ライフサイエンスコーパス: morphine dependence

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1 ing full analgesic potency, does not promote morphine dependence.

2 pamine immediately prior to the induction of morphine dependence.

3 is known about the role of AMPA receptors in morphine dependence.

4 ety and autoreceptor function only following morphine dependence.

5 other gelatinase, MMP-2, is not involved in morphine dependence.

6 her methadone, an opioid often used to treat morphine dependence, also exhibited 5-HT(3) receptor ant

7 We compared the effects of acute morphine dependence and social isolation in non-anxious

8 Morphine dependence and withdrawal result in profound ne

9 he roles of D1 receptors, CREB, and GluR1 in morphine dependence are not well understood.

10 eptor antagonists than is the development of morphine dependence as assessed by naloxone-precipitated

11 hat orexin knock-out mice develop attenuated morphine dependence, as indicated by a less severe antag

12 Furthermore, oxytocin neurones develop morphine dependence by a mechanism which is distinct fro

13 h) attenuated the development of acute (3 h) morphine dependence (i.e., decreased naloxone-precipitat

14 the cAMP response element (CRE), we induced morphine dependence in CRE-LacZ transgenic mice and perf

15 chemical methods in NAc tissue, we show that morphine dependence increases cell surface expression of

16 Morphine dependence is associated with long-term adaptiv

17 Morphine dependence is characterized by somatic and moti

18 acid (EAA) receptor antagonists to modulate morphine dependence is controversial, and very little is

19 l MMP-9 may contribute to the development of morphine dependence primarily through neuronal activatio

20 We use a rat model of morphine dependence to show that GluA1 subunits of AMPA

21 In a rat model of morphine dependence, we previously identified increased

22 e facilitated morphine tolerance and reduced morphine dependence without affecting morphine reward.

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