ライフサイエンスコーパス: mosquito

1 oduction of gametes following ingestion by a mosquito.

2 at spore dosages as low as one conidium per mosquito.

3 ith asthma and/or rhinitis and sensitized to mosquito.

4 or approaches that limit ZIKV replication in mosquitoes.

5 nhibitors of the larval ACE activity of both mosquitoes.

6 ibit development of Plasmodium falciparum in mosquitoes.

7 r productive DENV infection of Aedes aegypti mosquitoes.

8 nd subsequently enhances viral prevalence in mosquitoes.

9 induce increased attractiveness of humans to mosquitoes.

10 s of Pfs25 block sexual-stage development in mosquitoes.

11 cy, and no permethrin resistance among local mosquitoes.

12 s during ammonia metabolism of Aedes aegypti mosquitoes.

13 dscapes underpinning human interactions with mosquitoes.

14 to bind to its intracellular target and kill mosquitoes.

15 block malaria transmission to all anopheline mosquitoes.

16 egulated but also induced in resistant Culex mosquitoes.

17 d from salivary glands of infected Colombian mosquitoes.

18 vitro and in mice and blocks transmission to mosquitoes.

19 nfrastructure specific to Wolbachia-infected mosquitoes.

20 hei expressing PfCelTOS in Anopheles gambiae mosquitoes.

21 mes from infected patients and Aedes aegypti mosquitoes.

22 oocyst infection and prevalence in Anopheles mosquitoes.

23 r, is an arbovirus transmitted by Haemagogus mosquitoes.

24 aling facilitates the arboviral infection of mosquitoes.

25 ial larvicides used widely to control vector mosquitoes.

26 es at higher abundance than those from Aedes mosquitoes.

27 is the first report of the V410L mutation in mosquitoes.

28 ratio released by subject N did not attract mosquitoes.

29 alciparum gametocytes to Anopheles stephensi mosquitoes.

30 on limited evidence, abating transmission to mosquitoes.

31 n to time since treatment and infectivity to mosquitoes.

32 t blocking strategy, allowing a reduction of mosquito 18s rRNA gene sequences by more than 80% for th

33 ed against a human landing catch during peak mosquito abundance, this "host decoy" trap caught nearly

34 Asian tiger mosquito (Ae. albopictus) HP-I peptides potently activat

35 The yellow fever mosquito Aedes aegypti forms aerial swarms that serve as

36 The mosquito Aedes aegypti is a major vector of numerous vir

37 as been transferred from Drosophila into the mosquito Aedes aegypti, where it can block the transmiss

38 which are transmitted by the disease vector mosquito Aedes aegypti.

39 The yellow fever mosquito, Aedes aegypti, particularly in Neotropical reg

40 be an interkingdom cue for the yellow fever mosquito, Aedes aegypti, seeking blood-meals as well as

41 Culex quinquefasciatus, and the yellow fever mosquito, Aedes aegypti.

42 uated 10-del ZIKV was incapable of infecting mosquitoes after oral feeding of spiked-blood meals, rep

43 Saliva and muscle-derived mosquito allergens have been purified and characterized.

44 ns may play a role in the pathophysiology of mosquito allergy in the tropics, and some of them might

45 y required during its navigation through the mosquito and vertebrate host tissues.

46 CHIKV is transmitted by Aedes species of mosquitoes and is capable of an epidemic, urban transmis

47 ERPRETATION: The new design houses had fewer mosquitoes and were cooler than modified and unmodified

48 The primary cycle involves mosquitoes and wild and domestic ruminant hosts.

49 d-borne virus transmitted primarily by Aedes mosquitos and is major cause of disease in tropical and

50 sed by the mean number of oocysts in control mosquitoes) and antibody titers.

51 rains: wAlbB (isolated from Aedes albopictus mosquitoes) and wStri (isolated from the planthopper Lao

52 detectable by microscopy), attractiveness to mosquitoes, and modified sleeping behaviour.

53 rapidly through populations of Aedes aegypti mosquitoes, and strongly inhibits infection with key hum

54 Mosquitoes are hematophagous insects that carry-on and t

55 isms responsible for collective behaviour in mosquitoes are not well understood.

56 ion, dissemination and transmission rates in mosquitoes are only mildly affected by coinfection.

57 Aedes and Culex mosquitoes are the main culprits, spreading infection wh

58 Anopheles mosquitoes are vectors of the human malaria parasite, Pl

59 cific promoter increased fungal lethality to mosquitoes at spore dosages as low as one conidium per m

60 Malaria parasites are thought to influence mosquito attraction to human hosts, a phenomenon that ma

61 compounds were identified that can influence mosquito behaviour, including 2- and 3-methylbutanal, 3-

62 hich contribute to a better understanding of mosquito biology and are thus a key to finding new strat

63 Here we show that infection via mosquito bite delays ZIKV replication to peak viral load

64 Zika virus (ZIKV) can be transmitted by mosquito bite or sexual contact.

65 se it more closely mimics human infection by mosquito bite than needle-based inoculations.

66 es preventing every sporozoite inoculated by mosquito bite: a major challenge for Plasmodium falcipar

67 Anecdotes related to preferential mosquito bites are very common, but to date there is no

68 t of two people systematically receives more mosquito bites than the other when both are equally acce

69 prophylactic measures to reduce or eliminate mosquito bites, including the use of insect repellents.

70 mosquitoes than children, they received more mosquito bites, thus balancing their contribution to the

71 Chikungunya virus (CHIKV), a mosquito-borne alphavirus, causes febrile disease, muscl

72 The re-emergence of Zika virus (ZIKV), a mosquito-borne and sexually transmitted flavivirus circu

73 Zika virus (ZIKV) is a mosquito-borne and sexually transmitted flavivirus that

74 Mosquito-borne arboviruses are a major source of human d

75 Malaria is a mosquito-borne disease affecting millions of people ever

76 Dengue is a mosquito-borne disease that threatens over half of the w

77 ed harmonic models to compare seasonality of mosquito-borne diseases on a continent-wide scale.

78 appropriate and timely control measures for mosquito-borne diseases.

79 ical outbreaks as well as future patterns of mosquito-borne diseases.

80 sed as a template for the analysis of future mosquito-borne epidemics.

81 The impact of mosquito-borne flavivirus infections worldwide is signif

82 West Nile virus (WNV) is a mosquito-borne flavivirus that causes epidemics of encep

83 Zika virus (ZIKV) is a mosquito-borne flavivirus that emerged recently as a glo

84 Zika virus (ZIKV) is a mosquito-borne flavivirus that has rapidly extended its

85 RTANCE Recent outbreaks of ZIKV, a neglected mosquito-borne flavivirus, have identified sexual transm

86 The four dengue virus (DENV) serotypes are mosquito-borne flaviviruses responsible for dengue fever

87 West Nile virus (WNV) is a major cause of mosquito-borne illness in the United States.

88 Mosquito-borne infections are increasing in number and a

89 INTERPRETATION: Cache Valley virus, a mosquito-borne orthobunyavirus, has only been identified

90 Dengue virus is a mosquito-borne pathogen that causes up to about 100 mill

91 ZIKV is a rapidly spreading mosquito-borne pathogen that has been linked to Guillain

92 Malaria is caused by mosquito-borne Plasmodium spp. parasites that must infec

93 eplication within red blood cells, while its mosquito-borne transmission depends on intra-erythrocyti

94 developing effective strategies to minimize mosquito-borne transmission of human diseases.

95 Dengue is the most common mosquito-borne viral disease in human beings, and vector

96 es (DENV-1 to DENV-4), is a highly prevalent mosquito-borne viral disease in humans.

97 Zika, a mosquito-borne viral disease that emerged in South Ameri

98 In many regions of the world, mosquito-borne viruses pose a growing threat to human he

99 ith ecological modifications that may favour mosquito breeding.

100 early stages of Plasmodium infection in the mosquito, but it has a strong deleterious effect on spor

101 ansmission following the bite of an infected mosquito, but the contribution of individual cell types

102 ommon mechanisms underlying the infection of mosquitoes by these viruses.

103 Wolbachia infection of mosquitoes can block dengue virus infection and is teste

104 Mosquitoes carrying the more-susceptible allele (2L+(a))

105 ght nearly ten times the number of Anopheles mosquitoes caught by a human collector.

106 The overall reduction of all mosquitoes caught was highest in the double-storey build

107 arameters we selected four mammalian and two mosquito cell lines, and further characterised these as

108 of the effects of Wolbachia in Aedes aegypti mosquito cells and midgut.

109 A single dose of EILV/CHIKV produced in mosquito cells elicited rapid (within 4 d) and long-last

110 rus was rescued that replicates in human and mosquito cells with growth kinetics representative of wi

111 m the planthopper Laodelphax striatellus) in mosquito cells.

112 anges can be substantial, such that the same mosquito colony may be considered fully susceptible or h

113 he role of forest disturbance in shaping the mosquito community structure, and to identify the ecolog

114 tion and transmission rates in Aedes aegypti mosquitoes comparable to those of the primary isolate an

115 evelopment and protecting ookinetes from the mosquito complement-like response, respectively.

116 Mosquito control programs aimed at limiting transmission

117 New mosquito control strategies are vitally needed to addres

118 Mosquito control, vaccine, and therapeutics are 3 potent

119 mosquitoes poses a significant challenge to mosquito control.

120 var gene during parasite development in the mosquito correlates with the presence of low levels of H

121 human patients, raising the likelihood that mosquitoes could be exposed to multiple arboviruses duri

122 , host-seeking females of the southern house mosquito, Culex quinquefasciatus, and the yellow fever m

123 l northeastern Tanzania, Africa, to decrease mosquito density and regulate indoor climate.

124 Instead, host responses to mosquito-derived factors have an important influence on

125 the most used insecticides to control Aedes mosquitoes, despite the development of pyrethroid resist

126 previously unknown but essential process for mosquito development.

127 ssing Cas9 have been developed for the major mosquito disease vector Aedes aegypti Here, we describe

128 asite development when ingested by Anopheles mosquitoes during blood meals.

129 aegypti, as prophylactic immunity to protect mosquitoes during the vulnerable stages of each molt.

130 CLIPA14 kd mosquitoes elicited a potent melanization response again

131 Mosquitoes exhibit unusual wing kinematics; their long,

132 nced by the host species upon which infected mosquitoes feed.

133 malaria transmission potential based on 1209 mosquito feeding assays in endemic areas of Burkina Faso

134 25 antibodies for transmission blocking upon mosquito feeding.

135 hat reported gametocyte counts or results of mosquito-feeding assays.

136 A genetic variation within mosquito fibrinogen related-protein 30 (FBN30) was previ

137 sor is compatible with "gold-standard" adult mosquito field-collection protocols and generates electr

138 (NS1) facilitates flavivirus acquisition by mosquitoes from an infected mammalian host and subsequen

139 o test the hypothesis that leg loss inhibits mosquitoes from biting and reproducing, mosquitoes with

140 sfully employed in southeast Asia to prevent mosquitos from entering and cooling the house, could be

141 modification (SSM) is an important tool for mosquito functional genomics and comparative gene expres

142 Both strategies lasted more than 500 mosquito generations (or about 40 years) in 24% of runs,

143 e as an essential bacterial gene product for mosquito growth.

144 Mosquitoes harbor a high diversity of RNA viruses, inclu

145 iotic bacterium Wolbachia into Aedes aegypti mosquitoes has the potential to greatly reduce the publi

146 orogonic development of P. falciparum in the mosquito host.

147 Mosquitoes host communities of microbes in their digesti

148 and played a pivotal role in dissecting the mosquito immune response against infection.

149 of this parasite to study the impact of the mosquito immune response on human malaria transmission.

150 um falciparum, protecting ookinetes from the mosquito immune response.

151 Understanding the mechanisms underlying mosquito immunity could provide new tools to control arb

152 quiring 45% fewer spores to kill half of the mosquitoes in 5 days as single toxin strains.

153 reated nets or indoor residual sprays target mosquitoes in human dwellings.

154 The percentages of infected mosquitoes in the different surveys ranged from 0.05 (4/

155 o exposing a human subject's arm to infected mosquitoes in the standard arm-in-cage assay.

156 rticosterone, enhance bird susceptibility to mosquitoes in ways that enhance rates of co-infection?

157 We find that, in these mosquitoes, in contrast to what has been found in many o

158 pellents are effective against ZIKV-infected mosquitoes, in part because of the ethical concerns rela

159 with the gut bacterial microbiome, which, in mosquitoes, increases dramatically soon after a blood me

160 dium sporozoite infection in field-collected mosquitoes indicates that the prevalence and intensity o

161 ped a blood meal substitute specifically for mosquitoes infected with the wMel Wolbachia strain.

162 mosquito populations in release trials, and mosquitoes infected with these strains show markedly red

163 Importantly, in mosquito-infected animals ZIKV tissue distribution was l

164 ng analysis reveals that ZIKV populations in mosquito-infected monkeys show greater sequence heteroge

165 The absence of LIMP reduces initial mosquito infection by 50%, impedes salivary gland invasi

166 ht represent an important source of human-to-mosquito infection due to frequent parasite carriage wit

167 Mosquito infection ensues upon oocyst development that f

168 lation method and two observed data streams, mosquito infection rates and reported human WNV cases.

169 quito midgut epithelium and establishment of mosquito infection.

170 ction of P. berghei P47 in Anopheles gambiae mosquito infections.

171 at FREP1-mediated Plasmodium transmission to mosquitoes is a conserved pathway and that targeting the

172 rstanding the interaction between humans and mosquitoes is a critical area of study due to the phenom

173 Plasmodium invasion of anopheline mosquitoes is an obligatory step for malaria transmissio

174 erception of itchiness and attractiveness to mosquitoes is driven, at least in part, by the genetic d

175 The control of mosquitoes is threatened by the appearance of insecticid

176 their ability to kill homozygous susceptible mosquitoes) is high and exposure (the proportion of mosq

177 In adult mosquitoes, it is essential for maturation of the ovary

178 and PBCHO, both of which are toxic to Culex mosquito larvae, can be further metabolized by CYP9M10/C

179 PBCOOH, which is considerably less toxic to mosquito larvae.

180 d exposure often seen in laboratory tests is mosquito leg loss, a condition that has thus far been as

181 R-277) plays an important role in regulating mosquito lipid metabolism.

182 However, mosquito-malaria molecular interactions in nature are no

183 function of these elements in the genome of mosquitoes may lead to epidemiological interventions.

184 a severely impaired capacity to traverse the mosquito midgut and transform to oocysts.

185 tein that promotes parasite traversal of the mosquito midgut epithelium and establishment of mosquito

186 llows ookinete invasion and traversal of the mosquito midgut epithelium.

187 terization of PIMMS2 (Plasmodium invasion of mosquito midgut screen candidate 2), a Plasmodium berghe

188 Mosquito midgut stages of the malaria parasite present a

189 Anopheles ovaries that stably colonizes the mosquito midgut, female ovaries, and male accessory glan

190 and reducing FBN30 expression by RNAi makes mosquitoes more susceptible to P. berghei.

191 the simulated biting rates to be largest and mosquito mortality rates and extrinsic incubation period

192 ertebrate blood meal for reproduction, these mosquitoes need a lot of energy; therefore, understandin

193 Although many species of mosquitoes never take a blood meal, identifying genes th

194 iours emerge in groups of more than six male mosquitoes, occurring to a greater degree than predicted

195 l and reproductive aspects of the biology of mosquitoes of epidemiological importance.

196 ion and attractiveness to Anopheles coluzzii mosquitoes of skin odours from participants that were in

197 bachia strain (wAnga-Mali) was identified in mosquitoes of the Anopheles gambiae complex collected in

198 Mosquitoes of the Anopheles gambiae complex were identif

199 f how environmental variations influence the mosquito phenotype.

200 We used a dynamic mosquito population and virus transmission model driven

201 kening the integration of this strain into a mosquito population relative to that of other Wolbachia

202 The direct monitoring of mosquito populations in field settings is a crucial inpu

203 to be able to spread to high frequencies in mosquito populations in release trials, and mosquitoes i

204 We propose that comparisons of EVEs across mosquito populations may explain differences in vector c

205 criptomics analysis of 12 Western Australian mosquito populations structured by species and geographi

206 development of pyrethroid resistance in many mosquito populations worldwide.

207 to effectively introduce such bacteria into mosquito populations.

208 ted that this is a viable strategy to modify mosquito populations.

209 essory glands and spreads rapidly throughout mosquito populations.

210 rge-scale Wolbachia transformations of urban mosquito populations.

211 Behavioral resilience in mosquitoes poses a significant challenge to mosquito con

212 e dehydrogenase-1 silencing in Aedes aegypti mosquitoes promotes a blood feeding-induced adulticidal

213 populations and block virus transmission by mosquitoes, providing an important approach to dengue co

214 The spread of blood-borne pathogens by mosquitoes relies on their taking a blood meal; if there

215 showing that CLIPA2 knockdown (kd) enhances mosquito resistance to infections with fungi, bacteria,

216 symbiotic bacteria have been shown to render mosquitoes resistant to the parasite, the challenge rema

217 The ingestion of blood by mosquitoes resulted in robust GABA production from gluta

218 sodium channel gene in pyrethroid resistant mosquitoes revealed evidences of alternative splicing ev

219 for flavivirus infection of human cells and mosquitoes: RPLP1 and RPLP2 (RPLP1/2).

220 to reduce arbovirus disease is to reduce the mosquito's ability to transmit virus.

221 The mosquito's innate immune system controls both Plasmodium

222 uch diseases, the pathogen must traverse the mosquito salivary gland (SG) for transmission to a new h

223 arasites that must infect and survive within mosquito salivary glands (SGs) prior to host transmissio

224 generate 110 ZIKV genomes from clinical and mosquito samples from 10 countries and territories, grea

225 te conditions may increase the length of the mosquito season in many locations, projected increases i

226 f prestinA and NDAE1 in interactions between mosquito SGs and Plasmodium, and suggest the need for fu

227 in reduction of the number of sporozoites in mosquito SGs.

228 XDH1-deficient mosquitoes showed a persistence of serine proteases in t

229 characterized IgE-binding proteins from the mosquito species Aedes aegypti.

230 , particularly to the Americas, where native mosquito species are capable of virus transmission.

231 NAs and that, with four multiplexed gRNAs, a mosquito species could potentially be suppressed on a co

232 We previously reported that several mosquito species, including Aedes aegypti, do not develo

233 Among Anopheles mosquito species, these phenotypic differences include v

234 e survey a wide range of medically important mosquito species, to quantitatively demonstrate how acou

235 n the genomes of Aedes, Culex, and Anopheles mosquito species.

236 tor control tools that target indoor-resting mosquitoes, such as bednets and insecticides, are curren

237 Thus, we establish a new paradigm for mosquito surveillance that takes advantage of the existi

238 To further evaluate their functions in mosquito survival and parasite infection, these genes we

239 Blood-fed mosquitoes synthesized [(13)C] metabolites in mainly 2 c

240 in gene expression occur after adult female mosquitoes take a blood meal and use the nutrients for e

241 te occurs in an unpredictable moment, when a mosquito takes a blood meal.

242 ocyte carriers attracted almost 2 times more mosquitoes than children who were parasite free, harbore

243 though adults transmitted fewer parasites to mosquitoes than children, they received more mosquito bi

244 substitution in the ZIKV FSS13025 strain in mosquitoes that acquired ZIKV from a viraemic C57BL/6 mo

245 The wMel-infected mosquitoes that are field-reared have even greater relat

246 ne if there are universal nonbiting genes in mosquitoes that could be manipulated as a means to contr

247 oes) is high and exposure (the proportion of mosquitoes that encounter the insecticide) is low.

248 s of pyrethroid efficacy should not discount mosquitoes that survive insecticide exposure with fewer

249 alized community of macroinvertebrates (e.g. mosquitoes) that feed on microbial communities associate

250 a-specific physiological changes in infected mosquitoes, that allows the bacterium to spread, and blo

251 blood meals enhance arbovirus replication in mosquitoes through activation of the GABAergic system.

252 Thus, we here expose Ae. aegypti mosquitoes to chikungunya, dengue-2 or Zika viruses, bot

253 n reduce the permissiveness of Aedes aegypti mosquitoes to disseminated arboviral infections.

254 he rearing and releasing of large numbers of mosquitoes to eliminate or modify local Aedes population

255 sed susceptibility of field-reared wild-type mosquitoes to infection than laboratory-reared counterpa

256 some of the most important components of the mosquito transcriptome, and we identified 19 new virus s

257 im to control malaria by inhibiting human-to-mosquito transmission show considerable promise though t

258 e of, and risk from, these modes compared to mosquito transmission.

259 ied ten ApiAP2 genes that were essential for mosquito transmission: four were critical for the format

260 Hematophagous female mosquitoes transmit numerous devastating human diseases,

261 The Aedes aegypti mosquito transmits arboviruses, including dengue, chikun

262 the phenomenal burdens on public health from mosquito-transmitted diseases.

263 West Nile virus (WNV) is a mosquito-transmitted flavivirus that can cause debilitat

264 Zika virus (ZIKV) is an emerging mosquito-transmitted flavivirus that now causes epidemic

265 Examples of mosquito-transmitted flaviviruses include dengue, yellow

266 Plasmodium sporozoites, the mosquito-transmitted forms of the malaria parasite, firs

267 Re-emergence of chikungunya virus, a mosquito-transmitted pathogen, is of serious public heal

268 on.IMPORTANCE CHIKV is a globally spreading, mosquito-transmitted virus that causes debilitating acut

269 In particular, mosquito-transmitted viruses, such as those that cause Z

270 , unlike other flaviviruses, is sexually and mosquito-transmitted, and an increase in the incidence o

271 We show that, although mosquitoes use familiar separated flow patterns, much of

272 Mosquitoes use their antennae as hearing organs to locat

273 ions where minimally-trained users map local mosquitoes using their personal phones.

274 primarily transmitted by Aedes aegypti, the mosquito vector also important in transmission of the fl

275 equisite for transmission from humans to the mosquito vector and has emerged as a target for interven

276 amics in that it is concurrently spread by a mosquito vector and through sexual contact.

277 luding increased air travel and uncontrolled mosquito vector populations.

278 irus (ZIKV) that explicitly includes two key mosquito vector species: Aedes aegypti and Aedes albopic

279 dition, was essential for development in the mosquito vector.

280 es, called gametocytes, are infective to the mosquito vector.

281 iency of malaria parasite development within mosquito vectors (sporogony) is a critical determinant o

282 ansion is attributed to the success of Aedes mosquito vectors, but local epidemiological drivers are

283 n monitoring and modeling arboviruses within mosquito vectors.

284 al prevalence of dengue virus (DENV) and its mosquito vectors.

285 al network connecting flaviviruses and their mosquito vectors.

286 ion is the effective management of Anopheles mosquito vectors.

287 glutamic acid increased virus acquisition by mosquitoes via activation of the GABAergic system.

288 ort to describe the extent and nature of the mosquito virome, little is known about how these viruses

289 regarding this fascinating interplay between mosquito, virus, and the mammalian host.

290 ships, we were able to distinguish potential mosquito viruses from those present in coinfecting bacte

291 on against ZIKV-infected and old noninfected mosquitoes was achieved with 5% DEET, which corresponds

292 The odds of transmission to mosquitoes were also lower in AL treatment groups (OR 0.

293 Mosquitoes were collected in central Panama at immature

294 ed in all study houses with data loggers and mosquitoes were collected indoors and outdoors using Fur

295 Mosquitoes were fed a blood meal supplemented with [1,2-

296 promotes ZIKV infectivity and prevalence in mosquitoes, which could have facilitated transmission du

297 Here we report free-flight mosquito wing kinematics, solve the full Navier-Stokes e

298 We observed that genetically modified mosquitoes with increased immune activity in the midgut

299 bits mosquitoes from biting and reproducing, mosquitoes with one, two, or six legs were evaluated for

300 hing of blood from 1094 wild-caught bloodfed mosquitoes with that of humans resident in the same hous