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1 e inhibited by Purkinje cells and excited by mossy fibres.
2 well as direct vestibular afferent inputs as mossy fibres.
7 cted to graded ribbon-type synapses and that mossy fibres are well suited for transmitting broad-band
8 sential for the lamina-specific targeting of mossy fibre axons onto CA3 pyramidal neurons in the deve
9 ce demonstrate that synaptic excitation from mossy fibres becomes more effective at increasing the ra
10 epticus, there were significant increases in mossy fibre bouton size, faster rates of action potentia
11 We also analysed the ultrastructure of rat mossy fibre boutons using transmission electron microsco
12 tch-clamp recordings in vivo from cerebellar mossy fibre boutons-the primary source of synaptic input
13 synapses with CA3 pyramidal cells via large mossy-fibre boutons, but rather to all synapses formed b
14 e anatomical and physiological properties of mossy fibre-CA3 interneurone synapses have been previous
18 s, the effect of KA on glutamate release and mossy fibre-CA3 synaptic transmission was occluded by th
21 nce of Purkinje cell activity can facilitate mossy fibre-driven spiking by CbN cells, in turn driving
27 monstrate that high-frequency stimulation of mossy fibres in rat hippocampal slices, in the presence
28 e cerebellar cortex, controlling the flow of mossy fibre information to other cells via granule cells
29 glutamate, which is released from excitatory mossy fibres, inhibits GABA release from Golgi cell term
30 l that movement is accompanied by changes in mossy fibre input rate that drive membrane potential dep
32 ls were driven with bursts of high-frequency mossy fibre input, as observed in vivo, larger inhibitio
34 nly a few quantal EPSCs, and yet spontaneous mossy fibre inputs triggered spikes only when inhibition
36 ble to reverse the selective deficit seen in mossy fibre-long-term potentiation (MF-LTP) in Cplx2-/-
37 dent LTD and LTP at other cortical synapses, mossy fibre LTD occurs when synaptic conditions are insu
41 increase in neurotransmitter release during mossy fibre LTP may be mediated by a unitary mechanism t
42 naptic vesicle protein Rab3A is required for mossy fibre LTP, but the protein kinase A substrates rab
43 artate) receptors, whereas the other, called mossy fibre LTP, has a principal presynaptic component.
44 NMDA receptors but prevents the induction of mossy fibre LTP, which is independent of NMDA receptors.
46 chanisms occurs in the CA3 hippocampus where mossy fibre (MF) axons of dentate gyrus granule cells ta
50 arboxycyclopropyl)glycine (DCG-IV) depressed mossy fibre (MF)-evoked excitatory drive to these intern
51 hat repetitive activation of the hippocampal mossy fibre pathway, which is associated with high-affin
53 pocampus, where repetitive activation of the mossy-fibre pathway generates a slow, kainate-receptor-m
54 sicular release and recycling in hippocampal mossy fibre presynaptic boutons, we used (i) two-photon
55 Measurements of intracellular Ca2+ in single mossy-fibre presynaptic terminals indicate that nAChRs c
57 amidal cell layer, with spreading of the CA3 mossy fibre projection to an ectopic infrapyramidal posi
58 me topographical organisation within the LRN mossy fibre projection to certain cerebellar cortical zo
63 ells constitute the input layer, translating mossy fibre signals into parallel fibre input to Purkinj
64 cally effective antidepressant treatments on mossy fibre sprouting in the rat dentate gyrus using Tim
65 so ineffective, indicating that induction of mossy fibre sprouting is not a common property of effect
67 ketamine dissociated these effects such that mossy fibre sprouting was attenuated significantly, whil
68 degree of hippocampal cell proliferation and mossy fibre sprouting was quantified using the Timm stai
69 of the hippocampus and co-localization with mossy fibre sprouting, a feature of temporal lobe epilep
72 l glutamatergic synaptic transmission at the mossy-fibre synapse because the amplitude, input-output
73 f short-term plasticities are normal, LTP at mossy fibre synapses is abolished in mice lacking the sy
74 f glutamate receptor, but LTP at hippocampal mossy fibre synapses is independent of NMDA receptors an
76 ne vesicle mobility at excitatory cerebellar mossy fibre synapses which sustain transmission over a b
77 Mossy fibre LTP is expressed in hippocampal mossy fibre synapses, cerebellar parallel fibre synapses
78 ansmitter release is enhanced at hippocampal mossy fibre synapses, the concentration of glutamate inc
80 recordings to show the relationship between mossy fibre synaptic currents evoked by somatosensory st
81 ay be related to the long-term depression of mossy fibre synaptic responses that has recently been sh
82 ibre synaptic transmission, these details of mossy fibre synaptic transmission should provide insight
83 ven the central role of CA3 interneurones in mossy fibre synaptic transmission, these details of moss
87 e investigate transmission at the cerebellar mossy fibre terminal, which can fire at over 200 Hz for
88 ve approximately 10 times more synapses from mossy fibres than do principal neurones has led to the r
89 euregulins are concentrated in glutamatergic mossy fibres that innervate granule cells in the interna
91 These findings suggest that the relay from mossy fibre to granule cell can act in a 'detonator' fas
93 se (during high frequency stimulation of the mossy fibres), tonic inhibition and superfused ACh are 1
95 hether the other major cerebellar input, the mossy fibres (which generate simple spikes within Purkin
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