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1 re, locomotion, and susceptibility to limbic motor seizures.
2 physiological, and behavioral adaptations to motor seizures.
3 which protects against focally evoked limbic motor seizures.
4 one of the SwHi rats, had spontaneous limbic motor seizures 5 weeks following pilocarpine-induced sta
5 ypical group (4 cases presenting with simple motor seizures and a tendency for more frequent generali
6 contralateral hemisphere following the first motor seizure, and the pattern of its expression reflect
7 nly a few seizures, 1 had more than 30 focal motor seizures, and 1 had 4 witnessed generalized tonic-
8 e neurological disorder resulting in ataxia, motor seizures, and behavioral absence seizures resembli
9 cognised because they usually present as non-motor seizures, and can overlap with other symptoms of t
11 (200 pmol) into STN protected against limbic motor seizures evoked either by intravenous bicuculline
12 substantia nigra, protected rats from limbic motor seizures evoked focally from area tempestas, an ep
13 ttent EEGs with behavioural observations for motor seizures failed to demonstrate spontaneous seizure
14 rats exhibited a decreased latency to limbic motor seizures following acute pilocarpine administratio
17 directly related to the suppression of tonic motor seizures in the electroshock model of epilepsy.
21 d ECS treatment caused progressively shorter motor seizures (tolerance) in both rats and wild-type mi
24 ons of the percentage change in frequency of motor seizures were done with a Mann-Whitney U test.
25 ilepticus (n = 25), including 15 with subtle motor seizures, were more likely to die than those with
26 e day prior to his admission, he had a focal motor seizure with rotation of the head and eyes to the
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