ライフサイエンスコーパス: monocyte infiltration

1 (BBB), and are cleared without neutrophil or monocyte infiltration.

2 neurysm in ApoE(-/-) mice partly by reducing monocyte infiltration.

3 ctions in microglial activation and cerebral monocyte infiltration.

4 tor, and chemokines mediating neutrophil and monocyte infiltration.

5 liver injury, steatosis, and proinflammatory monocyte infiltration.

6 d with a dramatic decrease in neutrophil and monocyte infiltration.

7 stic of HIV-1 that influences HAD, increased monocyte infiltration.

8 eactive responses of retinal glial cells and monocyte infiltration.

9 n between chemokine expression and placental monocyte infiltration.

10 l malaria and were associated with placental monocyte infiltration.

11 s (GA) characterized by recipient T cell and monocyte infiltration.

12 osis, are characterized by compartmentalized monocyte infiltration.

13 chemoattractants associated with glomerular monocyte infiltration.

14 We previously showed that host monocyte infiltration and activation within the graft dr

15 l CCR2/CCR5 inhibitor, cenicriviroc) reduces monocyte infiltration and APAP-induced liver injury (AIL

16 however, the spatiotemporal distribution of monocyte infiltration and its correlation to prognostic

17 DCs accumulated in the heart coincident with monocyte infiltration and loss of resident reparative em

18 Monocyte infiltration and macrophage formation are pivot

19 ow that TLR5 agonist, flagellin, can promote monocyte infiltration and osteoclast maturation directly

20 Monocyte infiltration and subsequent differentiation int

21 utaneous collagen mRNA, which is preceded by monocyte infiltration and the up-regulation of cutaneous

22 proliferative glomerulonephritis with marked monocyte infiltration and, at times, intracapillary thro

23 d less crescent formation, tubular dilation, monocyte infiltration, and interstitial renal fibrosis.

24 d that VEGF regulates vascular permeability, monocyte infiltration, and scar-associated macrophages f

25 pment by decreasing steatosis, liver damage, monocyte infiltration, and the production of inflammator

26 d.nNOS had a particularly striking impact on monocyte infiltration; as early as 24 hours after gene t

27 Inhibition of monocyte infiltration at the induction phase of experime

28 macrophages has reinforced the concept that monocyte infiltration dictates macrophage buildup.

29 To test the hypothesis that monocyte infiltration during atherogenesis is MCP-1 depe

30 To elucidate the regulation of this monocyte infiltration, expression of monocyte chemoattra

31 to the RPE layer, followed by (2) subsequent monocyte infiltration from the retinal vasculature into

32 At 3 sites of monocyte infiltration/giant cell formation (granulation

33 ed significantly impaired CCL2 secretion and monocyte infiltration in an experimental model of perito

34 The impaired monocyte infiltration in MVO regions could be related to

35 ion of rPSGL-1-Ig abolished antibody-induced monocyte infiltration in the allograft, but had little e

36 n markedly increased neutrophil, T-cell, and monocyte infiltration in the diseased brain.

37 -1(-/-) mice exhibited significantly reduced monocyte infiltration in wire injury-induced neointima a

38 st the impact of PM(2.5) in eliciting direct monocyte infiltration into fat, we rendered FVBN mice ex

39 d less monocyte-derived cells, less Ly6c(hi) monocyte infiltration into lesions, and lower levels of

40 hemotactic protein-1 (MCP-1) is critical for monocyte infiltration into the arterial wall and neointi

41 Monocyte infiltration into the CNS is a hallmark of seve

42 G3 should have a greater capacity to trigger monocyte infiltration into the graft than IgG2 or IgG4 d

43 Interestingly, blockade of CCR2-dependent monocyte infiltration into the heart reduced soluble MER

44 mulate pathobiological processes involved in monocyte infiltration into the mesangium.

45 Recently, monocyte infiltration into the placental intervillous sp

46 ils and two stages of GR1+CD68+ inflammatory monocyte infiltration into the site of inoculation.

47 Monocyte infiltration into the vessel wall, a key initia

48 Monocyte infiltration into these areas appeared 24 h lat

49 Monocyte infiltration is implicated in a variety of dise

50 nded on the level of chemokine secretion and monocyte infiltration; low-level MCP-1 secretion with mo

51 -inflammatory cytokine signaling, peripheral monocyte infiltration, microglial activation, and hypoth

52 e histologically analyzed for neutrophil and monocyte infiltration, neovascularization and epithelial

53 n of endothelial adhesion molecules limiting monocyte infiltration of the artery wall.

54 CFU of Brucella spp. display neutrophil and monocyte infiltration of the joint space and surrounding

55 Low density lipoprotein (LDL) oxidation and monocyte infiltration of the vessel wall underlie athero

56 cyte infiltration (CD43-positive cells), and monocyte infiltration (RAM-11-positive cells).

57 ation; low-level MCP-1 secretion with modest monocyte infiltration resulted in tumor formation, where

58 phase of NASH development by promoting blood monocyte infiltration through the production of IP-10 an

59 Notably, limiting monocyte infiltration via genetic Ccl2 reduction prolong

60 In the absence of MVO, monocyte infiltration was more intense in MI regions wit

61 ce imaging performed in series revealed that monocyte infiltration was spatially inhomogeneous in rep

62 The distribution patterns of monocyte infiltration were correlated to the presence of

63 Atherosclerotic lesions are characterized by monocyte infiltration, which may be regulated by the che

64 terized histologically by tubular injury and monocyte infiltration, while the stable posttransplant s