitor, and selective serotonin reu
5 side effects commonly seen with traditional monoamine oxidase inhibitor antidepressants were not obs
9 selective serotonin reuptake inhibitors and monoamine oxidase inhibitors did not show significant pr
10 ive to standard acute-phase treatment with a monoamine oxidase inhibitor for outpatients with major d
12 f smoking behavior in humans and presence of monoamine oxidase inhibitors in tobacco smoke, we hypoth
13 reated with beta-blockers, ACE inhibitors or monoamine oxidase inhibitors, in children under 5 years
14 covery of a new class of compounds acting as monoamine oxidase inhibitors (MAO-Is) and bearing a 6'-s
15 ibitor (SSRI) fluoxetine (10 mg/kg), and the monoamine oxidase inhibitor (MAOI) phenelzine (10 mg/kg)
16 Hamsters were treated chronically with the monoamine oxidase inhibitor (MAOI), clorgyline, and then
19 n of LSD1 or inhibition of its activity with monoamine oxidase inhibitors (MAOIs) results in the accu
20 been treated with tricyclic antidepressants, monoamine oxidase inhibitors (MAOIs), fluoxetine, or pla
21 is a small body of evidence indicating that monoamine oxidase inhibitors may be effective in at leas
25 e inhibitors desipramine and reboxetine, the monoamine oxidase inhibitor pargyline, and the atypical
26 Our lab has found opposing effects of the monoamine oxidase inhibitor phenelzine and the tricyclic
27 group therapy (CBGT) and treatment with the monoamine oxidase inhibitor phenelzine sulfate for socia
29 n has also been introduced in a study of the monoamine oxidase inhibitor rasagiline, demonstrating a
30 selective serotonin reuptake inhibitor or a monoamine oxidase inhibitor rather than a tricyclic anti
32 total of 341 patients received the selective monoamine oxidase inhibitor selegiline (10 mg a day), al
33 clusively among PD patients treated with the monoamine oxidase inhibitor selegiline (L-deprenyl).
35 ects on outcomes compared with waitlist were monoamine oxidase inhibitors (SMD -1.01, 95% credible in
36 cal depression are more likely to respond to monoamine oxidase inhibitors than to tricyclic antidepre
37 n by RNAi in human erythroid cells or by the monoamine oxidase inhibitor tranylcypromine in human ery
38 uoxetine) or norepinephrine (desipramine), a monoamine oxidase inhibitor (tranylcypromine), and elect
40 studies of the neuroprotective properties of monoamine oxidase inhibitors, we found that phenelzine p
42 ibitor) and tranylcypromine (10 mg/kg, i.p., monoamine oxidase inhibitor) with each drug being inject
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