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1 , with prefibrotic myelofibrosis or reactive monocytosis.
2 levels and subsequent hyperlipidemia-induced monocytosis.
3 morphological features of myelodysplasia or monocytosis.
4 pment of severe anemia with macrocytosis and monocytosis.
5 ype mice with artificially induced Ly-6C(hi) monocytosis.
6 ral infections like Epstein-Barr virus cause monocytosis.
7 cing panels that effectively identify clonal monocytosis and complement clinical prognostic scoring s
10 nant condition that presents with persistent monocytosis and is often associated with leukocytosis, l
12 oth CCL2 and CCL7 are required for efficient monocytosis and monocyte accumulation in the CNS, only C
13 )/(-) mice fed a chow or Western- type diet, monocytosis and neutrophilia developed in association wi
15 KO) BM-transplanted Ldlr(-/-) mice displayed monocytosis and neutrophilia in the absence of hematopoi
16 ases in IL-23 and IL-17A and proinflammatory monocytosis and neutrophilia that precedes development o
18 ted with a blunting of hyperglycemia-induced monocytosis and reduced monocyte recruitment to the plaq
19 phage proinflammatory responses, Ly-6C(high) monocytosis, and atherosclerosis in the settings of hype
21 y via amelioration of hyperlipidemia-induced monocytosis, and can be augmented with a synthetic CXCR7
23 ow myeloid progenitor cell proliferation and monocytosis, as well as reduced atherosclerosis and a st
27 2 mediates highly selective peripheral blood monocytosis during WNV infection of mice and that this i
29 Defined by a persistent peripheral blood monocytosis >/=1 x 10(9)/L and monocytes accounting for
31 e/monocyte inflammatory activation patterns (monocytosis, high-inflammatory gene expression, raised g
32 reatment-induced resolution of lymphopaenia, monocytosis, hypercytokinaemia, and hyperchemokinaemia.
35 which prompts the question as to whether the monocytosis in childhood obesity contributes to atheroge
36 aim of this study was to test whether blood monocytosis in mice with atherosclerosis affects infarct
37 ) mice induced a strong and highly selective monocytosis in peripheral blood that was absent in Ccr2(
38 nk4b(fl/fl)-LysMcre mice develop nonreactive monocytosis in the peripheral blood accompanied by incre
39 neoplasm characterized by myelodysplasia and monocytosis, including but not limited to, chronic myelo
41 asm (MPN) overlap disorders characterized by monocytosis, myelodysplasia, and a characteristic hypers
42 autonomously to control HSPC proliferation, monocytosis, neutrophilia, and monocyte accumulation in
43 oduction, glomerulonephritis, and the unique monocytosis of BXSB males, were severely reduced or abse
44 th healthy donors and patients with reactive monocytosis or another hematologic malignancy, CMML pati
45 ced frequencies of marginal zone B cells and monocytosis, renal disease, and premature morbidity.
46 atherosclerotic mice with chronic Ly-6C(hi) monocytosis results in impaired healing, underscoring th
48 that WNV infection induces a CCR2-dependent monocytosis that precedes monocyte migration into the CN
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