コーパス検索結果 (1語後でソート)
通し番号をクリックするとPubMedの該当ページを表示します
1 iocytosis is a rare disorder associated with monoclonal gammopathy.
2 or hereditary amyloidosis with a concomitant monoclonal gammopathy.
3 into bone marrow and the presence of an IgM monoclonal gammopathy.
4 inosis, extracutaneous manifestations, and a monoclonal gammopathy.
5 coni syndrome (FS) is a rare complication of monoclonal gammopathy.
6 from the sera of individuals with a form of monoclonal gammopathy.
7 a cell leukemia (PCL) is the most aggressive monoclonal gammopathy.
8 ot analysis, primarily in presence of an IgM monoclonal gammopathy.
9 utcomes of eosinophilic fasciitis-associated monoclonal gammopathy.
10 for histology in patients who do not have a monoclonal gammopathy.
11 proportion of patients will have persistent monoclonal gammopathy.
12 anscriptional reorganization of AL and other monoclonal gammopathies.
13 , University College London, and Registry of Monoclonal Gammopathies.
14 irected or B cell clone-directed therapy for monoclonal gammopathies.
15 um and glutamate than patients with indolent monoclonal gammopathies.
16 by CD138(+) cells across the progression of monoclonal gammopathies.
17 n has been implicated in the pathogenesis of monoclonal gammopathies.
18 se-associated gammopathies and some sporadic monoclonal gammopathies.
19 Gaucher's disease have an increased risk of monoclonal gammopathies.
20 irus (EBV) replication in the persistence of monoclonal gammopathy.1 It has been known for some time
22 vated erythropoietin and erythrocytosis; (3) monoclonal gammopathy; (4) perinephric fluid collections
24 se characterized by urticarial exanthema and monoclonal gammopathy accompanied by systemic symptoms s
25 yeloma (CMM) are preceded by an asymptomatic monoclonal gammopathy (AMG), classified as either monocl
27 a B-cell malignancy characterized by an IgM monoclonal gammopathy and bone marrow (BM) infiltration
28 symptoms are always preceded by a detectable monoclonal gammopathy and by elevated biomarkers of orga
30 We review the causal association between monoclonal gammopathy and neuropathy, and critically rev
31 fection is unknown, but in many patients the monoclonal gammopathy and other B-cell abnormalities can
32 HIV infection have an increased incidence of monoclonal gammopathy and plasma cell dyscrasias.2,3 The
33 o indicate a causal relationship between the monoclonal gammopathy and the renal damage and because t
35 me is characterized by an urticarial rash, a monoclonal gammopathy, and clinical, histological, and b
36 One patient with C3G had GN unrelated to the monoclonal gammopathy, and one with PGNMID did not compl
37 olyneuropathy, organomegaly, endocrinopathy, monoclonal gammopathy, and skin changes (POEMS) syndrome
38 olyneuropathy, organomegaly, endocrinopathy, monoclonal gammopathy, and skin changes) syndrome is a r
44 aracterized by recurrent urticarial rash and monoclonal gammopathy, associated with clinical and biol
50 que in kidney pathology for the diagnosis of monoclonal gammopathy-associated nephropathies, and coul
52 f amyloid organ involvement in patients with monoclonal gammopathies at higher risk to develop the di
53 characteristics of human disease, including monoclonal gammopathy, BM infiltration with lytic bone l
55 pared with 2% of age-matched controls, had a monoclonal gammopathy characterized as IgG lambda in 9 p
56 ion disease (MIDD) is a rare complication of monoclonal gammopathy characterized by deposition of mon
59 ion and test results led us to consider that monoclonal gammopathy could be the cause of this entity.
60 NA methylation in the immunoglobulin M (IgM) monoclonal gammopathy disease spectrum remains poorly un
62 mune cells in 305 patients with asymptomatic monoclonal gammopathy enrolled in S0120 under the auspic
63 sed on clinical and laboratory findings with monoclonal gammopathy evaluation and, if indicated, TTR
64 tologists can screen subjects who have known monoclonal gammopathy for amyloid organ dysfunction and
65 ent of peripheral neuropathy associated with monoclonal gammopathies has been advanced by recent clin
66 e spectrum of kidney lesions associated with monoclonal gammopathy has significantly expanded over th
67 ) and associated skeletal destruction, serum monoclonal gammopathy, immune suppression, and end-organ
69 ocytic leukemia/small lymphocytic lymphoma), monoclonal gammopathy in 20%, and multiple myeloma in 6%
70 phritic factor in child GP patients and with monoclonal gammopathy in adult GP patients, who frequent
72 a cell dyscrasias.2,3 The exact mechanism of monoclonal gammopathy in patients with HIV infection is
74 trosome amplification was detected in 67% of monoclonal gammopathies, including monoclonal gammopathy
75 onal immunoglobulin in 33% of sporadic human monoclonal gammopathies is also specific for the lysolip
76 e that clinical progression in patients with monoclonal gammopathies is associated with an acquired b
77 -stage renal disease, the persistence of the monoclonal gammopathy is associated with high rates of r
82 al population, the presence of non-malignant monoclonal gammopathy (MG) can be causally associated wi
84 he rate and predictors of finding lesions of monoclonal gammopathy (MG) of renal significance (MGRS)
88 biopsy in only 15% of patients, included 21 monoclonal gammopathies of renal significance; 15 multip
89 l population can be identified as in myeloma/monoclonal gammopathies of undetermined significance (MG
90 nalysis of bone marrow trephine samples from monoclonal gammopathies of undetermined significance (MG
91 y modified paraprotein target (paratargs) in monoclonal gammopathies of undetermined significance (MG
92 from 61 patients (29 AL, 23 MM, and 9 MGUS [monoclonal gammopathies of undetermined significance]) w
93 noglobulin light-chain (AL) amyloidosis, and monoclonal gammopathy of clinical significance (MGCS), a
99 tion was found in 89% of patients, including monoclonal gammopathy of renal significance (65%) and sy
105 PGNMID) is classified within the spectrum of monoclonal gammopathy of renal significance (MGRS).
109 luding multiple myeloma, AL amyloidosis, and monoclonal gammopathy of renal significance, face a high
110 underwent bone marrow biopsy which revealed Monoclonal Gammopathy of renal significance, triggering
111 tion process of normal plasma cells (PCs) to monoclonal gammopathy of uncertain significance (MGUS) a
112 tribution was determined in 63 patients with monoclonal gammopathy of uncertain significance (MGUS) a
113 hat healthy human controls and patients with monoclonal gammopathy of uncertain significance expresse
115 from 56 subjects representing premalignant (monoclonal gammopathy of uncertain significance), early,
116 performed CGH on 25 cases of MM, 4 cases of monoclonal gammopathy of uncertain significance, and 1 c
117 as an indolent course of disease that mimics monoclonal gammopathy of undermined significance, wherea
118 ltiple myeloma (SMM) bridges the gap between monoclonal gammopathy of undetermined significance (a mo
119 34 patients with WM and 10 patients with IgM monoclonal gammopathy of undetermined significance (IgM
121 amples from 58 patients with WM, 77 with IgM monoclonal gammopathy of undetermined significance (IgM-
123 er in these patients is more consistent with monoclonal gammopathy of undetermined significance (MGUS
125 demonstrate an increased fracture risk with monoclonal gammopathy of undetermined significance (MGUS
127 the original clone, referred to as secondary monoclonal gammopathy of undetermined significance (MGUS
128 le myeloma (MM) patients (n = 8740) and 5652 monoclonal gammopathy of undetermined significance (MGUS
130 istently preceded by the precursor states of monoclonal gammopathy of undetermined significance (MGUS
132 ich evolves from a premalignant stage called monoclonal gammopathy of undetermined significance (MGUS
137 ilial clustering of the precursor condition, monoclonal gammopathy of undetermined significance (MGUS
139 pesticide applicators to assess the risk of monoclonal gammopathy of undetermined significance (MGUS
143 in the etiology of multiple myeloma (MM) and monoclonal gammopathy of undetermined significance (MGUS
144 e marrow PCs from subjects with MM (n = 16), monoclonal gammopathy of undetermined significance (MGUS
145 ve cases of multiple myeloma, three cases of monoclonal gammopathy of undetermined significance (MGUS
146 The factors that determine progression from monoclonal gammopathy of undetermined significance (MGUS
149 21) in more than 500 untreated patients with monoclonal gammopathy of undetermined significance (MGUS
151 thylation is relatively common and occurs in monoclonal gammopathy of undetermined significance (MGUS
152 of bone marrow specimens from patients with monoclonal gammopathy of undetermined significance (MGUS
154 studies have reported a higher prevalence of monoclonal gammopathy of undetermined significance (MGUS
155 in 67% of monoclonal gammopathies, including monoclonal gammopathy of undetermined significance (MGUS
156 s of premalignant non-immunoglobulin M (IgM) monoclonal gammopathy of undetermined significance (MGUS
157 ppa or lambda immunoglobulin light chains in monoclonal gammopathy of undetermined significance (MGUS
158 rease in CD4+ CD25+ T cells in patients with monoclonal gammopathy of undetermined significance (MGUS
160 d for multiple myeloma (MM) and premalignant monoclonal gammopathy of undetermined significance (MGUS
161 receptors on plasma cells from patients with monoclonal gammopathy of undetermined significance (MGUS
162 ed in the early pathogenesis of premalignant monoclonal gammopathy of undetermined significance (MGUS
163 The events leading the transformation of the monoclonal gammopathy of undetermined significance (MGUS
165 ression was detected in 5 of 5 patients with monoclonal gammopathy of undetermined significance (MGUS
166 been made to quantitate adverse outcomes of monoclonal gammopathy of undetermined significance (MGUS
168 previously untreated active myeloma, 14 had monoclonal gammopathy of undetermined significance (MGUS
170 omal aberrations have been identified in the monoclonal gammopathy of undetermined significance (MGUS
171 ewly diagnosed multiple myeloma (MM), 5 with monoclonal gammopathy of undetermined significance (MGUS
172 arises from a common benign PC tumor called Monoclonal Gammopathy of Undetermined Significance (MGUS
173 al similarities to multiple myeloma (MM) and monoclonal gammopathy of undetermined significance (MGUS
174 V-8 may play a role in the transformation of monoclonal gammopathy of undetermined significance (MGUS
175 ifferentially expressed in plasma cells from monoclonal gammopathy of undetermined significance (MGUS
176 ic cells from two out of eight patients with monoclonal gammopathy of undetermined significance (MGUS
177 e multiple myeloma to a benign form known as monoclonal gammopathy of undetermined significance (MGUS
178 g clonal cells in the blood of patients with monoclonal gammopathy of undetermined significance (MGUS
180 l factors are suggested to jointly influence monoclonal gammopathy of undetermined significance (MGUS
181 uman multiple myeloma (MM) and its precursor monoclonal gammopathy of undetermined significance (MGUS
183 es investigate the outcomes of patients with monoclonal gammopathy of undetermined significance (MGUS
184 itions smoldering multiple myeloma (SMM) and monoclonal gammopathy of undetermined significance (MGUS
189 ently preceded by an asymptomatic condition, monoclonal gammopathy of undetermined significance (MGUS
190 ple myeloma (MM) is consistently preceded by monoclonal gammopathy of undetermined significance (MGUS
191 recursor conditions recognized clinically as monoclonal gammopathy of undetermined significance (MGUS
192 festation of monoclonal gammopathies such as monoclonal gammopathy of undetermined significance (MGUS
195 d studies indicate a possible association of monoclonal gammopathy of undetermined significance (MGUS
196 ht-chain (AL) amyloidosis who present with a monoclonal gammopathy of undetermined significance (MGUS
197 s of multiple myeloma (MM) and its precursor monoclonal gammopathy of undetermined significance (MGUS
199 Multiple myeloma is consistently preceded by monoclonal gammopathy of undetermined significance (MGUS
200 (MM) are preceded by precursor states termed monoclonal gammopathy of undetermined significance (MGUS
201 sorting exosomes from multiple myeloma (MM), monoclonal gammopathy of undetermined significance (MGUS
203 olves from a premalignant condition known as monoclonal gammopathy of undetermined significance (MGUS
204 peculated that the clinical progression from monoclonal gammopathy of undetermined significance (MGUS
205 M transformation from immunoglobulin-M (IgM) monoclonal gammopathy of undetermined significance (MGUS
206 of multiple myeloma and its precursor state, monoclonal gammopathy of undetermined significance (MGUS
207 n premalignant plasma cell dyscrasia such as monoclonal gammopathy of undetermined significance (MGUS
208 onsistently preceded by the precursor state, monoclonal gammopathy of undetermined significance (MGUS
209 Blood, Farr et al showed that patients with monoclonal gammopathy of undetermined significance (MGUS
210 lonal gammopathy (AMG), classified as either monoclonal gammopathy of undetermined significance (MGUS
211 ressed in bones of a subset of patients with monoclonal gammopathy of undetermined significance (MGUS
214 esson et al study the risk of progression of monoclonal gammopathy of undetermined significance (MGUS
217 was absent in 90% of immunoglobulin M (IgM) monoclonal gammopathy of undetermined significance (MGUS
218 old woman with AL-amyloidosis secondary to a monoclonal gammopathy of undetermined significance (MGUS
219 nce interval [CI], 1.31-17.86; P = .018) and monoclonal gammopathy of undetermined significance (OR,
220 plasma cells (B cells, normal plasma cells, monoclonal gammopathy of undetermined significance [MGUS
221 with the marrow in preneoplastic gammopathy (monoclonal gammopathy of undetermined significance [MGUS
222 ldenstrom macroglobulinemia [WM], 6 with IgM monoclonal gammopathy of undetermined significance [MGUS
224 ping phenotypic profiles between AL and both monoclonal gammopathy of undetermined significance and M
225 indeterminate potential (CHIP), analogous to monoclonal gammopathy of undetermined significance and m
226 rasias, including asymptomatic states called monoclonal gammopathy of undetermined significance and s
227 rates from precursor states of MM, including monoclonal gammopathy of undetermined significance and s
229 olution from precursor conditions, including monoclonal gammopathy of undetermined significance and s
231 ias encompass a spectrum from the precursors monoclonal gammopathy of undetermined significance and s
232 attern of bone marrow involvement similar to monoclonal gammopathy of undetermined significance at di
233 Inclusion criteria were newly diagnosed MM, monoclonal gammopathy of undetermined significance at hi
236 those from healthy donors and patients with monoclonal gammopathy of undetermined significance or ot
237 ded patients who were 18 years or older with monoclonal gammopathy of undetermined significance or sm
238 as asymptomatic precursor conditions-either monoclonal gammopathy of undetermined significance or sm
239 multiple myeloma are dichotomised as having monoclonal gammopathy of undetermined significance or sm
240 icroenvironment cells in transformation from monoclonal gammopathy of undetermined significance or sm
241 ecreased serum adiponectin concentrations in monoclonal gammopathy of undetermined significance patie
242 The PANGEA models improved prediction of monoclonal gammopathy of undetermined significance progr
244 ver different disease stages, from 23.66% in monoclonal gammopathy of undetermined significance to 3.
245 right-skewed model of genetic evolution from monoclonal gammopathy of undetermined significance to MM
246 in a pattern that parallels progression from monoclonal gammopathy of undetermined significance to MM
249 n biomarkers can help identify patients with monoclonal gammopathy of undetermined significance who a
250 sionally may be the presenting feature); and monoclonal gammopathy of undetermined significance with
251 molecular characteristics that evolves from monoclonal gammopathy of undetermined significance, a hi
252 nguish POEMS from neuropathy associated with monoclonal gammopathy of undetermined significance, addi
253 h light chain amyloidosis, multiple myeloma, monoclonal gammopathy of undetermined significance, and
255 progression from the non-malignant disorder monoclonal gammopathy of undetermined significance, are
256 ight chain amyloidosis, multiple myeloma and monoclonal gammopathy of undetermined significance, immu
258 er than in plasma cells from healthy donors, monoclonal gammopathy of undetermined significance, smol
259 evidence of paraproteinemia in a setting of monoclonal gammopathy of undetermined significance, smol
260 tive solution to delineate the complexity of monoclonal gammopathy of undetermined significance, smou
261 increased in both multiple myeloma (MM) and monoclonal gammopathy of undetermined significance, sugg
262 ong 5652 patients with IgG/IgA (but not IgM) monoclonal gammopathy of undetermined significance, supp
263 re akin to MM, whereas nonprogressor SMM has monoclonal gammopathy of undetermined significance-like
264 neuropathy, myeloproliferative disorder, and monoclonal gammopathy of undetermined significance.
265 ge-progressive premalignant condition termed monoclonal gammopathy of undetermined significance.
266 e marrow myeloma cells, even in asymptomatic monoclonal gammopathy of undetermined significance.
267 apheresis have shown limited efficacy in IgM monoclonal gammopathy of undetermined significance.
268 induced increase in MM cell proliferation in monoclonal gammopathy of undetermined significance/smold
269 higher levels of BM and circulating TPO than monoclonal gammopathy of undetermined significance/smold
271 0%) methylation of SOCS1 and SYK; (5) MM and monoclonal gammopathy of unknown significance (MGUS) had
272 ra from patients with MM, chronic leukemias, monoclonal gammopathy of unknown significance (MGUS), PB
273 dults have the precursor malignant condition monoclonal gammopathy of unknown significance (MGUS).
274 ith multiple myeloma (MM) and its precursor, monoclonal gammopathy of unknown significance (MGUS).
276 but not in the limited plasma cell disorder monoclonal gammopathy of unknown significance or in nonm
277 onsecutive patients with multiple myeloma or monoclonal gammopathy of unknown significance sequential
278 tomatic MM, 5 with smoldering MM, and 4 with monoclonal gammopathy of unknown significance) and 20 in
283 hat proceeds through a premalignant state of monoclonal gammopathy of unknown significance; however,
284 e cohorts were at high risk for developing a monoclonal gammopathy on the basis of Black race or a fa
287 eatures an unexpected low rate of detectable monoclonal gammopathy, questioning the reality of an und
289 s introduced by the International Kidney and Monoclonal Gammopathy Research Group (IKMG) in 2012.
290 thology Society and International Kidney and Monoclonal Gammopathy Research Group jointly tasked a wo
291 gkin lymphoma (RR, 2.35; 95% CI, 1.96-2.82), monoclonal gammopathy (RR, 1.90; 95% CI, 1.55-2.32), mye
292 athy (PPK) is a rare ocular manifestation of monoclonal gammopathies such as monoclonal gammopathy of
293 AL) and multiple myeloma (MM) are 2 distinct monoclonal gammopathies that involve the same cellular c
294 ficance (MGRS) was introduced to distinguish monoclonal gammopathies that result in the development o
295 IgG deposits, a form of renal involvement by monoclonal gammopathy that mimics immune-complex glomeru
297 ith end-stage renal disease and known benign monoclonal gammopathy underwent kidney transplantation a
300 rdless of age, is frequently associated with monoclonal gammopathies, which often recognize various m