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1 iated inflammatory mediators (in particular, monocyte chemoattractant protein-1).
2 eta), tumor necrosis factor (TNF), IL-6, and monocyte chemoattractant protein 1.
3 ression of proinflammatory interleukin 6 and monocyte chemoattractant protein 1.
4 ut increased amounts of gamma interferon and monocyte chemoattractant protein 1.
5 necrosis factor-beta, interferon-gamma, and monocyte chemoattractant protein-1.
6 ming systemic inflammation was the chemokine monocyte chemoattractant protein-1.
7 their migration in response to the chemokine monocyte chemoattractant protein-1.
8 nd secretion of the proinflammatory cytokine monocyte chemoattractant protein-1.
9 th-regulated oncogene-alpha (GRO-alpha), and monocyte chemoattractant protein-1.
10 splanted visceral fat pad and reduced plasma monocyte chemoattractant protein-1.
11 cated in endogenous fat inflammation such as monocyte chemoattractant protein-1.
12 f intercellular cell adhesion molecule-1 and monocyte chemoattractant protein-1.
13 a-like ligand 4, apelin, angiopoietin-2, and monocyte chemoattractant protein-1.
14 nitric oxide synthase 3, thrombomodulin, and monocyte chemoattractant protein-1.
15 5%, P < 0.002; interleukin-6, 13%, P < 0.01; monocyte chemoattractant protein-1, 10%, P < 0.0006) and
16 or necrosis factor-alpha (-16% versus +12%), monocyte chemoattractant protein-1 (-13% versus +0.2%),
17 creases in interleukin-6 (21%; P < 0.02) and monocyte chemoattractant protein 1 (14% decrease at 4 wk
18 ange messenger RNA: interleukin-1beta = 7.6, monocyte chemoattractant protein-1 = 15.3, and tumor nec
19 ations of inflammatory biomarkers, including monocyte chemoattractant protein-1 (adjusted OR 9.0 [95%
20 ls of IL-6, tumor necrosis factor-alpha, and monocyte chemoattractant protein-1 after stimulation wit
21 increased CD68, tumor necrosis factor alpha, monocyte chemoattractant protein-1, alpha-smooth muscle
22 remote myocardium (e.g., 12-fold increase of monocyte chemoattractant protein-1), although levels wer
23 increases in CC chemokine ligand 2 (CCL2, or monocyte chemoattractant protein-1), an important macrop
25 diponectin and leptin while reducing that of monocyte chemoattractant protein 1 and interleukin-8 by
26 eotaxin, IL-2, and IL-12 and the chemokines monocyte chemoattractant protein 1 and keratinocyte-deri
27 alveolar epithelial cells produced excessive monocyte chemoattractant protein 1 and reactive oxygen s
30 flammatory factors (interleukin-6 and -8 and monocyte chemoattractant protein-1 and -2), and mobiliza
31 n the vascular wall (decreased production of monocyte chemoattractant protein-1 and activation of p38
32 ukin-6, tumor necrosis factor) and adaptive (monocyte chemoattractant protein-1 and CXCL10 chemokines
33 plasia and pro-inflammatory gene expression (monocyte chemoattractant protein-1 and cytokine-induced
34 or necrosis factor-alpha, interleukin-6, and monocyte chemoattractant protein-1 and decreased M2 mark
35 ry and proproliferative mediators, including monocyte chemoattractant protein-1 and hypoxia-inducible
36 Seeded BMCs secreted significant amounts of monocyte chemoattractant protein-1 and increased early m
37 d at the promoters of the inflammatory genes monocyte chemoattractant protein-1 and interleukin-6 in
38 in significantly reduced production of serum monocyte chemoattractant protein-1 and interleukin-6 lev
39 lial selectin surface expression and reduced monocyte chemoattractant protein-1 and interleukin-6 pro
40 -infected MSKO mouse livers had: (1) greater monocyte chemoattractant protein-1 and macrophage inflam
42 ased expression of the proinflammatory gene, monocyte chemoattractant protein-1 and matrix metallopro
43 NA levels of tumor necrosis factor-alpha and monocyte chemoattractant protein-1 and reduced mRNA and
44 25(OH)(2)D(3) supplementation also inhibited monocyte chemoattractant protein-1 and stimulated adipon
45 macrophages showed reduced migration toward monocyte chemoattractant protein-1 and transmigration th
46 uction of key proatherogenic factors such as monocyte chemoattractant protein-1 and tumor necrosis fa
48 okine (C-C motif) ligand 5 and expression of monocyte chemoattractant protein-1 and vascular cell adh
49 oliferating cell nuclear antigen+ cells, and monocyte chemoattractant protein-1 and vascular cell adh
50 sAPPbeta) and two neuroinflammatory markers (monocyte chemoattractant protein-1 and YKL-40) were meas
51 he TLR9 (toll-like receptor 9), IFNG, MCP-1 (monocyte chemoattractant protein-1) and GM-CSF genes, an
52 ed with increased renal expression of MCP-1 (monocyte chemoattractant protein-1) and VLA-4 (very-late
53 necrosis factor alpha, interleukin-6, CCL2 (monocyte chemoattractant protein 1), and CCL5 (RANTES).
54 F-beta, connective tissue growth factor, and monocyte chemoattractant protein 1), and epithelial-to-m
55 induced inflammation (tumor necrosis factor, monocyte chemoattractant protein 1, and chemokine [C-X-C
56 s of myeloperoxidase, tumor necrosis factor, monocyte chemoattractant protein 1, and gamma interferon
57 eukin 1beta (IL-1beta), IL-6, CXCL1/KC, CCL2/monocyte chemoattractant protein 1, and granulocyte colo
58 terleukin-6, keratinocyte-derived chemokine, monocyte chemoattractant protein 1, and interleukin-10),
59 e, with extremely high mRNA levels for IL-8, monocyte chemoattractant protein 1, and macrophage infla
60 asminogen activator receptor, interleukin-6, monocyte chemoattractant protein 1, and matrix metallopr
61 ene and protein expression of tissue factor, monocyte chemoattractant protein-1, and cyclooxygenase-2
62 ated lipocalin, tumor necrosis factor-alpha, monocyte chemoattractant protein-1, and fractional hypox
63 on- gamma , interleukin [IL]-4, IL-10, IL-6, monocyte chemoattractant protein-1, and growth-regulated
64 dependent increase in release of IL-6, IL-8, monocyte chemoattractant protein-1, and growth-regulated
65 with statistically different levels of IL-6, monocyte chemoattractant protein-1, and heat-shock prote
66 ed secretion of tumor necrosis factor-alpha, monocyte chemoattractant protein-1, and interleukin-12.
67 lecule-1, intercellular adhesion molecule-1, monocyte chemoattractant protein-1, and interleukin-17A;
68 increased expression of inflammatory genes, monocyte chemoattractant protein-1, and interleukin-6, a
69 pha, macrophage inflammatory protein-1alpha, monocyte chemoattractant protein-1, and keratinocyte-der
70 irculating levels of P-selectin, E-selectin, monocyte chemoattractant protein-1, and macrophage conte
71 atory cytokines tumor necrosis factor-alpha, monocyte chemoattractant protein-1, and macrophage infla
72 lating factor, keratinocyte chemoattractant, monocyte chemoattractant protein-1, and macrophage infla
75 L-8, IL-10, interferon-inducible protein-10, monocyte chemoattractant protein-1, and tumor necrosis f
76 roinflammatory cytokines (interleukin-1beta, monocyte chemoattractant protein-1, and tumor necrosis f
77 neutrophil gelatinase-associated lipocalin, monocyte chemoattractant protein-1, and tumor necrosis f
78 neutrophil gelatinase-associated lipocalin, monocyte chemoattractant protein-1, and tumor necrosis f
79 ession of intercellular adhesion molecule-1, monocyte chemoattractant protein-1, and vascular endothe
80 NA levels of tumor necrosis factor-alpha and monocyte chemoattractant protein-1 as 129/B6-ApoE(-/-) c
81 r alpha and interleukin-6) and the chemokine monocyte chemoattractant protein 1 at levels indistingui
82 ficantly with matrix metalloproteinase-3 and monocyte chemoattractant protein-1 at baseline, biomarke
83 iR-132 and demonstrated that miR-132 induces monocyte chemoattractant protein-1 at least in part via
84 ellular protease plasmin cleaves mouse MCP1 (monocyte chemoattractant protein 1) at lysine 104, relea
85 m levels of CD40 ligand, serum amyloid A and monocyte chemoattractant protein-1, (b) limited evidence
86 from WKY rats synthesized significantly more monocyte chemoattractant protein-1 basally and after sti
87 7/BL6 mice, markedly augmented the levels of monocyte chemoattractant protein-1 bound to RBCs, which
88 macrophage inflammatory protein-1alpha, and monocyte chemoattractant protein-1, but it did not exert
89 of tumor necrosis factor, interleukin-6, and monocyte chemoattractant protein 1 by spleen cells was a
90 osis factor-alpha, IL-1beta, IL-6, IL-8, and monocyte chemoattractant protein-1 by co-cultured dendri
91 IL-8, granulocyte colony-stimulating factor, monocyte chemoattractant protein-1, C-reactive protein,
92 ssive neuroblastoma, repressed expression of monocyte chemoattractant protein-1/CC chemokine ligand 2
93 terferon [IFN-gamma], and IL-6), chemokines (monocyte chemoattractant protein 1/CCL-2, macrophage inf
95 ll alpha chemoattractant (I-TAC/CXCL11), and monocyte chemoattractant protein 1 (CCL2) were measured
96 CD163 (sCD163) and soluble CD14 (sCD14), and monocyte chemoattractant protein 1 (CCL2) with subclinic
97 d PGE(2)-induced production of the chemokine monocyte chemoattractant protein-1 (CCL2), which was lin
98 an IC50 of 22.8 nM but was inactive against monocyte chemoattractant protein-1 (CCL2)-mediated calci
99 d-brain barrier leakage, induction of MCP-1 (monocyte chemoattractant protein 1) (CCL2), ICAM-1 (inte
100 of histamine, cytokines, and the chemokines monocyte chemoattractant protein 1/CCL2, macrophage infl
101 n stimulation with CXCL16 astrocytes release monocyte chemoattractant protein-1/CCL2 and (2) the neur
102 crosis factor-alpha) and chemokines (such as monocyte chemoattractant protein-1/ chemokine (C-C motif
103 biomarkers (glial fibrillary acidic protein, monocyte chemoattractant protein 1/chemokine (C-C motif)
104 glial fibrillary acidic protein (p = 0.002), monocyte chemoattractant protein 1/chemokine (C-C motif)
105 leukin-8/C-X-C motif chemokine ligand 8, and monocyte chemoattractant protein-1/chemokine ligand 2 in
106 8/C-X-C motif chemokine ligand 8 P=0.04, and monocyte chemoattractant protein-1/chemokine ligand 2 P=
107 ts contained increased leptin, resistin, and monocyte chemoattractant protein-1 compared with plasma
108 12p40, interferon-inducible protein 10, and monocyte chemoattractant protein 1 concentrations, where
110 growth factor, hepatocyte growth factor and monocyte chemoattractant protein-1, contributing to deve
111 enes such as tumor necrosis factor-alpha and monocyte chemoattractant protein-1, decreased AT inflamm
112 of THP-1 monocytes to migrate toward MCP-1 (monocyte chemoattractant protein 1) depended upon Par3 a
113 nside-out activation of beta(2) integrins by monocyte chemoattractant protein-1 did not change IL-13-
115 in 6, macrophage inflammatory protein 2, and monocyte chemoattractant protein 1 expression as determi
116 y reduced hepatic inflammation, particularly monocyte chemoattractant protein-1 expression and macrop
117 ell as vascular cell adhesion molecule-1 and monocyte chemoattractant protein-1 expression in endothe
118 n, normal T cell expressed and secreted) and monocyte chemoattractant protein-1 expression induced by
121 proinflammatory mediators interleukin-6 and monocyte chemoattractant protein-1, fibroblast growth fa
123 nterferon gamma-inducible protein of 10 kDa, monocyte chemoattractant protein 1, growth-related oncog
124 binding oligomerization domain containing-2, monocyte chemoattractant protein-1, IL-2, and IL-12p40 i
125 micked by stimulation of Hmox1(+/+) SCs with monocyte chemoattractant protein-1, IL-6, IL-1beta, and
126 tate dehydrogenase, as well as expression of monocyte chemoattractant protein-1, IL-6, IL-1beta, and
127 NA levels of tumor necrosis factor-alpha and monocyte chemoattractant protein 1 in lumbar spinal cord
128 hyper-IgE syndrome generated lower levels of monocyte chemoattractant protein 1 in response to the pr
129 okines, including keratinocyte chemokine and monocyte chemoattractant protein 1 in toxin A-exposed mi
130 n, normal T cell expressed and secreted) and monocyte chemoattractant protein-1 in a mouse model of o
131 tic lesion areas and decreased expression of monocyte chemoattractant protein-1 in the aorta as compa
134 GE and other proinflammatory genes including monocyte chemoattractant protein-1, interferon-gamma-ind
135 accumulation, apoptosis, necrotic cores, and monocyte chemoattractant protein 1, interleukin 1beta, i
136 inflammatory response through alteration of monocyte chemoattractant protein-1, interleukin-1beta, a
137 of cytokines keratinocyte-derived chemokine, monocyte chemoattractant protein-1, interleukin-6 (IL-6)
138 lasminogen activator inhibitor-1, TNF-alpha, monocyte chemoattractant protein-1, interleukin-6, and k
139 m TK-/- mice exhibited blunted production of monocyte chemoattractant protein-1, interleukin-6, and m
140 oduction of cytokines and chemokines, namely monocyte chemoattractant protein-1, interleukin-6, and m
141 icantly higher levels of expression of cKIT, monocyte chemoattractant protein-1, interleukin-6, strom
142 ion, as denoted by the reduced expression of monocyte chemoattractant protein-1, intracellular adhesi
144 cytokines interleukin-6, interleukin-8, and monocyte chemoattractant protein-1, is markedly increase
145 ial protein expression of interleukin-18 and monocyte chemoattractant protein-1, key mediators of car
146 ction was strongly correlated with increased monocyte chemoattractant protein 1 levels (r = 0.396, P
147 eased plasma tumor necrosis factor-alpha and monocyte chemoattractant protein-1 levels in Tg-hCBS apo
148 igation and puncture, and interleukin 10 and monocyte chemoattractant protein-1 levels were higher in
151 ing factor were significantly reduced, while monocyte chemoattractant protein 1, macrophage inflammat
152 es were induced to produce chemokines (e.g., monocyte chemoattractant protein 1, macrophage inflammat
153 protein-1alpha, and C-reactive protein, and monocyte chemoattractant protein-1, macrophage inflammat
155 terferon-gamma inducible protein-10 [IP-10], monocyte chemoattractant protein-1, macrophage inflammat
156 ngII-induced expression of cyclooxygenase-2, monocyte chemoattractant protein-1, macrophage inflammat
157 ing tumor necrosis factor alpha (TNF-alpha); monocyte chemoattractant protein 1; macrophage inflammat
158 heal had higher tumor necrosis factor-alpha, monocyte chemoattractant protein-1, matrix metallopeptid
159 ssion of the fibrocyte recruiting chemokines monocyte chemoattractant protein 1 (MCP-1) and CXCL12, a
160 ed, there was a significant decrease in CCL2/monocyte chemoattractant protein 1 (MCP-1) and inflammat
161 stability of tumor necrosis factor alpha and monocyte chemoattractant protein 1 (MCP-1) but strongly
162 low-density lipoprotein (ox-LDL)-stimulated monocyte chemoattractant protein 1 (MCP-1) from macropha
163 d various levels of interleukin-8 (IL-8) and monocyte chemoattractant protein 1 (MCP-1) in response t
164 lammatory cytokines interleukin-8 (IL-8) and monocyte chemoattractant protein 1 (MCP-1) in response t
165 ssion of the inflammatory mediators CD36 and monocyte chemoattractant protein 1 (MCP-1) in the brain
173 ar adhesion molecule 1 (ICAM-1), E-selectin, monocyte chemoattractant protein 1 (MCP-1), and interleu
175 ter infection (day 2), interleukin 6 (IL-6), monocyte chemoattractant protein 1 (MCP-1), macrophage i
176 anulocyte colony-stimulating factor (G-CSF), monocyte chemoattractant protein 1 (MCP-1), macrophage i
177 s of IFN-gamma-inducible protein 10 (IP-10), monocyte chemoattractant protein 1 (MCP-1), macrophage i
178 IL-12, and IL-18; chemokines, such as IL-8, monocyte chemoattractant protein 1 (MCP-1), RANTES, and
179 rogenase, and inflammatory mediators such as monocyte chemoattractant protein 1 (MCP-1), TNF-alpha, a
182 , we show that T. cruzi strongly upregulates monocyte chemoattractant protein 1 (MCP-1)/CCL2 and frac
183 l-derived factor 1alpha (SDF-1alpha)/CXCL12, monocyte chemoattractant protein 1 (MCP-1)/CCL2, and vas
185 , S1P stimulated secretion of the chemokine, monocyte chemoattractant protein 1 (MCP-1/CCL2), from th
186 ctants were induced in the kidney, including monocyte chemoattractant protein 1 (MCP-1/CCL2), macroph
189 ion, associated with 38% reduced circulating monocyte chemoattractant protein-1 (MCP-1) and 36% lower
190 6), tumor necrosis factor-alpha (TNF-alpha), monocyte chemoattractant protein-1 (MCP-1) and C-reactiv
192 ited TNF-alpha-induced inflammatory factors, monocyte chemoattractant protein-1 (MCP-1) and interleuk
193 so increased levels of inflammatory cytokine monocyte chemoattractant protein-1 (MCP-1) and MCP-1 ind
194 trong association between the stimulation of monocyte chemoattractant protein-1 (MCP-1) and the anabo
195 h as interleukin (IL)-6, IL-1beta, IL-8, and monocyte chemoattractant protein-1 (MCP-1) and the secre
196 d and is accompanied by increases in mRNA of monocyte chemoattractant protein-1 (MCP-1) and tumor nec
197 VSMC stimulated by TGF-beta/AdSmad3 revealed monocyte chemoattractant protein-1 (MCP-1) as a likely f
198 pirfenidone impaired macrophage migration to monocyte chemoattractant protein-1 (MCP-1) as well as IL
199 an obligate dimeric mutant of the chemokine monocyte chemoattractant protein-1 (MCP-1) by substituti
201 uced Src and STAT3 tyrosine phosphorylation, monocyte chemoattractant protein-1 (MCP-1) expression an
202 JNK as the exclusive mediator of FFA-induced monocyte chemoattractant protein-1 (MCP-1) expression in
204 mation, in association with higher levels of monocyte chemoattractant protein-1 (MCP-1) expression.
206 atment with p38 MAPK inhibitor reduced renal monocyte chemoattractant protein-1 (MCP-1) levels, the n
207 a murine model of an arteriovenous fistula, monocyte chemoattractant protein-1 (MCP-1) mRNA and prot
213 ile inducing inflammatory cytokine (IL-6 and monocyte chemoattractant protein-1 (MCP-1)) expression.
214 mobility mass spectrometry (IMMS) to analyze monocyte chemoattractant protein-1 (MCP-1), a CC chemoki
216 in HK-2 cells to stimulate the production of monocyte chemoattractant protein-1 (MCP-1), a key chemok
217 -1beta (IL-1beta), the cytokines IL-8, IL-6, monocyte chemoattractant protein-1 (MCP-1), and growth-r
218 yperactivation of ERK and p38 in response to monocyte chemoattractant protein-1 (MCP-1), and increase
219 phage accumulation, diminished expression of monocyte chemoattractant protein-1 (MCP-1), and lower le
220 activation-regulated chemokine (TARC), IL-8, monocyte chemoattractant protein-1 (MCP-1), and murine b
221 Stroke outcome, expression of brain CD36, monocyte chemoattractant protein-1 (MCP-1), CCR2, and pl
224 d beta2-integrins, cyclooxygenase-2 (COX-2), monocyte chemoattractant protein-1 (MCP-1), interleukin-
225 We have previously shown that the chemokine, monocyte chemoattractant protein-1 (MCP-1), is a mediato
227 ge inflammatory protein-1alpha (MIP-1alpha), monocyte chemoattractant protein-1 (MCP-1), regulated on
228 n-gamma-inducible protein of 10 kDa (IP-10), monocyte chemoattractant protein-1 (MCP-1), tumor necros
229 netic basis of circulating concentrations of monocyte chemoattractant protein-1 (MCP-1), we conducted
236 chemotaxis can be signaled by the chemokine monocyte chemoattractant protein-1 (MCP-1)/CCL2 (CC chem
237 that interact with the oligomeric chemokine Monocyte Chemoattractant Protein-1 (MCP-1)/CCL2 with dif
238 rated increased expression of iNOS, C1r, and monocyte chemoattractant protein-1 (MCP-1); MCP-1 expres
239 RK) to mechanically trigger the secretion of monocyte chemoattractant protein-1 (MCP-1, also known as
240 ort the expression profile of the chemokine, monocyte chemoattractant protein-1 (MCP-1, CCL2), during
242 to chronic pain includes the upregulation of monocyte chemoattractant protein-1 (MCP-1/CCL2) and its
243 proinflammatory cytokine IL-6 and chemokine monocyte chemoattractant protein-1 (MCP-1/CCL2) in respo
244 keratinocytes up-regulated the expression of monocyte chemoattractant protein-1 (MCP-1/CCL2), TNFalph
247 mor necrosis factor alpha (TNF-alpha), CCL2 (monocyte chemoattractant protein 1 [MCP-1]), and CCL5 (R
249 ines (interleukin 6 [IL-6], IL-8, IL-1alpha, monocyte chemoattractant protein 1 [MCP-1], and colony-s
250 atory cytokines (interleukin-6 [IL-6], IL-8, monocyte chemoattractant protein 1 [MCP-1], and IL-1beta
251 we show that TXA(2) mimetic, I-BOP, induced monocyte chemoattractant protein -1(MCP-1)/chemokine (C-
252 ce was observed, certain chemokines (RANTES, monocyte chemoattractant protein 1[MCP-1], and IP-10) we
253 eoprotegerin (OPG) expression and increasing monocyte chemoattractant protein 1 (MCP1) expression in
254 rleukin-6 (Il-6), interleukin-1beta (Il-1b), monocyte chemoattractant protein 1 (Mcp1), and fibrosis-
255 ic pain, and mice overexpressing its ligand, monocyte chemoattractant protein-1 (MCP1; also known as
256 ential of mouse melanoma cells in HDAC3- and monocyte chemoattractant protein 1-(MCP1)-dependent mann
257 CI and other wild type CC chemokines, MCP-1 (monocyte chemoattractant protein-1), MIP-1beta, and RANT
258 10]), and proinflammatory cytokines (MCP-1 [monocyte chemoattractant protein 1], MIP-1alpha/beta [ma
259 essed and secreted, T-cell activation-3, and monocyte chemoattractant protein-1 mRNAs were lower comp
263 kin-6 (P=0.01), isoprostanes (P=0.0002), and monocyte chemoattractant protein-1 (P=0.008); SAT only r
264 proteinase 9, metalloproteinase inhibitor 1, monocyte chemoattractant protein-1, P-selectin, fibrinog
265 Expression of tumor necrosis factor-alpha, monocyte chemoattractant protein-1, plasminogen activato
266 e (C-C motif) ligand 2 (CCL2), also known as monocyte chemoattractant protein-1, plays a critical rol
267 with increased lipopolysaccharide-stimulated monocyte chemoattractant protein 1 production by JR-CSF
268 educed IL-4, IL-5, IL-13, eotaxin, IL-8, and monocyte chemoattractant protein 1 production without af
269 found a 3-fold increase in interleukin-6 and monocyte chemoattractant protein-1 production by G2A(-/-
270 Pio also did not attenuate Ang II-induced monocyte chemoattractant protein-1 production in PPARgam
271 responses to TLR2 and TLR4 ligands, reduced monocyte chemoattractant protein-1 production, and preve
272 ammation, limits neutrophils recruitment and monocyte chemoattractant protein-1 production, thus redu
274 cts of Klotho signaling on interleukin-8 and monocyte chemoattractant protein-1 promoter recruitment
276 isoprostanes, R2 0.07 versus 0.10, P=0.002; monocyte chemoattractant protein-1, R2 0.07 versus 0.08,
277 ed with the changes of tubular expression of monocyte chemoattractant protein-1, RANTES (regulated up
278 sis included higher levels of interleukin-8, monocyte chemoattractant protein-1, resistin, soluble in
279 ophage inflammatory protein 1alpha/beta, and monocyte chemoattractant protein 1, respectively, as det
280 and 1/lymphotactin and CC chemokine ligand 2/monocyte chemoattractant protein 1 reveal that invariant
281 Given its unique role, future studies into monocyte chemoattractant protein-1's exact role during s
282 hly associated with urinary isoprostanes and monocyte chemoattractant protein-1 (SAT versus VAT compa
283 sociated phospholipase A2 mass and activity, monocyte chemoattractant protein-1, soluble endothelial
284 methylarginine, tumor necrosis factor-alpha, monocyte chemoattractant protein-1, soluble vascular cel
285 toreceptor cultures exposed to starvation or monocyte chemoattractant protein-1-stimulated (MCP-1-sti
286 yed similar up-regulation of miR-132/212 and monocyte chemoattractant protein-1, supporting in vivo r
287 This was associated with marked increase in monocyte chemoattractant protein-1 synthesis in WKY glom
288 emokine, C-C motif chemokine ligand 2 (CCL2; monocyte chemoattractant protein 1), termed mNOX-E36, in
289 erestingly, higher levels of interleukin 22, monocyte chemoattractant protein 1, TNF-alpha, and IP-10
290 in systolic BP, heart rate variability, and monocyte chemoattractant protein-1, together with reduce
291 nes for interleukin (IL)-1beta, IL-6, IL-10, monocyte chemoattractant protein-1, tumor necrosis facto
293 ray indicated that the chemokine CCL2/MCP-1 (monocyte chemoattractant protein-1) was strongly induced
294 vels of IFN-gamma, interleukin-6 (IL-6), and monocyte chemoattractant protein 1 were detected in the
295 ), monokine induced by interferon-gamma, and monocyte chemoattractant protein 1 were quantified as me
296 itive cells) and production of the chemokine monocyte chemoattractant protein-1 were significantly bl
297 , intercellular cell adhesion molecule-1 and monocyte chemoattractant protein-1, were also determined
298 idenced by the upregulation of ephrin B2 and monocyte chemoattractant protein-1, which are 2 stretch-
299 oprotein 1 stimulates macrophages to secrete monocyte chemoattractant protein-1, which then activates
300 nd decreased production of oxidant-inducible monocyte chemoattractant protein-1, which we have previo
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