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1 population arising from a common granulocyte/monocyte progenitor.
2 risk cases revealed expansion of granulocyte-monocyte progenitors.
3 Cs, multipotent progenitors, and granulocyte-monocyte progenitors.
4 ting with a greater expansion of granulocyte-monocyte progenitors.
5 (SCF)-dependent EML cell line to granulocyte/monocyte progenitors.
6 used to document the distribution pattern of monocyte progenitors.
7 nal differentiation of committed granulocyte/monocyte progenitors.
8 karyocytic-erythroid progenitor, granulocyte-monocyte progenitor and hematopoietic stem cell bone mar
9 cells, which correlated with an increase in monocyte progenitors and a decrease in granulocyte proge
10 rated an increase in the growth potential of monocyte progenitors and a significant decrease in granu
12 absence of dramatic increases in bone marrow monocyte progenitors and was independent of chemokine (C
13 pool (common myeloid progenitor, granulocyte-monocyte progenitor, and megakaryocyte-erythroid progeni
14 n from the common myeloid to the granulocyte/monocyte progenitor but is not required beyond this stag
15 ha is required for generation of granulocyte-monocyte progenitors, but the subsequent role of C/EBPal
16 proinflammatory cytokines, overt granulocyte/monocyte progenitor cell apoptosis, and failure to recon
17 rrow progenitors and demonstrate that common monocyte progenitor cells express high levels of APOBEC1
18 nd osteoclasts are derived from monocytes or monocyte progenitor cells, the ways in which they are fo
19 that Ly6C(hi) monocytes develop from common monocyte progenitors (cMoPs) and reside in the bone marr
21 lineage, the common myeloid and granulocyte-monocyte progenitors (CMP/GMP), have been shown to prote
22 ghly purified common myeloid and granulocyte-monocyte progenitors (CMPs/GMPs) accelerated myeloid rec
23 resulted in an expansion of the granulocyte/monocyte progenitor compartment and was associated with
26 ause in addition to lymphoid and granulocyte-monocyte progenitors, FLT3(-) Mk- and E-restricted downs
27 lls (HSCs), short-term HSCs, and granulocyte-monocyte progenitors from individuals with high-risk (-7
28 ate mapping revealed that yolk-sac and fetal monocyte progenitors gave rise to the majority of cardia
29 sP3KB null mice, the bone marrow granulocyte monocyte progenitor (GMP) population was expanded, and G
30 expansion and differentiation of granulocyte/monocyte progenitor (GMP) populations, which is due in p
31 , there was a strong skew toward granulocyte-monocyte progenitor (GMP) production at the expense of e
32 lpha then directs the LMP to the granulocyte-monocyte progenitor (GMP) stage, while inhibiting lympho
33 on myeloid progenitors (CMP) and granulocyte-monocyte progenitors (GMP) protects against death follow
34 hGM-CSFR-expressing (hGM-CSFR+) granulocyte/monocyte progenitors (GMPs) and megakaryocyte/erythrocyt
36 llows us to separate oligopotent granulocyte-monocyte progenitors (GMPs) and their lineage-committed
39 n myeloid progenitors (CMPs) and granulocyte/monocyte progenitors (GMPs)], which have a distinct gene
42 ignificantly altered distribution profile of monocyte progenitors in norepinephrine-depleted mice com
44 of mature monocytes and myeloid granulocyte-monocyte progenitors in the bone marrow and spleen of hy
49 g common myeloid progenitors and granulocyte-monocyte progenitors, resulting in lower rates of myeloi
50 Flow cytometric analysis of early and late monocyte progenitors showed a significantly altered dist
51 alysis of early (ER-MP12) and late (ER-MP20) monocyte progenitors showed an increase in monocyte line
52 the bone marrow starting at the granulocyte-monocyte progenitor stage and reduced systemic expansion
53 d each of these fates or whether specialized monocyte progenitor subsets exist before inflammation.
54 olony-stimulating factor receptor density in monocyte progenitors was assessed by 125I macrophage col
56 cription factor expression, these neutrophil/monocyte progenitors were reprogrammed to take on erythr
57 the commitment of these cells to granulocyte/monocyte progenitors, whereas dominant-negative Stat5 co
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