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1 switching disinfectant from free chlorine to monochloramine.
2 isinfectant changeover from free chlorine to monochloramine.
3 understood, in particular in the presence of monochloramine.
4 and in UV and UV/H(2)O(2) in the presence of monochloramine.
5 serves cellular viability in the presence of monochloramine.
6  IEC-18 cells showed increased resistance to monochloramine.
7 re observed only in C2/AS cells treated with monochloramine.
8                     The protective effect of monochloramine against legionella should be confirmed by
9 rformed of the virucidal efficacy of PAA and monochloramine against murine norovirus (MNV) and MS2 ba
10 ed at CT values of less than 50 mg-min/L for monochloramine and 200 mg-min/L for PAA.
11  higher than N-chloroacetamide under typical monochloramine and acetaldehyde concentrations.
12 ino acid and alpha-monochloramine, and alpha-monochloramine and aldehyde were confirmed by high press
13                   The effects of pH, initial monochloramine and bromide ion concentrations, phosphate
14 ation and decomposition from the reaction of monochloramine and bromide ion.
15 ion rate constants were then used to predict monochloramine and bromochloramine concentration profile
16 sulting product, nitrite, can be oxidized by monochloramine and hypochlorous acid (HOCl), potentially
17                         The reaction between monochloramine and ranitidine followed second order kine
18  monochloramine penetration by reacting with monochloramine and reduced its concentration within biof
19  of pH, disinfectant type (free chlorine and monochloramine), and chlor(am)ine residual (CR) were exa
20 tionships between alpha-amino acid and alpha-monochloramine, and alpha-monochloramine and aldehyde we
21  8.5 in the presence of sodium hypochlorite, monochloramine, and chlorine dioxide.
22 more, reaction of N-CNTs with free chlorine, monochloramine, and ozone generated byproduct NDMA at yi
23 ing water exhibit differential resistance to monochloramine, and that the disinfection process select
24 our byproducts were identified in UV-C, UV-C/monochloramine, and UV/H(2)O(2)/monochloramine oxidation
25 res' disease than those that used water with monochloramine as a residual disinfectant (odds ratio 10
26 n extended model consisting of reactions for monochloramine autodecomposition, the decay of bromamine
27 es were exposed to varying concentrations of monochloramine, bromide, and iodide in both synthetic an
28  similar to those of ammonia metabolism, and monochloramine cometabolism accounted for 30% of the obs
29            The current research investigated monochloramine cometabolism by nitrifying mixed cultures
30                                    Recently, monochloramine cometabolism by the pure culture ammonia-
31                                              Monochloramine cometabolism kinetics were similar to tho
32 nking water distribution systems; therefore, monochloramine cometabolism may be a significant contrib
33              These results demonstrated that monochloramine cometabolism occurred in mixed cultures s
34  after installation (T0), in the presence of monochloramine concentration between 1.5 and 2 mg/L, 10/
35 ifferent byproducts depending on the applied monochloramine concentration.
36 e to nitrogen mass ratio, and (3) decreasing monochloramine concentration.
37                      Results showed that the monochloramine decay rate increased with decreasing pH a
38  reductive dissolution, primarily induced by monochloramine decomposition, and that of chloropyromorp
39                                  Addition of monochloramine decreased the degradation rates of tramad
40 Nitrosomonas europaea, was shown to increase monochloramine demand.
41                                              Monochloramine derivatives are long lived physiological
42 in biofilms was also spatially influenced by monochloramine diffusion and reaction within biofilms, s
43 e of 1 year in 11 fixed sites, the impact of monochloramine disinfection on Legionella, heterotrophic
44                                The impact of monochloramine disinfection on the complex bacterial com
45 re the changes that EPS underwent during the monochloramine disinfection process.
46 ial community structure were detected during monochloramine disinfection using PMA-pyrosequencing, wh
47                            The efficiency of monochloramine disinfection was dependent on the quantit
48                          In contrast, a high monochloramine dose (250 mg L(-1) as Cl2) at pH 7.0 (the
49 full-scale drinking water filters exposed to monochloramine for a range of contact times.
50 h about 25% of municipalities in the USA use monochloramine for disinfection of drinking water, the e
51 th drinking water might not have occurred if monochloramine had been used instead of free chlorine fo
52 r polymeric substances (EPS) in biofilms, as monochloramine has a selective reactivity with proteins
53      The membrane-impermeant oxidant taurine monochloramine, however, had no effect on whole cell cur
54 ever, although the transport and reaction of monochloramine in biofilm could be observed, the specifi
55                   Free chlorine or preformed monochloramine in feedwater led to the production of nit
56  values for 1-log10 MS2 reduction by PAA and monochloramine in MWW were 1254 and 1228 mg-min/L, respe
57 ing disinfection by peracetic acid (PAA) and monochloramine in secondary wastewater (WW) and phosphat
58 ed the bactericidal effectiveness of PAA and monochloramine in wastewater, but limited information is
59  different reactivity of EPS components with monochloramine influenced disinfectant penetration, biof
60 bility of IEC-18 cells to withstand oxidant (monochloramine) injury.
61 peroxidase system or HOCl, generating stable monochloramines instead.
62   The results of the present study show that monochloramine is a promising disinfectant that can prev
63 ous study, we reported that the transport of monochloramine is affected by the extracellular polymeri
64 eophilic substitution between ranitidine and monochloramine led to byproducts that are critical inter
65 abolism may be a significant contribution to monochloramine loss during nitrification episodes in dri
66 metabolism accounted for 30% of the observed monochloramine loss.
67 us acid (HOCl), potentially leading to rapid monochloramine loss.
68  putida EPS multiplied both the time and the monochloramine mass required to achieve a full biofilm p
69  UV treatment in the presence and absence of monochloramine, NDMA formation potential can be halved.
70                                              Monochloramine (NH(2)Cl) is a membrane-permeant oxidant
71 yrrole-containing heterocycles revealed that monochloramine (NH(2)Cl) is an excellent reagent for thi
72  defined the cellular actions of the oxidant monochloramine (NH(2)Cl) on anion secretion in human col
73 formation, quantified the stoichiometries of monochloramine (NH2Cl) and aqueous O2 consumption, deriv
74 arrestment through simultaneously increasing monochloramine (NH2Cl) and chlorine to nitrogen mass rat
75 studies have shown that autodecomposition of monochloramine (NH2Cl) can cause lead release from PbO2
76 f NH4(+) promotes conversion of the residual monochloramine (NH2Cl) in the permeate to dichloramine (
77           The effects of chemically prepared monochloramine (NH2Cl) on protein kinase C (PKC) and PKC
78                                 In excess of monochloramine, nucleophilic substitution between raniti
79 isinfection of drinking water, the effect of monochloramine on the occurrence of Legionnaires' diseas
80 phy (OCT) during three months of exposure to monochloramine or free chlorine.
81 acids can result in the formation of organic monochloramines or organic dichloramines, depending on t
82 disinfectant exposure, the mean stiffness of monochloramine- or free-chlorine-treated biofilms was 4
83 n UV-C, UV-C/monochloramine, and UV/H(2)O(2)/monochloramine oxidation of tramadol using MS(3) capabil
84 tes on bacterial cells, protein EPS hindered monochloramine penetration by reacting with monochlorami
85 sets representing the effect of pH, bromide, monochloramine, phosphate, and excess ammonia.
86 xidation by hydroxyl radicals in UV/H(2)O(2)/monochloramine process mineralized some of the byproduct
87                                              Monochloramine quickly reacts with chloroacetaldehyde, a
88                         However, the oxidant monochloramine rapidly decreased TER and increased manni
89 d and bicarbonate ion were estimated for the monochloramine reaction.
90 mate carbonate buffer rate constants for the monochloramine reaction.
91 omoted biofilm cell viability by obstructing monochloramine reactive sites on bacterial cells, protei
92 and kBromine/BP-3, respectively, whereas low monochloramine reactivity was observed (kNH2Cl/BP-3 = 0.
93                                              Monochloramine reacts with acetaldehyde, a common ozone
94 ginosa) suggested that currently recommended monochloramine residual levels may underestimate the ris
95 min/L for PAA and 6, 13, and 28 mg-min/L for monochloramine, respectively.
96 water quality models to simulate nitrite and monochloramine's fate.
97 e less toxic and more stable oxidant taurine monochloramine (TauNHCl).
98 uffer demonstrated that in waters containing monochloramine, the presence of bromide ion enhanced NDM
99 to acetonitrile and (2) was oxidized (k3) by monochloramine to produce N-chloroacetamide.
100 o-1-(chloroamino)ethanol is also oxidized by monochloramine to produce the previously unreported DBP
101                                              Monochloramine transport profiling measured by a chloram
102  that increased in relative abundance during monochloramine treatment include Legionella, Escherichia
103   Epithelial resistance to oxidant injury by monochloramine was determined by (51)Cr release.
104  EPS composition on bacteria disinfection by monochloramine was qualitatively determined using both w
105 survival under conditions of oxidant stress (monochloramine) was determined using (51)Cr release in h
106 tivity and contribution to susceptibility to monochloramine, we investigated the bacteria disinfectio
107 lso showed different levels of inhibition by monochloramine when tested.
108  begins with generation of an unstable alpha-monochloramine, which subsequently decomposes to yield a
109  the rate constants of chlorine, bromine and monochloramine with BP-3 were determined at various pH l
110 V exhibited comparable resistance to PAA and monochloramine with CT values for 2 log10 RT-qPCR reduct
111 action of disinfectants such as chlorine and monochloramine with organic matter may cause bladder can
112 sults suggested significant reactions of the monochloramine with peptide fragments of proteins that a

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