ライフサイエンスコーパス: morphogenetic

1 ew, we discuss recent findings regarding the morphogenetic and molecular processes required for intes

2 s that play essential roles in diverse plant morphogenetic and physiological responses to light.

3 l lethality and distinct neurodevelopmental, morphogenetic, and metabolic alterations.

4 However, the cellular control of morphogenetic apoptosis is poorly understood, notably th

5 der wall, represents an exquisite example of morphogenetic apoptosis, requiring the receptor protein

6 hat the cognate proteins are involved in the morphogenetic assembly pathway from bicones to mature cy

7 odermal cells in the primitive streak as the morphogenetic basis underlying the pathogenesis of neura

8 ment cell interactions, and an unanticipated morphogenetic behavior contributing to a striking differ

9 contractility is sufficient to explain many morphogenetic behaviors, which depend on cell cluster si

10 cells transduce ECM properties into complex morphogenetic behaviors.

11 he cAMP pulses that coordinate Dictyostelium morphogenetic cell movement and is highly expressed at t

12 es rise to a range of defects, from aberrant morphogenetic cell movements to failure to correctly ori

13 ment, devoid of patterning signals and major morphogenetic cell movements.

14 nscriptional thresholds required for driving morphogenetic cell-fate decisions.

15 To understand the molecular and morphogenetic changes associated with loss of this key m

16 mizes imaging performance during large-scale morphogenetic changes in living organisms.

17 and adult stage, allowed us to describe the morphogenetic changes shaping the adult SEZ.

18 into the root vascular cylinder, triggering morphogenetic changes to induce galls, de novo formed 'p

19 This study provides a global view of morphogenetic circuitry governing a key virulence trait,

20 uirements of the encoded amino acids and the morphogenetic consequences of mutation.

21 ning of transcription is crucial for driving morphogenetic conversions in the fungal pathogen C. albi

22 tworks directed by tissue-resident cells and morphogenetic cues.

23 ular dysfunction, including those related to morphogenetic defects.

24 This morphogenetic difference is evident by approximately 5 y

25 How such morphogenetic differences are genetically encoded and wh

26 results suggest that KNOXs trigger different morphogenetic effects through interplay between tissue c

27 C. elegans embryonic elongation is a morphogenetic event driven by actomyosin contractility a

28 in neighbor cells that is necessary for the morphogenetic event of apoptotic extrusion to occur.

29 be used to dynamically image and analyze key morphogenetic events during embryonic stages X to 11.

30 Out-of-plane tissue deformations are key morphogenetic events during plant and animal development

31 One of the first morphogenetic events in the vertebrate brain is the form

32 on is a change in cell shape that drives key morphogenetic events including gastrulation and neural t

33 present, our understanding of the timing and morphogenetic events leading to the formation of the hum

34 , and they are also essential drivers in the morphogenetic events of development.

35 directed by forces outside the gut, but the morphogenetic events that generate anatomical asymmetry

36 yonic phenomenon involves a complex array of morphogenetic events that require coordinated proliferat

37 be that then undergoes multiple simultaneous morphogenetic events to obtain its mature shape.

38 tify the generic rules that may govern these morphogenetic events, we developed a 3D-modeling framewo

39 determine large-scale deformations and other morphogenetic events.

40 sue behaviors during early vertebrate embryo morphogenetic events.

41 tion and could be applied to a wide range of morphogenetic events.

42 ng stromal mesenchyme to orchestrate complex morphogenetic events.

43 of reproducible shapes, through stereotyped morphogenetic events.

44 e biochemically controlled to robustly drive morphogenetic events.

45 etween these tissues is key to understanding morphogenetic evolution.

46 ver, the mechanisms by which these different morphogenetic factors are coordinated and how they may b

47 ll be useful for decoupling other downstream morphogenetic factors from hyphal growth.

48 ell understood, with hypotheses ranging from morphogenetic fields to the clone theory.

49 ndings, we hypothesize that Toll genes had a morphogenetic function in embryo elongation in the last

50 Understanding this morphogenetic function of YAP could facilitate the use o

51 prevent retinal differentiation by blocking morphogenetic furrow (MF) progression and R8 specificati

52 G1 arrest and neuronal specification at the morphogenetic furrow.

53 However, the nature of the AR-regulated morphogenetic genes and the mechanisms whereby AR contro

54 (AR) is thought to control the expression of morphogenetic genes in inductive UGS mesenchyme, which p

55 along the tooth row has been described as a 'morphogenetic gradient' in mammal, and more specifically

56 This configuration is also equivalent to a morphogenetic gradient, finally pointing to a mechanism

57 This morphogenetic map is expected to find widespread applica

58 ssue conflict resolution provides a flexible morphogenetic mechanism for generating shape diversity i

59 Convergent extension (CE) is a fundamental morphogenetic mechanism that underlies numerous processe

60 ween kidney architecture and the fundamental morphogenetic mechanisms that guide development.

61 Our results provide a novel perspective on morphogenetic mechanisms, which arise from cell-fate het

62 s and is, therefore, a novel and fundamental morphogenetic motif widespread in embryonic development.

63 ithelial cell properties required for proper morphogenetic movement and pattern formation.

64 LHX1 enables the morphogenetic movement of cells that accompanies the for

65 ion in early amniotes generally assumed that morphogenetic movement reflected migration relative to a

66 ions including knowledge of how GRNs control morphogenetic movements and how GRNs evolve.

67 Often these morphogenetic movements are accomplished by the coordina

68 iversification, cell-fate specification, and morphogenetic movements establishes the generation of ex

69 to completion of gastrulation coordinate the morphogenetic movements underlying the organization of t

70 pared with the contractile events that drive morphogenetic movements.

71 yostelia always initiates at the site of the morphogenetic organizer.

72 patial organization of forces determines the morphogenetic output.

73 ay alter the flux of intermediates through a morphogenetic pathway.

74 human CAKUT and shed light on distinct renal morphogenetic pathways that were identified as relevant

75 f extracellular signals that trigger crucial morphogenetic pathways.

76 of mechanics and chemistry combine to drive morphogenetic pattern formation.

77 und-leaf species to promote an exit from the morphogenetic phase of tomato leaf development.

78 iciency causes an abnormal renal ciliary and morphogenetic phenotype.

79 Drosophila dorsal closure (DC), a morphogenetic process in which an extraembryonic tissue

80 This morphogenetic process is powered by cell intercalation,

81 lation constitutes a fundamental yet diverse morphogenetic process of metazoan development.

82 ic axis elongation is a complex multi-tissue morphogenetic process responsible for the formation of t

83 cles in the heart undergo a poorly described morphogenetic process that results into a solidified com

84 the otic vesicle case exemplifies a generic morphogenetic process where spatial and temporal cues re

85 Craniofacial development is a complex morphogenetic process, disruptions in which result in hi

86 variants (CNVs) are strongly associated with morphogenetic processes and common neurodevelopmental di

87 ys controls the complexity and order of lens morphogenetic processes and lens transparency.

88 demonstrating how the aforementioned mechano-morphogenetic processes are coordinated to generate a bo

89 at recapitulates some of the important early morphogenetic processes during organ development.

90 mbryo is one of the most intensively studied morphogenetic processes in animal development [1-4].

91 trated a startling array of regenerative and morphogenetic processes in this single-celled organism,

92 y (WNT/PCP) pathway plays important roles in morphogenetic processes in vertebrates.

93 Current approaches to recreate morphogenetic processes in vitro rely on poorly controll

94 and molecular pathways with the cellular and morphogenetic processes of palatal shelf growth, pattern

95 y nonmuscle myosin II (NMY-2) drive cellular morphogenetic processes such as cytokinesis.

96 One of the key morphogenetic processes used during development is the c

97 s a combination of directed differentiation, morphogenetic processes, and the intrinsically driven se

98 ian secondary palate involves highly dynamic morphogenetic processes, including outgrowth of palatal

99 tissues, the embryo also undergoes dramatic morphogenetic processes, including the cell movements of

100 sues and how forces are involved in specific morphogenetic processes.

101 ebrate mutants are related to other types of morphogenetic processes.

102 traints and used to describe a wide range of morphogenetic processes.

103 the forces that drive cell- and tissue-scale morphogenetic processes.

104 This genetic framework parallels the morphogenetic program of shoot apical meristems and may

105 arkers and induces elements of the core hair morphogenetic program, including ectodysplasin A recepto

106 ress in the understanding of tissue-specific morphogenetic programs.

107 cells to switch between Rac1- and RhoA-like morphogenetic programs.

108 despread strategy to ensure the integrity of morphogenetic programs.

109 n synovial MSCs transduced with Bmp7 display morphogenetic properties by patterning a joint-like orga

110 system essential for generating the hallmark morphogenetic properties of pancreatic islets.

111 We found that modulation of bone morphogenetic protein (BMP) and WNT signaling combined w

112 the context of reduced ShcA levels, the bone morphogenetic protein (BMP) antagonist chordin-like 1 (C

113 We have recently shown that the bone morphogenetic protein (BMP) antagonist Gremlin 2 (Grem2)

114 gene aberrant in neuroblastoma (DAN), a bone morphogenetic protein (BMP) antagonist we detected by an

115 t CHRDL1 encodes Ventroptin, a secreted bone morphogenetic protein (BMP) antagonist, the molecular me

116 Hemojuvelin is a bone morphogenetic protein (BMP) co-receptor.

117 The bone morphogenetic protein (Bmp) family of secreted molecules

118 The Bone Morphogenetic Protein (BMP) family reiteratively signals

119 homeostasis by direct interaction with bone morphogenetic protein (BMP) ligands to induce hepcidin e

120 injury stimulates the production of two bone morphogenetic protein (BMP) ligands, Dpp and Gbb, which

121 cing (ChIP-seq) data, we identified the bone morphogenetic protein (BMP) pathway as a potential regul

122 Here we demonstrate that the bone morphogenetic protein (BMP) pathway can also be regulate

123 At diagnosis, deregulation of the bone morphogenetic protein (BMP) pathway is involved in LSC a

124 rticular the dual modulation of Wnt and bone morphogenetic protein (BMP) pathway signaling, this dire

125 e transforming growth factor (TGF)-beta/bone morphogenetic protein (BMP) pathway, which is one of the

126 f endodermal development, including the Bone morphogenetic protein (Bmp) pathway.

127 orming growth factor beta (TGFbeta) and bone morphogenetic protein (BMP) pathways.

128 Beta-catenin/Tcf and the TGF-beta bone morphogenetic protein (BMP) provide critical molecular s

129 The bone morphogenetic protein (BMP) receptor Tkv localizes to mi

130 7 of CRIM1 (cysteine-rich transmembrane bone morphogenetic protein (BMP) regulator 1).

131 eogenic hair follicles, which triggered bone morphogenetic protein (BMP) signaling and then activatio

132 We showed previously that SMOC inhibits bone morphogenetic protein (BMP) signaling downstream of its

133 ss of the growth-suppressive effects of bone morphogenetic protein (BMP) signaling has been demonstra

134 t of differential regulation of Wnt and bone morphogenetic protein (BMP) signaling in these two bone

135 een constructed and, here, we show that bone morphogenetic protein (BMP) signaling is essential for t

136 Here we show that hippocampal bone morphogenetic protein (BMP) signaling is modulated by an

137 The bone morphogenetic protein (BMP) signaling pathway comprises

138 Ras signaling and may also activate the bone morphogenetic protein (BMP) signaling pathway in colorec

139 ulation factor fibrinogen activates the bone morphogenetic protein (BMP) signaling pathway in oligode

140 Hepcidin expression is induced via the bone morphogenetic protein (BMP) signaling pathway that prefe

141 that RNAi silencing of a member of the Bone Morphogenetic Protein (BMP) signaling pathway, Decapenta

142 se mutants target the components of the Bone Morphogenetic Protein (BMP) signaling pathway, revealing

143 is established and patterned by Wnt and bone morphogenetic protein (BMP) signaling pathways, respecti

144 A morphogen gradient of Bone Morphogenetic Protein (BMP) signaling patterns the dorso

145 en transforming growth factor beta1 and bone morphogenetic protein (BMP) signaling plays an important

146 The family of TGF-beta and bone morphogenetic protein (BMP) signaling proteins has numer

147 Bone morphogenetic protein (BMP) signaling was activated with

148 Although antagonists of bone morphogenetic protein (BMP) signaling, such as Noggin, p

149 enewing SCs, Foxc1 activates Nfatc1 and bone morphogenetic protein (BMP) signaling, two key mechanism

150 notable for roles in Wingless (Wnt) and bone morphogenetic protein (BMP) signaling, were differential

151 the Drosophila ovary where it enhances bone morphogenetic protein (BMP) signaling.

152 eta and activin signals while enhancing bone morphogenetic protein (BMP) signaling.

153 ess-related integration site (WNT), and bone morphogenetic protein (BMP) signalling interactions capa

154 cardial trabeculation through Erbb2 and bone morphogenetic protein (BMP) signalling, we discover that

155 ion and signaling are up-regulated, and bone morphogenetic protein (BMP) signals are impaired.

156 Wnt/beta-catenin) or share (TGFbeta and bone morphogenetic protein (BMP)) core signaling components.

157 This data shows that Wnt, bone morphogenetic protein (BMP), and fibroblast growth facto

158 pidermal growth factor receptor (EGFR), bone morphogenetic protein (BMP), Jun kinase (JNK), JAK/STAT,

159 commonly increase signaling of the Wnt, bone morphogenetic protein (BMP), or Ras/ERK pathways, conver

160 s specified by sequential inhibition of bone morphogenetic protein (BMP), transforming growth factor-

161 ells (recell-dTBs); 3) dTBs seeded with bone morphogenetic protein (BMP)-2 (dTB-BMPs); and 4) freshly

162 Bone morphogenetic protein (BMP)-2 is used clinically for ske

163 factor (TGF)-beta family, TGF-beta1 and bone morphogenetic protein (BMP)-2, in synovial fibroblasts f

164 The bone morphogenetic protein (BMP)-SMAD signaling pathway has a

165 pathway and activation of the parallel bone morphogenetic protein (BMP)/Smad1/5 axis (recently ident

166 accompanied by changes in expression of bone morphogenetic protein (BMP)/sons of mothers against deca

167 c activity, and six were members of the bone morphogenetic protein (BMP)/TGF-beta pathway.

168 e gene encoding hepcidin (HAMP) via the bone morphogenetic protein (BMP)6 signaling to SMAD.

169 Bone morphogenetic protein (BMP)9 is a circulating growth fac

170 VBA, with and without recombinant human bone morphogenetic protein (rhBMP)-2, under space-making tita

171 Mutations in bone morphogenetic protein 1 (BMP1) in humans or deletion of

172 e cleavage of C-terminal procollagen by bone morphogenetic protein 1 (BMP1).

173 wth differentiation factor 9 (GDF9) and bone morphogenetic protein 15 (BMP15) are secreted during fol

174 s of osteoblastic differentiation using bone morphogenetic protein 2 (BMP-2) added to the medium.

175 Although bone morphogenetic protein 2 (BMP-2) is known to stimulate os

176 The expressions of bone morphogenetic protein 2 (BMP2) and BMP6; sex-determining

177 y, we discovered the double deletion of bone morphogenetic protein 2 (Bmp2) and bone morphogenetic pr

178 Bone morphogenetic protein 2 (BMP2) in chromosomal region 20p

179 Bone morphogenetic protein 2 (BMP2) promotes PF formation by

180 nanostructures amplified signalling of bone morphogenetic protein 2 significantly more than the natu

181 bone morphogenetic protein 2 (Bmp2) and bone morphogenetic protein 4 (Bmp4) in the dental epithelium

182 onse to transient (24-36 h) exposure to bone morphogenetic protein 4 (BMP4) plus inhibitors of ACTIVI

183 tein 1 (GLG1) expression and downstream bone morphogenetic protein 4 (BMP4) signaling and also reduce

184 fferent concentrations of activin A and bone morphogenetic protein 4 (BMP4) to polarize cells into me

185 TGF-beta signaling mediated by pSMAD2, bone morphogenetic protein 4 (BMP4), EGF, or PDGF was unaffec

186 Consequently, bone morphogenetic protein 4 (BMP4), or ectopic expression of

187 o promote beta-cell function, including bone morphogenetic protein 4 (BMP4).

188 Contributions of bone morphogenetic protein 4 and transforming growth factor (

189 otocol, using defined medium containing bone morphogenetic protein 4 by which human pluripotent stem

190 Bone morphogenetic protein 4 up-regulated alphaB-crystallin,

191 Bone morphogenetic protein 6 (BMP6) contributes to the iron-d

192 Lack of either bone morphogenetic protein 6 (BMP6) or the BMP coreceptor hem

193 Bone morphogenetic protein 6 (BMP6) signaling in hepatocytes

194 these molecules intersect in vivo with bone morphogenetic protein 6 (BMP6)/mothers against decapenta

195 gland fluid secretion, is regulated by bone morphogenetic protein 6.

196 rming growth factor beta (TGFbeta), and bone morphogenetic protein 7 (BMP7), CD1c(+) dendritic cells

197 We further identify bone morphogenetic protein 7 as one of them.

198 -A, VEGF-C, VEGF-D, VEGF-A isoform 121, bone morphogenetic protein 7, macrophage colony-stimulating f

199 nto endocrine cell types by exposure to bone morphogenetic protein 7.

200 M) in the presence of human recombinant bone morphogenetic protein 7/human recombinant fibroblast gro

201 ng fibroblast growth factor 21 (FGF21), bone morphogenetic protein 8b (BMP8b), growth differentiation

202 caused by loss-of-function mutations in bone morphogenetic protein 9 (BMP9)-ALK1-Smad1/5/8 signaling,

203 e kinase 1 (ALK1), endoglin, Smad4, and bone morphogenetic protein 9 (BMP9).

204 naling did not significantly affect the bone morphogenetic protein activity.

205 negatively regulates the activities of bone morphogenetic protein and phosphoinositide 3-kinase (PI3

206 address whether elevated activities of bone morphogenetic protein and PI3K/AKT signaling pathways we

207 es demonstrated activation of canonical bone morphogenetic protein and Wnt/beta-catenin signaling and

208 ogenic fate of CNC(kit) is regulated by bone morphogenetic protein antagonism, a signaling pathway ac

209 ession of the gene encoding mesenchymal bone morphogenetic protein antagonist, GREM1.

210 nels regulate release of the Drosophila bone morphogenetic protein Dpp in the developing fly wing and

211 Treatment with bone morphogenetic protein or SHH pathway inhibitors decrease

212 (rs3072), which encodes a ligand in the bone morphogenetic protein pathway, and TBX5 (rs2701108), whi

213 analyzed the effect of Irf8 on TGF-beta/bone morphogenetic protein pathway-specific genes in DCs and

214 ocaine due to severe down-modulation of bone morphogenetic protein receptor (BMPR) axis: the anti-pro

215 ggests that serotonin, mutations in the bone morphogenetic protein receptor (BMPR) II gene, and estro

216 ations leading to reduced expression of bone morphogenetic protein receptor (BMPR) II, these mutation

217 The effect of a mutation in the bone morphogenetic protein receptor 2 (BMPR2) gene on right v

218 se decreased expression and function of bone morphogenetic protein receptor 2 (BMPR2) is observed in

219 ed by endothelial dysfunction, impaired bone morphogenetic protein receptor 2 (BMPR2) signaling, and

220 hyperactivating mutations of the type I bone morphogenetic protein receptor ACVR1 (Activin type 1 rec

221 er, transforming growth factor beta and bone morphogenetic protein receptor II signaling, and hypertr

222 of transforming growth factor beta and bone morphogenetic protein receptor II signaling, human RV an

223 ential transforming growth factor beta, bone morphogenetic protein receptor II signaling, or cardiac

224 Expression of bone morphogenetic protein receptor type 2 (BMPR2) and its ta

225 gous mutations in the gene encoding the bone morphogenetic protein receptor type 2 (BMPR2) are the co

226 eritable PAH caused by mutations in the bone morphogenetic protein receptor type 2 (BMPR2) gene and p

227 ertension with germline mutation in the bone morphogenetic protein receptor type 2 (BMPR2) gene, righ

228 te clinical and molecular similarity to bone morphogenetic protein receptor type 2 mutation-associate

229 Mutations in the gene encoding the bone morphogenetic protein receptor type II (BMPR2) are the c

230 aggregation; 2) rs11202221, in BMPR1A (bone morphogenetic protein receptor type1A), replicated in Af

231 In addition, the reduction in bone morphogenetic protein signaling and Shotgun expression o

232 However, inhibition of bone morphogenetic protein signaling caused a significant att

233 ), transforming growth factor-beta, and bone morphogenetic protein signaling pathways affect cardiomy

234 /Erk1/2 signaling and downregulation of bone morphogenetic protein signaling, with negative and posit

235 ther by erythroferrone or by inhibiting bone morphogenetic protein signaling.

236 ed that loss of PEAT modestly increases bone morphogenetic protein target gene expression and also el

237 ntially expressed genes were related to bone morphogenetic protein type 2 receptor (BMPR2) signaling.

238 HPAH patients inherit mutations in the bone morphogenetic protein type 2 receptor gene (BMPR2), but

239 Mutations in the bone morphogenetic protein type-II receptor (BMPR-II) are the

240 Heterozygous germ-line mutations in the bone morphogenetic protein type-II receptor (BMPR-II) gene un

241 ion and new bone formation through WNT, bone morphogenetic protein, and Notch signaling pathways.

242 of Hedgehog, fibroblast growth factor, bone morphogenetic protein, and Wnt signaling in the genital

243 ian hedgehog, fibroblast growth factor, bone morphogenetic protein, and Wnt signaling pathways in var

244 ing pathways, including Sonic hedgehog, bone morphogenetic protein, fibroblast growth factor, transfo

245 signalling pathways, specifically Wnt, bone morphogenetic protein, Notch and epidermal growth factor

246 The four members of the family: bone morphogenetic protein-1 (BMP-1), mammalian tolloid (mTLD

247 growth factor A (VEGFA), interleukin-2, bone morphogenetic protein-10, VEGFC, and 2 (FGF2) were marke

248 y, sustained in vivo delivery of active bone morphogenetic protein-2 (BMP-2) protein to responsive ta

249 fibroblast growth factor-2 (FGF-2) and bone morphogenetic protein-2 (BMP-2) work synergistically to

250 ECFCs produced bone morphogenetic protein-2 (BMP-2), a potent osteoinductive

251 es differentiation of OCs downstream of bone morphogenetic protein-2 (BMP-2)-stimulated osteoblast-co

252 h a collagen scaffold soaked in saline, bone morphogenetic protein-2 (BMP-2; 200 ng), 1 muM CGS21680

253 d temporal release of recombinant human bone morphogenetic protein-2 (BMP2) and vascular endothelial

254 Although bone morphogenetic protein-2 (BMP2) has demonstrated extraord

255 mesenchymal stem cells (MSCs), whereas bone morphogenetic protein-2 (BMP2) promotes osteogenic diffe

256 teoblasts, CypA is necessary for BMP-2 (Bone Morphogenetic Protein-2)-induced Smad phosphorylation.

257 In the present study, bone morphogenetic protein-2/BMP-2-directed osteogenic differ

258 Bone morphogenetic protein-4 (BMP4) plays a key role in regul

259 r hESC differentiated to TB by means of bone morphogenetic protein-4 and inhibitors of activin A and

260 isms involved revealed the induction of bone morphogenetic protein-7 (BMP7) expression, a critical re

261 We evaluated the effects of THR-184, a bone morphogenetic protein-7 agonist, in patients at high ris

262 r objective is to determine circulating Bone morphogenetic protein-9(BMP-9) levels in subjects with M

263 y, wild-type (WT) animals, in which the bone morphogenetic protein-mothers against decapentaplegic ho

264 ress driven and downstream of canonical bone morphogenetic protein-SMAD signaling.

265 th sLR11 inhibits thermogenesis via the bone morphogenetic protein/TGFbeta signalling pathway and red

266 Exogenous bone morphogenetic proteins (Bmp) are well known to induce ec

267 Bone morphogenetic proteins (BMP), part of the TGFbeta superf

268 al serine-threonine kinase receptor for bone morphogenetic proteins (BMPs) 9 and 10.

269 ds are specified by mesenchymal-derived bone morphogenetic proteins (BMPs) and fibroblast growth fact

270 Bone morphogenetic proteins (BMPs) are growth factors that pr

271 Bone morphogenetic proteins (BMPs) are secreted cytokines/gro

272 Bone morphogenetic proteins (BMPs) are secreted growth factor

273 Bone morphogenetic proteins (BMPs) are TGF-beta family member

274 Since the discovery of bone morphogenetic proteins (BMPs) as pluripotent cytokines e

275 Subsequent treatment with bone morphogenetic proteins (BMPs) enhanced differentiation t

276 In Xenopus laevis, bone morphogenetic proteins (Bmps) induce expression of the t

277 s the inhibitory effect of lung-derived bone morphogenetic proteins (BMPs) on self-renewal and theref

278 Bone Morphogenetic Proteins (BMPs) pattern the dorsal-ventral

279 Bone morphogenetic proteins (BMPs) play key roles in the regu

280 Bone morphogenetic proteins (BMPs) regulate diverse cellular

281 cultures, such as those involving Wnt, bone morphogenetic proteins (BMPs), Notch, and Hedgehog (Hh).

282 ing by Sonic hedgehog (SHH) followed by Bone morphogenetic proteins (BMPs), regulate a dynamic expres

283 ntiate into preosteoblasts that produce bone morphogenetic proteins (BMPs).

284 bsequent transcriptional suppression of bone morphogenetic proteins 5 and 7.

285 Bone morphogenetic proteins 9 and 10 (BMP9/BMP10) are circula

286 te locally (e.g., Wingless/Ints [Wnts], Bone Morphogenetic Proteins [BMPs], and Hedgehogs [Hhs]) and

287 ns as an anchor for cell-cell signalling and morphogenetic proteins involved in spore development.

288 gulated transforming growth factor beta/bone morphogenetic proteins signaling and that this imbalance

289 and that ligandless activity in the TGF/bone morphogenetic proteins signaling pathway contributes to

290 is essential for development and has been a morphogenetic puzzle.

291 tations were pleiotropic, affecting multiple morphogenetic reactions.

292 cans morphogenesis and identify 102 negative morphogenetic regulators and 872 positive regulators, hi

293 nformation is recognized and integrated into morphogenetic responses.

294 l glycosyltranferase enzymes, which play key morphogenetic roles in bacterial cell wall synthesis.

295 Sonic hedgehog (SHH) is an essential morphogenetic signal that dictates cell fate decisions i

296 use of proteins engineered with adhesive and morphogenetic solid-binding peptides is a promising rout

297 pment in mice, but its role in these dynamic morphogenetic steps has been inferred from fixed tissues

298 omplex subunit Hir1 decreases sensitivity to morphogenetic stimuli.

299 ures, bacterial lifestyles, and/or bacterial morphogenetic strategies.

300 e fungal pathogen Candida albicans undergoes morphogenetic switching from the yeast to the filamentou