ライフサイエンスコーパス: monocytic leukemia

1 on was detected in one individual with acute monocytic leukemia.

2 retroviral insertional mutagenesis in murine monocytic leukemias.

3 between proliferation and differentiation in monocytic leukemias.

4 Cluster F (n = 33) was dominated by monocytic leukemias (97% of cases), also showing increas

5 ding WM164 melanoma, WM35 melanoma, U937 pro-monocytic leukemia, and HT1080 fibrosarcoma cells, XAF1

6 in the presence of OPN increased human acute monocytic leukemia cell line (THP-1) monocyte binding, w

7 LXA4 also induced a rise in [Ca2+]i in the monocytic leukemia cell line (time to reach maximum = 15

8 E. sennetsu, but not HGE agent, in the acute monocytic leukemia cell line THP-1 almost completely inh

9 nduce proinflammatory cytokines in the human monocytic leukemia cell line THP-1 and bone marrow-deriv

10 phospholipid surfaces and THP-1 (human acute monocytic leukemia cell line) monocytes.

11 ing in control of differentiation in a human monocytic leukemia cell line, THP-1 cells were transient

12 n of the E. chaffeensis inclusion in a human monocytic leukemia cell line, THP-1 cells, implying that

13 man peripheral blood monocytes and the acute monocytic leukemia cell line, THP-1.

14 the binding of phospholipid vesicles to the monocytic leukemia cell lines THP-1 and J774A.1 with a f

15 ion to paclitaxel-induced apoptosis in human monocytic leukemia cells (U937).

16 ctin ligands in leukocytes and myelocytic or monocytic leukemia cells are carried by transmembrane gl

17 lso induced in primary macrophages and THP-1 monocytic leukemia cells by the phorbol ester 12-O-tetra

18 ochondrial dysfunction and apoptosis in U937 monocytic leukemia cells exposed to the proteasome inhib

19 1WAF1/CIP1 on the apoptotic response of U937 monocytic leukemia cells to 1-beta-D-arabinofuranosylcyt

20 Exposure of U937 monocytic leukemia cells to a marginally toxic concentra

21 Exposure of U937 monocytic leukemia cells to minimally toxic concentratio

22 Simultaneous exposure (30 hours) of U937 monocytic leukemia cells to minimally toxic concentratio

23 Here, we show that acute exposure of THP-1 monocytic leukemia cells to the phorbol ester 12-O-tetra

24 e demonstrated that treatment of THP-1 human monocytic leukemia cells with Z-LLL-CHO, a reversible pr

25 cium ([Ca2+]i) in PBM and THP-1 cells (acute monocytic leukemia cells) as well as on the functional r

26 atic ductal epithelial-like cells, and THP-1 monocytic leukemia cells).

27 This suggests that in monocytic leukemia cells, an antagonism exists between t

28 In monocytes and monocytic leukemia cells, integrin-mediated adhesion res

29 In THP-1 monocytic leukemia cells, phorbol ester-induced stabiliz

30 compound inhibited sLe(X) formation in U937 monocytic leukemia cells, suggesting that it had inhibit

31 titutively higher H(2)O(2) content than U937 monocytic leukemia cells.

32 trated in two variants derived from the U937 monocytic leukemia in the absence of exogenous inhibitor

33 Acute monocytic leukemia is a distinct subtype of acute myeloi

34 In patient 3, acute monocytic leukemia-M3 with t(15;17) arose 18 months afte

35 Human fetal RPE and monocytic leukemia macrophage (THP-1) cell lines were cu

36 oliferator-activated receptor-alpha in human monocytic leukemia THP-1 cells and human aortic endothel

37 detected in E. chaffeensis cultured in human monocytic leukemia THP-1 cells.

38 Ca2+-independent activity of CaM-KII in the monocytic leukemia, U937.

39 the t(8;16)(p11;p13) translocation in acute monocytic leukemia with erythrophagocytosis that fuses M

40 The monocytic leukemia zinc finger (MOZ) gene encodes a larg

41 The human monocytic leukemia zinc finger (MOZ) protein is an essen

42 Monocytic leukemia zinc finger (MOZ)/KAT6A is a MOZ, Ybf

43 Here, we use deletion of the monocytic leukemia zinc finger gene (Moz/Kat6a/Myst3) to

44 Using stage-specific deletion of monocytic leukemia zinc finger protein (MOZ), a histone

45 V-1 Tat interactive protein Tip60, the human monocytic leukemia zinc finger protein MOZ and the yeast

46 ransferase complexes, where the gene of MOZ (monocytic leukemia zinc finger protein) was first identi

47 in reaction (RT-PCR) that the genes for MOZ, monocytic leukemia zinc finger protein, and TIF2, transc

48 to 72.1 kb within the third intron of MORF (monocytic leukemia zinc finger protein-related factor or

49 man oncogenic histone acetyltransferase MOZ (monocytic leukemia zinc finger) is required for specifyi

50 MORF [MOZ (monocytic leukemia zinc-finger protein)-related factor]