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1 mulated KG1 cells (phenotypically similar to monocyte-derived dendritic cells).
2 e rise to a subset of monocytes that yielded monocyte-derived dendritic cells.
3 by LRP-expressing U87-MG cells and by human monocyte-derived dendritic cells.
4 d upregulation strategy was applied in human monocyte-derived dendritic cells.
5 rnalization was observed in freshly isolated monocyte-derived dendritic cells.
6 5aR2 expression in pulmonary eosinophils and monocyte-derived dendritic cells.
7 LT-induced activation of human monocytes and monocyte-derived dendritic cells.
8 Migration assays were performed using human monocyte-derived dendritic cells.
9 g-fusion protein of BTN2A1 bound to immature monocyte-derived dendritic cells.
10 governing recognition by DC-SIGN on immature monocyte-derived dendritic cells.
11 N-gamma in response to self-CD1 expressed on monocyte-derived dendritic cells.
12 lls, NK cells, and mature, but not immature, monocyte-derived dendritic cells.
13 against infected monocytes, macrophages, and monocyte-derived dendritic cells.
14 essing Mtb39 (adenoMtb39) was used to infect monocyte-derived dendritic cells.
15 ass II-depleted cell cultures by adding back monocyte-derived dendritic cells.
16 of CD11b(+) conventional dendritic cells and monocyte-derived dendritic cells.
17 ced expression of nitric oxide synthase 2 in monocyte-derived dendritic cells.
18 n TLR stimulation compared with donor-paired monocyte-derived dendritic cells.
19 ction of the interleukin-12 (IL-12) in human monocyte-derived dendritic cells.
20 were CCR2-bearing monocytes/macrophages and monocyte-derived dendritic cells.
21 to be required for the infection of immature monocyte-derived dendritic cells.
22 ticles and fusion to primary CD4 T cells and monocyte-derived dendritic cells.
23 cytic cell line (THP-1) and in human primary monocyte-derived dendritic cells.
24 he differentiation and the function of human monocyte-derived dendritic cells.
25 m this method using peripheral cells such as monocytes derived dendritic cells.
26 BK virus peptide libraries loaded or not on monocytes-derived dendritic cells.
29 We use our methodology to study how human monocyte-derived dendritic cells alert neighboring cells
31 om transcriptomic data measuring response of monocyte derived dendritic cells and A549 epithelial cel
32 mpletely inhibited FVIII endocytosis by both monocyte-derived dendritic cells and bone marrow-derived
33 In this study, the interaction between human monocyte-derived dendritic cells and C. jejuni was studi
34 rophages (MDMs), which differed from that in monocyte-derived dendritic cells and CD4 T cells, withou
36 GN is a major receptor for infection of both monocyte-derived dendritic cells and interstitial dermal
37 mbination of alpha-galactosylceramide-loaded monocyte-derived dendritic cells and low-dose lenalidomi
38 a strongly reduced internalization by human monocyte-derived dendritic cells and macrophages, as wel
39 wn to elicit inflammatory responses by human monocyte-derived dendritic cells and macrophages, includ
40 se- and ligand dose-dependent manner in both monocyte-derived dendritic cells and monocyte-derived ma
42 ne production probably via the activation of monocyte-derived dendritic cells and the TLR, TLR2, and
45 IPSE/alpha-1 was confirmed in human primary monocyte-derived dendritic cells and was found to be a c
46 ing antigens in common with blood monocytes, monocyte-derived dendritic cells, and macrophages, NLCs
48 ivated plasmacytoid dendritic, TLR-activated monocyte-derived dendritic cells, and on B cells stimula
49 ophages, peripheral blood mononuclear cells, monocyte-derived dendritic cells, and plasmacytoid dendr
50 ease from PBMCs, LPS-triggered maturation of monocyte-derived dendritic cells, and tetanus toxoid-ind
53 lso inhibited the production of IL-12 p70 by monocyte-derived dendritic cells, as well as the product
54 e expression of the above antigens occurs on monocyte-derived dendritic cells, because these molecule
55 DC is highly expressed in macrophages and in monocyte-derived dendritic cells, but not in monocytes,
57 addition of TLF to human peripheral blood or monocyte-derived dendritic cell cultures resulted in cel
58 ls, have both been associated with arrest of monocyte-derived dendritic cell (DC) differentiation and
60 ne receptor CCR2 was implicated in mediating monocyte-derived dendritic cell (DC) recruitment into th
61 ribe a phenotypically and functionally novel monocyte-derived dendritic cell (DC) subset, designated
62 responses, we developed an in vitro model of monocyte-derived dendritic cell (DC)-dependent, human na
65 of CMV pp65 messenger RNA-loaded autologous monocyte-derived dendritic cells (DC) as a cellular vacc
66 ting step in the process of generating human monocyte-derived dendritic cells (DC) for clinical appli
67 s spectrometry) of microfilaria (mf)-exposed monocyte-derived dendritic cells (DC) indicated that mul
69 and C5a did not suppress IL-12 production by monocyte-derived dendritic cells (DC) stimulated with CD
75 ces a semimature, tolerogenic state on human monocyte-derived dendritic cells (DCs) activated by a pr
76 gonist, FP7, in vitro on human monocytes and monocyte-derived dendritic cells (DCs) and in vivo durin
78 nses via surface TLR2, which is expressed on monocyte-derived dendritic cells (DCs) and NK cells.
79 affect trafficking of monocytes/macrophages, monocyte-derived dendritic cells (DCs) and T-helper cell
82 s were used to test protein binding to human monocyte-derived dendritic cells (DCs) by flow cytometry
83 f cytokine regulation were observed in human monocyte-derived dendritic cells (DCs) costimulated with
84 er the immunostimulatory properties of human monocyte-derived dendritic cells (DCs) could be enhanced
87 igate whether iNKT cells can be activated by monocyte-derived dendritic cells (DCs) exposed to lipid
91 e expression patterns were measured in human monocyte-derived dendritic cells (DCs) infected in vitro
95 wn to promote chemotactic migration of human monocyte-derived dendritic cells (DCs) toward the chemok
99 roducing Ly6C(hi) inflammatory monocytes and monocyte-derived dendritic cells (DCs), suggesting that
105 so expressed on circulating dendritic cells, monocyte-derived dendritic cells, dendritic cells in lym
106 , mannose receptor, or DC-SIGN expression in monocyte-derived dendritic cells did not prevent FVIII u
107 fection from relapsers and observed impaired monocyte-derived dendritic cell differentiation, a reduc
109 ial challenge assays using keratinocytes and monocyte-derived dendritic cells established distinct IL
110 cyte-derived human macrophages (MDM) but not monocyte-derived dendritic cells express basal levels of
111 When pulsed with targeted liposomes, human monocyte-derived dendritic cells expressing CD169/Sn act
113 pregulation of CD40, CD80, CD83, and CD86 on monocyte-derived dendritic cells from allergic patients
115 reduced costimulatory molecule expression on monocyte-derived dendritic cells from healthy volunteers
118 fied myeloid dendritic cells, monocytes, and monocyte-derived dendritic cells (GM-CSF/IL-4/TGF-beta).
119 e- and genome-wide analyses in primary human monocyte-derived dendritic cells here showed that TLR8 s
121 e found on endothelial cells, human immature monocyte-derived dendritic cells (iDCs) bound rBRAK with
123 SP-D enhanced the binding of HIV to immature monocyte derived dendritic cells (iMDDCs) and was also f
124 ons were demonstrated in LGTV-infected human monocyte-derived dendritic cells, important target cells
126 e HCV-specific CD8(+) T cells and autologous monocyte-derived dendritic cells in the absence or prese
127 contained significantly more macrophages and monocyte-derived dendritic cells in the dermis after VAC
128 ses, it stimulated strong IL-12 responses by monocyte-derived dendritic cells in the presence of IFN-
130 rleukin-18 (IL-18)-dependent manner, whereas monocyte-derived dendritic cells induced NK activation t
131 ounters between human T cells and allogeneic monocyte-derived dendritic cells induces durable, profou
132 s maintained or induced during dermal DC and monocyte-derived dendritic cell/inflammatory dendritic e
133 overexpression in cell lines and in primary monocyte-derived dendritic cells inhibits the replicatio
134 ether proinflammatory cytokine expression by monocyte-derived dendritic cells is affected by the indu
135 oma, infects three types of dendritic cells: monocyte-derived dendritic cells, Langerhans cells, and
137 this antiviral activity were cocultured with monocyte-derived dendritic cells matured with CD40 ligan
139 IFN-alpha) and inducible chemokines by human monocyte-derived dendritic cells (mDCs) and plasmacytoid
140 gamma interferon (IFN-gamma) dependent, and monocyte-derived dendritic cells (mDCs) promote IgG2 pro
141 l miRNA-protein networks that regulate human monocyte-derived dendritic cell (MDDC) differentiation.
145 d cytokine secretion was diminished in human monocyte-derived dendritic cells (MDDC) from rs7282490 I
146 ed to monocyte-derived macrophages (MDM) and monocyte-derived dendritic cells (MDDC) in virus-like pa
147 ycoproteins on virus uptake by primary human monocyte-derived dendritic cells (MDDC) in vitro and on
148 we investigated the effects of PMT on human monocyte-derived dendritic cells (MDDC) in vitro and sho
149 We have previously shown that HHV-8 enters monocyte-derived dendritic cells (MDDC) through DC-SIGN,
150 es and cells with the morphology of immature monocyte-derived dendritic cells (MDDC) were observed.
151 de more TNF, IL-6, and pro-IL-1beta than did monocyte-derived dendritic cells (MDDC), despite similar
154 profile of gene expression of human immature monocyte-derived dendritic cells (MDDCs) and peripheral
155 ously showed that galectin-1 activates human monocyte-derived dendritic cells (MDDCs) and triggers a
156 NAg), but not deglycosylated PNAg, activated monocyte-derived dendritic cells (MDDCs) as measured by
159 a hypothesis that mature rhesus monkey (Rh) monocyte-derived dendritic cells (MDDCs) modified by gen
160 ediates the binding and transfer of HIV from monocyte-derived dendritic cells (MDDCs) to permissive T
161 , leaving the immune system and specifically monocyte-derived dendritic cells (MDDCs) understudied.
162 on of adenosine receptors expressed by human monocyte-derived dendritic cells (MDDCs) was performed w
163 alpha, upregulate costimulatory molecules in monocyte-derived dendritic cells (MDDCs), enabling effec
165 t of Vpr for facilitating HIV-1 infection of monocyte-derived dendritic cells (MDDCs), one of the fir
166 nd CD4(+) T-cells and compared it to that of monocyte-derived dendritic cells (MDDCs), which are less
170 oid cells, including monocytes, macrophages, monocyte-derived dendritic cells (mo-DC), and dendritic
171 ic inflammatory monocytes differentiate into monocyte-derived dendritic cells (MO-DCs), which are CD1
174 ired recruitment of Ly6C(high) monocytes and monocyte-derived dendritic cells (moDC) and lower moDC c
175 and LAMP-2 (CD107b) on the surface of human monocyte-derived dendritic cells (MoDC) and show only LA
176 vivo administration of IS on the ability of monocyte-derived dendritic cells (MoDC) to differentiate
178 suppression of IL6R-alpha signaling in human monocyte-derived dendritic cells (moDCs) and T cells, ou
179 and adult monocytes were differentiated into monocyte-derived dendritic cells (MoDCs) and TLR agonist
180 ch human cytomegalovirus (HCMV) infection of monocyte-derived dendritic cells (moDCs) contribute to i
183 induced activation of neonatal monocytes and monocyte-derived dendritic cells (MoDCs) have not been r
184 This PD-L1 upregulation was observed in monocyte-derived dendritic cells (MoDCs) obtained from e
185 s stimulated ex vivo using either autologous monocyte-derived dendritic cells (moDCs) or HLA-A2-Ig-ba
186 P[13] replicated more extensively in porcine monocyte-derived dendritic cells (MoDCs) than did HRV Wa
187 -ICs in human B cells and in CD23-expressing monocyte-derived dendritic cells (moDCs) that represent
190 es in IFN induction and the role of DI RNAs, monocyte-derived dendritic cells (moDCs) were infected w
191 naling pathway when porcine peripheral blood monocyte-derived dendritic cells (MoDCs) were treated wi
192 uce a rapid (after 1 d) accumulation of host monocyte-derived dendritic cells (moDCs) without any inc
193 cyte-derived macrophages (MDMs), (iii) bound monocyte-derived dendritic cells (MoDCs), and (iv) trans
194 , Ly6c(hi) monocyte-derived Mphis (moMphis), monocyte-derived dendritic cells (moDCs), and myeloid-de
195 eated (bafilomycin and cytochalasin D) human monocyte-derived dendritic cells (moDCs), bone marrow-de
196 panded using HLA-mismatched immature, mature monocyte-derived dendritic cells (moDCs), or PBMCs.
201 In vitro cultures of different cell types (monocyte-derived dendritic cells [moDCs], PBMCs [periphe
202 onocytes were up to 40-fold more potent than monocyte-derived dendritic cells or CD2- monocytes at in
203 genes are both expressed in LCs, but not in monocyte-derived dendritic cells, or in blood CD1c(+) or
204 aryotic sources, when transfected into human monocyte-derived dendritic cell precursors, induces high
207 flammatory cytokines in PBMCs, maturation of monocyte-derived dendritic cells (rendering their profil
208 macrophages, but detection on the surface of monocyte-derived dendritic cells required stimulation wi
209 evels of mutant CARD9 protein, the patients' monocyte-derived dendritic cells responded poorly to CAR
210 s of the Ag-presenting function of CD1+CD83+ monocyte-derived dendritic cells showed that such cells
211 report that primary human CD1c(+) as well as monocyte-derived dendritic cells significantly upregulat
212 production by both autologous monocytes and monocyte-derived dendritic cells than either LT-IIa- or
215 ndritic cells and transfer of infection from monocyte-derived dendritic cells to CD4+ T cells were me
224 can express TL1A, fresh blood monocytes and monocyte-derived dendritic cells were stimulated with va
226 itioned NK cells triggered the maturation of monocyte-derived dendritic cells, which promoted T cell
227 pendent manner, leading to the generation of monocyte-derived dendritic cells with a tolerogenic cyto
228 ignificant shift in the ratio of circulating monocyte-derived dendritic cells with significantly diff
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