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1 an inhibitor of myosin function (butanedione monoxime).
2 ersed by addition of 10 mM 2,3-butanedione 2-monoxime.
3 and by the myosin inhibitor 2,3-butanedione monoxime.
4 yosin by cytochalasin D or 2,3-butanedione-2-monoxime.
5 ncreases were not blocked by 2,3-butanedione monoxime.
6 ng) were estimated by use of 2,3-butanedione monoxime.
8 ly inhibited a slow phase, while butanedione monoxime, a myosin ATPase inhibitor, inhibited both the
9 on in the presence of 7.5 mM 2,3-butanedione monoxime, an inhibitor of actin-myosin cross-bridge cycl
11 thase kinase 3 (GSK3) inhibitor indirubin-3'-monoxime and GSK3beta short interfering RNAs, demonstrat
13 esponse was affected by both 2,3-butanedione monoxime and sarcomere length in the same manner as acti
14 rs monoisonitrosoacetone and 2,3-butanedione monoxime and shows potential for further refinement.
15 slope of the restitution relation (diacetyl monoxime and verapamil) prevent the induction of VF and
18 enosine triphosphatase using 2,3-butanedione monoxime (BDM) and found that phagocytosis was inhibited
20 h 0.5 mM AlF4- or with 30 mM 2,3 butanedione-monoxime (BDM) during measurements of the Ca2+ dependenc
22 of the chemical phosphatase 2,3-butanedione monoxime (BDM) on glycine current (IGly) of murine ventr
23 activated eggs treated with 2,3-butanedione monoxime (BDM) undergo a dramatic large-scale torsion, w
24 was conducted to evaluate 2,3-butanedione 2-monoxime (BDM), a compound that interferes with acto-myo
25 ing 20 nM dopamine, dopamine+2,3-butanedione monoxime (BDM), a MgATPase-inhibitor, or the calcium-sen
26 s of a chemical phosphatase, 2,3-butanedione monoxime (BDM), on endogenous and expressed Ca2+ channel
27 The myosin ATPase inhibitor, butanedione monoxime (BDM), reversibly inhibited gliding motility ac
28 (pCa 4.5) exposed to 10 mM 2,3-butanedione 2-monoxime (BDM), stretch was accompanied by a lattice com
29 ere insensitive to 5-10 mM 2,3-butanedione 2-monoxime (BDM), which inhibited active contractions.
32 mol/L egtazic acid; 3) 5 mmol/L butane-dione-monoxime (BDM); or 4) 50 mmol/L ammonium chloride (NH4Cl
33 investigation was to utilize 2,3-butanedione monoxime (BDM; an inhibitor of contractile activation) t
35 idodiacetyl reaction between 2,3-butanedione monoxime (diacetylmonoxime) and the de novo carbamido co
36 viously that the small molecule indirubin-3'-monoxime (I3MO) prevents vascular smooth muscle cell (VS
37 ddition of the neutral oxime 2,3-butanedione monoxime increases the rate of reactivation of hCE1 from
38 tochalasin D) and myosin ATPase (butanedione monoxime), indicating that they rely on an actinomyosin
40 the cardioplegic agent BDM (2,3-butanedione monoxime), its effects on the force-velocity properties
41 rates with myosin inhibitors 2,3-butanedione monoxime or KT5926 caused the reduction of both vinculin
42 Cross-bridge inhibition (with butanedione monoxime) or augmentation (with 2 deoxy-ATP) had no grea
43 concentrations of phosphate, 2,3-butanedione monoxime, or adenosine triphosphate at a fixed calcium c
44 he myosin ATPase inhibitor 2,3-butanedione-2-monoxime perturbed NF translocation within NB2a/d1 axona
45 ubation with the GSK3 inhibitor indirubin-3'-monoxime protected against this decrease in activity.
46 , genistein, wortmannin, and 2,3-butanedione monoxime-sensitive pathway and replicated within phagoly
48 azapine, cytochalasin D, and 2,3-butanedione monoxime significantly inhibited (P < 0.05) in vitro inf
49 ed by the myosin inhibitor 2,3-butanedione-2-monoxime, suggesting that tension promotes tyrosine kina
50 abolished in the presence of 2,3-butanedione monoxime, suggesting that the basal level of contractile
51 nce of the tension inhibitor 2,3-butanedione monoxime suggests that regulatory light chain (RLC) phos
52 oss-bridge (inorganic phosphate, butanedione monoxime, trifluoperazine, and blebbistatin) are modifie
54 ng crossbridge inhibition by 2,3-butanedione monoxime when near-neighbour thin filament RU interactio
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