ライフサイエンスコーパス: motor cortex

1 sterior parietal, somatosensory, visual, and motor cortex.

2 been on the connectivity and activity of the motor cortex.

3 movement responses within shared networks of motor cortex.

4 ere altered during infusion of ZD7288 within motor cortex.

5 a brain-machine interface using neurons from motor cortex.

6 neurons in the spinal cord, brain stem, and motor cortex.

7 significant disconnection of dMSNs from the motor cortex.

8 and physiological inhibition in the primary motor cortex.

9 nts triggered by gamma tACS over the primary motor cortex.

10 ing to speech evokes neural responses in the motor cortex.

11 by neural activation patterns within primary motor cortex.

12 ic representations of speech articulators in motor cortex.

13 ween head and forelimb areas of peri-infarct motor cortex.

14 oop were connected through the cerebellum or motor cortex.

15 transcription factors persist in the mature motor cortex.

16 stic motor deficits and neuronal loss in the motor cortex.

17 through connectivity with the cerebellum or motor cortex.

18 the superior and inferior regions of ventral motor cortex.

19 in both layer 5A and layer 5B of the mature motor cortex.

20 ion neurons (SPNs) of the mouse auditory and motor cortex.

21 n depends critically on the integrity of the motor cortex.

22 cerebello-thalamo-cortical loop through the motor cortex.

23 n both anaesthetized and awake mouse sensori-motor cortex.

24 fter focal cortical stroke in mouse forelimb motor cortex.

25 ortant role for sensory input in controlling motor cortex.

26 polysynaptic inhibition from the STN to the motor cortex.

27 ve locations of head and hand regions in the motor cortex.

28 idual layer V pyramidal neurons in the mouse motor cortex.

29 d low gamma desynchronization in the primary motor cortex.

30 letion resulted in structural changes in the motor cortex.

31 their corresponding articulatory gestures in motor cortex.

32 ocalized to ALM or widely distributed within motor cortex.

33 d to the cerebellum before a test pulse over motor cortex.

34 at the ultrastructural level in peri-infarct motor cortex.

35 -30 Hz) coupled to electrodes C3/C4 close to motor cortex.

36 sterior relative to ALM, including vibrissal motor cortex.

37 vation has identified movements that require motor cortex.

38 lidocortical projections from the GPe to the motor cortex.

39 EEG was recorded over leg and hand area of motor cortex.

40 ions modulates GABAA inhibition in the human motor cortex.

41 e local field potential of non-human primate motor cortex.

42 nges in cortical circuits in the ipsilateral motor cortex.

43 BDNF in the nigrostriatal system and primary motor cortex.

44 ile previous observations on the function of motor cortex.

45 lection) altered neural responses in primary motor cortex ~65 ms after the limb disturbance, and then

46 In macaque motor cortex, a large sample of pyramidal neurons were n

47 These results suggest that motor cortex acquires skillful control by leveraging bot

48 the axonal arbors of multiple neurons in the motor cortex across a single mouse brain.

49 However, the reduced motor cortex activity during more automated behaviors su

50 Here we interface leg motor cortex activity with epidural electrical stimulati

51 We provide evidence that the motor cortex acts according to this principle: cortical

52 We performed optogenetic mapping of motor cortex after a cervical SCI that interrupts most c

53 connectivity between the putamen and primary motor cortex after levodopa intake during movement suppr

54 underlies functional vicariation in residual motor cortex after motor cortical infarcts.SIGNIFICANCE

55 Neurons in the mouse anterior lateral motor cortex (ALM) have been shown to have selective per

56 Activity in the mouse anterior lateral motor cortex (ALM) instructs directional movements, ofte

57 This bias is gradually consolidated in RA, a motor cortex analogue downstream of LMAN.

58 sists of subdural electrodes placed over the motor cortex and a transmitter placed subcutaneously in

59 We hypothesized that pairing stimulation of motor cortex and cervical spinal cord would strengthen m

60 sitive fibers were observed in the secondary motor cortex and cingulate cortex, and sparse hrGFP-posi

61 increased c-Fos expression in the secondary motor cortex and cingulate cortex, but decreased c-Fos e

62 connectivity increased between ipsilesional motor cortex and contralesional premotor cortex after th

63 organization, inducing an expansion of trunk motor cortex and forepaw sensory cortex into the deaffer

64 ticospinal tract (CST) that originate in the motor cortex and innervate the spinal cord.

65 ity increases only in the left supplementary motor cortex and left postcentral gyrus/precuneus after

66 a significant partial coherence between left motor cortex and lip movements and this partial coherenc

67 ty between corticospinal (CS) neurons in the motor cortex and muscles in mice.

68 of signals along different pathways between motor cortex and muscles.

69 nce of excitatory/inhibitory activity in the motor cortex and potentially has implications for disord

70 raphe nuclei (phase II), followed by primary motor cortex and precerebellar nuclei (phase III) and fi

71 compared with controls in the right primary motor cortex and right caudate.

72 Repeated pairing of motor cortex and spinal cord stimulation caused lasting

73 hen tested the effect of repeated pairing of motor cortex and spinal stimulation.

74 Months-long recordings from motor cortex and striatum made and analyzed with our sys

75 of beta-phase to gamma-amplitude in primary motor cortex and that deep brain stimulation facilitates

76 We placed epidural electrodes over motor cortex and the dorsal cervical spinal cord in rats

77 normal interaction between pre-supplementary motor cortex and the inferior frontal gyrus was absent i

78 N channels contribute to the organization of motor cortex and to skilled motor behaviour during a for

79 N channels contribute to the organization of motor cortex and to skilled motor behaviour during a for

80 microelectrode array in the leg area of the motor cortex and with a spinal cord stimulation system c

81 psilateral activity suppresses contralateral motor cortex and, accordingly, that inhibiting ipsilater

82 icroelectrode arrays in the hand area of his motor cortex, and 4 months and 9 months later received a

83 al controls retain cellular integrity in the motor cortex, and CA1 pyramidal neurons show abnormaliti

84 neurons and astrocytes in somatosensory and motor cortex, and hippocampus, 8 weeks post-TBI.

85 m an oversynchronization of afferents to the motor cortex, and that these symptoms are treatable usin

86 sed metabolism in the preoptic area, primary motor cortex, and the amygdala, and decreased metabolism

87 Furthermore, the BF and the motor cortex-another source of movement-related activity

88 The cerebellum and the motor cortex appear to be critical for our ability to le

89 s suggest that neural population dynamics in motor cortex are invariant to the number of recorded neu

90 ay epoch, object location was represented in motor cortex areas that are medial and posterior relativ

91 Recent theories of limb control emphasize motor cortex as a dynamical system, with planning settin

92 is mechanism supports a viewpoint of primary motor cortex as a site of dynamic integration for behavi

93 has been made using rodents to establish the motor cortex as an adaptive structure that supports moto

94 on of complex multiple forelimb movements in motor cortex as assessed by intracortical microstimulati

95 om supplementary motor area and left primary motor cortex as the source of signal modification in aud

96 o the position of the larynx area within the motor cortex, as well as its connections with the phonat

97 y 30 min over the hand representation of the motor cortex at an interstimulus interval mimicking the

98 Motor cortex, basal ganglia (BG), and thalamus are arran

99 A motor cortex-based brain-computer interface (BCI) create

100 Changes in neural activity occur in the motor cortex before movement, but the nature and purpose

101 How the motor cortex behaves during this state remains unknown.

102 idal and gigantopyramidal neurons in primary motor cortex between 11 carnivore and 9 primate species.

103 iated with a narrowband gamma oscillation in motor cortex between 60 and 90 Hz, a similar, though wea

104 was placed over contralateral or ipsilateral motor cortex, bihemispheric stimulation yielded substant

105 are strongly and consistently represented in motor cortex but can be accounted for to improve BCI per

106 rebello-thalamo-cortical circuit through the motor cortex (but not the cerebellum).

107 not induce abnormal LFP activity in sensori-motor cortex, but did label astrocytes, and may thus be

108 accompanied by widespread changes within the motor cortex, but it is unknown whether these changes ar

109 MSNs show an increased connectivity with the motor cortex, but only after a severe dopaminergic lesio

110 We demonstrate that the motor cortex can reestablish output to the limbs longitu

111 of the iCSP from the contralesional primary motor cortex (cM1) hand/arm area to spinal levels C5-T1

112 troduce a novel method to measure cerebellar-motor cortex connectivity during movement preparation.

113 ontext of learning, modulation of cerebellar-motor cortex connectivity is somatotopically specific to

114 ve learning with the hand affects cerebellar-motor cortex connectivity, not only for the trained hand

115 l current stimulation of the rat (all males) motor cortex consisting of a continuous subthreshold sin

116 cortex, dorsal premotor cortex, and primary motor cortex contralateral to the acting hand.

117 creatine (Cr) levels were measured from the motor cortex contralateral to the greater functional def

118 al circuits, probably from somatosensory and motor cortex, contribute to sensory gating in the iS1 du

119 al circuits, probably from somatosensory and motor cortex, contribute to sensory gating in the iS1 du

120 ing that a tonic inhibition state in primary motor cortex could prevent the affordance from provoking

121 We propose that greater GABA capacity in the motor cortex counteracts an intrinsically more excitable

122 nnectivity between the contralateral STN and motor cortex decreased.

123 gical inhibition of HCN channels in forelimb motor cortex decreases reaching accuracy and increases a

124 eparation evokes substantial activity in the motor cortex despite no apparent movement.

125 es a decrease in hyperdirect inputs from the motor cortex directly to the STN and that rescuing this

126 l population responses revealed that the rat motor cortex displays similar modulation as other mammal

127 Motor cortex does not contain articulatory representatio

128 es of layer 5 pyramidal neurons in the mouse motor cortex during development and motor learning.

129 the population activity patterns produced by motor cortex during different behaviors determine the se

130 that can emulate changes seen in the primary motor cortex during learning.

131 The structure of responses in motor cortex during listening was organized along acoust

132 ce of the behavioral procedure to engage the motor cortex during motor control studies, gait rehabili

133 ere we show that tonic disinhibition of left motor cortex during prism adaptation enhances consolidat

134 his gap, we examined population responses in motor cortex during reach preparation and movement.

135 ow that the encoding of hindlimb features in motor cortex dynamics is comparable in rats and cats.

136 Cortical stimulations in motor cortex elicited muscle contractions.

137 Magnetothermal genetic stimulation in the motor cortex evoked ambulation, deep brain stimulation i

138 l stimulation produced large augmentation in motor cortex evoked potentials if they were timed to con

139 Motor cortex-evoked muscle potentials showed parallel ch

140 release into the dorsal striatum and speech motor cortex exerts direct modulation of neuronal activi

141 We found that neurons in primary motor cortex exhibited a change in the amplitude of thei

142 During overground locomotion, the motor cortex exhibited consistent neuronal population re

143 The motor cortex exhibits structural changes in response to

144 ode either over contralateral or ipsilateral motor cortex facilitated motor learning nearly twice as

145 elated words (e.g., "write"), but in ventral motor cortex for face-related words ("talk").

146 ulation therapies in addition to the primary motor cortex for patients who do not respond adequately

147 NA-seq analysis of the BA4 (Brodmann area 4) motor cortex from seven human HD brains and seven contro

148 ferior olive, including projections from the motor cortex, generate rapid excitatory potentials follo

149 stimulation of a control region-the primary motor cortex-had no effect on adaptive control.

150 forelimb are generated from the same area of motor cortex have remained elusive.

151 forelimb are generated from the same area of motor cortex have remained elusive.

152 can also be readily detected in the sensory motor cortex, hippocampus and cerebellum of post-mortem

153 metabolite profiles were generated from the motor cortex, hippocampus, cerebellum and liver tissue o

154 PFC hypoconnectivity and motor cortex hyperconnectivity correlated in patients, s

155 o afferent inputs from the pre-supplementary motor cortex; (ii) atomoxetine can enhance downstream mo

156 DCS) over both contralateral and ipsilateral motor cortex in a between-group design during 4 d of uni

157 array to record multiunit activity from the motor cortex in a study participant with quadriplegia fr

158 We performed optogenetic mapping of motor cortex in channelrhodopsin-2 expressing mice to as

159 etal cortical connections with the laryngeal motor cortex in humans compared with nonhuman primates t

160 Conclusion NAA/Cr levels decreased in the motor cortex in patients with CSM 6 months after success

161 intracerebral electrodes sampling the human motor cortex in pharmacoresistant epileptic patients, we

162 The motor cortex in the brain tracks lip movements to help w

163 The role of the primary motor cortex in the formation of a comprehensive network

164 Despite the facultative role of the motor cortex in the production of locomotion in rats, th

165 by previous noninvasive TMS studies of human motor cortex indicating a reduction of corticospinal exc

166 Yet, when and how motor cortex influences muscle activity during movement

167 matosensory cortex, premotor cortex, primary motor cortex, insula and posterior parietal cortex, as w

168 w brain activation in a network of cortical (motor cortex, insula, cingulate, amygdala) and sub-corti

169 MENT Connectivity between the cerebellum and motor cortex is a critical pathway for the integrity of

170 The motor cortex is far from a stable conduit for motor comm

171 able asymmetries in arm movements before the motor cortex is functionally linked to the spinal cord,

172 d unveiled time-grained evoked activities of motor cortex layer V neurons that show high-frequency sp

173 Motor cortex lesions prior to training, however, rendere

174 The laryngeal motor cortex (LMC) is essential for the production of le

175 rete premotor signals that drive the primary motor cortex M1 to reflect the movement of the grasping

176 to cause dyskinesia, alterations in primary motor cortex (M1) activity are also prominent during dys

177 Here we asked how both primary motor cortex (M1) and PMd represented reach direction du

178 Here, we report that the primary motor cortex (M1) and primary somatosensory cortex (S1),

179 Different modules within PPC and motor cortex (M1) appear to control various motor behavi

180 e movement-related activation of the primary motor cortex (M1) are thought to be a major contributor

181 The traditional classification of primary motor cortex (M1) as an agranular area has been challeng

182 ranial magnetic stimulation (TMS) to primary motor cortex (M1) at specific intervals to induce plasti

183 These connections with motor cortex (M1) can be estimated as cerebellar inhibit

184 The proximal representation in the primary motor cortex (M1) controls the arm for reaching, while t

185 emotor cortex onto the contralateral primary motor cortex (M1) during the preparation of a complex bi

186 l interpretations of the function of primary motor cortex (M1) emphasize its lack of the granular lay

187 d communicates those programs to the primary motor cortex (M1) for execution.

188 Sensory inputs reaching the primary motor cortex (M1) from the somatosensory cortex (S1) are

189 ting current stimulation (tACS) over primary motor cortex (M1) has opposite effects on motor performa

190 current stimulation (tDCS) over the primary motor cortex (M1) has resulted in improved performance i

191 ion (TDCS) of the cerebellum and the primary motor cortex (M1) have been found to improve visuomotor

192 ence point to a critical role of the primary motor cortex (M1) in consolidation.

193 The arm region in the primary motor cortex (M1) is assumed to control reaching, while

194 The primary motor cortex (M1) is highly influenced by premotor/motor

195 ue ventral premotor cortex (PMv) and primary motor cortex (M1) is modulated by the observation of ano

196 The primary motor cortex (M1) is strongly influenced by several fron

197 anatomical networks that include the primary motor cortex (M1) of both hemispheres.

198 yzed neuronal activity recorded from primary motor cortex (M1) of monkeys performing a 3D arm posture

199 ents is by modulating or shaping the primary motor cortex (M1) outputs to hand muscles.

200 nsity between 0.5 and 2.0 mA on left primary motor cortex (M1) plasticity, as well as the impact of i

201 we analyze data from invasive human primary motor cortex (M1) recordings from patients with Parkinso

202 ly in the striatum and all layers of primary motor cortex (M1) through a muscarinic-receptor mediated

203 tion (TMS) over the hand area of the primary motor cortex (M1) when human participants (50% females)

204 tion (TMS) over the hand area of the primary motor cortex (M1) when humans tracked with the eyes a vi

205 nnectivity pattern of the stimulated primary motor cortex (M1), without changing overall local activi

206 In our case, the target was the primary motor cortex (M1).

207 scriminate four different tasks in the human motor cortex (M1).

208 matched domains in premotor cortex (PMC) and motor cortex (M1).

209 complete body representation in the primary motor cortex (M1).

210 e dominant oscillatory activity in the human motor cortex (M1).

211 from primary visual cortex (V1) and primary motor cortex (M1).

212 left or right DLPFC or left or right primary motor cortex (M1).

213 to reversibly deactivate selected regions of motor cortex (M1).

214 processes in the cerebellum (CB) and primary motor cortex (M1).

215 ry), cortical function (greater ipsilesional motor cortex [M1] activation), and cortical connectivity

216 rodents, the medial aspect of the secondary motor cortex (M2) is known by other names, including med

217 Two-photon calcium imaging of secondary motor cortex (M2) revealed different ensemble activity s

218 extends rostrally to the posterior secondary motor cortex (M2), suggesting a functional corticocortic

219 vo tractography, in addition to the cerebral motor cortex, major portions of CPC streamlines leave th

220 This manifold-based view of motor cortex may lead to a better understanding of how t

221 Alternatively, the ipsilateral motor cortex may play an active role in the control and/

222 However, until now, no one has studied the motor cortex (MC) and its possible role in classical eye

223 connections between separate neural sites in motor cortex (MC).

224 ld potential (LFP) activity with activity in motor cortex (MCx) and medial prefrontal cortex (mPFC),

225 substantia nigra pars reticulata (SNpr) and motor cortex (MCx) in the hemiparkinsonian rat during tr

226 hine produced increased ventral striatum and motor cortex metabolism in females, and increased ventra

227 orsal premotor cortex (PMd, area 6), primary motor cortex (MI, area 4), and posterior parietal cortex

228 y kinematics, muscle synergies, and hindlimb motor cortex modulation in freely moving rats performing

229 However, the extent of motor cortex modulations appears linked to the degree of

230 a motivation and reward area located in the motor cortex more strongly than did adults in response t

231 t influence of movement target separation on motor cortex neural activity, mediated by lateral intera

232 Motor cortex neural patterns during listening were subst

233 cospinal axons originating in caudal primary motor cortex ("new M1") and area 3a make monosynaptic co

234 caused by microelectrode implantation in the motor cortex of healthy rats compared to non-implanted c

235 distinct classes of cortical neurons in the motor cortex of hSOD1(G93A) mice of both sexes and found

236 both the brains of RS model mice and in the motor cortex of human RS autopsy samples.

237 Delivery of the Cal-Light components to the motor cortex of mice by viral vectors labels a subset of

238 e longitudinal metabolite alterations in the motor cortex of patients with cervical spondylotic myelo

239 The influence of motor cortex on muscles during different behaviors is in

240 d rats by either conditional knockout in the motor cortex or monoclonal antibody infusion resulted in

241 The motor cortex orchestrates simple to complex motor behavi

242 ling of the number of neurons in the primary motor cortex over the brainstem and spinal cord motor ne

243 ea and functional anticorrelation to primary motor cortex (p < 0.001).

244 s associated with dynamic changes in primary motor cortex physiology, to date, there are no published

245 An understanding of motor cortex plasticity has been advanced greatly using

246 functional MR imaging signal in the primary motor cortex (PMC), as false-negative blood oxygen level

247 We found that the activation of hindlimb motor cortex preceded gait initiation.

248 Synaptic projections from the motor cortex preferentially target the principal olivary

249 prepared to swallow showed increases in the motor cortex, prefrontal cortices, posterior parietal co

250 Thus, in rat motor cortex, pyramidal cells have long duration action

251 , we show that a posterior part of secondary motor cortex receives corticocortical axons from the ros

252 emonstrate that pretarget neural activity in motor cortex reflects the monkey's internal model of vis

253 edication (OFF) showed hypoactivation of the motor cortex relative to healthy controls, even when con

254 ent for existing models that seek to explain motor cortex responses.

255 immediately after lesions in the adult mouse motor cortex restored damaged cortical pathways.

256 immediately after lesions in the adult mouse motor cortex restored damaged motor cortical pathways.

257 tral portion of the precentral gyrus (speech motor cortex) resulted in the manipulation of speech tim

258 lesions of the caudal forelimb region of the motor cortex, resulting in nearly complete loss of grasp

259 song learning in the avian analog of primary motor cortex (robust nucleus of the arcopallium, RA) in

260 +/- SEM, Sham = 92.29 +/- 5.56%, P < 0.01), motor cortex showed only a significant increase, in NPY

261 In rat motor cortex, SMI32-postive pyramidal neurons expressing

262 The P25/N33 receives inputs from the motor cortex, so we also examined the contribution of di

263 KEY POINTS: Pairing motor cortex stimulation and spinal cord epidural stimul

264 We recorded myogenic MEPs after transcranial motor cortex stimulation in 6 lambs aged 1-2 days.

265 to coincide with the descending volley from motor cortex stimulation, MEPs were more than doubled.

266 us pallidus (GPi), external globus pallidus, motor cortex, thalamus, or cerebellum.

267 diated by synaptic plasticity in a region of motor cortex that, before lesions, is not essential for

268 Furthermore, the primary motor cortex, the ventral lateral premotor cortex, and t

269 of each hemisphere are involved: the primary motor cortex, the ventral lateral premotor cortex, the s

270 Motor cortex therefore represents sensory, motor, and ou

271 hen used in layers I-IV of slices of the rat motor cortex to determine the percent contribution of HS

272 medial striatum and sensorimotor inputs from motor cortex to dorsolateral striatum, we show that asso

273 euronal population responses in the hindlimb motor cortex to hindlimb kinematics and hindlimb muscle

274 anscranial magnetic stimulation over primary motor cortex to measure long-interval cortical inhibitio

275 However, the importance of the motor cortex to regain locomotion after neurological dis

276 ncreased intracortical inhibition in primary motor cortex under high working memory load.

277 yer V pyramidal neurons in the mouse primary motor cortex using two-photon microscopy.

278 expression in sections from macaque and rat motor cortex, using two different antibodies (NeuroMab,

279 to a superficial cortical vein overlying the motor cortex via catheter angiography and demonstrate ne

280 The primary vibrissae motor cortex (vM1) is responsible for generating whiskin

281 sensory cortex, vS1, together with vibrissal motor cortex, vM1 (a frontal cortex target of vS1), whil

282 In this study, using rat motor cortex, we found that tACS effects are highly vari

283 Gigantopyramidal neurons in the motor cortex were extremely large, confirming the observ

284 MEPs evoked from motor cortex were robustly augmented with spinal epidura

285 nd LICI in the frontal cortex-but not in the motor cortex-were indicators of remission of suicidal id

286 igated how this isolation is achieved in the motor cortex when macaques received visual feedback sign

287 r portion corresponding to the region of the motor cortex where the mouth and orofacial movements are

288 ) is extensively deposited in progressive MS motor cortex, where regulation of fibrinolysis appears p

289 iffering PlexA1 expression in layer 5 of the motor cortex, which is strong in wild-type mice but weak

290 For this modulation they use their motor cortex, which, via its corticobulbar fibers, has d

291 Here we imaged activity across motor cortex while mice performed a whisker-based object

292 we recorded multi-scale neural activity from motor cortex while three macaques performed a novel neur

293 dapting internal model of visuomotor gain in motor cortex while two macaques performed a reaching tas

294 ndicated that short-latency pathways linking motor cortex with spinal motor neurons are selectively a

295 al magnetic stimulation over the ipsilateral motor cortex with the coil in the posterior-anterior (PA

296 structural connectivity of the supplementary motor cortex with the sensorimotor putamen predicted mor

297 dorsolateral prefrontal cortex and the left motor cortex, with the latter acting as a control site.

298 (CST) neurons in the subdivision of primary motor cortex within the central sulcus ("new M1") and ar

299 icant functional disconnection of dMSNs from motor cortex without changes in iMSN connectivity.

300 tigate the function of mouse whisker primary motor cortex (wM1), a frontal region defined by dense in