ライフサイエンスコーパス: monosynaptic

1 majority of responses from area 3a were non-monosynaptic.

2 ents, bypass local circuitry and have direct monosynaptic access to neurons projecting to brainstem a

3 e via mutual projections that give each area monosynaptic access to the other area's CSPs.

4 ional transmission was chiefly restricted to monosynaptic actions.

5 ation induced unmasking and strengthening of monosynaptic Adelta drive to lamina I NK1R(+) neurons ma

6 ofold) and magnitude (by 75% in a subset) of monosynaptic Adelta-fiber but not monosynaptic C-fiber-e

7 ut (69%) with a smaller proportion receiving monosynaptic Adelta-fiber input (28%).

8 s revealed that a proportion received silent monosynaptic Adelta-fiber input, suggesting that these m

9 sults of the present study indicate that the monosynaptic afferent input to mitral cells depends on t

10 lomerulus revealed that mitral cells receive monosynaptic afferent inputs.

11 virus in a transgenic Gpr151-Cre mouse line, monosynaptic afferents of habenular and thalamic Gpr151-

12 l activity by timing the neuronal firing via monosynaptic afferents, thalamic nuclei act as a relay s

13 rograde labeling in the amgBST compared with monosynaptic and 48-hour cases, whereas MEAad-injected c

14 nism that depends on the activity balance of monosynaptic and disynaptic pathways to inhibitory neuro

15 d and new M1 generated putative long-latency monosynaptic and non-monosynaptic effects; the majority

16 The sensory to motor pathways are monosynaptic and oligosynaptic in this system, thus prov

17 ns in three anesthetized monkeys revealed no monosynaptic and only one weak oligosynaptic EPSP after

18 ulation of neurons in the mPFC that received monosynaptic and orthodromic inputs from the BLA demonst

19 st, the amplitudes of intracortically evoked monosynaptic and polysynaptic GABAergic inhibitory synap

20 (R)-CPP, GluN2A/B antagonists, blocked both monosynaptic and polysynaptic NMDA EPSCs initiated by pr

21 lamina III projection cells receive powerful monosynaptic and polysynaptic nociceptive input.

22 tion of synaptic NMDA receptors expressed in monosynaptic and polysynaptic pathways from peripheral s

23 Communication capacity via monosynaptic and polysynaptic pathways, in aggregate, la

24 and confirms their functional connection via monosynaptic and polysynaptic pathways.

25 mulation of excitatory afferents evoked both monosynaptic and polysynaptic responses distributed in t

26 Using monosynaptic and polysynaptic viral tracers, we found th

27 here were no significant differences between monosynaptic and PRV cases in the subnuclear distributio

28 in the central and medial extended amygdala, monosynaptic and transneuronal viral tracing experiments

29 ere observed on both sides, although middle (monosynaptic) and late (long latency) responses were mor

30 n receptor, which allowed a demonstration of monosynaptic anterograde tracing from defined cells.

31 These synaptic responses were mediated by monosynaptic as well as recurrent polysynaptic input.

32 mined with network analysis tools applied to monosynaptic association (within one side) and commissur

33 Although monosynaptic bulbospinal projections to phrenic motoneur

34 ted that CFA inflammation reduced ADS in the monosynaptic C fiber input to lamina I neurokinin 1 rece

35 Control neurons predominantly received monosynaptic C-fiber input (69%) with a smaller proporti

36 subset) of monosynaptic Adelta-fiber but not monosynaptic C-fiber-evoked responses.

37 , possibly via the release of glutamate from monosynaptic C1 inputs.

38 I (ECII)-->dentate gyrus-->CA3-->CA1 and the monosynaptic circuit ECIII-->CA1 have been considered th

39 The monosynaptic circuit revealed here provides a neural sub

40 st, light inhibits pumping via the I2 neuron monosynaptic circuit.

41 opamine currents were separated into a fast, monosynaptic component and a slower-rising and decaying

42 that part of this locomotor drive involved a monosynaptic component coming directly from the lumbar l

43 ation (LTF) [6], which can be induced in the monosynaptic connection between Aplysia sensory and moto

44 gly, we uncovered a hitherto uncharacterized monosynaptic connection between cranial sensory neurons

45 ally assisted circuit mapping demonstrates a monosynaptic connection between these cholinergic neuron

46 rthermore, interneurons receiving a putative monosynaptic connection from a simultaneously recorded p

47 e of a direct Cajal-Retzius cell-interneuron monosynaptic connection.

48 - to fourfold higher than the probability of monosynaptic connections among corticocortical or cortic

49 We first found that the frequency of monosynaptic connections among corticostriatal pyramidal

50 -induced intramembrane proteolysis to reveal monosynaptic connections arising from genetically labele

51 Monosynaptic connections between muscle spindle (Ia) aff

52 gated the NMDA receptor-mediated currents at monosynaptic connections between pairs of layer 5 pyrami

53 d with two-photon microscopy to characterize monosynaptic connections between the Purkinje cells of j

54 ss-correlations in these preparations showed monosynaptic connections from 16/19 (84%) of EBSNs, but

55 ndicating that NTS CA neurons receive direct monosynaptic connections from afferent terminals.

56 le-cell patch recording was used to test for monosynaptic connections from hilar GPHNs to granule cel

57 , but not the reverse, reflecting the direct monosynaptic connections from the PFC to STR.

58 ll types within the S1 cortex receive direct monosynaptic connections from these input sources.

59 found that corticoclaustral afferents formed monosynaptic connections onto both ClaC neurons and PV i

60 uron seen in nonprimates and the fast direct monosynaptic connections present in new M1.

61 even cortico-motoneuronal cells, which make monosynaptic connections to alpha-motoneurons, can becom

62 om vibrissa sensory afferent fibers and send monosynaptic connections to facial nucleus motoneurons t

63 ticospinal tract in Old World primates makes monosynaptic connections to motoneurons; previous anatom

64 Monosynaptic connections were confirmed by measures of t

65 Stretch-sensitive Ia afferent monosynaptic connections with motoneurons form the stret

66 s in the primary motor cortex (M1) that make monosynaptic connections with motoneurons innervating sh

67 cells in the primary motor cortex (M1) have monosynaptic connections with motoneurons.

68 dorsal and middle hippocampus with putative monosynaptic connections with place cells recorded on th

69 LGN afferents made monosynaptic connections with pyramidal (Pyr) and fast-s

70 ne cortico-motoneuronal (CM) cells that make monosynaptic connections with the motoneurons of the inj

71 s (up to 90), helped us to identify apparent monosynaptic connections, confirming the excitatory and

72 e lOFC sends requisite input to the BLA, via monosynaptic connections, to promote CS-induced cocaine-

73 dition to putative excitatory and inhibitory monosynaptic connections, we uncovered prominent millise

74 ory afferents and motoneurons typically form monosynaptic connections, while neurons innervating anta

75 3 pyramidal cells at a latency indicative of monosynaptic connections.

76 uction in detected excitatory and inhibitory monosynaptic connections.

77 the subcortical targets with which they make monosynaptic connections.

78 in Sema3E and its receptor Plexin-D1 prevent monosynaptic connectivity in the cutaneous maximus muscl

79 the sensory response properties and upstream monosynaptic connectivity of cells mediating the CC or C

80 al rs-fMRI connectivity patterns that mirror monosynaptic connectivity with isocortex.

81 ncy (<20 ms) interneuron spiking, indicating monosynaptic connectivity, but firing probability progre

82 trains confirmed a fast and precisely timed monosynaptic connectivity, supporting the notion that SI

83 with the precise timing and rare failures of monosynaptic contacts on second-order neurons.

84 ary motor cortex ("new M1") and area 3a make monosynaptic cortico-motoneuronal connections with limb

85 ays afforded by the preceding elaboration of monosynaptic corticobulbar tracts, giving rise to enhanc

86 reveal any postspike effects consistent with monosynaptic corticomotoneuronal connections.

87 with weaker interneuron activity and reduced monosynaptic coupling between excitatory and inhibitory

88 Strong monosynaptic coupling between pyramidal neurons and near

89 count for this, we find that, in addition to monosynaptic coupling, PV-MSN interactions are mediated

90 Optogenetic activation of this monosynaptic craniofacial-to-PBL projection induced robu

91 significant glycinergic inputs for mediating monosynaptic crossover inhibition.

92 We compared monosynaptic CST amplitude input to segmental circuits w

93 e coronal slices containing V1, and recorded monosynaptic currents from excitatory and inhibitory neu

94 Here, we examine the distribution of monosynaptic currents generated in the superficial SC by

95 CTect impulses generate potent, fast-rising monosynaptic currents in the SC similar to those generat

96 ation-specific interactions predicted by the monosynaptic distribution of horizontal connections.

97 spinal fibers from old M1 generate weak late monosynaptic effects in motoneurons.

98 d putative long-latency monosynaptic and non-monosynaptic effects; the majority of responses from are

99 A few PVN-projecting NTS neurons had monosynaptic EPSC characteristics.

100 asted 200-500 ms and consisted of an initial monosynaptic EPSC followed by a series of late EPSCs sup

101 ine significantly increased the amplitude of monosynaptic EPSCs evoked from the dorsal root and the f

102 nd long-lasting increase in the amplitude of monosynaptic EPSCs evoked from the dorsal root in approx

103 of glutamatergic mEPSCs and the amplitude of monosynaptic EPSCs evoked from the dorsal root were sign

104 th oxotremorine-M decreased the amplitude of monosynaptic EPSCs in approximately 67% of neurons but i

105 /M4 double-KO mice, oxotremorine-M inhibited monosynaptic EPSCs in significantly fewer neurons ( appr

106 but increased the paired pulse ratio of the monosynaptic EPSCs in SNr GABA neurons, indicating a pre

107 rements exploiting the voltage dependence of monosynaptic EPSCs similarly indicated dominant expressi

108 inine on glycinergic IPSCs but not on evoked monosynaptic EPSCs.

109 t to prevent excessive depolarization by the monosynaptic EPSP and multiple action potential firings.

110 responses 5-HT did not affect the descending monosynaptic EPSP conditioned by ventrolateral column (V

111 ve fields anywhere on the head evoked large, monosynaptic EPSPs ( approximately 5-20 mV) in tINs, at

112 a substantial increase in the amplitudes of monosynaptic EPSPs evoked by Ia afferents in MNs as meas

113 inal neurones in the caudal medulla revealed monosynaptic EPSPs in all groups of motoneurones, with t

114 ul inhibition by Purkinje cells or by direct monosynaptic excitation from the inferior olive.

115 nto CA3 pyramidal cells (PCs) provide strong monosynaptic excitation that exhibit prominent facilitat

116 al and electrosensory afferents elicit local monosynaptic excitation, quickly followed by inhibition

117 Monosynaptic excitatory connections from pyramidal cells

118 al retrosplenial cortex (RSC) and these form monosynaptic excitatory connections onto a wide spectrum

119 Axons from RSC formed monosynaptic excitatory connections onto M2 pyramidal ne

120 lls born before SE form functional recurrent monosynaptic excitatory connections with other granule c

121 nto BG output neurons sometimes precedes the monosynaptic excitatory drive from cortical afferents.

122 Islet and central cells had monosynaptic excitatory inputs exclusively from C-affere

123 In contrast, radial and vertical cells had monosynaptic excitatory inputs from both A delta- and C-

124 eptor activation on segmental and descending monosynaptic excitatory inputs to frog lumbar motoneuron

125 Hence, monosynaptic excitatory lemniscal TC connections onto la

126 SNAP significantly reduced the amplitude of monosynaptic excitatory postsynaptic currents (EPSCs) ev

127 ts in small DRG neurons and the amplitude of monosynaptic excitatory postsynaptic currents of dorsal

128 ased HVACC currents in small DRG neurons and monosynaptic excitatory postsynaptic currents of spinal

129 s, although electrical stimulation generated monosynaptic excitatory postsynaptic potentials that wer

130 Two sites of monosynaptic excitatory projection in the cord were iden

131 tion of corticoclaustral afferents generated monosynaptic excitatory responses as well as disynaptic

132 ls of double-projecting vCA1 neurons induced monosynaptic excitatory responses in both the mPFC and b

133 nes in the principal olivary nucleus receive monosynaptic extra-somatic glutamatergic input from the

134 ysis, an approach that enables assessment of monosynaptic extracellular currents generated in differe

135 that layer 2/3 Pyr cells receive excitatory monosynaptic FF and FB inputs, which are opposed by disy

136 (2) changes in the size of the corticospinal monosynaptic field potential in the spinal cord.

137 Cord dorsum recordings of monosynaptic field potentials evoked by electrical skin

138 increased both the terminal excitability and monosynaptic fields with similar time courses.

139 e obtained from layer V pyramidal cells, and monosynaptic GABA(A) receptor-mediated IPSCs were elicit

140 Neither CRF nor Ucn I altered monosynaptic GABA(A)-mediated IPSCs before or after chro

141 n cells in specific layers and elicit robust monosynaptic GABAergic and nicotinic postsynaptic curren

142 ing activation of MS axons, we observed fast monosynaptic GABAergic IPSPs in the majority (>60%) of f

143 Photostimulation of MCH projections evoked a monosynaptic glutamate release in the LS.

144 ulation of cholinergic interneurons triggers monosynaptic glutamate- and acetylcholine-mediated curre

145 parafascicular inputs evokes suprathreshold monosynaptic glutamatergic excitation in NGF interneuron

146 ation, to demonstrate that the activation of monosynaptic glutamatergic projections from anterior ins

147 Chemogenetic disconnection of monosynaptic glutamatergic vCA1 to mPFC projections usin

148 ntifies a novel neural pathway through which monosynaptic glutamatergic ventral hippocampal field CA1

149 re, we found that the mPFC provides a direct monosynaptic, glutamatergic drive to both DRN 5-HT and G

150 tINs produce small ( approximately 2-6 mV), monosynaptic, glutamatergic EPSPs in the hindbrain retic

151 vation of BLA terminals in the vHPC provided monosynaptic, glutamatergic inputs to vHPC pyramidal neu

152 Thirteen dorsal horn interneurons with monosynaptic group II input were characterized electroph

153 and pyramidal tract (PT) dendrites, whereas monosynaptic hippocampal input primarily targeted IT, bu

154 the surround suppression through long-range monosynaptic horizontal spread.

155 types of spinal interneurons (propriospinal, monosynaptic Ia-excitatory, reciprocal Ia-inhibitory, Re

156 mitted across first-order synapses, probably monosynaptic, in the spinal cord.

157 f the substantia nigra that provide a robust monosynaptic inhibition of DA neurons.

158 OFF cone BCs mediate monosynaptic inhibition of rod BCs via motif C3 driven b

159 ncing widespread, GABA(B) receptor-mediated, monosynaptic inhibition.

160 collaterals; but reveal both excitatory and monosynaptic inhibitory currents in the ventral pallidum

161 thermore, continuous connections can undergo monosynaptic inhibitory LTP, independent of excitatory d

162 fference in the amplitude of stimulus-evoked monosynaptic inhibitory postsynaptic potentials (IPSPs)

163 ic SACs produced a GABA(A) receptor-mediated monosynaptic inhibitory response, followed by DA-D(1)-li

164 In contrast, tetanization elicited LTP of monosynaptic inhibitory responses in continuous, but not

165 m spiny projection neuron (MSN) activity via monosynaptic inhibitory signaling.

166 ell (NAcSh) MSNs was fine-tuned by GABAergic monosynaptic innervation from adjacent fast-spiking inte

167 d disynaptic pathways to inhibitory neurons: Monosynaptic input causes more powerful oscillations whe

168 stablish that inhibitory SG neurones receive monosynaptic input from a subset of unmyelinated primary

169 rons, which included a greater prevalence of monosynaptic input from low-threshold A-fibers when prec

170 e of OFC neurons, although neurons receiving monosynaptic input from MD were less affected and some p

171 emonstrate that mitral cells receive direct, monosynaptic input from olfactory receptor neurons.

172 ML interneurons received a direct excitatory monosynaptic input from the entorhinal cortex via the pe

173 , a ventral striatum structure that receives monosynaptic input from the olfactory bulb, is uniquely

174 n though some of these cells received potent monosynaptic input from the same LGNd neurons whose rate

175 hibits feeding when excited by touch-induced monosynaptic input from the second type of interneuron,

176 the accessory olfactory bulb (AOB), receive monosynaptic input from the sensory periphery and alread

177 hilus and stratum lucidum receive this dual monosynaptic input on MF stimulation.

178 n to receive high-threshold (Adelta/C fiber) monosynaptic input, whereas lamina III NK1R+ neurons rec

179 neurons received low-threshold (Abeta fiber) monosynaptic input.

180 ying the same muscle, there were also strong monosynaptic inputs from Ia afferents supplying unrelate

181 rd, we show that connections compatible with monosynaptic inputs from mechanosensory Rohon-Beard neur

182 rons in all layers of MEC receive convergent monosynaptic inputs from PrS and PaS and second, that el

183 TS second-order relay neurones which receive monosynaptic inputs from the SARs.

184 (TC) recipient areas receive weak but direct monosynaptic inputs from the thalamus.

185 ) to selectively identify and quantify their monosynaptic inputs in vivo.

186 To understand how monosynaptic inputs onto adult-born dentate granule cell

187 s were explained by changes in the weight of monosynaptic inputs received by interneurons from new py

188 d a strategy to genetically target and trace monosynaptic inputs to a single neuron in vitro and in v

189 Here we describe the monosynaptic inputs to a subpopulation of mouse S1 inhib

190 ieved to not project to CA2, send functional monosynaptic inputs to CA2 pyramidal cells through abund

191 tion, to characterize the firing patterns of monosynaptic inputs to dopamine neurons while mice perfo

192 DTAM(IX-X) neurons provide excitatory monosynaptic inputs to laryngeal motor neurons and mixed

193 Furthermore, we obtain brain-wide maps of monosynaptic inputs to MCH and OH cells, and demonstrate

194 ole-brain light-sheet imaging, we mapped the monosynaptic inputs to midbrain dopamine neurons project

195 e a powerful system for revealing the direct monosynaptic inputs to specific cell types in Cre-expres

196 cally targeted viral tracing to identify the monosynaptic inputs to the projection neurons of layer I

197 e, we comprehensively identified each area's monosynaptic inputs using the rabies virus.

198 c insult that generates prominent excitatory monosynaptic inputs, both local recurrent and widespread

199 eurons, profoundly affected their pattern of monosynaptic inputs.

200 ies virus results in unambiguous labeling of monosynaptic inputs.

201 ead transsynaptically and label their direct monosynaptic inputs.

202 defined neuronal cell types and their direct monosynaptic inputs.

203 mined laminar distribution does not preclude monosynaptic interaction with neurons located in deeper

204 On finer timescales, putative monosynaptic interactions reflected short-term plasticit

205 9% of sites), minimal focal shocks activated monosynaptic IPSCs at fixed latency (low jitter) that of

206 However, LSP4-2022 also reduced evoked monosynaptic IPSCs in CA1 pyramidal cells and, in contra

207 ation of GABAergic LH --> PVH fibers induced monosynaptic IPSCs in PVH neurons, and potently increase

208 No monosynaptic IPSPs could be recorded in the presence of

209 nsmission, we evoked GABAA receptor-mediated monosynaptic IPSPs in deep cerebellar nuclei neurons by

210 rons in the LGN in a pattern consistent with monosynaptic labeling of koniocells, rather than disynap

211 57) occurred at latencies compatible with a monosynaptic linkage, including in motoneurons projectin

212 r long-latency monosynaptic (n = 108) or non-monosynaptic linkages (n = 108).

213 rogressive and phase-dependent modulation of monosynaptic (middle) and long-latency (late) stimulatio

214 Monosynaptic mossy fiber inputs to fast-spiking basket c

215 of oculomotor activation, comparable to the monosynaptic motor-evoked potential evoked by TMS of pri

216 es as likely mediated by either long-latency monosynaptic (n = 108) or non-monosynaptic linkages (n =

217 Antidromic (n=35), monosynaptic (n=2), di-or tri-synaptic (n=18) and long-l

218 trol scheme by which the transmission in the monosynaptic neural circuits is modulated in all leg mus

219 ith activation of Ia afferent fibres through monosynaptic neural circuits since they were inhibited w

220 enetic activation of this projection elicits monosynaptic nicotinic and GABAergic currents in glomeru

221 N2B antagonist, caused modest suppression of monosynaptic NMDA EPSC amplitudes, but had a widely vari

222 In contrast, there is no evidence of monosynaptic nonvisual inputs to the superficial layers.

223 Excitatory postsynaptic currents (EPSCs) in monosynaptic nTS neurons were recorded in the horizontal

224 ET cells receive monosynaptic olfactory nerve input and drive the major i

225 in external tufted (ET) cells in response to monosynaptic (ON) inputs.

226 ST shocks activated EPSCs along monosynaptic or polysynaptic pathways.

227 ssed by application of serotonin leaving the monosynaptic output of GABAergic cells unaffected.

228 ortex and sensory pathways and, in turn, has monosynaptic outputs to spinal motorneurons.

229 > CA3 --> CA1 --> entorhinal cortex) and the monosynaptic pathway (entorhinal cortex --> CA1 --> ento

230 e identified and characterized a nigro-vagal monosynaptic pathway in rats that controls gastric tone

231 ynaptic pathway is dispensable and the short monosynaptic pathway is sufficient for incremental spati

232 uential, particularly compared with a direct monosynaptic pathway linking piriform cortex and OFC.

233 specifically those OVLT neurones that form a monosynaptic pathway to the PVN.

234 pathway while preserving transmission in the monosynaptic pathway.

235 n the trigeminal motor nucleus, suggesting a monosynaptic, possibly proprioceptive, pathway.

236 Here we report the discovery of a monosynaptic prefrontal cortex (predominantly anterior c

237 Hence, we hypothesized that monosynaptic projections between these brain regions med

238 The connections and monosynaptic projections of muscle spindle afferents of

239 the whether any contribution is via direct, monosynaptic projections, or the direction of informatio

240 Monosynaptic rabies tracing reveals that CC neurons pref

241 ion segregation in these circuits, we used a monosynaptic rabies virus system to generate brain-wide

242 We used the monosynaptic rabies virus system, in conjunction with mi

243 We used a monosynaptic rabies virus to define the circuit's functi

244 Using a monosynaptic rabies virus-based tracing technique, we st

245 Using a modified monosynaptic rabies virus-based transsynaptic tracing st

246 Using a monosynaptic rabies viruses-based transneuronal tracing

247 ults are therefore critical for interpreting monosynaptic rabies-based tracing in the sensory system.

248 crucial prerequisite of spoken language: (i) monosynaptic refinement of the projections of motor cort

249 l circuits leads to hyperexcitability of the monosynaptic reflex.

250 g systems, inducing hyperexcitability of the monosynaptic reflex.

251 amplitudes (75%, 62%), and also facilitated monosynaptic reflexes evidenced by an increase of the H/

252 vides the opportunity to study modulation of monosynaptic reflexes for multiple muscles simultaneousl

253 macological sensitivity, our results suggest monosynaptic release of both GABA and ACh.

254 lation of SuM(vgat/vglut2) terminals elicits monosynaptic release of both glutamate and GABA onto den

255 In turn, the multisegmental monosynaptic responses (MMR) technique provides the oppo

256 We found that commissural inputs evoked monosynaptic responses in both intratelencephalic (IT) a

257 ient (24 of 25) neurons generated axonal and monosynaptic responses in layer 4 and/or 6 of the aligne

258 administered drugs that increase excitatory monosynaptic responses in the brain.

259 ated a rapid and substantial decrease in the monosynaptic responses recorded at the first central sta

260 ectroporation of DNA to target infection and monosynaptic retrograde spread of a genetically modifiab

261 nitial rabies virus infection and subsequent monosynaptic retrograde spread.

262 Using rabies virus -mediated monosynaptic retrograde tracing to label the inputs and

263 n to demonstrate cell-specific infection and monosynaptic retrograde transport of virus, which strong

264 analyses show diminished EPSP amplitudes in monosynaptic sensory-motor circuits in these mutants.

265 neurons is required for synapse formation in monosynaptic sensory-motor circuits.

266 oper synapse formation in the development of monosynaptic sensory-motor circuits.

267 anisms underlying synapse formation in these monosynaptic sensory-motor connections are unknown.

268 o GTPase Cdc42 controls synapse formation in monosynaptic sensory-motor connections in presynaptic, b

269 ctive role in the establishment of patterned monosynaptic sensory-motor connections.

270 ded that the organization of the longissimus monosynaptic spindle input favours relatively tonic and

271 d that firing of individual CHIs resulted in monosynaptic spontaneous inhibitory post-synaptic curren

272 rgent ST afferent inputs (22% two; 14% three monosynaptic ST-EPSCs).

273 -CTB neuronal transport was target specific, monosynaptic, stable for several weeks, and reproducible

274 and weak oscillations; in contrast, stronger monosynaptic stimulation (e.g., suppressive contextual s

275 el predicted characteristic contributions to monosynaptic stLFP signatures both for the regular-spiki

276 s finding suggests that twitches trigger the monosynaptic stretch reflex and, by doing so, contribute

277 ico-striatal pathways, emerges primarily via monosynaptic structural connections.

278 m rs-fMRI in female mice with the underlying monosynaptic structural connectome as provided by the Al

279 bellum identified brainstem neurons that are monosynaptic targets of inhibition from the cerebellar f

280 rt and non-alert EEG states, we examined the monosynaptic TC responses and short-term synaptic dynami

281 no state-related changes in the amplitude of monosynaptic TC responses when TC spikes with similar pr

282 However, monosynaptic TC synaptic transmission has not been direc

283 To probe monosynaptic thalamic activation of cortical postsynapti

284 f the focal LFP signature of the single-axon monosynaptic thalamocortical connection as measured by s

285 than 1.2 ms were classified as definitively monosynaptic; these were seen only after stimulation wit

286 eling of contralateral LVST neurons within a monosynaptic time window all indicate an overwhelmingly

287 Glycoprotein-deleted rabies virus-mediated monosynaptic tracing has become a standard method for ne

288 Rabies virus-based monosynaptic tracing has been used to identify neuronal

289 vering DNA plasmids that allowed retrograde, monosynaptic tracing of each neuron's presynaptic inputs

290 ion of genetically specified neurons and (4) monosynaptic tracing of neuronal inputs.

291 The development of retrograde monosynaptic tracing vectors has enabled visualization o

292 rus (RABV) has been the reagent of choice in monosynaptic tracing, since it permits the mapping of sy

293 Using rabies virus monosynaptic tracing, we mapped cocaine-induced global c

294 Using in vivo electroporation and monosynaptic tracing, we show that postnatal-born granul

295 Finally, using rabies-virus-assisted monosynaptic tracing, we show that the GPh is embedded i

296 efficient and prolonged photostimulation of monosynaptic transmission at the neuromuscular junction

297 o demonstrate a progressive delay of C fiber monosynaptic transmission to the spinal cord that is sim

298 , including both muscle stretch encoding and monosynaptic transmission, could be separated from other

299 Monosynaptic transsynaptic rabies tracing indicated the

300 reflexes (SRs) in ventral roots, presumably monosynaptic, were evoked by electrical stimulation of a