ライフサイエンスコーパス: monocytic cell

1 igration (85%), with similar results in U937 monocytic cells.

2 thelial cell lines (HeLa and C33A) and THP-1 monocytic cells.

3 that was reversed by GSK3beta suppression in monocytic cells.

4 ity as well as transendothelial migration of monocytic cells.

5 This was confirmed in primary human monocytic cells.

6 selected mRNAs in interferon-gamma-activated monocytic cells.

7 an increased accumulation of S100A9-positive monocytic cells.

8 Ly6C(low) granulocytic and Ly6G(-)Ly6C(high) monocytic cells.

9 anscripts were identified in endothelial and monocytic cells.

10 ryo-1 up-regulates IFN-gammaR2 expression in monocytic cells.

11 ssion and regulation of IFN-gammaR chains in monocytic cells.

12 th IL-4 in enhanced CCL2 production in human monocytic cells.

13 expression in human PMA-differentiated THP-1 monocytic cells.

14 -mediated IL-1beta secretion requires ASC in monocytic cells.

15 ceptors (TLRs) ligands has been described in monocytic cells.

16 mpaired nuclear import of HIV-1 DNA in human monocytic cells.

17 related cytokines from differentiated human monocytic cells.

18 lysaccharide-induced TNF-alpha production in monocytic cells.

19 transcription initiation between T cells and monocytic cells.

20 atory host cells such as T cells and myeloid/monocytic cells.

21 porter gene in Jurkat T cells, but not THP-1 monocytic cells.

22 of cell surface ICAM-1 expressions in THP-1 monocytic cells.

23 the nucleus in human neutrophils, but not in monocytic cells.

24 rcellular infiltrate of atypical myeloid and monocytic cells.

25 and ERK with slightly different kinetics in monocytic cells.

26 ot contribute to IL-1beta release from human monocytic cells.

27 t to the secretome and the proteome of human monocytic cells.

28 ection with the activation status of porcine monocytic cells.

29 ge conversion from band-stage neutrophils to monocytic cells.

30 tective role for Panx1 in endothelial and/or monocytic cells.

31 ntly suppressed exogenous viral infection in monocytic cells.

32 yristate acetate (PMA) activated THP-1 human monocytic cells.

33 PRRSV is ideal for deciphering how monocytic cell activation statuses interact with antivir

34 at this contributes significantly to reduced monocytic cell adhesion to TNF-alpha-activated HAEC.

35 dothelial APP was also involved in mediating monocytic cell adhesion.

36 expression of adhesion molecules, leading to monocytic cell adhesion.

37 d that FKN was a major contributor to T- and monocytic-cell adhesion to HIMECs.

38 subsequent LPS challenge and is achieved by monocytic cells after prolonged exposure to LPS.

39 nd that HCV infects both CD14(+), CD16(+)(+) monocytic cells and CD14(+)(+), CD16(+)(+) monocytic cel

40 (AAT) decreases HIV replication in PBMCs and monocytic cells and decreases NF-kappaB activity, we pos

41 nd 15-prostaglandin dehydrogenase in primary monocytic cells and in a human Th2-related lung disease.

42 alis strain A7436 induces cell death in THP1 monocytic cells and in human primary peripheral blood ma

43 -1 transcription and replication in cultured monocytic cells and in primary monocyte-derived macropha

44 s (PRRSV), which directly infects subsets of monocytic cells and interferes with antiviral responses.

45 mation, we examined the interaction of THP-1 monocytic cells and isolated peripheral blood monocytes

46 (Gal3), a pleiotropic lectin, is produced by monocytic cells and macrophages.

47 association with the cytoskeleton in myeloid/monocytic cells and modulates IL-1beta processing, NF-ka

48 re that oxLDL induced TF expression in human monocytic cells and monocytes.

49 R-297 and -299 are endogenously expressed in monocytic cells and negatively regulate VEGFA expression

50 We treated human THP-1 monocytic cells and primary human blood monocytes with R

51 During differentiation of U937 monocytic cells and primary human CD14(+) monocytes, ST6

52 ty and proinflammatory cytokine secretion in monocytic cells and primary murine macrophages.

53 ucing expression of carbonic anhydrase II in monocytic cells and promoting acidification of the extra

54 o HIV gp120, for the neurotoxic phenotype of monocytic cells and subsequent toxin-initiated neuronal

55 nity, because it directly infects subsets of monocytic cells and subverts overall immune responses.

56 Cooperative responses between monocytic cells and tissue cells are likely to be crucia

57 creases TNF-alpha secretion in cultured U937 monocytic cells and TNF-alpha levels in the blood of typ

58 RANKL induced osteoclastogenesis in these monocytic cells, and curcumin inhibited both RANKL- and

59 af7, is expressed by resting primary myeloid/monocytic cells, and its expression in these cells is re

60 , survival and properties of agrin-deficient monocytic cells, and occur at stages later than stem cel

61 cell surface RON expression in HIV-infected monocytic cells, and Tat-mediated degradation of RON pro

62 owever, the presence and function of PKD1 in monocytic cells are currently unknown.

63 e LPS-induced TNF-alpha and TF expression in monocytic cells are currently unknown.

64 intact, demonstrating that G-CSFR signals in monocytic cells are sufficient to induce HSPC mobilizati

65 at short-term stimulation of human and mouse monocytic cells as well as mouse osteoblasts with P2RX7

66 cytokines, human TNFalpha, and IL-8 in THP-1 monocytic cells as well as mouse TNFalpha and MIP-2 in R

67 luble CD163 likely originates from activated monocytic cells, as serum from stroke patients stimulate

68 tion, variola was disseminated by means of a monocytic cell-associated viremia.

69 ills proliferating and nonproliferating U937 monocytic cells at a comparable specific activity, appro

70 Importantly, porcine monocytic cells at different activation states were susc

71 se pathways in uninfected and PRRSV-infected monocytic cells at different activation states.

72 Although it induces chemotaxis of monocytic cells at high concentrations, its physiologica

73 , we demonstrated that TM knockdown in human monocytic cells attenuated LPS-induced signaling pathway

74 d that miR-146a is critical for the in vitro monocytic cell-based endotoxin tolerance.

75 t miR-146a plays a crucial role for in vitro monocytic cell-based endotoxin-induced cross-tolerance.

76 from lipopolysaccharide (LPS)-treated THP-1 monocytic cells bind to and are internalized by human en

77 ) monocytic cells and CD14(+)(+), CD16(+)(+) monocytic cells but not CD14(+)(+), CD16- cells in indiv

78 ral proteins implicated in driving cancer in monocytic cells but only harbor limited activity in epit

79 This site exists in T cells and monocytic cells, but not in B cells or fibroblasts.

80 rs transcriptional activation in T cells and monocytic cells, but not in B cells.

81 cell proliferation and ERK1/2 activation in monocytic cells, but the integrin binding-defective R74E

82 nhanced immune response that is dependent on monocytic cells, but this hyperimmune phenotype and its

83 we present evidence that upon stimulation of monocytic cells by M. tuberculosis a unique TNF-alpha en

84 type 1 (HIV-1) infection in noncycling human monocytic cells by reducing the intracellular deoxynucle

85 ssion with small interference RNA in myeloid/monocytic cells caused a dramatic increase in NFkappaB a

86 We observed that monocytic cells cocultured (but not in contact) with ECs

87 d TLR4 surface expression in HIV-infected U1 monocytic cells compared to the uninfected parental U937

88 A forms and induced less TNF-alpha in THP-1 monocytic cells compared with LOS from group 1.

89 se of the autofluorescent signal recorded in monocytic cells could be correlated with the IR detectio

90 norrhoeae infection promotes NLRP3-dependent monocytic cell death via pyronecrosis, a recently descri

91 solution of inflammation is characterized by monocytic cell-dependent production of proangiogenic fac

92 feasibility and efficacy of IU injections of monocytic cells (derived from normal marrow) in feline a

93 s relayed by the MKK6-p38MAPK cascade induce monocytic cell differentiation from band-stage neutrophi

94 Monocytic cell differentiation potential was retained in

95 C/EBPalpha, which enabled the induction of a monocytic cell differentiation program.

96 d the effects of ex vivo-derived exosomes on monocytic cell differentiation/activation using THP-1 ce

97 ns, overexpression of wild-type ILK in human monocytic cells diminishes beta(1) integrin/vascular cel

98 fect of Panx1 deletion in endothelial and/or monocytic cells during atherogenesis is counterbalanced

99 We show that the donor monocytic cells engraft and persist (for up to 125 days)

100 Monocytic cells exposed to Borrelia burgdorferi, through

101 Monocytic cells express several P2Y receptors but the ro

102 elate with inefficient HIV-1 transmission by monocytic cells expressing DC-SIGN.

103 pon primary but not secondary stimulation of monocytic cells (FimA tolerance).

104 X-2 expression and PGE(2) production in THP1 monocytic cells following infection with Porphyromonas g

105 We found that migration of monocytic cells, formation of L. monocytogenes-containin

106 In particular, miR-10a was transferred to monocytic cells from EC-EVs and could repress inflammato

107 re normal in KLF4(-/-) chimeras, bone marrow monocytic cells from KLF4(-/-) chimeras expressed lower

108 erant, and there is increasing evidence that monocytic cells from patients with systemic inflammatory

109 mportant skin-based factor regulating CD11b+ monocytic cell function in the acute post-ultraviolet pe

110 In monocytic cells, geldanamycin (GA), an Hsp90 inhibitor,

111 present study was to examine changes in the monocytic cell gene-expression profile in response to C.

112 A new population of monocytic cells has been found to express LYVE-1 in norm

113 Monocytic cells have been found to be an important part

114 been implicated in neurotoxin production by monocytic cells (i.e., macrophages and microglia), as we

115 The reduction of monocytic cells in Ccr2(-/-) mice or after Gr-1 antibody

116 r, our study reveals a dynamic modulation of monocytic cells in response to varying dosages of endoto

117 7 increases the number of mature myeloid and monocytic cells in spleen and peripheral blood.

118 pports a role for IL-27 in the activation of monocytic cells in terms of inflammatory responses.

119 ompanied by increased numbers of myeloid and monocytic cells in the bone marrow resembling the myelop

120 -type mice is associated with marked loss of monocytic cells in the bone marrow.

121 ecreted by primary human monocytes and THP-1 monocytic cells in the presence of alcohol in a concentr

122 aneously measure the mechanical stiffness of monocytic cells in three rotational and two translationa

123 in the gastrointestinal tract) and lymphoid/monocytic cells in tonsils and Peyer's patches (explaini

124 in the gastrointestinal tract) and lymphoid/monocytic cells in tonsils and Peyer's patches (explaini

125 ctures in the ECM, providing a substrate for monocytic cells in vitro, whereas lung fibroblasts from

126 ts of DNA and DNA-coated beads by U937 human monocytic cells, in vitro.

127 g studies revealed SAMHD1 tetramers in human monocytic cells, in which it strongly restricts HIV-1 in

128 Monocytic cells, including macrophages and dendritic cel

129 Activation statuses of monocytic cells, including monocytes, macrophages (Mvarp

130 We found that cross-linking of CD13 on U937 monocytic cells induced phosphorylation of a number of p

131 hat M. tuberculosis infection of human THP-1 monocytic cells induced the down-regulation of CatG mRNA

132 n cellular iron release was examined in U937 monocytic cells induced to differentiate to the macropha

133 The monocytic cells infected with viable bacteria markedly d

134 We observed spontaneous monocytic-cell infiltration in the lungs of Serpine2-def

135 model in which G-CSFR signals in bone marrow monocytic cells inhibit the production of trophic factor

136 n a mixed culture containing fibroblasts and monocytic cells, interferon-gamma stimulated Trx release

137 tein by gamma interferon (IFN-gamma) in U937 monocytic cells is halted by a delayed translational sil

138 We showed that in monocytic cells KLF4 has to be repressed to allow their

139 g various phases of HIV-1 infection using U1 monocytic cells (latently infected U937 cells with HIV-1

140 stimulation of TLR2 in human epithelial and monocytic cells leads to rapid and transient activation

141 High glucose treatment of THP-1 monocytic cells led to a significant three- to fivefold

142 tional changes was carried out using a human monocytic cell line (THP-1 cells) in response to differe

143 to induce inflammatory responses in a human monocytic cell line (THP-1 cells).

144 ed with TLR2/4-mediated responses in a human monocytic cell line (THP-1) and in human primary monocyt

145 a fully endotoxin tolerant state in a human monocytic cell line (THP-1) and mouse bone marrow-derive

146 ecretion were conducted with the human THP-1 monocytic cell line and corroborated in primary human pe

147 r of cytokine signaling 1 (SOCS1) in a human monocytic cell line and in HEK293-TLR4/MD2 cells stably

148 The THP-1 human monocytic cell line and purified recombinant lipidated O

149 Human THP-1 monocytic cell line cells were infected with C. pneumoni

150 Also, overexpression of miR-155 in the THP1 monocytic cell line decreases PU.1 protein levels and DC

151 d IL-12 production in a NEMO knockdown human monocytic cell line following LPS treatment.

152 T. gondii, we genetically engineered a human monocytic cell line for NALP1 gene knockdown by RNA inte

153 We report the establishment of a monocytic cell line latently infected with KSHV (KSHV-TH

154 we knocked down these proteins in the human monocytic cell line Mono Mac 6 (MM6).

155 of the pro-inflammatory cytokine IL-6 in the monocytic cell line Mono Mac 6, induction of the Toll-in

156 When used to prime the human monocytic cell line MonoMac 6, the production of TNF-alp

157 We show here that infection of the murine monocytic cell line P388D1 with either Pichinde virus va

158 Similar regulation was observed in the monocytic cell line RAW264.7.

159 In the THP-1 human monocytic cell line stably expressing the HIV-LTR-lucife

160 the interaction of C. jejuni with the human monocytic cell line THP-1 and show that a range of proin

161 y, we observe that depletion of BICD2 in the monocytic cell line THP-1 results in an induction of IFN

162 CD1d ligation on the human monocytic cell line THP-1 similarly specifically stimula

163 Here, we investigated the adhesion of the monocytic cell line THP-1 to a surface coated with inter

164 The human monocytic cell line THP-1 treated with lipopolysaccharid

165 -triggered antimicrobial activity, the human monocytic cell line THP-1 was infected with M. tuberculo

166 s required for ehrlichial infection of human monocytic cell line THP-1 were characterized.

167 nalysis revealed that treatment of the human monocytic cell line THP-1 with LPS induced a rapid and t

168 IL-18BPa secretion in cultures of the human monocytic cell line THP-1, as measured by specific ELISA

169 nhibited CCL2-driven chemotaxis of the human monocytic cell line THP-1, CCL3-driven chemotaxis of per

170 Using the monocytic cell line THP-1, we show that angiocidin induc

171 were highly and equally active in the human monocytic cell line THP-1, which has high constitutive e

172 phabeta signaling in human APC and the human monocytic cell line THP-1, which provides a model for th

173 ing on E. chaffeensis infection of the human monocytic cell line THP-1.

174 osa-phagocyte interaction by using the human monocytic cell line THP-1.

175 to lipid rafts in blood monocytes and in the monocytic cell line THP-1.

176 q and bacA mutants was followed in the human monocytic cell line THP-1.

177 A3A in CD14(+)-enriched primary cells or the monocytic cell line THP-1.

178 To model systemic effects, we used the human monocytic cell line THP-1; to model effects in the centr

179 vity in a time-dependent manner in the human monocytic cell line THP1.

180 broma cells stimulated chemotaxis of a human monocytic cell line to a greater extent than conditioned

181 uses a minimal defect in growth in the human monocytic cell line U937 and the environmental host Acan

182 trophils and diminished calcium signaling in monocytic cell line U937 transfected with the C5a recept

183 human cells, we stably transfected the human monocytic cell line U937 with murine IL-4R(alpha) cDNA b

184 creases TET2 protein expression in the human monocytic cell line U937.

185 The THP-1 human monocytic cell line was coincubated for 3 h with medium

186 A human monocytic cell line was genetically engineered using len

187 Knocking down DJ-1 (siRNA) in THP-1 (human monocytic cell line) and polymorphonuclear cells from pa

188 p47phox were increased in THP-1 cells (human monocytic cell line) under HG (15 mmol/l glucose) condit

189 cytokine induced IL-6 expression in a human monocytic cell line, and (3) AhR-deficient mice are resi

190 In the THP-1 human monocytic cell line, reducing AIRE expression resulted i

191 In peripheral blood monocytes and the THP-1 monocytic cell line, SAA induces the expression of IL-12

192 We used a human monocytic cell line, THP-1, and dendritic cells to study

193 ome, but not the plasma membrane, in a human monocytic cell line, THP-1, and primary human monocyte-d

194 We have found that infection of the human monocytic cell line, THP-1, resulted in reduced cellular

195 In the human monocytic cell line, THP-1, SHIP-1 became tyrosine-phosp

196 Using primary mouse microglia and the human monocytic cell line, THP-1, we have begun investigating

197 r in stably transfected CHO cells and in the monocytic cell line, THP-1.

198 man airway smooth muscle cells and the human monocytic cell line, THP-1.

199 Using a monocytic cell line, THP1, we show that LPS activates en

200 itself, on adhesion of THP-1 cells, a human monocytic cell line, to vascular endothelial cells (ECs)

201 At the molecular level, in a human monocytic cell line, U937 IL-10 suppressed LPS-induced m

202 Using a human monocytic cell line, we demonstrate that Porphyromonas g

203 MCs from patients with APECED syndrome and a monocytic cell line.

204 stimulated potent calcium flux in the human monocytic cell line.

205 tic differentiation using RAW264.7, a murine monocytic cell line.

206 eoclastogenesis in RAW 264.7 cells, a murine monocytic cell line.

207 ]) in both normal monocytes and in the THP-1 monocytic cell line.

208 1-dependent IL-1beta generation in the THP-1 monocytic cell line.

209 ester accumulation in foam cells of the THP1 monocytic cell line.

210 ased consequent late cell death of the human monocytic cell line.

211 an undifferentiated, normally nonpermissive monocytic cell line.

212 effects on migration of THP-1 cells, a human monocytic cell line.

213 own in primary human microglia and the human monocytic cells line THP-1 cells, using diverse cell sti

214 induces less inflammatory signaling in human monocytic cell lines and murine macrophages than the par

215 lial cells and measured the binding of human monocytic cell lines and peripheral blood monocytes.

216 s of vitamin A, repress HIV-1 replication in monocytic cell lines and primary macrophages by blocking

217 vatives of the A549 lung carcinoma and THP-1 monocytic cell lines and used these to study T cell resp

218 neal exudate cells, and adoptive transfer of monocytic cell lines engineered to express the mutant CD

219 revealed that Bryo-1 treatment of the human monocytic cell lines MonoMac6 and THP-1 and human monocy

220 observed similar recombination rates in two monocytic cell lines regardless of the differentiation s

221 IRF-1 from undifferentiated, uninfected monocytic cell lines was modified during extraction to p

222 een A1PI expression in primary monocytes and monocytic cell lines, and inflammatory cytokine expressi

223 nduced by P. gingivalis LPS in a human THP.1 monocytic cell lines.

224 cycle in primary human T lymphocytes and in monocytic cell lines.

225 uence of HIV on global lncRNAs expression in monocytic cells lines.

226 ntiviral infection with activation status of monocytic cells may provide a means of potentiating anti

227 y in differentiation into dendritic cells of monocytic cells migrating into ultraviolet-exposed skin.

228 Although monocytic cell migration peaked at 1-3 d after ultraviol

229 protein annexin 1 (ANXA1) on the process of monocytic cell migration was studied using transfected U

230 sue factor procoagulant activity (TF PCA) on monocytic cells more potently than other stimuli that de

231 ured by flow cytometry using uptake by THP-1 monocytic cells of fluorescent beads coated with gp120,

232 ortunately, the activation status of porcine monocytic cells or how cell activation status functional

233 tory cytokine release was evaluated in THP-1 monocytic cells or THP-1 cells lacking the inflammasome

234 ganism has been detected in peripheral blood monocytic cells (PBMCs).

235 The discovery of this novel adipose tissue monocytic cell population provides advances toward under

236 Selective depletion of this monocytic cell population, using either clodronate-lipos

237 abetes resulted in increased accumulation of monocytic cells positive for S100A9, a proinflammatory b

238 These monocytic cells represent one of the best-defined human

239 cation by phorbol ester in latently infected monocytic cells requires the posttranscriptional inducti

240 ntal antagonism of miR-452 in differentiated monocytic cells resulted in increased expression of MMP1

241 gest that M. tuberculosis infection of human monocytic cells results in a "cathepsin switch" with dow

242 f infected human, primary, neuronal stem and monocytic cells revealed effects on neurodevelopment and

243 peripheral-blood mononuclear cells and THP-1 monocytic cells secreted TNF- alpha in response to cervi

244 Thus, incubation of U937 monocytic cells stably expressing L-selectin (U937LAM) w

245 ll-like receptor (TLR) 2 in human and murine monocytic cells stimulated with Borrelia burgdorferi.

246 s expression of APOL1 in differentiated U937 monocytic cells stimulated with IFN-gamma resulted in a

247 ng, IL-1F7 was localized in human peripheral monocytic cells, suggesting its role in immune regulatio

248 otein (AP)-1 was reduced in TLR2-neutralized monocytic cells, suggesting that AP-1 plays a role in th

249 plasminogen activator inhibitor-1 (PAI-1) by monocytic cells, suggesting that TSP1-induced macrophage

250 Enforced expression of human ABIN-3 in human monocytic cells suppressed the cytoplasmic degradation o

251 ) greater opsonization of these IEs by human monocytic cells than IgGs from malaria-exposed Malian me

252 linically characterized by overproduction of monocytic cells that can infiltrate organs, including th

253 ldren characterized by the overproduction of monocytic cells that infiltrate the spleen, lung, and li

254 ation was seen especially in lymphocytes and monocytic cells, the natural hosts for HIV-1 infection.

255 thia, stimulate cytokine production in human monocytic cells (THP-1) through Toll-like receptor 9 (TL

256 Monocytic cells (THP-1) were cultured in the presence of

257 , we showed that Slit2 inhibited adhesion of monocytic cells to activated human endothelial cells, as

258 investigate the activation status of porcine monocytic cells to determine the intricate interaction o

259 nfluence the antigen presenting phenotype of monocytic cells to determine the nature of T cell respon

260 th ALF significantly reduced the adhesion of monocytic cells to immobilized P-selectin.Fc.

261 rom erythroid cells, activated MNC and THP-1 monocytic cells to induce LT production.

262 ower concentrations of human GzmA stimulated monocytic cells to secrete proinflammatory cytokines (in

263 tease thrombin in regulating the adhesion of monocytic cells to smooth muscle cells producing an infl

264 n and the recruitment of bone marrow-derived monocytic cells to the hypothalamus; in addition, this i

265 rom CD11c(dim)CD11b(int)Gr1(-) lung-resident monocytic cells transformed by KRAS(G12D) In contrast, B

266 is 5 x 5 array, which included separation of monocytic cells (U937) or human T cell leukemia virus ty

267 human epithelial pulmonary cells (A549) and monocytic cells (U937) upon IAV infection using synchrot

268 Cultured human monocytic cells, U937, were differentiated into macropha

269 pression and mechanism of their induction in monocytic cells under high-glucose conditions.

270 A and IL-15Ralpha appeared on the surface of monocytic cells upon activation.

271 These data combine to suggest that monocytic cells use a cytoplasmic retention mechanism to

272 apable of extensive replication within human monocytic cell vacuoles and induces apoptotic death via

273 ion-channel function of ATP receptor P2X7 in monocytic cells via nAChR containing alpha9 and alpha10

274 -alpha (TNF-alpha) and tissue factor (TF) in monocytic cells via the activation of the transcription

275 Inflammatory signaling in human THP-1 monocytic cells was much greater with pathogenic than wi

276 Furthermore, KYNA-triggered adhesion of monocytic cells was reduced by short hairpin RNA-mediate

277 study antiviral responses in PRRSV-infected monocytic cells, we characterized inflammatory cytokine

278 When extracts of human monocytic cells were chromatographed on a CRID affinity

279 ions of pyrin and caspase-1 from THP-1 human monocytic cells were consistent with the interaction of

280 At 48 hpf, monocytic cells were evident in both the ICM and circula

281 CD19+ B cells and CD11c+ monocytic cells were found in the spleen, lung, liver, a

282 Inflammatory monocytic cells were not observed infiltrating the skin

283 the two genomovars was preserved when human monocytic cells were stimulated with purified LPS.

284 ll types, primary human CD4 T-cells and U937 monocytic cells, were used to compare differences in cyt

285 t in apoptosis in many cell types, including monocytic cells, whereas transient activation of P2RX7 i

286 te receptor ET-BR in Kupffer cells and THP-1 monocytic cells, which also involved HIF-1alpha activati

287 and other galactose-related genes in myeloid-monocytic cells, which could affect energy utilization a

288 rst genome-wide analysis of TNF tolerance in monocytic cells, which differentially inhibits NF-kappaB

289 Using RAW 264.7 mouse monocytic cells, which express endogenous MCH receptor,

290 n, it is dispensable in B, T, dendritic, and monocytic cells, which is in contrast with an essential

291 receptor, purinergic receptor X7 (P2RX7), on monocytic cells, which promotes the processing/release o

292 Infection of human THP-1 monocytic cells with a MYXV construct deleted for the M0

293 Treatment of monocytic cells with ALF for 2 h significantly reduced t

294 se to CCL5 demonstrated that pretreatment of monocytic cells with ALF reduced migration (p = 0.0004)

295 paB proteins before and after stimulation of monocytic cells with bacterial lipopolysaccharide (LPS).

296 ae on host messenger RNA expression in human monocytic cells with complement DNA microarrays was stud

297 upon T. gondii infection was not observed in monocytic cells with NALP1 knockdown.

298 vels are stable upon infection of human THP1 monocytic cells with Pg but decrease after cytokine indu

299 sialylation in macrophages, we treated U937 monocytic cells with PMA, which stimulates both macropha

300 pe 1 (HIV-1) coreceptors (CCR5 and CXCR4) by monocytic cells within human genital ulcers.