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1 iated inflammatory mediators (in particular, monocyte chemoattractant protein-1).
2 eta), tumor necrosis factor (TNF), IL-6, and monocyte chemoattractant protein 1.
3 ression of proinflammatory interleukin 6 and monocyte chemoattractant protein 1.
4 ut increased amounts of gamma interferon and monocyte chemoattractant protein 1.
5  necrosis factor-beta, interferon-gamma, and monocyte chemoattractant protein-1.
6 ming systemic inflammation was the chemokine monocyte chemoattractant protein-1.
7 their migration in response to the chemokine monocyte chemoattractant protein-1.
8 nd secretion of the proinflammatory cytokine monocyte chemoattractant protein-1.
9 th-regulated oncogene-alpha (GRO-alpha), and monocyte chemoattractant protein-1.
10 splanted visceral fat pad and reduced plasma monocyte chemoattractant protein-1.
11 cated in endogenous fat inflammation such as monocyte chemoattractant protein-1.
12 f intercellular cell adhesion molecule-1 and monocyte chemoattractant protein-1.
13 a-like ligand 4, apelin, angiopoietin-2, and monocyte chemoattractant protein-1.
14 nitric oxide synthase 3, thrombomodulin, and monocyte chemoattractant protein-1.
15 5%, P < 0.002; interleukin-6, 13%, P < 0.01; monocyte chemoattractant protein-1, 10%, P < 0.0006) and
16 or necrosis factor-alpha (-16% versus +12%), monocyte chemoattractant protein-1 (-13% versus +0.2%),
17 creases in interleukin-6 (21%; P < 0.02) and monocyte chemoattractant protein 1 (14% decrease at 4 wk
18 ange messenger RNA: interleukin-1beta = 7.6, monocyte chemoattractant protein-1 = 15.3, and tumor nec
19 ations of inflammatory biomarkers, including monocyte chemoattractant protein-1 (adjusted OR 9.0 [95%
20 ls of IL-6, tumor necrosis factor-alpha, and monocyte chemoattractant protein-1 after stimulation wit
21 increased CD68, tumor necrosis factor alpha, monocyte chemoattractant protein-1, alpha-smooth muscle
22 remote myocardium (e.g., 12-fold increase of monocyte chemoattractant protein-1), although levels wer
23 increases in CC chemokine ligand 2 (CCL2, or monocyte chemoattractant protein-1), an important macrop
24 adhesion molecule 1, soluble E-selectin, and monocyte chemoattractant protein 1 and cancer risk.
25 diponectin and leptin while reducing that of monocyte chemoattractant protein 1 and interleukin-8 by
26  eotaxin, IL-2, and IL-12 and the chemokines monocyte chemoattractant protein 1 and keratinocyte-deri
27 alveolar epithelial cells produced excessive monocyte chemoattractant protein 1 and reactive oxygen s
28                   Systemic LDL oxidation and monocyte chemoattractant protein 1 and the expression of
29      In RAW264.7 cells, allosamidin elevated monocyte chemoattractant protein 1 and tumor necrosis fa
30 flammatory factors (interleukin-6 and -8 and monocyte chemoattractant protein-1 and -2), and mobiliza
31 n the vascular wall (decreased production of monocyte chemoattractant protein-1 and activation of p38
32 ukin-6, tumor necrosis factor) and adaptive (monocyte chemoattractant protein-1 and CXCL10 chemokines
33 plasia and pro-inflammatory gene expression (monocyte chemoattractant protein-1 and cytokine-induced
34 or necrosis factor-alpha, interleukin-6, and monocyte chemoattractant protein-1 and decreased M2 mark
35 ry and proproliferative mediators, including monocyte chemoattractant protein-1 and hypoxia-inducible
36  Seeded BMCs secreted significant amounts of monocyte chemoattractant protein-1 and increased early m
37 d at the promoters of the inflammatory genes monocyte chemoattractant protein-1 and interleukin-6 in
38 in significantly reduced production of serum monocyte chemoattractant protein-1 and interleukin-6 lev
39 lial selectin surface expression and reduced monocyte chemoattractant protein-1 and interleukin-6 pro
40 -infected MSKO mouse livers had: (1) greater monocyte chemoattractant protein-1 and macrophage inflam
41                 Unlike WT mice, induction of monocyte chemoattractant protein-1 and macrophage migrat
42 ased expression of the proinflammatory gene, monocyte chemoattractant protein-1 and matrix metallopro
43 NA levels of tumor necrosis factor-alpha and monocyte chemoattractant protein-1 and reduced mRNA and
44 25(OH)(2)D(3) supplementation also inhibited monocyte chemoattractant protein-1 and stimulated adipon
45  macrophages showed reduced migration toward monocyte chemoattractant protein-1 and transmigration th
46 uction of key proatherogenic factors such as monocyte chemoattractant protein-1 and tumor necrosis fa
47 h plaque stabilization and downregulation of monocyte chemoattractant protein-1 and ubiquitin.
48 okine (C-C motif) ligand 5 and expression of monocyte chemoattractant protein-1 and vascular cell adh
49 oliferating cell nuclear antigen+ cells, and monocyte chemoattractant protein-1 and vascular cell adh
50 sAPPbeta) and two neuroinflammatory markers (monocyte chemoattractant protein-1 and YKL-40) were meas
51 he TLR9 (toll-like receptor 9), IFNG, MCP-1 (monocyte chemoattractant protein-1) and GM-CSF genes, an
52 ed with increased renal expression of MCP-1 (monocyte chemoattractant protein-1) and VLA-4 (very-late
53  necrosis factor alpha, interleukin-6, CCL2 (monocyte chemoattractant protein 1), and CCL5 (RANTES).
54 F-beta, connective tissue growth factor, and monocyte chemoattractant protein 1), and epithelial-to-m
55 induced inflammation (tumor necrosis factor, monocyte chemoattractant protein 1, and chemokine [C-X-C
56 s of myeloperoxidase, tumor necrosis factor, monocyte chemoattractant protein 1, and gamma interferon
57 eukin 1beta (IL-1beta), IL-6, CXCL1/KC, CCL2/monocyte chemoattractant protein 1, and granulocyte colo
58 terleukin-6, keratinocyte-derived chemokine, monocyte chemoattractant protein 1, and interleukin-10),
59 e, with extremely high mRNA levels for IL-8, monocyte chemoattractant protein 1, and macrophage infla
60 asminogen activator receptor, interleukin-6, monocyte chemoattractant protein 1, and matrix metallopr
61 ene and protein expression of tissue factor, monocyte chemoattractant protein-1, and cyclooxygenase-2
62 ated lipocalin, tumor necrosis factor-alpha, monocyte chemoattractant protein-1, and fractional hypox
63 on- gamma , interleukin [IL]-4, IL-10, IL-6, monocyte chemoattractant protein-1, and growth-regulated
64 dependent increase in release of IL-6, IL-8, monocyte chemoattractant protein-1, and growth-regulated
65 with statistically different levels of IL-6, monocyte chemoattractant protein-1, and heat-shock prote
66 ed secretion of tumor necrosis factor-alpha, monocyte chemoattractant protein-1, and interleukin-12.
67 lecule-1, intercellular adhesion molecule-1, monocyte chemoattractant protein-1, and interleukin-17A;
68  increased expression of inflammatory genes, monocyte chemoattractant protein-1, and interleukin-6, a
69 pha, macrophage inflammatory protein-1alpha, monocyte chemoattractant protein-1, and keratinocyte-der
70 irculating levels of P-selectin, E-selectin, monocyte chemoattractant protein-1, and macrophage conte
71 atory cytokines tumor necrosis factor-alpha, monocyte chemoattractant protein-1, and macrophage infla
72 lating factor, keratinocyte chemoattractant, monocyte chemoattractant protein-1, and macrophage infla
73 , soluble intercellular adhesion molecule-1, monocyte chemoattractant protein-1, and P-selectin.
74                         Suppression of ED-1, monocyte chemoattractant protein-1, and RANTES expressio
75 L-8, IL-10, interferon-inducible protein-10, monocyte chemoattractant protein-1, and tumor necrosis f
76 roinflammatory cytokines (interleukin-1beta, monocyte chemoattractant protein-1, and tumor necrosis f
77  neutrophil gelatinase-associated lipocalin, monocyte chemoattractant protein-1, and tumor necrosis f
78  neutrophil gelatinase-associated lipocalin, monocyte chemoattractant protein-1, and tumor necrosis f
79 ession of intercellular adhesion molecule-1, monocyte chemoattractant protein-1, and vascular endothe
80 NA levels of tumor necrosis factor-alpha and monocyte chemoattractant protein-1 as 129/B6-ApoE(-/-) c
81 r alpha and interleukin-6) and the chemokine monocyte chemoattractant protein 1 at levels indistingui
82 ficantly with matrix metalloproteinase-3 and monocyte chemoattractant protein-1 at baseline, biomarke
83 iR-132 and demonstrated that miR-132 induces monocyte chemoattractant protein-1 at least in part via
84 ellular protease plasmin cleaves mouse MCP1 (monocyte chemoattractant protein 1) at lysine 104, relea
85 m levels of CD40 ligand, serum amyloid A and monocyte chemoattractant protein-1, (b) limited evidence
86 from WKY rats synthesized significantly more monocyte chemoattractant protein-1 basally and after sti
87 7/BL6 mice, markedly augmented the levels of monocyte chemoattractant protein-1 bound to RBCs, which
88  macrophage inflammatory protein-1alpha, and monocyte chemoattractant protein-1, but it did not exert
89 of tumor necrosis factor, interleukin-6, and monocyte chemoattractant protein 1 by spleen cells was a
90 osis factor-alpha, IL-1beta, IL-6, IL-8, and monocyte chemoattractant protein-1 by co-cultured dendri
91 IL-8, granulocyte colony-stimulating factor, monocyte chemoattractant protein-1, C-reactive protein,
92 ssive neuroblastoma, repressed expression of monocyte chemoattractant protein-1/CC chemokine ligand 2
93 terferon [IFN-gamma], and IL-6), chemokines (monocyte chemoattractant protein 1/CCL-2, macrophage inf
94                                              Monocyte chemoattractant protein 1 (CCL2) is a recently
95 ll alpha chemoattractant (I-TAC/CXCL11), and monocyte chemoattractant protein 1 (CCL2) were measured
96 CD163 (sCD163) and soluble CD14 (sCD14), and monocyte chemoattractant protein 1 (CCL2) with subclinic
97 d PGE(2)-induced production of the chemokine monocyte chemoattractant protein-1 (CCL2), which was lin
98  an IC50 of 22.8 nM but was inactive against monocyte chemoattractant protein-1 (CCL2)-mediated calci
99 d-brain barrier leakage, induction of MCP-1 (monocyte chemoattractant protein 1) (CCL2), ICAM-1 (inte
100  of histamine, cytokines, and the chemokines monocyte chemoattractant protein 1/CCL2, macrophage infl
101 n stimulation with CXCL16 astrocytes release monocyte chemoattractant protein-1/CCL2 and (2) the neur
102 crosis factor-alpha) and chemokines (such as monocyte chemoattractant protein-1/ chemokine (C-C motif
103 biomarkers (glial fibrillary acidic protein, monocyte chemoattractant protein 1/chemokine (C-C motif)
104 glial fibrillary acidic protein (p = 0.002), monocyte chemoattractant protein 1/chemokine (C-C motif)
105 leukin-8/C-X-C motif chemokine ligand 8, and monocyte chemoattractant protein-1/chemokine ligand 2 in
106 8/C-X-C motif chemokine ligand 8 P=0.04, and monocyte chemoattractant protein-1/chemokine ligand 2 P=
107 ts contained increased leptin, resistin, and monocyte chemoattractant protein-1 compared with plasma
108  12p40, interferon-inducible protein 10, and monocyte chemoattractant protein 1 concentrations, where
109                                              Monocyte chemoattractant protein-1 concentrations in nas
110  growth factor, hepatocyte growth factor and monocyte chemoattractant protein-1, contributing to deve
111 enes such as tumor necrosis factor-alpha and monocyte chemoattractant protein-1, decreased AT inflamm
112  of THP-1 monocytes to migrate toward MCP-1 (monocyte chemoattractant protein 1) depended upon Par3 a
113 nside-out activation of beta(2) integrins by monocyte chemoattractant protein-1 did not change IL-13-
114            In vitro, IL-6 markedly increased monocyte chemoattractant protein-1-driven monocyte-to-my
115 in 6, macrophage inflammatory protein 2, and monocyte chemoattractant protein 1 expression as determi
116 y reduced hepatic inflammation, particularly monocyte chemoattractant protein-1 expression and macrop
117 ell as vascular cell adhesion molecule-1 and monocyte chemoattractant protein-1 expression in endothe
118 n, normal T cell expressed and secreted) and monocyte chemoattractant protein-1 expression induced by
119 tin on vascular cell adhesion molecule-1 and monocyte chemoattractant protein-1 expression.
120 ating vascular cell adhesion molecule -1 and monocyte chemoattractant protein-1 expressions.
121  proinflammatory mediators interleukin-6 and monocyte chemoattractant protein-1, fibroblast growth fa
122                                          The monocyte chemoattractant protein 1 gene (MCP-1) is regul
123 nterferon gamma-inducible protein of 10 kDa, monocyte chemoattractant protein 1, growth-related oncog
124 binding oligomerization domain containing-2, monocyte chemoattractant protein-1, IL-2, and IL-12p40 i
125 micked by stimulation of Hmox1(+/+) SCs with monocyte chemoattractant protein-1, IL-6, IL-1beta, and
126 tate dehydrogenase, as well as expression of monocyte chemoattractant protein-1, IL-6, IL-1beta, and
127 NA levels of tumor necrosis factor-alpha and monocyte chemoattractant protein 1 in lumbar spinal cord
128 hyper-IgE syndrome generated lower levels of monocyte chemoattractant protein 1 in response to the pr
129 okines, including keratinocyte chemokine and monocyte chemoattractant protein 1 in toxin A-exposed mi
130 n, normal T cell expressed and secreted) and monocyte chemoattractant protein-1 in a mouse model of o
131 tic lesion areas and decreased expression of monocyte chemoattractant protein-1 in the aorta as compa
132 amma, tumor necrosis factor-alpha, IL-8, and monocyte chemoattractant protein-1 in the spleen.
133       Five inflammatory markers (IL-6, IL-8, monocyte chemoattractant protein-1, interferon-gamma-ind
134 GE and other proinflammatory genes including monocyte chemoattractant protein-1, interferon-gamma-ind
135 accumulation, apoptosis, necrotic cores, and monocyte chemoattractant protein 1, interleukin 1beta, i
136  inflammatory response through alteration of monocyte chemoattractant protein-1, interleukin-1beta, a
137 of cytokines keratinocyte-derived chemokine, monocyte chemoattractant protein-1, interleukin-6 (IL-6)
138 lasminogen activator inhibitor-1, TNF-alpha, monocyte chemoattractant protein-1, interleukin-6, and k
139 m TK-/- mice exhibited blunted production of monocyte chemoattractant protein-1, interleukin-6, and m
140 oduction of cytokines and chemokines, namely monocyte chemoattractant protein-1, interleukin-6, and m
141 icantly higher levels of expression of cKIT, monocyte chemoattractant protein-1, interleukin-6, strom
142 ion, as denoted by the reduced expression of monocyte chemoattractant protein-1, intracellular adhesi
143                                              Monocyte chemoattractant protein-1 is a chemokine recrui
144  cytokines interleukin-6, interleukin-8, and monocyte chemoattractant protein-1, is markedly increase
145 ial protein expression of interleukin-18 and monocyte chemoattractant protein-1, key mediators of car
146 ction was strongly correlated with increased monocyte chemoattractant protein 1 levels (r = 0.396, P
147 eased plasma tumor necrosis factor-alpha and monocyte chemoattractant protein-1 levels in Tg-hCBS apo
148 igation and puncture, and interleukin 10 and monocyte chemoattractant protein-1 levels were higher in
149  indicated Cr elevations correlated with CSF monocyte chemoattractant protein-1 levels.
150 ed plasma interleukin-6, interleukin-10, and monocyte chemoattractant protein-1 levels.
151 ing factor were significantly reduced, while monocyte chemoattractant protein 1, macrophage inflammat
152 es were induced to produce chemokines (e.g., monocyte chemoattractant protein 1, macrophage inflammat
153  protein-1alpha, and C-reactive protein, and monocyte chemoattractant protein-1, macrophage inflammat
154                              Upregulation of monocyte chemoattractant protein-1, macrophage inflammat
155 terferon-gamma inducible protein-10 [IP-10], monocyte chemoattractant protein-1, macrophage inflammat
156 ngII-induced expression of cyclooxygenase-2, monocyte chemoattractant protein-1, macrophage inflammat
157 ing tumor necrosis factor alpha (TNF-alpha); monocyte chemoattractant protein 1; macrophage inflammat
158 heal had higher tumor necrosis factor-alpha, monocyte chemoattractant protein-1, matrix metallopeptid
159 ssion of the fibrocyte recruiting chemokines monocyte chemoattractant protein 1 (MCP-1) and CXCL12, a
160 ed, there was a significant decrease in CCL2/monocyte chemoattractant protein 1 (MCP-1) and inflammat
161 stability of tumor necrosis factor alpha and monocyte chemoattractant protein 1 (MCP-1) but strongly
162  low-density lipoprotein (ox-LDL)-stimulated monocyte chemoattractant protein 1 (MCP-1) from macropha
163 d various levels of interleukin-8 (IL-8) and monocyte chemoattractant protein 1 (MCP-1) in response t
164 lammatory cytokines interleukin-8 (IL-8) and monocyte chemoattractant protein 1 (MCP-1) in response t
165 ssion of the inflammatory mediators CD36 and monocyte chemoattractant protein 1 (MCP-1) in the brain
166                                        While monocyte chemoattractant protein 1 (MCP-1) is known to b
167                                              Monocyte chemoattractant protein 1 (MCP-1) mediates acut
168                       Although the M-CSF and monocyte chemoattractant protein 1 (MCP-1) mRNA levels w
169                                              Monocyte chemoattractant protein 1 (MCP-1) peaked postdi
170                                              Monocyte chemoattractant protein 1 (MCP-1) plays a pivot
171                    We showed previously that monocyte chemoattractant protein 1 (MCP-1) was a critica
172            Recently, increased expression of monocyte chemoattractant protein 1 (MCP-1) was reported
173 ar adhesion molecule 1 (ICAM-1), E-selectin, monocyte chemoattractant protein 1 (MCP-1), and interleu
174                  Of the 21 cytokines tested, monocyte chemoattractant protein 1 (MCP-1), keratinocyte
175 ter infection (day 2), interleukin 6 (IL-6), monocyte chemoattractant protein 1 (MCP-1), macrophage i
176 anulocyte colony-stimulating factor (G-CSF), monocyte chemoattractant protein 1 (MCP-1), macrophage i
177 s of IFN-gamma-inducible protein 10 (IP-10), monocyte chemoattractant protein 1 (MCP-1), macrophage i
178  IL-12, and IL-18; chemokines, such as IL-8, monocyte chemoattractant protein 1 (MCP-1), RANTES, and
179 rogenase, and inflammatory mediators such as monocyte chemoattractant protein 1 (MCP-1), TNF-alpha, a
180 lammatory mediators, including TNF-alpha and monocyte chemoattractant protein 1 (MCP-1).
181 gration via increased expression of IL-6 and monocyte chemoattractant protein 1 (MCP-1).
182 , we show that T. cruzi strongly upregulates monocyte chemoattractant protein 1 (MCP-1)/CCL2 and frac
183 l-derived factor 1alpha (SDF-1alpha)/CXCL12, monocyte chemoattractant protein 1 (MCP-1)/CCL2, and vas
184                                              Monocyte chemoattractant protein 1 (MCP-1/CCL2) is a pro
185 , S1P stimulated secretion of the chemokine, monocyte chemoattractant protein 1 (MCP-1/CCL2), from th
186 ctants were induced in the kidney, including monocyte chemoattractant protein 1 (MCP-1/CCL2), macroph
187                 Recently, we have identified monocyte chemoattractant protein 1 (MCP-1; CCL2) as a pr
188                                              Monocyte chemoattractant protein-1 (MCP-1 or CCL2) regul
189 ion, associated with 38% reduced circulating monocyte chemoattractant protein-1 (MCP-1) and 36% lower
190 6), tumor necrosis factor-alpha (TNF-alpha), monocyte chemoattractant protein-1 (MCP-1) and C-reactiv
191                   Very high plasma levels of monocyte chemoattractant protein-1 (MCP-1) and interleuk
192 ited TNF-alpha-induced inflammatory factors, monocyte chemoattractant protein-1 (MCP-1) and interleuk
193 so increased levels of inflammatory cytokine monocyte chemoattractant protein-1 (MCP-1) and MCP-1 ind
194 trong association between the stimulation of monocyte chemoattractant protein-1 (MCP-1) and the anabo
195 h as interleukin (IL)-6, IL-1beta, IL-8, and monocyte chemoattractant protein-1 (MCP-1) and the secre
196 d and is accompanied by increases in mRNA of monocyte chemoattractant protein-1 (MCP-1) and tumor nec
197 VSMC stimulated by TGF-beta/AdSmad3 revealed monocyte chemoattractant protein-1 (MCP-1) as a likely f
198 pirfenidone impaired macrophage migration to monocyte chemoattractant protein-1 (MCP-1) as well as IL
199  an obligate dimeric mutant of the chemokine monocyte chemoattractant protein-1 (MCP-1) by substituti
200                                              Monocyte chemoattractant protein-1 (MCP-1) directs migra
201 uced Src and STAT3 tyrosine phosphorylation, monocyte chemoattractant protein-1 (MCP-1) expression an
202 JNK as the exclusive mediator of FFA-induced monocyte chemoattractant protein-1 (MCP-1) expression in
203                      15(S)-HETE also induced monocyte chemoattractant protein-1 (MCP-1) expression vi
204 mation, in association with higher levels of monocyte chemoattractant protein-1 (MCP-1) expression.
205                                              Monocyte chemoattractant protein-1 (MCP-1) is involved i
206 atment with p38 MAPK inhibitor reduced renal monocyte chemoattractant protein-1 (MCP-1) levels, the n
207  a murine model of an arteriovenous fistula, monocyte chemoattractant protein-1 (MCP-1) mRNA and prot
208                                              Monocyte chemoattractant protein-1 (MCP-1) plays a role
209                                              Monocyte chemoattractant protein-1 (MCP-1) plays an impo
210                         These cells produced monocyte chemoattractant protein-1 (MCP-1) upon PIM trea
211                                The chemokine monocyte chemoattractant protein-1 (MCP-1) was cooperati
212                                  Previously, monocyte chemoattractant protein-1 (MCP-1) was demonstra
213 ile inducing inflammatory cytokine (IL-6 and monocyte chemoattractant protein-1 (MCP-1)) expression.
214 mobility mass spectrometry (IMMS) to analyze monocyte chemoattractant protein-1 (MCP-1), a CC chemoki
215                                              Monocyte chemoattractant protein-1 (MCP-1), a CC-motif c
216 in HK-2 cells to stimulate the production of monocyte chemoattractant protein-1 (MCP-1), a key chemok
217 -1beta (IL-1beta), the cytokines IL-8, IL-6, monocyte chemoattractant protein-1 (MCP-1), and growth-r
218 yperactivation of ERK and p38 in response to monocyte chemoattractant protein-1 (MCP-1), and increase
219 phage accumulation, diminished expression of monocyte chemoattractant protein-1 (MCP-1), and lower le
220 activation-regulated chemokine (TARC), IL-8, monocyte chemoattractant protein-1 (MCP-1), and murine b
221    Stroke outcome, expression of brain CD36, monocyte chemoattractant protein-1 (MCP-1), CCR2, and pl
222                   The role of the chemokine, monocyte chemoattractant protein-1 (MCP-1), elevated in
223                            The expression of monocyte chemoattractant protein-1 (MCP-1), intercellula
224 d beta2-integrins, cyclooxygenase-2 (COX-2), monocyte chemoattractant protein-1 (MCP-1), interleukin-
225 We have previously shown that the chemokine, monocyte chemoattractant protein-1 (MCP-1), is a mediato
226                     Analysis of mice lacking monocyte chemoattractant protein-1 (MCP-1), MCP-3, MCP-5
227 ge inflammatory protein-1alpha (MIP-1alpha), monocyte chemoattractant protein-1 (MCP-1), regulated on
228 n-gamma-inducible protein of 10 kDa (IP-10), monocyte chemoattractant protein-1 (MCP-1), tumor necros
229 netic basis of circulating concentrations of monocyte chemoattractant protein-1 (MCP-1), we conducted
230 n WAT and an increase of the proinflammatory monocyte chemoattractant protein-1 (MCP-1).
231 appaB pathway, which increased production of monocyte chemoattractant protein-1 (MCP-1).
232  expression of the proinflammatory cytokine, monocyte chemoattractant protein-1 (MCP-1).
233 high levels of interleukin (IL)-6, IL-8, and monocyte chemoattractant protein-1 (MCP-1).
234  and selective induction of the CC chemokine monocyte chemoattractant protein-1 (MCP-1).
235 tion, as measured by decreased expression of monocyte chemoattractant protein-1 (MCP-1).
236  chemotaxis can be signaled by the chemokine monocyte chemoattractant protein-1 (MCP-1)/CCL2 (CC chem
237  that interact with the oligomeric chemokine Monocyte Chemoattractant Protein-1 (MCP-1)/CCL2 with dif
238 rated increased expression of iNOS, C1r, and monocyte chemoattractant protein-1 (MCP-1); MCP-1 expres
239 RK) to mechanically trigger the secretion of monocyte chemoattractant protein-1 (MCP-1, also known as
240 ort the expression profile of the chemokine, monocyte chemoattractant protein-1 (MCP-1, CCL2), during
241                         Genetic deletions of monocyte chemoattractant protein-1 (MCP-1, CCL2), fracta
242 to chronic pain includes the upregulation of monocyte chemoattractant protein-1 (MCP-1/CCL2) and its
243  proinflammatory cytokine IL-6 and chemokine monocyte chemoattractant protein-1 (MCP-1/CCL2) in respo
244 keratinocytes up-regulated the expression of monocyte chemoattractant protein-1 (MCP-1/CCL2), TNFalph
245  CCR2 on its interactions with the chemokine monocyte chemoattractant protein-1 (MCP-1/CCL2).
246                                              Monocyte chemoattractant protein-1 (MCP-1; CCL2)-mediate
247 mor necrosis factor alpha (TNF-alpha), CCL2 (monocyte chemoattractant protein 1 [MCP-1]), and CCL5 (R
248 6), and chemokine (C-C motif) ligand (CCL2) (monocyte chemoattractant protein 1 [MCP-1]).
249 ines (interleukin 6 [IL-6], IL-8, IL-1alpha, monocyte chemoattractant protein 1 [MCP-1], and colony-s
250 atory cytokines (interleukin-6 [IL-6], IL-8, monocyte chemoattractant protein 1 [MCP-1], and IL-1beta
251  we show that TXA(2) mimetic, I-BOP, induced monocyte chemoattractant protein -1(MCP-1)/chemokine (C-
252 ce was observed, certain chemokines (RANTES, monocyte chemoattractant protein 1[MCP-1], and IP-10) we
253 eoprotegerin (OPG) expression and increasing monocyte chemoattractant protein 1 (MCP1) expression in
254 rleukin-6 (Il-6), interleukin-1beta (Il-1b), monocyte chemoattractant protein 1 (Mcp1), and fibrosis-
255 ic pain, and mice overexpressing its ligand, monocyte chemoattractant protein-1 (MCP1; also known as
256 ential of mouse melanoma cells in HDAC3- and monocyte chemoattractant protein 1-(MCP1)-dependent mann
257 CI and other wild type CC chemokines, MCP-1 (monocyte chemoattractant protein-1), MIP-1beta, and RANT
258  10]), and proinflammatory cytokines (MCP-1 [monocyte chemoattractant protein 1], MIP-1alpha/beta [ma
259 essed and secreted, T-cell activation-3, and monocyte chemoattractant protein-1 mRNAs were lower comp
260               SkMCs released IL-6, IL-8, and monocyte chemoattractant protein-1 on Hsp60 stimulation.
261 llular adhesion molecule-1 (p < 0.0001), and monocyte chemoattractant protein-1 (p = 0.0004).
262 cellular adhesion molecule-1 (p = 0.01), and monocyte chemoattractant protein-1 (p = 0.01).
263 kin-6 (P=0.01), isoprostanes (P=0.0002), and monocyte chemoattractant protein-1 (P=0.008); SAT only r
264 proteinase 9, metalloproteinase inhibitor 1, monocyte chemoattractant protein-1, P-selectin, fibrinog
265   Expression of tumor necrosis factor-alpha, monocyte chemoattractant protein-1, plasminogen activato
266 e (C-C motif) ligand 2 (CCL2), also known as monocyte chemoattractant protein-1, plays a critical rol
267 with increased lipopolysaccharide-stimulated monocyte chemoattractant protein 1 production by JR-CSF
268 educed IL-4, IL-5, IL-13, eotaxin, IL-8, and monocyte chemoattractant protein 1 production without af
269 found a 3-fold increase in interleukin-6 and monocyte chemoattractant protein-1 production by G2A(-/-
270    Pio also did not attenuate Ang II-induced monocyte chemoattractant protein-1 production in PPARgam
271  responses to TLR2 and TLR4 ligands, reduced monocyte chemoattractant protein-1 production, and preve
272 ammation, limits neutrophils recruitment and monocyte chemoattractant protein-1 production, thus redu
273 yte/macrophage migration rather than reduced monocyte chemoattractant protein-1 production.
274 cts of Klotho signaling on interleukin-8 and monocyte chemoattractant protein-1 promoter recruitment
275                                              Monocyte chemoattractant protein-1 provides independent
276  isoprostanes, R2 0.07 versus 0.10, P=0.002; monocyte chemoattractant protein-1, R2 0.07 versus 0.08,
277 ed with the changes of tubular expression of monocyte chemoattractant protein-1, RANTES (regulated up
278 sis included higher levels of interleukin-8, monocyte chemoattractant protein-1, resistin, soluble in
279 ophage inflammatory protein 1alpha/beta, and monocyte chemoattractant protein 1, respectively, as det
280 and 1/lymphotactin and CC chemokine ligand 2/monocyte chemoattractant protein 1 reveal that invariant
281   Given its unique role, future studies into monocyte chemoattractant protein-1's exact role during s
282 hly associated with urinary isoprostanes and monocyte chemoattractant protein-1 (SAT versus VAT compa
283 sociated phospholipase A2 mass and activity, monocyte chemoattractant protein-1, soluble endothelial
284 methylarginine, tumor necrosis factor-alpha, monocyte chemoattractant protein-1, soluble vascular cel
285 toreceptor cultures exposed to starvation or monocyte chemoattractant protein-1-stimulated (MCP-1-sti
286 yed similar up-regulation of miR-132/212 and monocyte chemoattractant protein-1, supporting in vivo r
287  This was associated with marked increase in monocyte chemoattractant protein-1 synthesis in WKY glom
288 emokine, C-C motif chemokine ligand 2 (CCL2; monocyte chemoattractant protein 1), termed mNOX-E36, in
289 erestingly, higher levels of interleukin 22, monocyte chemoattractant protein 1, TNF-alpha, and IP-10
290  in systolic BP, heart rate variability, and monocyte chemoattractant protein-1, together with reduce
291 nes for interleukin (IL)-1beta, IL-6, IL-10, monocyte chemoattractant protein-1, tumor necrosis facto
292                                              Monocyte chemoattractant protein-1 was measured at basel
293 ray indicated that the chemokine CCL2/MCP-1 (monocyte chemoattractant protein-1) was strongly induced
294 vels of IFN-gamma, interleukin-6 (IL-6), and monocyte chemoattractant protein 1 were detected in the
295 ), monokine induced by interferon-gamma, and monocyte chemoattractant protein 1 were quantified as me
296 itive cells) and production of the chemokine monocyte chemoattractant protein-1 were significantly bl
297 , intercellular cell adhesion molecule-1 and monocyte chemoattractant protein-1, were also determined
298 idenced by the upregulation of ephrin B2 and monocyte chemoattractant protein-1, which are 2 stretch-
299 oprotein 1 stimulates macrophages to secrete monocyte chemoattractant protein-1, which then activates
300 nd decreased production of oxidant-inducible monocyte chemoattractant protein-1, which we have previo

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