ライフサイエンスコーパス: monocytic

1 dhesion molecule-1 expression to increase EC-monocytic adhesion, but the Ser45/Thr41-phosphorylated b

2 ling pathways, leading to myelomonocytic and monocytic AML cell differentiation.

3 are suggestive of a novel mechanism by which monocytic AML cells evade cell-mediated immunity.

4 therapeutic potential in myelomonocytic and monocytic AMLs.

5 ogenitors, which produced CD11b(+) Ly-6C(hi) monocytic and CD11b(+) Ly-6G(hi) granulocytic cells loca

6 ased early type I IFN transcription in human monocytic and epithelial cell lines, but this was surpri

7 possibly via interacting with NLRC3 in human monocytic and epithelial cells.

8 infection, there was an increase in Ly6C(hi) monocytic and Gr-1(hi) neutrophilic cells in lymphoid or

9 Of the expanded monocytic and granulocytic cell populations of MDSCs, th

10 The cells are known to descend from immature monocytic and granulocytic cells, respectively, which ar

11 Specifically, cells of the monocytic and granulocytic lineages increased nearly 60%

12 aused by excessive proliferation of cells of monocytic and granulocytic lineages.

13 These cell subsets include monocytic and granulocytic myeloid-derived suppressor ce

14 etate (TPA), and trichostatin A (TSA), in U1 monocytic and J-Lat 10.6 lymphocytic latently infected c

15 CXCR3 is associated with monocytic and lymphocytic infiltration of inflamed or tu

16 block in T-lineage specification and induced monocytic and plasmacytoid dendritic cell differentiatio

17 MDSCs consist of two major subsets, monocytic and polymorphonuclear (PMN).

18 Human monocytic and professional antigen-presenting cells have

19 latelet-like particles with activated THP-1, monocytic, and endothelial cells led to visual and funct

20 dividuals, were found to express epithelial, monocytic, and endothelial protein markers and were obse

21 The cellular response was predominantly monocytic, and focal fibroplasia was observed at the air

22 ensable for the development of granulocytic, monocytic, and megakaryocytic cells.

23 eptor 2 on classical monocytes, resulting in monocytic beta1- and beta2-integrin conformational chang

24 letion of common lymphoid progenitors, and a monocytic bias in comparison with the granulocytic bias

25 ed reduces the immunosuppressive activity of monocytic (CD11b(+), Ly6G(-), Ly6C(high)) MDSC.

26 granulocytic (CD11b(+)Ly6G(+)Ly6C(low)) and monocytic (CD11b(+)Ly6G(-)Ly6C(high)) myeloid-derived su

27 Monocytic, CD11b(+) Ly6C(hi) Ly6G(-) cells but not granu

28 Although the monocytic, CD11b(+) Ly6C(hi) Ly6G(-) cells were able to

29 howed previously that cross-linking of human monocytic CD13 with activating Abs induces strong adhesi

30 PRRSV is ideal for deciphering how monocytic cell activation statuses interact with antivir

31 s relayed by the MKK6-p38MAPK cascade induce monocytic cell differentiation from band-stage neutrophi

32 Monocytic cell differentiation potential was retained in

33 C/EBPalpha, which enabled the induction of a monocytic cell differentiation program.

34 d the effects of ex vivo-derived exosomes on monocytic cell differentiation/activation using THP-1 ce

35 to induce inflammatory responses in a human monocytic cell line (THP-1 cells).

36 ed with TLR2/4-mediated responses in a human monocytic cell line (THP-1) and in human primary monocyt

37 ecretion were conducted with the human THP-1 monocytic cell line and corroborated in primary human pe

38 d IL-12 production in a NEMO knockdown human monocytic cell line following LPS treatment.

39 We report the establishment of a monocytic cell line latently infected with KSHV (KSHV-TH

40 we knocked down these proteins in the human monocytic cell line Mono Mac 6 (MM6).

41 of the pro-inflammatory cytokine IL-6 in the monocytic cell line Mono Mac 6, induction of the Toll-in

42 Similar regulation was observed in the monocytic cell line RAW264.7.

43 y, we observe that depletion of BICD2 in the monocytic cell line THP-1 results in an induction of IFN

44 nhibited CCL2-driven chemotaxis of the human monocytic cell line THP-1, CCL3-driven chemotaxis of per

45 A3A in CD14(+)-enriched primary cells or the monocytic cell line THP-1.

46 vity in a time-dependent manner in the human monocytic cell line THP1.

47 trophils and diminished calcium signaling in monocytic cell line U937 transfected with the C5a recept

48 creases TET2 protein expression in the human monocytic cell line U937.

49 A human monocytic cell line was genetically engineered using len

50 Knocking down DJ-1 (siRNA) in THP-1 (human monocytic cell line) and polymorphonuclear cells from pa

51 In the THP-1 human monocytic cell line, reducing AIRE expression resulted i

52 We used a human monocytic cell line, THP-1, and dendritic cells to study

53 man airway smooth muscle cells and the human monocytic cell line, THP-1.

54 ester accumulation in foam cells of the THP1 monocytic cell line.

55 ased consequent late cell death of the human monocytic cell line.

56 an undifferentiated, normally nonpermissive monocytic cell line.

57 effects on migration of THP-1 cells, a human monocytic cell line.

58 MCs from patients with APECED syndrome and a monocytic cell line.

59 induces less inflammatory signaling in human monocytic cell lines and murine macrophages than the par

60 lial cells and measured the binding of human monocytic cell lines and peripheral blood monocytes.

61 vatives of the A549 lung carcinoma and THP-1 monocytic cell lines and used these to study T cell resp

62 neal exudate cells, and adoptive transfer of monocytic cell lines engineered to express the mutant CD

63 een A1PI expression in primary monocytes and monocytic cell lines, and inflammatory cytokine expressi

64 cycle in primary human T lymphocytes and in monocytic cell lines.

65 We observed spontaneous monocytic-cell infiltration in the lungs of Serpine2-def

66 g various phases of HIV-1 infection using U1 monocytic cells (latently infected U937 cells with HIV-1

67 human epithelial pulmonary cells (A549) and monocytic cells (U937) upon IAV infection using synchrot

68 s (PRRSV), which directly infects subsets of monocytic cells and interferes with antiviral responses.

69 (Gal3), a pleiotropic lectin, is produced by monocytic cells and macrophages.

70 re that oxLDL induced TF expression in human monocytic cells and monocytes.

71 We treated human THP-1 monocytic cells and primary human blood monocytes with R

72 nity, because it directly infects subsets of monocytic cells and subverts overall immune responses.

73 Importantly, porcine monocytic cells at different activation states were susc

74 se pathways in uninfected and PRRSV-infected monocytic cells at different activation states.

75 Although it induces chemotaxis of monocytic cells at high concentrations, its physiologica

76 ral proteins implicated in driving cancer in monocytic cells but only harbor limited activity in epit

77 type 1 (HIV-1) infection in noncycling human monocytic cells by reducing the intracellular deoxynucle

78 We observed that monocytic cells cocultured (but not in contact) with ECs

79 A forms and induced less TNF-alpha in THP-1 monocytic cells compared with LOS from group 1.

80 se of the autofluorescent signal recorded in monocytic cells could be correlated with the IR detectio

81 fect of Panx1 deletion in endothelial and/or monocytic cells during atherogenesis is counterbalanced

82 In particular, miR-10a was transferred to monocytic cells from EC-EVs and could repress inflammato

83 Monocytic cells have been found to be an important part

84 The reduction of monocytic cells in Ccr2(-/-) mice or after Gr-1 antibody

85 r, our study reveals a dynamic modulation of monocytic cells in response to varying dosages of endoto

86 pports a role for IL-27 in the activation of monocytic cells in terms of inflammatory responses.

87 ecreted by primary human monocytes and THP-1 monocytic cells in the presence of alcohol in a concentr

88 aneously measure the mechanical stiffness of monocytic cells in three rotational and two translationa

89 in the gastrointestinal tract) and lymphoid/monocytic cells in tonsils and Peyer's patches (explaini

90 in the gastrointestinal tract) and lymphoid/monocytic cells in tonsils and Peyer's patches (explaini

91 ctures in the ECM, providing a substrate for monocytic cells in vitro, whereas lung fibroblasts from

92 We found that cross-linking of CD13 on U937 monocytic cells induced phosphorylation of a number of p

93 We showed that in monocytic cells KLF4 has to be repressed to allow their

94 uence of HIV on global lncRNAs expression in monocytic cells lines.

95 ntiviral infection with activation status of monocytic cells may provide a means of potentiating anti

96 sue factor procoagulant activity (TF PCA) on monocytic cells more potently than other stimuli that de

97 ured by flow cytometry using uptake by THP-1 monocytic cells of fluorescent beads coated with gp120,

98 ortunately, the activation status of porcine monocytic cells or how cell activation status functional

99 tory cytokine release was evaluated in THP-1 monocytic cells or THP-1 cells lacking the inflammasome

100 These monocytic cells represent one of the best-defined human

101 ntal antagonism of miR-452 in differentiated monocytic cells resulted in increased expression of MMP1

102 f infected human, primary, neuronal stem and monocytic cells revealed effects on neurodevelopment and

103 s expression of APOL1 in differentiated U937 monocytic cells stimulated with IFN-gamma resulted in a

104 ) greater opsonization of these IEs by human monocytic cells than IgGs from malaria-exposed Malian me

105 linically characterized by overproduction of monocytic cells that can infiltrate organs, including th

106 ldren characterized by the overproduction of monocytic cells that infiltrate the spleen, lung, and li

107 , we showed that Slit2 inhibited adhesion of monocytic cells to activated human endothelial cells, as

108 investigate the activation status of porcine monocytic cells to determine the intricate interaction o

109 nfluence the antigen presenting phenotype of monocytic cells to determine the nature of T cell respon

110 tease thrombin in regulating the adhesion of monocytic cells to smooth muscle cells producing an infl

111 n and the recruitment of bone marrow-derived monocytic cells to the hypothalamus; in addition, this i

112 rom CD11c(dim)CD11b(int)Gr1(-) lung-resident monocytic cells transformed by KRAS(G12D) In contrast, B

113 A and IL-15Ralpha appeared on the surface of monocytic cells upon activation.

114 These data combine to suggest that monocytic cells use a cytoplasmic retention mechanism to

115 ion-channel function of ATP receptor P2X7 in monocytic cells via nAChR containing alpha9 and alpha10

116 Inflammatory monocytic cells were not observed infiltrating the skin

117 , survival and properties of agrin-deficient monocytic cells, and occur at stages later than stem cel

118 luble CD163 likely originates from activated monocytic cells, as serum from stroke patients stimulate

119 nhanced immune response that is dependent on monocytic cells, but this hyperimmune phenotype and its

120 g studies revealed SAMHD1 tetramers in human monocytic cells, in which it strongly restricts HIV-1 in

121 Monocytic cells, including macrophages and dendritic cel

122 Activation statuses of monocytic cells, including monocytes, macrophages (Mvarp

123 ation was seen especially in lymphocytes and monocytic cells, the natural hosts for HIV-1 infection.

124 Cultured human monocytic cells, U937, were differentiated into macropha

125 study antiviral responses in PRRSV-infected monocytic cells, we characterized inflammatory cytokine

126 ll types, primary human CD4 T-cells and U937 monocytic cells, were used to compare differences in cyt

127 rst genome-wide analysis of TNF tolerance in monocytic cells, which differentially inhibits NF-kappaB

128 Using RAW 264.7 mouse monocytic cells, which express endogenous MCH receptor,

129 ot contribute to IL-1beta release from human monocytic cells.

130 t to the secretome and the proteome of human monocytic cells.

131 ection with the activation status of porcine monocytic cells.

132 ge conversion from band-stage neutrophils to monocytic cells.

133 tective role for Panx1 in endothelial and/or monocytic cells.

134 yristate acetate (PMA) activated THP-1 human monocytic cells.

135 ntly suppressed exogenous viral infection in monocytic cells.

136 igration (85%), with similar results in U937 monocytic cells.

137 mpaired nuclear import of HIV-1 DNA in human monocytic cells.

138 atory host cells such as T cells and myeloid/monocytic cells.

139 Hemangioblastic and monocytic cEPC levels were not correlated (Spearman corr

140 nvestigate the levels of hemangioblastic and monocytic cEPCs in patients with periodontitis and perio

141 A non-significant trend for higher levels of monocytic cEPCs in periodontitis was also observed.

142 EPCs were assessed using flow cytometry, and monocytic cEPCs were identified using immunohistochemist

143 sed untreated SLE patients shared a distinct monocytic chemokine signature, despite clinical heteroge

144 id not alter tumor incidence implicating the monocytic compartment as the functionally immunosuppress

145 gene-deficient mouse strain, we analyzed the monocytic component of the inflammatory infiltrate in th

146 ri W620 were carried out using human primary monocytic dendritic cells (moDC) and co-culture with aut

147 howed an increase of CD11c(+)Ly-6C(+)CCR2(+) monocytic dendritic cells within the popliteal lymph nod

148 uman AML cases exhibited reduced morphologic monocytic differentiation and inferior survival.

149 The 15 kDa isoform is a potent inducer of monocytic differentiation to dendritic cells, but the 9

150 In mice, Dicer1 deletion blocked monocytic differentiation, depleted macrophages, and cau

151 ity or immunopathology during infection with monocytic Ehrlichia.

152 Human monocytic ehrlichiosis (HME) is caused by a tick-borne o

153 crophages and causes potentially fatal human monocytic ehrlichiosis (HME) that mimics toxic-shock-lik

154 ia chaffeensis, the etiologic agent of human monocytic ehrlichiosis (HME), has been extensively studi

155 te intracellular bacterium that causes human monocytic ehrlichiosis (HME).

156 Human monocytic ehrlichiosis is one of the most prevalent tick

157 ckettsial organism, causes the disease human monocytic ehrlichiosis.

158 r tick-borne bacterium responsible for human monocytic ehrlichiosis.

159 -term rEC-hMPP-derived myeloid (granulocytic/monocytic, erythroid, megakaryocytic) and lymphoid (natu

160 Thus, IRF8 does not regulate granulocytic vs monocytic fate in GMPs, but instead acts downstream of l

161 ngiotensin-II infusion induced the uptake of monocytic fibroblast precursors that initiated the devel

162 mainly contributed by the CD11b(+)Ly6C(high) monocytic fraction of the myeloid cells and not the tumo

163 inding assays demonstrated that granulocytic/monocytic (G/M) commitment is marked by Runx1 suppressio

164 Coexpression of the monocytic growth factor colony-stimulating factor (CSF)-

165 ingle cell transcriptional profiles of THP-1 monocytic human leukemia cells.

166 o determine whether women with TMD exhibit a monocytic hyperinflammatory response compared with contr

167 res reduced iNKT proliferation and abrogated monocytic IL-12 production.

168 lammatory disease with characteristic lympho-monocytic infiltration in lungs, livers, and kidneys, al

169 ontrol women, and to examine associations of monocytic inflammatory responses with clinical pain.

170 hly susceptible to OPC, indicating that this monocytic influx is insufficient for host defense.

171 In the YS, EMPs gave rise to MFs without monocytic intermediates, while EMP seeding the FL upon t

172 nduce proinflammatory cytokines in the human monocytic leukemia cell line THP-1 and bone marrow-deriv

173 Human fetal RPE and monocytic leukemia macrophage (THP-1) cell lines were cu

174 Monocytic leukemia zinc finger (MOZ)/KAT6A is a MOZ, Ybf

175 Here, we use deletion of the monocytic leukemia zinc finger gene (Moz/Kat6a/Myst3) to

176 Using stage-specific deletion of monocytic leukemia zinc finger protein (MOZ), a histone

177 ransferase complexes, where the gene of MOZ (monocytic leukemia zinc finger protein) was first identi

178 MORF [MOZ (monocytic leukemia zinc-finger protein)-related factor]

179 The monocytic leukemic zinc finger (MOZ) histone acetyltrans

180 s in the spleen that phenotypically resemble monocytic-like (CD11b(+)Ly6C(high)) and granulocytic-lik

181 Histiocytes are white blood cells of the monocytic lineage and include macrophages and dendritic

182 megakaryocytic-erythroid versus granulocytic-monocytic lineage decision-making.

183 s), multinucleated giant cells (MGCs) of the monocytic lineage, is bone resorption.

184 allows HIV-1 strains to infect cells of the monocytic lineage.

185 absence of TLR4, IFN-gamma, or depletion of monocytic-lineage cells or CLEC-2 on platelets markedly

186 est the in vivo relevance of these findings, monocytic-lineage-specific GCN2KO mice were challenged w

187 ytes, natural killer (NK) cells, and myeloid-monocytic lineages.

188 to megakaryocytic-erythroid and granulocytic-monocytic lineages.

189 e-stage vaccine testing, we demonstrate that monocytic (M)-MDSCs and polymorphonuclear (PMN)-MDSCs ca

190 e.We report here a unique mechanism by which monocytic (M)-MDSCs are spared, allowing them to polariz

191 CD14(pos)HLA-DR(low/neg) monocytic (M)-MDSCs were expanded in intensive care unit

192 TING activation is immunosuppressive due to (monocytic) M-MDSC infiltration, which results in tumor r

193 ically expressed by osteoblasts, but not for monocytic markers.

194 and the other carrying both endothelial and monocytic markers.

195 cumulation of polymorphonuclear MDSC but not monocytic MDSC (M-MDSC), and decreased polymorphonuclear

196 mulation of granulocytic MDSC (grMDSCs) over monocytic MDSC (moMDSCs).

197 Two major MDSC subsets, including monocytic MDSC and granulocytic MDSC, have been describe

198 (+)Ly6-G(-)Ly6-C(+) monocytic subtypes, with monocytic MDSC exhibiting higher T cell-suppressive func

199 Moreover, monocytic MDSC have the plasticity to differentiate into

200 , peptibodies depleted both granulocytic and monocytic MDSC subsets.

201 ate cells (HSC) have been reported to induce monocytic-MDSC from mature CD14(+) monocytes in a contac

202 Frequency of DC-HIL(+) monocytic MDSCs (CD14(+)HLA-DR(no/low)) in blood and ski

203 of myeloid-derived suppressor cells (MDSCs), monocytic MDSCs (M-MDSCs) and polymorphonuclear MDSCs (P

204 s from severe liver damage by recruitment of monocytic MDSCs and maintaining a balance between IL-1be

205 ne a functional role of CREMalpha in hepatic monocytic MDSCs for the pathogenesis of immune-mediated

206 Notably, monocytic MDSCs in Spp1(-/-) mice were less suppressive

207 uiting both CCR4(+) Treg and CCR2(+)Ly-6C(+) monocytic MDSCs in this disease setting.

208 Monocytic MDSCs predictably suppressed antitumor immune

209 et CCR2-expressing cells, we show that these monocytic MDSCs regulate entry of activated CD8 T cells

210 that CCR4-deficient Treg and CCR2-deficient monocytic MDSCs were defective in glioma accumulation.

211 We demonstrated augmentation of monocytic MDSCs whose suppression of not only T-cell, bu

212 results argue that therapeutic targeting of monocytic MDSCs would enhance outcomes in immunotherapy.

213 cking the recruitment of CD4 lymphocytes and monocytic MDSCs, respectively.

214 -cell PD-1 expression and reduced numbers of monocytic MDSCs.

215 nt mice failed to maximally accrue Tregs and monocytic MDSCs.

216 We previously demonstrated that LPS binds to monocytic membrane-bound thrombomodulin (TM), but the ro

217 rophages by blocking the association between monocytic membrane-bound TM and CD14.

218 ulating anti-inflammatory cytokines, reduced monocytic MHC class II expression and attenuated cytokin

219 We demonstrate here that monocytic (mMDSC) and granulocytic (gMDSC) subsets of my

220 of mouse tumor-infiltrating granulocytic and monocytic (MO-MDSC) subsets compared with MDSCs from per

221 In human epithelial DLD-1 and monocytic Mono Mac 6 cells resveratrol decreased the exp

222 lation that could be further subdivided into monocytic (mononuclear) and granulocytic (polymorphonucl

223 d PD-1+CD8+; 95% CI, 1.13-8.35; P = .01) and monocytic myeloid derived suppressor cells (mMDSC) (95%

224 ILC2s, in turn, activate monocytic myeloid-derived suppressor cells (M-MDSCs) via

225 ved an infiltration of neutrophils, T cells, monocytic myeloid-derived suppressor cells (M-MDSCs), an

226 Similar to monocytic myeloid-derived suppressor cells (M-MDSCs), OC

227 Interestingly, a subset of monocytic myeloid-derived suppressor cells (MDSCs) also

228 Recruitment of monocytic myeloid-derived suppressor cells (MDSCs) and d

229 Con A induced the recruitment of CD49d(+) monocytic myeloid-derived suppressor cells (MDSCs) and r

230 C)-treated WT, but not IFNAR-deficient mice, monocytic myeloid-derived suppressor cells (MDSCs) were

231 arkers (CD11b+Gr1+Ly6C+Ly6G-F4/80(low)) with monocytic myeloid-derived suppressor cells (MDSCs), had

232 Monocytic myeloid-derived suppressor cells (mMDSC) are a

233 tinib abrogates cytokine-driven expansion of monocytic myeloid-derived suppressor cells (mMDSC) from

234 Cao et al. report increased numbers of monocytic myeloid-derived suppressor cells (Mo-MDSC) in

235 Recently, CD14(+) HLA-DR(-/low) monocytic myeloid-derived suppressor cells (Mo-MDSCs) ha

236 Human monocytic myeloid-derived suppressor cells (MO-MDSCs) wi

237 ure of the TME by decreasing the presence of monocytic myeloid-derived suppressor cells and increasin

238 2, which resulted in the recruitment of both monocytic myeloid-derived suppressor cells and macrophag

239 e to inflammatory cytokines, Ly-6C(hi)G(neg) monocytic myeloid-derived suppressor cells expressing in

240 ased ERK and P38 MAPK signaling responses in monocytic myeloid-derived suppressor cells, which was pa

241 , including macrophages and granulocytic and monocytic myeloid-derived suppressor cells.

242 cells that produce lymphoid and granulocytic-monocytic (myeloid) lineages is unclear.

243 tein colocalized with CD68-positive cells of monocytic origin and muscle-actin-specific-antibody (HHF

244 These results implicate a monocytic origin of CD11c(+) cells in inflamed islets an

245 read, the most prominent among them being of monocytic origin such as tumor-associated macrophages (T

246 a subpopulation of human suppressor cells of monocytic origin, referred to as human monocyte-derived

247 tinal RPE and lipid-filled cells of probable monocytic origin.

248 itors(GMPs) during viral infection, favoring monocytic over the granulocytic differentiation.

249 n MDMs differentiated in the presence of non-monocytic peripheral blood mononuclear cells due in part

250 Toll-like receptor (TLR)-hyper-inflammatory monocytic phenotype has been implicated as a mechanism o

251 al fluid (CSF) analysis revealed lymphocytic/monocytic pleocytosis, elevated protein concentration, a

252 New studies suggest that circulating monocytic populations may play a role in mediating the i

253 of myeloid lineages and reduced granulocytic/monocytic populations.

254 )CD62L(hi) progenitors had full lymphoid and monocytic potential but lacked erythroid potential.

255 he two DC subsets appeared to share the same monocytic precursor and to be developmentally related; b

256 trigger TLR-dependent signaling pathways in monocytic precursor cells but possibly also in other imm

257 We generated human Tip-DC from monocytic precursor cells of healthy individuals, atopic

258 ved F4/80(+) Mvarphi population, but not non-monocytic precursor-derived CD103(+) dendritic cells.

259 t differentiation of TAMs in tumor site from monocytic precursors was controlled by downregulation of

260 From human monocytic precursors, we differentiated MGCs that expres

261 liferation, and maturation of their Ly6C(hi) monocytic precursors.

262 e, we show that Cebpa-deficient granulocytic-monocytic progenitors were equally resistant to transfor

263 n survival assays, we found that myeloid and monocytic progenitors were sensitive to GM-CSF signal in

264 the binary cell fate choices of granulocytic-monocytic progenitors(GMPs) during viral infection, favo

265 f immature cells containing granulocytic and monocytic progenitors, which expand under nearly all inf

266 red bone marrow-derived myeloid cells and in monocytic RAW264.7 cells, the LPS-induced expression of

267 Enhanced monocytic receptor for advanced glycation end products e

268 matory phenotype, manifested by an increased monocytic release of cytokines after an inflammatory ins

269 ovide an overview of the mechanisms by which monocytic subpopulations contribute to angiogenesis in t

270 granulocytic cell populations of MDSCs, the monocytic subset was the predominant source of OPN.

271 nitric oxide production, reminiscent of the monocytic subtype of myeloid-derived suppressor cells pr

272 +) granulocytic and CD11b(+)Ly6-G(-)Ly6-C(+) monocytic subtypes, with monocytic MDSC exhibiting highe

273 ulation of cell death pathways, we found the monocytic suppressor-cell subset, but not the granulocyt

274 Monocytic suppressors mediate T cell suppression, wherea

275 Monocytic suppressors tolerate the absence of MCL-1 prov

276 e unequivocal identification of ABA within a monocytic T helper 1 (THP-1) cell line, using lumogallio

277 In monocytic THP-1 and primary airway smooth muscle cells,

278 ablation of miRs 135b, 190, and 422a in the monocytic THP-1 cell line.

279 molecule 1 expression, resulting in reduced monocytic THP-1 cell recruitment in vitro.

280 experiments or RNA knockdown indicated that monocytic THP-1 cells and primary human neutrophils requ

281 teract with the cyclin A-CDK1-CDK2 complexin monocytic THP-1 cells and primary monocyte-derived macro

282 article, we report that retreatment of human monocytic THP-1 cells and primary monocytes with pathoge

283 Human monocytic THP-1 cells stimulated with MRP8/MRP14 express

284 Unlike monocytic THP-1 cells with prominent nuclear localizatio

285 acts prepared from in vitro propagated human monocytic THP-1 cells, which were used as a model cell l

286 the production of various cytokines in human monocytic THP-1 cells.

287 fected human embryonic kidney 293T cells and monocytic THP-1 cells.

288 FRbeta(+) macrophages and FRbeta-transduced monocytic THP-1 cells.

289 x and Transwell CC receptor transfectant and monocytic THP-1 migration assays.

290 Conversely, overexpression of VentX in monocytic THP1 cells accelerated their differentiation t

291 Monocytic TLR-induced cytokine patterns were shared amon

292 e-bound thrombomodulin (TM), but the role of monocytic TM in LPS-induced inflammation remains unknown

293 To further investigate the function of monocytic TM in vivo, myeloid-specific TM-deficient mice

294 on and immunofluorescence assays showed that monocytic TM interacted with the LPS receptors, CD14 and

295 Therefore, we conclude that monocytic TM is a novel component in the CD14/TLR4/MD-2

296 man donors, but it is drastically reduced in monocytic tumor cells, THP-1, and U937, rendering them i

297 and nonsmoking control subjects, or of human monocytic U937 cells exposed to cigarette smoke extract

298 alpha5beta1 and biologically activate human monocytic U937 cells expressing both receptors.

299 human primary macrophages and differentiated monocytic U937 cells, in which occludin silencing result

300 pithelial MKN45 and AZ-521 but also in human monocytic U937 cells.