ライフサイエンスコーパス: morphological abnormality

1 n5l2 mutants that begins before the onset of morphological abnormality.

2 One scaffold did not show any morphological abnormality.

3 ngle SLK gene failed to condition an obvious morphological abnormality.

4 RD Cy/+), demonstrating obvious tubular cell morphological abnormalities.

5 cral regions correlating with the regions of morphological abnormalities.

6 mice are viable and fertile, with no obvious morphological abnormalities.

7 induced behavioral, electrophysiological and morphological abnormalities.

8 ar cAMP levels under conditions that promote morphological abnormalities.

9 t dispersed sites in the genome that lead to morphological abnormalities.

10 lop normally and have no gross structural or morphological abnormalities.

11 sfected p70s6k, failed to reverse any of the morphological abnormalities.

12 ally invaginating septa, which in turn cause morphological abnormalities.

13 ha 4-positive cells did not reveal any gross morphological abnormalities.

14 ys in these latter recipients showed no late morphological abnormalities.

15 criteria in combination with hematologic and morphological abnormalities.

16 ions in cortical growth and thickness induce morphological abnormalities.

17 al knockout mice before the onset of visible morphological abnormalities.

18 deficits related to locomotion without major morphological abnormalities.

19 r been ineffective or has caused pleiotropic morphological abnormalities.

20 sed roots was reflected by their significant morphological abnormalities.

21 mation accumulates relative to the burden of morphological abnormalities.

22 ce known cascades of nonspecific pathways or morphological abnormalities.

23 ly 10-fold, with quadruple knockouts showing morphological abnormalities.

24 inding that does not appear to be related to morphological abnormalities.

25 k to cytokinesis accompanied by a variety of morphological abnormalities.

26 ix mutants displayed strain-specific vacuole morphological abnormalities.

27 mice are viable and fertile without obvious morphological abnormalities.

28 sulted in a decreased growth rate and marked morphological abnormalities.

29 a high rate of postnatal mortality, runting, morphological abnormalities, a susceptibility to infecti

30 lo3(-/-) sperm exhibit a higher incidence of morphological abnormalities accentuated by hypotonic cha

31 pment did not show impaired viability, overt morphological abnormalities, alterations in motor behavi

32 n the pfs2-1 allele, the integuments display morphological abnormalities and 95% of the embryo sacs f

33 terval prolongation in the absence of T-wave morphological abnormalities and a steep QT/R-R relations

34 ploidy and tissue defects, and ultimately to morphological abnormalities and adult inviability.

35 trypanosomes to heat shock and caused severe morphological abnormalities and cell cycle disruption.

36 nt SOD1 and mitoKARS result in mitochondrial morphological abnormalities and cell toxicity.

37 Ultrastructural analysis revealed severe morphological abnormalities and defects in cell-cell adh

38 ns of Katnal1 mutant mice we reveal numerous morphological abnormalities and defects in neuronal migr

39 T procedures cumulatively increase placental morphological abnormalities and epigenetic perturbations

40 severe form of HCM characterized by striking morphological abnormalities and juvenile lethality.

41 ta-deficient cells resulted in resolution of morphological abnormalities and restoration of barrier f

42 significantly altered mRNA expression, clear morphological abnormalities, and reduced contractility u

43 Conversely, in TDP43(A315T) mice, morphological abnormalities appeared after the onset of

44 These morphological abnormalities appearing in cultured cells

45 for formation of the lesions underlying the morphological abnormalities are discussed.

46 ong-term reductions in brain volume and that morphological abnormalities are, in turn, associated wit

47 ose for 48 h exhibited a higher incidence of morphological abnormalities, as well as an increase in l

48 em to capture subtle, previously undetected, morphological abnormalities associated with maltreatment

49 A prr1 null mutant exhibited no morphological abnormalities at either pH; however, it lo

50 during ookinete formation and thus precedes morphological abnormalities by several days.

51 f the vacuolar enzyme carboxypeptidase Y and morphological abnormalities consistent with a defect in

52 xpress Dube3a in the eye or wing and display morphological abnormalities, dependent on the critical c

53 L-8 in fetal lung, intestine, and brain, and morphological abnormalities: e.g., disrupted lung alveol

54 n CFC cells, Ras/MAPK pathway activation and morphological abnormalities exhibited cell subtype-speci

55 ce, USP18(C61A/C61A) animals did not exhibit morphological abnormalities, fatal IFN hypersensitivity,

56 Several morphological abnormalities have been described in the s

57 Shh activity, increased cell cycle exit, and morphological abnormalities in ALNPs.

58 sibly venules), with the earliest detectable morphological abnormalities in AV connections by P8.

59 The morphological abnormalities in C3KO muscles are associat

60 tes of the mutant are reduced, and there are morphological abnormalities in conidiophores, leading to

61 is consistent with recent evidence of early morphological abnormalities in cortical neurons in HD.

62 Is) prevent peripheral nerve dysfunction and morphological abnormalities in diabetic animal models.

63 Morphological abnormalities in layer formation of the ce

64 Here, we show that morphological abnormalities in lens secondary fiber cell

65 ient for prevention of nerve dysfunction and morphological abnormalities in long-standing diabetes or

66 Some lineages never showed morphological abnormalities in MARCM clones, whereas oth

67 lasts from individuals with HGPS have severe morphological abnormalities in nuclear envelope structur

68 rated a significant increase in the rates of morphological abnormalities in offspring collected from

69 uted tomography can identify a wide range of morphological abnormalities in patients with cystic fibr

70 es of wild-type E. coli and at the poles and morphological abnormalities in PBP mutants.

71 ucose metabolism may have been confounded by morphological abnormalities in previously studied "PDAPP

72 t the authors previously reported to exhibit morphological abnormalities in schizophrenia.

73 embryos, concomitant with the appearance of morphological abnormalities in several organs which expr

74 Morphological abnormalities in SLOS resemble those seen

75 ctron microscopic analysis revealed no gross morphological abnormalities in striatal mitochondria of

76 Consistent with these results, we found morphological abnormalities in the architecture of the n

77 s behavioral defect is accompanied by subtle morphological abnormalities in the central nervous syste

78 ases in ER stress biomarkers and profound ER morphological abnormalities in the circumventricular sub

79 77, are required for female fertility, gross morphological abnormalities in the eggshell are observed

80 t show increased anxiety in association with morphological abnormalities in the length and complexity

81 use cardiomyopathy in cultured cells induces morphological abnormalities in the nuclear envelope and

82 The incidence of morphological abnormalities in the RAS population of Pse

83 no depletion of brain serotonin but produced morphological abnormalities in the serotonergic immunore

84 Morphological abnormalities in the thalamus, together wi

85 Two transplant recipients developed striking morphological abnormalities in their circulating neutrop

86 AN-depleted embryos do not display any gross morphological abnormalities in their nuclear envelopes o

87 ed deficits in 5-HTT KO mice, accompanied by morphological abnormalities in two key neural loci.

88 how that overexpression of CYFIP1 results in morphological abnormalities including cellular hypertrop

89 entia is caused by different cerebrovascular morphological abnormalities including large artery terri

90 o complete gastrulation and display distinct morphological abnormalities, including a decrease in ove

91 ype (WT), GOF TRPP2 more efficiently rescued morphological abnormalities, including curly tail and cy

92 Onset of a spectrum of morphological abnormalities, including defects in leaf s

93 of ducts and glands that exhibit a range of morphological abnormalities, including formation of hype

94 cells fuse but the resulting hyphae exhibit morphological abnormalities, including lack of properly

95 A series of morphological abnormalities, including peculiar caudal f

96 These strains also have morphological abnormalities, including shortened and bul

97 QT morphological abnormalities, including U waves and bifid

98 The frequency of AV node conduction and morphological abnormalities increased from no detectable

99 humidity-grown cpn1-1 mutants also exhibited morphological abnormalities, increased resistance to vir

100 m) functional lesions in the retina before a morphological abnormality is detectable.

101 Such morphological abnormalities may lead to future developme

102 ht into the variable respiratory defects and morphological abnormalities observed in mitochondria of

103 The homozygotes exhibited many of the morphological abnormalities observed in previously repor

104 onents downstream of Ste20p), the growth and morphological abnormalities of cells lacking Akr1p are n

105 All had neuropathic pain in the feet and morphological abnormalities of cutaneous innervation in

106 existed; however, skin biopsy results showed morphological abnormalities of epidermal nerve fibers.

107 n 113 CAG knock-in mice was characterized by morphological abnormalities of germ cell maturation, dec

108 ssion of Notch ICD mitigates the laminar and morphological abnormalities of migrating neurons in Reel

109 Moreover, loss of EphA4 function resulted in morphological abnormalities of NL suggestive of errors i

110 eas functional, metabolic, neurotrophic, and morphological abnormalities of peripheral diabetic neuro

111 tidine (7H) and Angiotensin III, rescued the morphological abnormalities of primary neurons different

112 tained from an affected patient demonstrated morphological abnormalities of the actin cytoskeleton an

113 Morphological abnormalities of the external ear and hear

114 hanol during early embryonic development had morphological abnormalities of the eye characterized by

115 gical analysis of mutant mice revealed gross morphological abnormalities of the inner ear, and also d

116 myopathy (HCM), cardiovascular MR can detect morphological abnormalities of the left ventricle (LV) n

117 Whether morphological abnormalities of the mitral valve represen

118 of cells from a triple A patient revealed no morphological abnormalities of the nuclei, nuclear envel

119 Morphological abnormalities of the peripheral nervous sy

120 atients with schizophrenia exhibit selective morphological abnormalities of the prefrontal cortex.

121 The severity of various morphological abnormalities of the strains was also atte

122 children without urinary tract functional or morphological abnormalities (OR, 8.0; 95% CI, 2.3-27.4;

123 n that alterations in axonal trafficking and morphological abnormalities precede and lead to senile p

124 the WAF of weathered slick oil of DWH caused morphological abnormalities predicted by a suite of adva

125 The morphological abnormalities reported here would be expec

126 Morphological abnormalities resembling cirrhosis develop

127 heterogeneous disease, and are an important morphological abnormality responsible for LV outflow obs

128 th an mTOR inhibitor (rapamycin) rescued the morphological abnormalities resulting from CYFIP1 overex

129 tter MTR (GM MTR) observed is trans-synaptic morphological abnormality secondary to demyelinating les

130 ndrial crystals (IMCs) are a well-documented morphological abnormality seen on transmission electron

131 We find that chs6Delta strains have morphological abnormalities similar to those of chs3 mut

132 t strains display other phenotypes including morphological abnormalities, stationary phase defects, a

133 Morphological abnormalities such as kinks and bends at t

134 However, morphological abnormalities suggested that syncytial emb

135 In fact, IKK1-/- mice display morphological abnormalities that are independent of its

136 f the SPN, and alveolin gene knockout causes morphological abnormalities that coincide with markedly

137 Morphological abnormalities that included ovoid or multi

138 cells deficient in p85alpha exhibited gross morphological abnormalities that were not evident in cel

139 onsistent with previously reported bilateral morphological abnormalities, the affected members showed

140 mation, reduced polysaccharide secretion and morphological abnormalities, was chosen for virulence st

141 ele of RHO3, RHO3(E129,A131), which causes a morphological abnormality, was expressed.

142 In SOD1(G93A) mice, mitochondrial morphological abnormalities were apparent at 15 days of

143 Surprisingly, by postnatal day 14 (P14), no morphological abnormalities were detectable in the corte

144 No morphological abnormalities were observed in diazoxide-

145 The earliest morphological abnormalities were observed in the embryon

146 No significant morphological abnormalities were observed, indicating th

147 Frequency and severity of morphological abnormalities were significantly greater i

148 ed protein body expansion but did not induce morphological abnormalities, which result from reduction

149 nt embryos prior to the development of overt morphological abnormalities, which suggests that Tbx4 re

150 6-harbouring cell line W12 shows progressive morphological abnormality with in vitro passage, culmina

151 yed overlapping but distinct biochemical and morphological abnormalities within the endosomal-lysosom