ライフサイエンスコーパス: mouse

1 e by breeding with a CaMKII-ERT2-Cre (FIGSKO mouse).

2 ously from five brain structures in an awake mouse.

3 ssive-compulsive disorder in human, dog, and mouse.

4 late the major events of meiotic prophase in mouse.

5 itor states that have only been described in mouse.

6 which is mutated in the BXH2 IRF8-deficient mouse.

7 he level of behavioural conditioning in each mouse.

8 transcriptional profiling of LmnaH222P/H222P mouse, a small animal model of LMNA cardiomyopathy, sugg

9 s) of these and other agonists in adult-type mouse AChRs having a mutation(s) at the transmitter-bind

10 nct roles in two sequential priming steps in mouse adrenal chromaffin cells.

11 nduced cardiac progenitor cells (iCPCs) from mouse adult fibroblasts using forced expression of Mesp1

12 elial barrier function in 16HBE cells and in mouse airways.

13 Similar changes are present in a mouse alpha-synucleinopathy model and in postmortem brai

14 Ube3a isoform 2 is conserved between mouse and human and known to play key roles in neuronal

15 In both mouse and human B cells, BCR ligands that deliver a TLR9

16 Here, we develop skin grafts from mouse and human epidermal progenitors that were engineer

17 traportal islet transplantation models using mouse and human islets, we demonstrated that alpha-1 ant

18 of rat neurons with validation in additional mouse and human neurons and multiple learning tests from

19 to initiate the in vitro differentiation of mouse and human PSCs into cardiac progenitor cells, foll

20 bservable phenotypic differences between the mouse and human skin and broadly informs on the prevaili

21 c hearing loss genes have been identified in mouse and human, but we remain ignorant of the full exte

22 primary neuronal culture, in brain slices of mouse and monkey, and in mouse brain in vivo.

23 nimal models of cholera pathogenesis (infant mouse and rabbit models).

24 Anti-hypoallergenic Cyp c 1 mutant mouse and rabbit sera were tested for their ability to i

25 clc loxP mouse with a thy1-cre mouse (NEGSKO mouse) and at a later age by breeding with a CaMKII-ERT2

26 ER-localized THBS1 is cytoprotective to rat, mouse, and human beta-cells exposed to cytokines or thap

27 er, MSA effectively inhibited the sprouts of mouse aortic rings and neoangiogenesis in chick embryo c

28 We used DAT-KO mouse as model for DTDS to explore the potential utility

29 ein injection (150 mug of each conjugate per mouse) at 1, 7 and 13 days post-injection.

30 the transcriptomes and proteomes of primary mouse B cells from wild-type and STAT6-deficient mice cu

31 Mouse BALF and human BALF had a strong positive correlat

32 Our data suggests that mouse behavior and EEG recordings are not sensitive to d

33 Spry2 deletion in the adult mouse beta-cell caused hyperglycemia and hypoinsulinemia

34 issues of human were calculated based on the mouse biodistribution.

35 ed in ABIN1[D485N] mice transplanted with WT mouse bone marrow.

36 etailed architecture of dendritic spines, in mouse brain circuitry.

37 of tricarboxylic acid cycle intermediates in mouse brain during ischemic stroke.

38 Primary SJL mouse brain endothelial cells (a target of MAV-1 in vivo

39 in brain slices of mouse and monkey, and in mouse brain in vivo.

40 precast gelatin-based molds in which a whole mouse brain is embedded, flash frozen, and cryosectioned

41 Acute knockdown of Myt1l in the developing mouse brain mimicked a Notch gain-of-function phenotype,

42 he present study, we established an in vitro mouse brain slice preparation that retains connectivity

43 noncoding, and intergenic RNAs in the mature mouse brain with RNA-Seq and validation with independent

44 ession remained unaltered in adult WT and KO mouse brain, SC, and kidneys.

45 ion of RTN1 and RTN3 is tightly regulated in mouse brains.

46 strema and nucleus tractus solitarius of the mouse brainstem.

47 METHODS AND In mouse carotid arteries with established intimal lesions

48 uced loss of function mutations in human and mouse cell lines measured by FLAER labelling.

49 e architecture, function, and development of mouse central visual pathways.

50 So far, the role of mouse Ces1/Ces1g deficiency in atherosclerosis is not el

51 gaging in social behaviors toward a juvenile mouse, compared with time spent in nonsocial behavior or

52 in basolateral plasma membrane exosomes from mouse cortical collecting duct cells.

53 racterization showed these cells, like their mouse counterparts, did not express PR domain containing

54 nistered with vehicle control or UFP (40 mug/mouse/day) for 3 days a week.

55 We generated a novel mutant mouse (Dclre1c(leaky)) that develops a LS phenotype.

56 The first cellular differentiation event in mouse development leads to the formation of the blastocy

57 Here, we showed that during mouse development, the histone-modifying enzyme histone

58 The Emu-TCL1 transgenic mouse develops a form of leukemia that is similar to the

59 CT-based indentation, the elastic modulus of mouse diaphragm was measured from changes in diaphragm t

60 Here, using the regenerating mouse digit tip as a mammalian model, we demonstrate tha

61 clei, dendrites and near dendritic spines of mouse dorsal hippocampal CA1 neurons.

62 Loss of Sin3a in mouse early foregut endoderm led to a specific and profo

63 for anterior-posterior axis formation of the mouse embryo and was shown to promote posterior neuroect

64 In the mouse embryo, global epigenetic changes occur during zyg

65 nd resolution of tetrads and rosettes in the mouse embryo, possibly in part by spatially restricting

66 Myc (c-Myc) or Oct4 during reprogramming of mouse embryonic fibroblasts into iPSCs.

67 ity, we established synchronized cultures of mouse embryonic stem cells as they exit the ground state

68 roduce a targeted autosome loss in aneuploid mouse embryonic stem cells with an extra human chromosom

69 and E13.5 Osr2(RFP/+) and Osr2(RFP/-) mutant mouse embryos and performed whole transcriptome RNA sequ

70 rogeneity was also evident in human ESCs and mouse embryos in vivo.

71 e performed transcriptome profiling on whole mouse embryos.

72 an epigenetic predictor of age in mice, the mouse epigenetic clock.

73 In VDR knockout mouse epithelial cells (KO), 1,25(OH)2D3 increased CYP24

74 al ischemia model, we developed a transgenic mouse expressing human CD39 (hCD39).

75 Sox6-null mouse fetuses present misshapen and nucleated erythrocyt

76 rican-green monkey kidney epithelial), 3T12 (mouse fibroblast), and RAW 264.7 (mouse macrophage) cell

77 gesia of 11, 22, and 26 was evaluated in the mouse formalin test (A3AR antagonist blocked and A3AR ag

78 a V(D)J passenger allele system to assay, in mouse GC B cells, sequence-intrinsic SHM-targeting rates

79 Using a conditional mouse genetic approach to disable nonredundant subunits

80 ed deletions at 17 sites in four loci of the mouse genome.

81 In contrast, in a mouse gut colonization model in which the natural microb

82 BBP-ZNF384 fusion altered differentiation of mouse hematopoietic stem and progenitor cells and also p

83 en gene network, which was down-regulated in mouse hepatocyte nuclear factor 4A knockout mice; were e

84 Ultradian rhythms in mouse hepatocytes in vivo have been published, and we va

85 examined the effect of maternal CB intake on mouse hippocampal interneurons largely focusing on chole

86 in vivo, we generated a human PR3 transgenic mouse (hPR3Tg).

87 es of Pn3P-specific CD4(+) T cells utilizing mouse immunization schemes that induce Pn3P-specific IgG

88 Using the continuously growing mouse incisor as a model of stem cell-based tissue renew

89 Using three mouse infection models, we show that the SIP signaling p

90 Throughout postnatal maturation of the mouse inner ear, cochlear hair cells display at least tw

91 Meg3 overexpression in a mouse insulin-secreting PNET cell line, MIN6, downregula

92 the behavioral phenotype of the ClockDelta19 mouse is characterized by hyperactivity, decreased anxie

93 DAT-Knockout (DAT-KO) mouse is currently the best animal model for this syndro

94 que marker implanted into the surface of the mouse jejunum, serving as a fiducial marker for precise

95 leated cells at the bone surface of inflamed mouse joints.

96 segment as an RV144 V2 Ab but paired with a mouse lambda L chain.

97 ecognition resulted in human VH pairing with mouse lambda L chains instead of allowing otherwise subd

98 romatin immunoprecipitation against Lmx1b in mouse limbs at embryonic day 12.5 followed by next-gener

99 lphaS pathology in another alphaS transgenic mouse line (M20), albeit less robustly in these mice.

100 Furthermore, a transgenic mouse line specifically labeling SL cells showed that th

101 We report the development of a transgenic mouse line where Cre-recombinase-induced expression of a

102 Using this mouse line, we establish a specific requirement for the

103 unction of H1 subtypes, we generated knockin mouse lines in which endogenous H1 subtypes are tagged.

104 crossed these drivers to conditional DREADD mouse lines to further characterize the functional contr

105 Mouse liver contains two natural killer (NK) cell popula

106 d progenitor cells in vitro and in embryonic mouse livers.

107 nce, we immunized BALB/c mice with senescent mouse lung fibroblasts and screened for antibodies that

108 3 phosphorylation and IL-6 expression in the mouse lungs, consistent with expression of ESAT-6, IL-6

109 L-6 and phosphorylated-STAT3 in Mtb-infected mouse lungs.

110 ding to platelets within thrombi in infected mouse lungs.

111 We observed that exosomes released during a mouse M. tuberculosis infection contribute significantly

112 al), 3T12 (mouse fibroblast), and RAW 264.7 (mouse macrophage) cell lines.

113 Using a unique mouse malaria model, here we investigated the mechanisms

114 y controls the mitosis-meiosis transition in mouse male germ cells.

115 Sex difference in mouse metabolic response to erythropoietin.

116 vestigate the regulatory mechanism of mIDH2 (mouse mitochondrial IDH2), we used lysine-to-glutamine (

117 Here, we generated a novel mouse model (eAA) with the erythroid-specific ablation o

118 ry of the disease in the Emu-Tcl1 (Tcl1) CLL mouse model and 68 CLL patients.

119 on in a high-fat diet (HFD) induced diabetic mouse model and a genetically engineered T2DM rat model.

120 tablished in vivo mild chronic hypoxia (MCH) mouse model and a new severe acute hypoxia (SAH) mouse m

121 ral characterization of the Chrna7 deficient mouse model appeared prudent.

122 s will be of potential use for monitoring HD mouse model disease progression and evaluating preclinic

123 red to a C3H/C57bl6 background to generate a mouse model for Alagille syndrome (Jag1(Ndr/Ndr) mice).

124 In a well-characterized mouse model for DM1 (HSALR mice), activation of AMPK sig

125 To test this hypothesis, we created a mouse model in which a portion of the sciatic nerve from

126 Using a transgenic mouse model in which FlnA is selectively depleted in mye

127 tinct cellular and molecular mechanisms in a mouse model of ACD.

128 sumption of canola oil on the phenotype of a mouse model of AD that develops both plaques and tangles

129 miR-29a significantly improved survival in a mouse model of aGVHD while retaining graft-versus-leukem

130 tions was low, microglial proliferation in a mouse model of Alzheimer's beta-amyloidosis was increase

131 nucleotide (FAD) in fresh brain samples of a mouse model of Alzheimer's disease (AD).

132 Further, in a mouse model of AMR, in which C57BL/6.

133 rain structure in a maternal Ube3a knock-out mouse model of AS.

134 ed PCSK9 hypomethylation and a translational mouse model of AUD showed that alcohol exposure leads to

135 e impact of granzyme A deficiency in the NOD mouse model of autoimmune diabetes.

136 t to directly investigate GFAP turnover in a mouse model of AxD that is heterozygous for a disease-ca

137 Using a mouse model of bacterial sepsis, we found that the numbe

138 of cellular intoxication and pathology in a mouse model of botulism.

139 -17 induces spontaneous immunopathology in a mouse model of CNS inflammation.

140 e model and a new severe acute hypoxia (SAH) mouse model of DWMI activates the initial step of the IS

141 Using a mouse model of foodborne L. monocytogenes infection, a r

142 in young and adult Fmr1 knock-out mice, the mouse model of fragile X syndrome (FXS).

143 Using a mouse model of inducible SOX2, which is broadly expresse

144 e putative prohemorrhagic effect of NAC in a mouse model of intracranial hemorrhage induced by in sit

145 aternal, dendrimer nanoparticle (DNAC), in a mouse model of intrauterine inflammation.

146 sed cone density by 30-40% in the Rpe65(-/-) mouse model of Leber congenital amaurosis and reduced th

147 le of oncogenic KRAS in CRC, we engineered a mouse model of metastatic CRC that harbors an inducible

148 Conditional deletion of Mof in a mouse model of MLL-AF9-driven leukemogenesis reduced tum

149 An EL4 mouse model of non-Hodgkin lymphoma and a B16 mouse mode

150 aling and cell death in RGCs, including in a mouse model of optic nerve injury, and show that the sam

151 dorsal root ganglia, spinal slices, and in a mouse model of pain induced by NaV1.7 activation, Pn3a a

152 es degeneration of dopaminergic neurons in a mouse model of Parkinson's disease using 1-methyl-4-phen

153 f senescence in vitro Here we show that in a mouse model of prostate cancer, SIN3B provides a barrier

154 In a transgenic mouse model of resectable PDAC, we investigated the coor

155 , mechanistic experiments were designed in a mouse model of resuscitated hemorrhagic shock and tissue

156 ouse model of non-Hodgkin lymphoma and a B16 mouse model of subcutaneous melanoma are used to extract

157 worsening of the AD phenotype in a relevant mouse model of the disease.

158 itis, as well as maintain normoglycemia in a mouse model of type 1 diabetes.

159 The ability to easily label pericytes in any mouse model opens the possibility of a broad range of in

160 ith an emphasis on exploitation of a "dirty" mouse model that better mimics the diverse infectious hi

161 A mouse model that recapitulates key histopathological fea

162 We used an HDM-driven asthma mouse model to compare the capacity of Jagged 1 and Jagg

163 unstable in humans, and we present a unique mouse model to study human circadian disorders with unst

164 Here, we develop a new mouse model to transfer genes specifically into the prim

165 ry to treat gastric bacterial infection in a mouse model using clarithromycin as a model antibiotic a

166 m healthy liver in an experimentally arrived mouse model using noninvasive technique.

167 es utilized the well-characterized Pax9(-/-) mouse model with a consistent cleft palate phenotype to

168 isease pathogenesis, we generated a knock-in mouse model with NB disruption mediated by 2 point mutat

169 rowth of MGC803 cells in vivo in a xenograft mouse model without observed toxicity.

170 arboring wild-type alleles of GBA, A53T-SNCA mouse model) were exposed to a brain-penetrant GCS inhib

171 SO7 had a similar effect in the BAC-Q72 SCA2 mouse model, and in both mouse models it normalized prot

172 LPS-induced endotoxemia, we developed a new mouse model, PMN(DTR) mice, in which injection of diphth

173 ph node dissection, both clinically and in a mouse model, results in a marked increase in the number

174 Using a glioma mouse model, we demonstrate that doxorubicin-loaded (D)C

175 With a mouse model, we examined the effect of neonatal exposure

176 th purified VacA proteins and infection of a mouse model, we show that H. pylori deregulates mitochon

177 and Cyp c 1-induced allergic symptoms in the mouse model.

178 dysfunction in a Duchenne muscular dystrophy mouse model.

179 zing a photoreceptor-specific, PKM2 knockout mouse model.

180 ive improvements in an AD APP/PS1 transgenic mouse model.

181 cy of CXCR4 in an apolipoprotein E-deficient mouse model.

182 trol airway inflammation in a humanized ILC2 mouse model.

183 Mouse models and genetic studies suggest the involvement

184 s been established in genetically engineered mouse models and human tumor cells.

185 T exon 1) underlies the disease pathology in mouse models and that the HTT exon 1 gene product can se

186 2040 metabolites in common, suggesting that mouse models can be used to interrogate human lung metab

187 response using bioengineered cell lines and mouse models containing either isoform of the gamma-subu

188 e model T126, was chosen to generate in vivo mouse models containing orthotopic breast tumors for in

189 Using mutant mouse models for the most common form of congenital deaf

190 Several mouse models have been generated to study OPMD; however,

191 line xenograft and patient-derived xenograft mouse models in vivo.

192 The study of both cancer-bearing mouse models in wild types and their corresponding contr

193 in the BAC-Q72 SCA2 mouse model, and in both mouse models it normalized protein levels of several SCA

194 in the absence of BACE1 S-palmitoylation in mouse models of AD amyloidosis.

195 genetically prone and experimentally induced mouse models of autoimmunity, increased serum levels of

196 l and vascular deficits in chronic and acute mouse models of CCM3 loss in vivo, significantly reducin

197 Novel mouse models of Chlamydia, Haemophilus influenzae, influ

198 of prostate 4 (STEAP4) was highly induced in mouse models of colitis and in IBD patients.

199 tion studies using DBA2/J and Nos3 (eNos) KO mouse models of diabetes, TEPP-46 treatment reversed met

200 We used genetically engineered mouse models of glioma and quantitative metabolomics to

201 endothelium and abrogate angiogenesis in the mouse models of oxygen-induced retinopathy and laser-ind

202 In mouse models of retroviral AML transplantation, as well

203 Mouse models of T. cruzi infection have been used to stu

204 Here we used mouse models to examine host determinants that affect H.

205 In this study, we created two RP mouse models to test whether dying, untreated rods negat

206 choroidal neovascularization, and transgenic mouse models with deficient or spontaneous retinal/choro

207 e (WT) mice were crossed to produce pregnant mouse models with or without adiponectin deficiency.

208 evel or abnormal behavioral responses in ASD mouse models, especially during an early developmental t

209 In both genetic mouse models, expression and phosphorylation of Sestrin2

210 cancer invasion in xenograft and orthotopic mouse models, respectively.

211 r severe OXPHOS deficiency comparing several mouse models, that will deepen our understanding, open a

212 Unlike in mouse models, the mechanisms underlying SAg-associated i

213 In xenograft and genetically engineered mouse models, the WDR4/PML axis elevates intratumoral Tr

214 In mouse models, TRX80 was associated with a proinflammator

215 Using mouse models, we demonstrate that melanoma growth is dra

216 Using in vitro cell culture and in vivo mouse models, we showed that COUP-TFII hinders myogenic

217 ens junctions, and confirm these findings in mouse models.

218 on, ameliorate skin fibrosis in experimental mouse models.

219 sfunction also contributes to the disease in mouse models.

220 protocol generically applicable to different mouse models.

221 ial necroptosis and remodeling using genetic mouse models.

222 of SPOP mutation in human prostate cancer or mouse models.

223 nditional knockout [cKO]) and naturally aged mouse models.

224 homologues of profibrotic genes expressed by mouse monocyte-derived alveolar macrophages during fibro

225 d Actgamma isoforms are expressed in primary mouse motoneurons and their transcripts are translocated

226 anted immediately after lesions in the adult mouse motor cortex restored damaged cortical pathways.

227 d cerebellar hypoplasia in GCp-specific Chd7 mouse mutants.

228 rce microscopy, we studied the elasticity of mouse myoblasts expressing a mutant form of the gene enc

229 ut not E2f3b, overexpression or knockdown in mouse NAc regulates cocaine-induced locomotor and place

230 lhm1 knockout, Panx1 knockout, and wild-type mouse nasal septal epithelial cells were grown at an air

231 y breeding a Gclc loxP mouse with a thy1-cre mouse (NEGSKO mouse) and at a later age by breeding with

232 tient-derived fibroblasts, which, similar to mouse neocortical progenitors, transiently arrest at pro

233 ve chromatin histone modification H3K4me2 in mouse neurons.

234 The induction of EBI3 protein expression in mouse NK cells is a late activation event.

235 in fertility, with a mean number of embryos/mouse of 4.4 +/- 1.3 compared to 7.8 +/- 0.5 for the uni

236 We report the identification in mouse of a previously unknown population of multipotent

237 art failure, Ser199 (equivalent to Ser200 in mouse) of cTnI (cardiac troponin I) is significantly hyp

238 ue, we used single glomerular stimulation in mouse olfactory bulb slices to measure the synaptic dyna

239 Ex vivo transduction of mouse organotypic explants identified Anc80L65 from a se

240 We found that human G9a (hG9a) unlike its mouse orthologue (mG9a) potently stimulated p53 transcri

241 Here, we show that mutation of the mouse orthologue of GPSM2 affects actin-rich stereocilia

242 istent colonization of mice by the EPEC-like mouse pathogen Citrobacter rodentium.

243 TRX80 also promotes the differentiation of mouse peritoneal and human macrophages toward a proinfla

244 PML3 gives rise to the varitint-waddler (Va) mouse phenotype.

245 a and RIM1beta are highly likely absent from mouse photoreceptors and that RIM2alpha is the major lar

246 xcellent in vitro properties and a promising mouse PK profile, making it suitable for in vivo testing

247 Human and mouse pluripotent stem cell-derived CMs (PSC-CMs) were t

248 Pkm-knockdown immortalized mouse podocytes had higher levels of toxic glucose metab

249 d short- and long-term Mef2c upregulation in mouse prefrontal projection neurons consistently resulte

250 find that TGF-beta signaling is operative in mouse primary keratinocytes in conventional cultures as

251 EAF2 knockout mouse prostate was also sensitized to gamma-irradiation.

252 Despite the species mismatch, the mouse Prox1-EGFP BAC enabled a reliable expression of EG

253 we generated rat-sized pancreata composed of mouse-PSC-derived cells.

254 performing the reverse experiment, injecting mouse PSCs into Pdx-1-deficient rat blastocysts, we gene

255 ectural differences in ERbeta neurons of the mouse PVN that are different from that previously descri

256 Expression of hNTCP in mouse, rat, and dog hepatocytes permits HDV infection bu

257 f aspartic acid/glutamic acid encoded in the mouse repertoire.

258 Using region-specific mouse reporter strains, we performed an RNA-seq screen,

259 In this study, we combine novel mouse reporters and single-cell RNA sequencing to reveal

260 Expression of mERbeta2 mRNA was detected in mouse reproductive tissues (ovary, testis, and prostate)

261 ending the predictive translational value of mouse research, with an emphasis on exploitation of a "d

262 etween RGCs and polyaxonal amacrine cells in mouse retina forms the synaptic mechanism responsible fo

263 3-expressing amacrine cells (VG3-ACs) in the mouse retina.

264 t a distinct set of feature detectors in the mouse retina.

265 cer into the inferior colliculus (IC) of the mouse revealed that most of the subcortical descending p

266 The Gria3A653T mouse showed significantly fewer brief bouts of activity

267 zed nuclear and whole cell transcriptomes in mouse single neurons and provided a normalization strate

268 asurements of circadian enzyme activities in mouse skeletal muscle confirmed that such timing separat

269 actions (MICs) on protein phosphorylation in mouse skeletal muscle.

270 WBP2 deletion in mouse skin results in reduced proliferation in neonatal

271 pathways, and shared seventeen genes with a mouse smoking model and twenty genes with previous emphy

272 Here, we describe a mouse strain lacking the E3 ubiquitin ligase RNF146 that

273 vity was addressed in vivo using 2 different mouse strains (BALB/c and C3H).

274 giogenic effect in both in vitro and in vivo mouse studies.

275 r differences among treatment groups for the mouse studies.

276 omical brain networks of human, macaque, and mouse, successfully predicting simulation and empirical

277 Mouse survival was enhanced by >13days compared to contr

278 Using transgenic mouse technology, we show that Shp2 is involved in oligo

279 t in both the brain and the periphery of the mouse to cause migraine-like photophobia by apparently d

280 n reparative dentinogenesis using an in vivo mouse tooth damage model.

281 Smooth-muscle cells from mouse tracheas were assayed in vitro for signaling pathw

282 currents in duck trigeminal ganglia than in mouse trigeminal ganglia.

283 or direct identification of binding sites in mouse TRPA1.

284 Here, we demonstrated that human SS and mouse tumors arising from SS18-SSX expression in the emb

285 ed a doxycycline suppressible Cre transgenic mouse under the regulation of the Nkx2.5 enhancer and sh

286 lial cell types in the developing and mature mouse ureter.

287 In mouse urothelial organoids, PPAR agonism is sufficient t

288 ression was about the same in both WT and KO mouse visceral yolk sac, brain, and spinal column.

289 egulated during functional maturation of the mouse visual cortex with miR-132/212 family being one of

290 In mouse, we identified more than 800 LTRs from ORR1, MT, M

291 When regulatory T cells (2 x 106/mouse) were intravenously administered immediately after

292 The C5aR2 knock-in mouse will help to reliably track and conditionally dele

293 thesis both at birth by breeding a Gclc loxP mouse with a thy1-cre mouse (NEGSKO mouse) and at a late

294 Human and mouse WNT10A mutant palmoplantar and tongue epithelia al

295 We established a mouse xenograft model of human acute myeloid leukemia (A

296 In intracranial mouse xenograft models of glioblastoma, inhibiting Wnt5a

297 In mouse xenograft models of human TNBC, administration of

298 patocellular carcinoma cells in vitro and in mouse xenografts.

299 class-I surface expression in vitro and in a mouse xenotransplantation model.

300 of A-to-I editing events are defined in the mouse, yet the functional impact of most is unknown.