ライフサイエンスコーパス: motion perception

1 sual processing (luminance, orientation, and motion perception).

2 cs and prior assumptions are critical for 3D motion perception.

3 ual motion information is important for self-motion perception.

4 le in visual/vestibular integration for self-motion perception.

5 ivalry, revealing a dissociation of form and motion perception.

6 y currently overlooked by Bayesian models of motion perception.

7 ive much of the group difference in coherent motion perception.

8 visual cortex thought to be involved in self-motion perception.

9 ctivity in the middle temporal (MT) area and motion perception.

10 areas had the greatest effect on biological motion perception.

11 usal relationships to deficits in biological motion perception.

12 deficits, with many involving impairments in motion perception.

13 ficits observed in ASD, including biological motion perception.

14 tion between pathways for heading and object motion perception.

15 global/contextual information, and impaired motion perception.

16 be associated with the preattentive illusory motion perception.

17 egration to downstream signals that underlie motion perception.

18 in addition to its well established roles in motion perception.

19 nship between cortical physiology and visual motion perception.

20 videnced by a sound-induced change in visual motion perception.

21 ontrast--a neural correlate of "reverse phi" motion perception.

22 AMO rearing results in asymmetric motion perception.

23 vidence that colour can indeed contribute to motion perception.

24 This identified brain areas responsible for motion perception.

25 l completion or 'filling in' during apparent motion perception.

26 18 lesion patients tested showed normal self-motion perception.

27 ooling of MT responses and the role of MT in motion perception.

28 eption in addition to its well known role in motion perception.

29 a primate region that is involved in visual motion perception.

30 t area MT has a basic role in structure-from-motion perception.

31 spectrally non-opponent, and supports normal motion perception.

32 then the case for V5/hMT+ serving multimodal motion perception.

33 nd the complex processes underlying auditory motion perception.

34 e body area) are not critical for biological motion perception.

35 rtant constraints on the roles of V6 in self-motion perception.

36 nuclei involved in postural control and self-motion perception.

37 obiological substrates underlying biological motion perception.

38 these areas are causally involved in tactile motion perception.

39 orm perception, adversely affects biological motion perception.

40 th damage to regions critical for biological motion perception.

41 le in visual-vestibular integration for self-motion perception.

42 the control of eye movements, attention, and motion perception.

43 ating areas of the visual cortex involved in motion perception.

44 typal example of global perception, coherent motion perception.

45 e often considered a consequence of impaired motion perception.

46 bed damage to ventral visual cortex impaired motion perception.

47 postural control and the computation of self-motion perception.

48 iddle temporal (MT) area, an area related to motion perception.

49 human homologs, i.e., area hMT and hMST, on motion perception.

50 nhibitory imbalance in the context of visual motion perception.

51 has not been considered critical for normal motion perception.

52 d eyes are ideally suited for fast panoramic motion perception.

53 BD) early in life permanently impairs global motion perception.

54 r and how these pathways interact to improve motion perception.

55 relation was found between VEP latencies and motion perception.

56 able attributes by examining dependencies in motion perception.

57 ion-making network known to support coherent motion perception.

58 and amygdala in a human disorder of abnormal motion perception.

59 neuronal signals in human MT+ support visual motion perception, (3) human MT+ is homologous to macaqu

60 e effect of prenatal drug exposure on global motion perception, a behavioural measure of processing w

61 We used motion perception, a percept with relatively well known

62 Overall, we report a pattern of motion perception abnormalities in ASD that includes sub

63 l visual area (MT) are crucially involved in motion perception, although it is not known exactly how

64 ew examines the acoustical basis of auditory motion perception and a wide range of psychophysical, el

65 ve demonstrated a close relationship between motion perception and activation of area V5, leading to

66 motion stimuli, which induce incorrect self-motion perception and eye movements, we explored the neu

67 cross space underlies many aspects of visual motion perception and has therefore received considerabl

68 is study provides impetus for future work in motion perception and its relationship to apraxia.

69 works that support functions, such as visual motion perception and language processing.

70 perform different functions, such as visual motion perception and language processing.

71 We capitalized on the strong link between motion perception and neural activity in the middle temp

72 of brain activity differed between states of motion perception and nonperception.

73 a stimulus, preserved reflexive saccades and motion perception and preserved autonomical and expressi

74 ing with a focus on the relationship between motion perception and smooth pursuit eye movements.

75 are the effect of feature-based attention on motion perception and smooth-pursuit eye movements in re

76 A critical step in self-motion perception and spatial awareness is the integrati

77 current static "snapshot" model of auditory motion perception and suggest a continuous process where

78 his is not predicted by any current model of motion perception and suggests that the visual cortex qu

79 nocellular-like sensory inputs necessary for motion perception and the computation of orienting movem

80 relevance of V5-V1 reentrant projections to motion perception and their plasticity.

81 ort the hypothesis that, at least for visual motion, perception and action are guided by inputs from

82 rast sensitivity), dynamic visual functions (motion perception), and VEPs were assessed repeatedly.

83 primary visual cortex (V1), a role in visual motion perception, and a suggested role in "blindsight."

84 ization of visual stimuli, poor saccades and motion perception, and poor emotional face perception wi

85 e is necessary for spatial orientation, self-motion perception, and postural control.

86 tween the integrity of the M pathway, visual motion perception, and reading ability.

87 and vestibular signals is important for self-motion perception, and such convergence has been observe

88 dition, cortical processes supporting global motion perception appear to be robust to fatigue.

89 time course of sensitivity changes in human motion perception are comparable to fly vision.

90 The neural underpinnings of biological motion perception are overlapping with brain regions inv

91 The brain systems that support motion perception are some of the most studied in the pr

92 to MT/V5 (the cortical area specialized for motion perception) are functionally distinct: the retino

93 that V5-V1 back projections, instrumental to motion perception, are functionally malleable.

94 e scanner, showing the involvement of action/motion perception areas.

95 sis, we investigated coherent and biological motion perception as well as coherent form perception in

96 We report a deficit in global motion perception at short viewing durations in ASC.

97 or areas are not only involved in biological motion perception, but also have causal relationships to

98 this information for three-dimensional (3D) motion perception, but here we consider a simpler strate

99 tical area MT plays a central role in visual motion perception, but models of this area have largely

100 the vestibular ocular reflex (VOR) and self-motion perception can be uncoupled both behaviourally an

101 Motion perception can ensue when the snapshots are diffe

102 These results reveal that motion perception cannot be isolated from position signa

103 To evaluate motion perception, contrast sensitivity for velocity dis

104 s of dazzle and hypercontrast, and a minimum motion perception (D(min)) and a motion-coherence task w

105 e an anatomical basis for the well-described motion perception deficits in congenital cataract patien

106 erception might depend on MT signals, global motion perception depends on mechanisms qualitatively di

107 Differences between groups on coherent motion perception did not remain significant when intell

108 patients frequently report visual problems, motion perception difficulties and abnormal depth percep

109 Global motion perception entails the ability to extract the cen

110 displayed widespread impairments in central motion perception even for non-form motion, for both slo

111 ectral sensitivity, contrast sensitivity and motion perception experiments confirmed that this patien

112 ports the idea that the role of MSTd in self-motion perception extends beyond optic flow processing.

113 Indeed, motion perception for typically suppressed stimuli was e

114 Behavioral tests of motion perception found no impairment and even better se

115 al and temporal processing underlying global motion perception has been elusive.

116 recent study of the asynchronous colour and motion perception has led to a new view of perceptual sy

117 Visual motion perception has long been associated with the dors

118 doxical interference effects of second-order motion perception imply that there are multiple forms of

119 We tested biological motion perception in a large group of unilateral stroke

120 of their actions fail to develop accurate 3D motion perception in a virtual reality environment, even

121 within and across modalities-to disambiguate motion perception in an ambiguous audiovisual display, w

122 ever, there was a substantial enhancement of motion perception in ASD: children with ASD exhibited a

123 These findings suggest that reduced global motion perception in autism is driven by an atypical res

124 inance-defined or Fourier) signals dominated motion perception in fish; edges or other features had l

125 o examine gamma oscillations during coherent motion perception in heavy cannabis users and controls.

126 ates of inhibitory function, we investigated motion perception in human children with ASD (n = 20) an

127 a strategy to deal with the complexities of motion perception in our natural environment.

128 psychophysical studies tend to characterize motion perception in terms of the statistical properties

129 eduction on their inputs; and (3) imply that motion perception in the cortex is consistent with ideas

130 V2 in motion processing, for contributing to motion perception in the dorsal pathway and/or for motio

131 ) to psychophysically characterize tuning of motion perception in the human visual system.

132 st that area V1 input contributes to complex motion perception in the mouse.

133 physiological studies suggest that chromatic motion perception in the primate brain may be performed

134 We measured motion perception in two groups of dyslexics, with and w

135 reported activations associated with tactile motion perception in visual motion area V5/hMT+, primary

136 eral aspects of visual perception, including motion perception, in patients with schizophrenia.

137 , and show multiple regions participating in motion perception, including V5, V3A, and a new area, th

138 fies how pathways for self-motion and object-motion perception interact, and (3) unifies the existing

139 rtical motion area MT (V5), and suggest that motion perception involves a dynamic interplay between M

140 Visual motion perception is critical to many animal behaviors,

141 l pattern of brain activation in response to motion perception is disrupted in DYT1 dystonia.

142 size limit of dmax, a breakdown of coherent motion perception is expected; however, in the presence

143 Visual motion perception is fundamental to many aspects of visu

144 findings provide support for the notion that motion perception is mediated by band-pass, spatial-freq

145 Perceptual studies suggest that visual motion perception is mediated by opponent mechanisms tha

146 idual stereo gratings so that the failure of motion perception is not due to inability to compute ste

147 3D motion perception is of central importance to daily life

148 Vestibular-motion perception is supported by a widespread white mat

149 , a cortical area whose importance to visual motion perception is well established.

150 15-27 years) performed a series of coherent motion perception judgements in which the amount of loca

151 Motion perception level may serve as a tool to assess th

152 suggests that recent models of binocular 3D motion perception may not reflect the strategies that hu

153 sized that dynamic visual processes, such as motion perception, may be more vulnerable to slowed cond

154 Although local motion perception might depend on MT signals, global mot

155 Similar to human motion perception, MT+ and V3A respond powerfully to all

156 t, that substantial neural loss specific for motion perception occurs during the processes of normal

157 imuli were measured to determine whether the motion perception of AMO monkeys was asymmetric, as pred

158 This enhanced motion perception of large high-contrast stimuli is cons

159 ly, the critical neural constraints limiting motion perception of large, high-contrast stimuli involv

160 atial suppression and, consequently, improve motion perception of large, moving patterns.

161 of suppressive center-surround mechanisms in motion perception of schizophrenic patients.

162 max) that can sustain perception of coherent motion; perception of relative speed; the amount of cohe

163 Recent computational models of biological motion perception operate on ambiguous two-dimensional r

164 ons are functionally critical for biological motion perception or are epiphenomenal remains unknown.

165 ural correlates of decision-making in global motion perception our findings suggest the global motion

166 ystonia were situated in proximity to normal motion perception pathways, suggesting that abnormalitie

167 g person rather than to represent biological motion perception per se.

168 s largely attributable to deficits of visual motion perception (R(2) adj = 0.57, P < 0.001).

169 how that a surprising perceptual error in 3D motion perception reflects the importance of prior proba

170 Visual motion perception relies on two opposing operations: int

171 atency remained significantly prolonged, and motion perception remained impaired throughout the 12-mo

172 Deficits in coherent motion perception seem to support this characterization,

173 hy volunteer subjects while they performed a motion perception task in which elliptical target trajec

174 ipants with autism took part in a biological motion perception task in which they classified observed

175 spects of task performance on the biological motion perception task were related to autism symptomato

176 In a series of forced-choice coherent motion perception tests, we parametrically varied a sing

177 er a more accurate and robust guide to human motion perception than any stimulus-based, statistical e

178 Within the hierarchy of motion perception, the dorsolateral middle superior temp

179 Motion perception therefore does not depend on a single

180 nly the dorsal visual stream is critical for motion perception, these novel findings implicate a more

181 ntion, and psychophysical measures of visual motion perception to all subjects.

182 xamined the impact of central vision loss on motion perception using random dot kinematograms to test

183 We found that global motion perception was impaired by prenatal exposure to a

184 Global motion perception was measured in one hundred and forty-

185 ese pathways normally mediate complex visual motion perception, we asked whether specific training in

186 regions in the ventral pathway to biological motion perception, we complement the behavioral findings

187 led for, but group differences in biological motion perception were more robust, remaining significan

188 TMS degraded visual motion perception when the evoked phosphene and the visu

189 through V1 is specialized for feature-based motion perception, whereas the retinocollicular pathway,

190 both groups exhibited similar impairments in motion perception with increasing stimulus size, reveali

191 tion principle and the breakdown of coherent motion perception with steps above an upper limit called

192 The hypothesis was that motion perception would be asymmetric with more sensitiv