ライフサイエンスコーパス: motor disorder

1 ugh impaired cholinergic pathway, leading to motor disorder.

2 ation of conversion disorder or dissociative motor disorder.

3 d different conclusions on the nature of the motor disorder.

4 understanding of the pathophysiology of the motor disorder.

5 r HIV-associated dementia or minor cognitive-motor disorder.

6 apeutic strategies for CBG-related vocal and motor disorders.

7 und to aid and assist recovery in functional motor disorders.

8 and aid in development of drugs for gastric motor disorders.

9 ommonly used to relief spasticity related to motor disorders.

10 s in the treatment of esophageal sensory and motor disorders.

11 ther studying, monitoring, and even treating motor disorders.

12 ory dysfunction as a component of esophageal motor disorders.

13 edical, and surgical therapies for childhood motor disorders.

14 regions during normal motor function and in motor disorders.

15 incidence of speech/language, scholastic, or motor disorders (829, 187, and 285 instances, respective

16 n on voluntary motor output in subjects with motor disorders affected by spasticity.

17 Parkinson's disease (PD) is the most common motor disorder affecting the elderly.

18 rget for enhancing voluntary motor output in motor disorders affecting the corticospinal tract.

19 Although defined as a motor disorder, ALS can arise concurrently with frontote

20 port widens the phenotype of dopa-responsive motor disorders and the range of young children with pri

21 insight into the pathogenesis of esophageal motor disorders and treatment of gastroesophageal reflux

22 chological impairment, 5 had minor cognitive/motor disorder, and 1 was demented.

23 ppear in the absence of any other sensory or motor disorder, and is sufficient to cause a literacy im

24 remains the most investigated and understood motor disorder, and the year's research focused heavily

25 vents that serve as signatures of esophageal motor disorders, and esophageal manometry retains its po

26 Importance: Speech/language, scholastic, and motor disorders are common in children.

27 While half of the mutations that cause motor disorders are positioned to affect enzyme activity

28 an their littermates and showed very obvious motor disorders, associated with a dramatic loss of larg

29 the vague and variable presentations of this motor disorder become appreciated.

30 ot only in patients with levodopa-responsive motor disorders, but also in patients with developmental

31 Rodent models of DYT1 dystonia, a motor disorder caused by a single gene mutation, demonst

32 on developed from 5 mo of age, a progressive motor disorder characterized at onset by impaired rota-r

33 mutations in humans lead to hyperekplexia, a motor disorder characterized by startle responses, the z

34 Although PD is primarily considered a motor disorder, cognitive impairment is often present at

35 More recent studies suggest instead that motor disorders derive from abnormal firing patterns, an

36 The childhood-onset motor disorder DYT6 dystonia is caused by loss-of-functi

37 ular interactors that probably bear on human motor disorders, especially those affecting speech.

38 Patients with functional motor disorder (FMD) including weakness and paralysis ar

39 nderlying various acquired and developmental motor disorders have been described, as well as the effe

40 This work reviews supranuclear ocular motor disorders, highlighting new data published during

41 r conversion disorder (DSM-IV), dissociative motor disorder (ICD-10) and 'psychogenic' paralysis.

42 known as congenital nystagmus, is an ocular motor disorder in humans characterized by spontaneous ey

43 entre international prospective study of the motor disorder in the largest cohort of patients studied

44 cracker" esophagus in two, and a nonspecific motor disorder in two patients.

45 dging the presence of additional sensory and motor disorders in certain individuals.

46 ciated with speech/language, scholastic, and motor disorders in offspring up to early adolescence.

47 Children with autism exhibit a host of motor disorders including poor coordination, poor tool u

48 deficiency virus type 1-associated cognitive-motor disorder, including the AIDS dementia complex, is

49 Mouse GAT1 (mGAT1) KO mice exhibit motor disorders, including gait abnormality, constant 25

50 ent of achalasia, the best-understood distal motor disorder, is resolving as the benefits and disadva

51 met diagnostic criteria for minor cognitive/motor disorder (MCMD) or HIV-associated dementia (HAD).

52 ile Parkinson's disease (PD) is considered a motor disorder, motor signs of PD can be exacerbated by

53 he pathogenesis and management of esophageal motor disorders occurred over the past year.

54 nfidence interval = 1.9-16.8; P = 0.002) and motor disorder (odds ratio = 3.3; confidence interval =

55 re provided, along with a summary of how the motor disorder of LND relates to current understanding o

56 The motor disorders of CP are frequently accompanied by impa

57 muscle in health, as well as in sensory and motor disorders of the esophagus.

58 ophagus in normal subjects and patients with motor disorders of the esophagus.

59 The concept that motor disorders of the gallbladder, cystic duct and sphi

60 ical relevance, because it helps explain why motor disorders of the LES and fundus frequently occur t

61 s a target antigen in a human paraneoplastic motor disorder [paraneoplastic opsoclonus-myoclonus atax

62 ) has expanded as an effective treatment for motor disorders, providing a valuable opportunity for in

63 t the mechanisms underlying dopamine-related motor disorders remain under debate.

64 (2.38) for speech/language, scholastic, and motor disorders, respectively.

65 development of therapeutics for upper airway motor disorders such as obstructive sleep apnoea.

66 of particular interest as it is elevated in motor disorders such as Parkinson's disease and modulate

67 vation and movement, and is dysfunctional in motor disorders such as Parkinson's disease.

68 The pathogenesis of complex ocular motor disorders, such as paradoxical pupillary constrict

69 ssociated dementia and related cognitive and motor disorders that affect 20 to 30% of treatment-naive

70 , epilepsy, and pyramidal and extrapyramidal motor disorders) that are primarily attributed to defici

71 For scholastic and motor disorders, there were no differences between offsp

72 An esophageal motor disorder was diagnosed only by 24-hour ambulatory

73 s-of-function mutations in Na(v)1.6 underlie motor disorders, with homozygous-null mutations causing