ライフサイエンスコーパス: mouse embryo

1 human NC cells (hNCCs) into the gastrulating mouse embryo.

2 ak, mesoderm and anterior mesendoderm of the mouse embryo.

3 nding cells diminishes bone formation in the mouse embryo.

4 ta-gonad-mesonephros (AGM) of the developing mouse embryo.

5 nce, the plane of cell division in the early mouse embryo.

6 r visceral endoderm (AVE) cells in the early mouse embryo.

7 ultiple roles of BMP signalling in the early mouse embryo.

8 he onset of epidermal differentiation in the mouse embryo.

9 raembryonic ectoderm (ExE) of the developing mouse embryo.

10 on in embryonic stem cells (ESCs) and in the mouse embryo.

11 ltipotent Pax3(+) cells in the somite of the mouse embryo.

12 ted during limb cartilage development in the mouse embryo.

13 pathway in the rapidly dividing cells of the mouse embryo.

14 ollowing transplantation into the developing mouse embryo.

15 ferentiated lineages in the pre-implantation mouse embryo.

16 d by two major routes, as illustrated by the mouse embryo.

17 it is the earliest expressed Hox gene in the mouse embryo.

18 enhancer activity and Shh expression in the mouse embryo.

19 early lineages in peri- and postimplantation mouse embryos.

20 from both wild-type and Tdg-deficient E11.5 mouse embryos.

21 to position the first inner cells of living mouse embryos.

22 n guiding post-crossing axon trajectories in mouse embryos.

23 s via injection into the cardiac crescent of mouse embryos.

24 to the primitive endoderm in both human and mouse embryos.

25 cardiomyocytes (CMs) derived from mESCs and mouse embryos.

26 th analysis of Fat1 expression in developing mouse embryos.

27 ampal neurons from wild-type and Ctnnd2 null mouse embryos.

28 ical properties in cell lines and transgenic mouse embryos.

29 ssion, respectively, in both C2C12 cells and mouse embryos.

30 nt of corneal avascularity in both chick and mouse embryos.

31 ary to induce LHX3(+)/LHX4(+) RP identity in mouse embryos.

32 ve hypertrophic chondrocytes in the bones of mouse embryos.

33 docardium by embryonic day 9.5 in transgenic mouse embryos.

34 d to prevent senescence in primary cells and mouse embryos.

35 which is required for activity in transgenic mouse embryos.

36 differences among 10 2-cell and five 4-cell mouse embryos.

37 at this gene is essential for development of mouse embryos.

38 upstream of Fgf8 that binds RAR isoforms in mouse embryos.

39 nriched remodeling complexes present in E8.5 mouse embryos.

40 m leading to vascular morphogenesis in early mouse embryos.

41 onal genetic transsynaptic tracing in female mouse embryos.

42 llary arch (primordium for the upper jaw) of mouse embryos.

43 equired for cell cleavage in preimplantation mouse embryos.

44 ull, results in elevated cellular hypoxia in mouse embryos.

45 on Goosecoid protein localization in staged mouse embryos.

46 adipogenesis in vitro and BAT development in mouse embryos.

47 e cells at different developmental stages of mouse embryos.

48 aled that these glycans are present on early mouse embryos.

49 y establish hematopoietic fate in Gata2Venus mouse embryos.

50 e performed transcriptome profiling on whole mouse embryos.

51 and generation of the pro-amniotic cavity in mouse embryos.

52 (+) cells in a subdomain of the NMP niche in mouse embryos.

53 racking individual cells in live analysis of mouse embryos.

54 ient incorporation of naive human cells into mouse embryos.

55 how germ cells differentiate into oocytes in mouse embryos.

56 ent was sufficient to extend trunk length in mouse embryos.

57 nd intact fluorescently-stained 12.5-day old mouse embryos.

58 nome activation timing between the human and mouse embryos.

59 es, and in the neural crest and limb buds of mouse embryos.

60 sues obtained from human fetuses with DS and mouse embryos.

61 ion has been studied extensively in frog and mouse embryos.

62 t state linger within the inner cell mass of mouse embryos?

63 Additionally, by investigating mouse embryo 5fC profiles in a tissue-specific manner, w

64 art field (pSHF) of Tbx5 and Osr1 transgenic mouse embryos, a time-course gene expression change duri

65 g with depletion of KDM6B in preimplantation mouse embryos abrogates CDX2 and GATA3 expression in the

66 image data sets of fruit fly, zebrafish and mouse embryos acquired with three types of fluorescence

67 In the mouse embryo, Adamts15 was expressed in the developing h

68 ibody inhibited embryo implantation in vivo, mouse embryo adhesion and spreading in vitro, as well as

69 ess in three different types of fibroblasts (mouse embryo, adult cardiac, and tail tip).

70 essential for any biologist working with the mouse embryo, although the last revision dates back to 1

71 ignificant 22q11DS phenotypic sites-in LgDel mouse embryos, an established 22q11DS model.

72 stem cells (TSCs) are derived from the early mouse embryo and can substantially contribute to placent

73 is challenging due to the small size of the mouse embryo and rapid heart rate.

74 percentage of pluripotent cells in the early mouse embryo and significantly reduces reprogramming eff

75 tion and morphogenesis: the pre-implantation mouse embryo and the developing mouse olfactory epitheli

76 The specimens are an early mouse embryo and two different cellular spheroids.

77 for anterior-posterior axis formation of the mouse embryo and was shown to promote posterior neuroect

78 endocrine progenitors from different staged mouse embryos and analyzed H3K27me3 genome-wide.

79 ogen signaling and target gene expression in mouse embryos and chick ex vivo assays.

80 nal and naive-like types) were injected into mouse embryos and cultured, some human cells survived bu

81 to the onset of the developmental program in mouse embryos and demonstrate a role for broad H3K4me3 d

82 Here, we show in mouse embryos and differentiating embryonic stem cells t

83 multiple imprinting control regions in early mouse embryos and embryonic stem (ES) cells.

84 d isolated populations of Cdh5(+) cells from mouse embryos and embryonic stem cells can be differenti

85 al and mesodermal cell fate determination in mouse embryos and ESCs.

86 ae, whole-mount chicken embryos, whole-mount mouse embryos and formalin-fixed paraffin-embedded human

87 Here, we use both developing mouse embryos and human embryonic stem cells (hESCs) to

88 ed autophagy in embryonic day 18.5 BAT3(-/-) mouse embryos and in mouse embryonic fibroblasts (MEFs)

89 the tooth bud mesenchyme in Osr2(-/-) mutant mouse embryos and is partially restored in the tooth mes

90 s enriched in HAND2 chromatin complexes from mouse embryos and limb buds.

91 ositioning during EMT in vivo, in developing mouse embryos and mammary gland, and in vitro, in cultur

92 o obtain high-speed image data from in utero mouse embryos and multi-angle, vector-flow algorithms we

93 and E13.5 Osr2(RFP/+) and Osr2(RFP/-) mutant mouse embryos and performed whole transcriptome RNA sequ

94 s (rsPSCs), can be efficiently obtained from mouse embryos and primate pluripotent stem cells, includ

95 when epigenetic reprogramming occurs: early mouse embryos and primordial germ cell development.

96 using single cells from Etv2-EYFP transgenic mouse embryos and reveal specific molecular pathways tha

97 the craniofacial mesenchyme of mid-gestation mouse embryos and that ablation of Pdgfrb in the neural

98 It is widely expressed in mouse embryos and tissues.

99 cause hearing loss, we evaluated Esrp1(-/-) mouse embryos and uncovered alterations in cochlear morp

100 nter-blastomere differences in 2- and 4-cell mouse embryos, and associate these differences with ICM/

101 in differentiating mouse ES cells, cultured mouse embryos, and developing zebrafish embryos.

102 BMP signaling activity in both zebrafish and mouse embryos, and excess BMP2 signaling in zebrafish em

103 lood and endothelial cell populations in the mouse embryo are specified independently, and that hemog

104 Geminin knockout mouse embryos are preimplantation lethal by the 32-cell

105 e region of active spinal neurulation in the mouse embryo as a prerequisite for successful NT closure

106 piblast-specific inactivation of Cubn in the mouse embryo as well as Cubn silencing in the anterior h

107 5/8 shows that BMP signaling is activated in mouse embryos as early as the 4-cell stage, and becomes

108 In the mouse embryo at embryonic day 6.5, cells located at the

109 uthors show that de novo polarisation of the mouse embryo at the 8-cell stage is directed by Phosphol

110 sl1-expressing cells in the urinary tract of mouse embryos at E10.5 and distributed in the bladder at

111 ally induces right ventricular dilatation in mouse embryos at embryonic day 16.5.

112 cted palatal epithelial cells from embryonic mouse embryos at various palate development stages.

113 In wild-type mouse embryos, beta-actin expression was prominent in th

114 a protocol supporting the development of the mouse embryo beyond the blastocyst stage in vitro.

115 llular heterogeneities detected in four-cell mouse embryos bias the process of cell fate acquisition

116 rces axial bias in skin spreading around the mouse embryo body.

117 ypertrophic and hypertrophic chondrocytes of mouse embryo bone cartilage.

118 aging and electrophysiological recordings in mouse embryo brainstem slices together with computationa

119 on of the MTHFD2L transcript is low in early mouse embryos but begins to increase at embryonic day 10

120 (ICM) and epiblast of the peri-implantation mouse embryo, but its function has not been investigated

121 enotypes and hepatitis in late organogenesis mouse embryos, but the molecular and cellular mechanisms

122 l forebrain angiogenesis and BBB function in mouse embryos, but the role of this receptor in adult an

123 cribes how to observe gastrulation in living mouse embryos by using light-sheet microscopy and comput

124 Otic tissues of mouse embryos carrying NCC lineage reporter transgenes w

125 Ablation of Dnmt1 in mouse embryos causes death at the post-gastrulation stag

126 We show that mutation of Fat1 in mouse embryos causes defects in cranial neural tube clos

127 In the CNS of embryonic day 10.5 mouse embryos, CD31(+)F4/80(+) hematopoietic lineage cel

128 Mouse embryos conditionally lacking Tgif1 and Tgif2 have

129 nail1/E-cadherin axis described in the early mouse embryo corresponds to Snail2/P-cadherin in the chi

130 Experiments involving mouse embryos deficient for both maternal and zygotic Oc

131 Here, we examined mouse embryos deficient for the chromatin remodeling pro

132 Accordingly, mouse embryos deficient in Orai1, Klf2, or Klf4 showed a

133 Immunohistochemistry of later-stage mouse embryos demonstrated tissue-specific expression in

134 Analysis of miR-34/449 knockout (KO) mouse embryos demonstrates significant spindle misorient

135 artially rescued the phenotypes in Chd7-null mouse embryos, demonstrating that p53 contributes to the

136 Wt1-null mouse embryos develop CDH but the mechanisms regulated b

137 In the absence of liver sinusoidal GATA4, mouse embryos developed hepatic capillaries with upregul

138 cover a crucial role of Brpf1 in controlling mouse embryo development and regulating cellular and gen

139 -h time-lapse sequences of post-implantation mouse embryo development with light-sheet microscopy.

140 iCPCs injected into the cardiac crescent of mouse embryos differentiated into cardiomyocytes.

141 Gata4(+/-);Tbx5(+/-) mouse embryos display decreased atrial and ventricular m

142 Surprisingly, Katnb1 null mutant mouse embryos display hallmarks of aberrant Sonic hedgeh

143 Placentas from Grhl2-deficient mouse embryos displayed defects in BCT cell polarity and

144 Depletion of Unkempt in mouse embryos disrupts the shape of migrating neurons, w

145 1 also confers severe anemia in midgestation mouse embryos due to defective definitive erythropoiesis

146 rnal provision of embryonic vitamin E to the mouse embryo during neural tube closure.

147 performed live confocal imaging of cultured mouse embryos during vessel remodeling.

148 In contrast to the published data from mouse embryos, during human pancreas development, we det

149 cord tissues after intraspinal injection of mouse embryos (E16).

150 Here we show that CLIC4-null mouse embryos exhibit impaired renal tubulogenesis.

151 nt role in placenta function as Tex19.1(-/-) mouse embryos exhibit intra-uterine growth retardation a

152 80% of Pax9(del/del);Wise(-/-) double-mutant mouse embryos exhibit rescued palatal shelf elevation/re

153 Setd5-deficient mouse embryos exhibit severe defects in neural tube form

154 nistic changes in early brain development in mouse embryos exposed to this maternal gene-environment

155 ne embryonic stem cell (ESC) line that, like mouse embryos, expresses functional GLUT2 transporters.

156 was expressed at higher levels in brains of mouse embryos expressing activated AKT3.

157 In Osr1 mutant mouse embryos, expression of Sox9 persisted in the devel

158 ontrol fetal human biopsies or in developing mouse embryos, FAT1 is expressed at lower levels in musc

159 by mild chromosome instability in genomes of mouse embryo fibroblast cells from Wwox-knockout mice.

160 ular structures of lamin A, C, B1, and B2 in mouse embryo fibroblast nuclei.

161 C12 activated apoptosis in mouse embryo fibroblasts (MEF) from both wild type (WT)

162 KSR1 disruption in mouse embryo fibroblasts (MEFs) abrogates growth factor-

163 se1 promote proteasomal turnover of Ube3a in mouse embryo fibroblasts (MEFs) and catalyze Ube3a ubiqu

164 Mouse embryo fibroblasts (MEFs) are convenient sources f

165 Accordingly, p19(Arf)-deficient mouse embryo fibroblasts (MEFs) arrest in response to ac

166 show that disruption of Cul9-p53 binding in mouse embryo fibroblasts (MEFs) by a knock-in mutation i

167 a, and kidney tumor and that PCBP4-deficient mouse embryo fibroblasts (MEFs) exhibit enhanced cell pr

168 Furthermore, mouse embryo fibroblasts (MEFs) from DUSP5(-/-) mice sho

169 In this report, we demonstrate that mouse embryo fibroblasts (MEFs) lacking all three isofor

170 In mouse embryo fibroblasts (MEFs) lacking CPEB, many mRNAs

171 Mouse embryo fibroblasts (MEFs) lacking Tm5NM1, which ha

172 rties of normal and vimentin-null (vim(-/-)) mouse embryo fibroblasts (mEFs) on substrates of differe

173 Furthermore, R137Q mouse embryo fibroblasts (MEFs) were more sensitive to D

174 was notably higher than in similarly treated mouse embryo fibroblasts (MEFs).

175 Tgif1; Tgif2-null embryos and in double-null mouse embryo fibroblasts (MEFs).

176 mental tumors prepared from HRAS-transformed mouse embryo fibroblasts and for primary brain tumor dev

177 us repressed Hh signaling in Ptch1-deficient mouse embryo fibroblasts and that repression was reverse

178 inally, we tested for PI establishment in 12 mouse embryo fibroblasts by natural selection.

179 Approximately 50% of reprogrammed mouse embryo fibroblasts displayed spontaneous beating a

180 Consistent with this, AK2(+/-) mouse embryo fibroblasts exhibit enhanced cell prolifera

181 d p53 activation were drastically reduced in mouse embryo fibroblasts harboring endogenous MDMX with

182 obal p53 transcriptional networks in primary mouse embryo fibroblasts in response to DNA damage.

183 we report that depletion of Mbnl proteins in mouse embryo fibroblasts leads to misregulation of thous

184 Genetic inactivation in mouse embryo fibroblasts or CUX2 knockdown in HCC38 cell

185 Here, we show that Rev1-deficiency in mouse embryo fibroblasts or mouse liver tissue is associ

186 at equal gene dosage in presenilin-deficient mouse embryo fibroblasts resulted in trans-dominant-nega

187 The loss of PRMT5 activity in mouse embryo fibroblasts results in almost complete loss

188 Although cultures of wild-type mouse embryo fibroblasts set Myc levels precisely, Myc l

189 ermore, cell-cycle progression of Rev3L(-/-) mouse embryo fibroblasts was arrested in late S/G2 follo

190 tes revealed that the majority of editing in mouse embryo fibroblasts was carried out by ADAR1.

191 in, paxillin is required for E6 to transform mouse embryo fibroblasts while HIC-5 is not.

192 ate ADAR1 p150 as the major A-to-I editor in mouse embryo fibroblasts, acting as a feedback suppresso

193 bryonic stem cells, epiblast stem cells, and mouse embryo fibroblasts, derived from mice of the same

194 ouse sarcoma (SA-NH), along with transformed mouse embryo fibroblasts, wild type or cells lacking fun

195 on of IFN-induced, RNA-mediated responses in mouse embryo fibroblasts.

196 ase in vitro and was capable of transforming mouse embryo fibroblasts.

197 ssociates with mitochondria in MAVS knockout mouse embryo fibroblasts.

198 In particular, while mouse embryos first exhibited sub-compartmentalization o

199 igital representations of the development of mouse embryos from the morula to early blastocyst stage,

200 ich further reveals differences in human and mouse embryo gene expression.

201 (RRRs) that are expressed normally at 2-cell mouse embryos generated by in vitro fertilization (IVF)

202 The first lineage segregation in the mouse embryo generates the inner cell mass (ICM), which

203 In the mouse embryo, global epigenetic changes occur during zyg

204 se, and whole-mount in situ hybridization in mouse embryos has shown that Yap1 is strongly expressed

205 MEFs derived from LCMT-1 knock-out mouse embryos have reduced levels of PP2A B regulatory s

206 Here we identify the mouse embryo hindbrain as a powerful model to study embr

207 vity in the cytoplasm of interphase two-cell mouse embryos (I2C).

208 ation from entire Drosophila, zebrafish, and mouse embryos imaged with confocal and light-sheet micro

209 e expression in both the wrist and digits of mouse embryos in patterns that are nearly indistinguisha

210 ere challenge this view and demonstrate that mouse embryos in the mitotic cell cycle can also directl

211 rogeneity was also evident in human ESCs and mouse embryos in vivo.

212 Caenorhabditis elegans, Xenopus laevis, and mouse embryos, in each species, it results in the restri

213 embryos have several features in common with mouse embryos, including a stage-related initiation of l

214 we generated a variety of editing schemes in mouse embryos, including indel (insertion/deletion) muta

215 niscent of those that cells exhibit in early mouse embryos, including symmetry breaking, axial organi

216 ic imprinting through studies of manipulated mouse embryos indicated that the paternal genome has a m

217 ay during preimplantation development of the mouse embryo is known to be essential for differentiatio

218 Here, we show that the first M-phase in the mouse embryo is significantly extended due to a delay in

219 for biochemical studies when cell number in mouse embryos is limiting.

220 e cells along the anterior-posterior axis of mouse embryos is responsible for left-right symmetry bre

221 orter, GLUT2 (SLC2A2), which is expressed by mouse embryos, is important for survival before embryoni

222 hough we have used this approach for imaging mouse embryos, it can be extended to imaging other types

223 Vangl2(Lp/Lp) mouse embryos lack PCP signalling and undergo almost com

224 fication of the cardiac conduction system in mouse embryos lacking all Endothelin signaling.

225 Here, we discovered that female mouse embryos lacking Coup-tfII (chicken ovalbumin upstr

226 Mouse embryos lacking intact maternal/zygotic CTNNB1 fro

227 Mouse embryos lacking interferon gamma (IFN-gamma) or IF

228 Here we report that mouse embryos lacking OLA1 have stunted growth, delayed

229 Here we show that mouse embryos lacking the chromatin-modifying enzyme his

230 Mutant mouse embryos lacking the heparan sulfotransferases Hs2s

231 Mouse embryos lacking the polycomb group gene member Yin

232 ablation of the entire Id (Id1-4) family in mouse embryos leads to failure of anterior cardiac proge

233 disruption of Wnt/beta-catenin signalling in mouse embryos led to conversion of fundic to antral epit

234 in lymphatic endothelial cells of developing mouse embryos led to defective lymphovenous valve format

235 rized cilia positioning that is essential to mouse embryo left-right asymmetry establishment.

236 committed preskeletal mesenchymal cells from mouse embryo limb buds and whole limb explants.

237 Treatment of mouse embryo limb mesenchymal micromass cultures with ex

238 In mouse embryos, Mab21l2 showed strong expression in the d

239 By contrast, Pou5f1-null mouse embryos maintained the expression of orthologous g

240 llular hierarchy during early development of mouse embryos, modeled the dynamic changes in gene expre

241 In mouse embryos, mutation of Prdm1 affects branchial arch

242 of both mixed-ratio (1:1 and 10:1) yeast and mouse embryo myogenesis proteomes.

243 re, we show that de novo polarisation of the mouse embryo occurs in two distinct phases at the 8-cell

244 ded the floor plate of Robo1/2 double mutant mouse embryos or Slit1/2/3 triple mutants.

245 Although Ikk2 deficiency is lethal in mouse embryos, our observations suggest a more restricte

246 We showed that in chicken and mouse embryos, PAPC expression is tightly regulated by t

247 Fkbp10 is expressed at low levels in E13.5 mouse embryos, particularly in skeletal tissues, and ste

248 nd resolution of tetrads and rosettes in the mouse embryo, possibly in part by spatially restricting

249 nct endodermal cell populations in the early mouse embryo remain ill defined.

250 f beta-catenin in the dorsal neural folds of mouse embryos represses the expression of the homeobox-c

251 on precursors in Cdhr23(-/-) and Cdhr15(-/-) mouse embryos, respectively, failed to enter the embryon

252 Hepatocyte-specific deletion of PR-SET7 in mouse embryos resulted in G2 phase arrest followed by ma

253 Vegfc gene deletion in mouse embryos results in failure of lymphangiogenesis, f

254 Analysis of mouse embryos revealed a localized domain of Zic1 expres

255 ad/mesonephros (AGM) regions of midgestation mouse embryos revealed a robust innate immune/inflammato

256 Real-time imaging of mouse embryos revealed EC movement from the DA to the CV

257 Determination of dNTP pools in mouse embryos revealed that inactivation of one SAMHD1 a

258 s-of-function manipulations in the chick and mouse embryo show that Neurog3 switches ventral progenit

259 he first five rounds of cell division in the mouse embryo, spindles assemble in the absence of centri

260 nal populations derived from preimplantation mouse embryos that can be propagated in vitro and, when

261 Here we show that mouse embryos that lack the small guanosine triphosphata

262 into regulation of gene expression in 1-cell mouse embryos that may confer a protective mechanism aga

263 We found that mouse embryos that were homozygous null for both ednra a

264 r certain samples, such as post-implantation mouse embryos, that expand significantly in size and are

265 w that, contrary to the current view, in the mouse embryo the patella initially develops as a bony pr

266 In the developing mouse embryo, the first hematopoietic stem cells (HSCs)

267 In R137Q knock-in mouse embryos, the BER efficiency was severely impaired,

268 rk that triggers de novo polarisation of the mouse embryo.The molecular trigger that establishes cell

269 l growth by promoting IGF2 production in the mouse embryo through mTORC2-catalyzed cotranslational IM

270 large cleared samples ranging from perinatal mouse embryos to adult organs, such as brains or kidneys

271 workflow with laser ablation of live-imaged mouse embryos to investigate the biomechanics of mammali

272 We show that exposure of mouse embryos to short-term gestational hypoxia induces

273 ggest that excessive 5-HT in MAO-A-deficient mouse embryos triggers cellular signaling cascades via 5

274 The early mouse embryo undertakes two types of cell division: symm

275 MO-mediated gene knockdown frog and knockout mouse embryos unearthed PCP/CE-related phenotypes as wel

276 pituitary, pancreas, lungs and oesophagus of mouse embryos using in situ hybridization.

277 fluorescent protein tags in human cells and mouse embryos using PCR fragments.

278 and nascent Flk1(+) mesoderm of gastrulating mouse embryos using single-cell RNA sequencing, represen

279 ted and free hsa-miR-30d was internalized by mouse embryos via the trophectoderm, resulting in an ind

280 y cilia on neuroepithelial cells in Pam(-/-) mouse embryos was also aberrant.

281 ough experiments conducted in both chick and mouse embryos we have developed a model explaining simul

282 approach to hematopoietic development in the mouse embryo, we map the progression of mesoderm toward

283 Using cell lines from Hat1+/+ and Hat1-/- mouse embryos, we demonstrate that Hat1 is not required

284 By analyzing intact mouse embryos, we demonstrate that the function of Tcf7l

285 Using high-resolution live imaging of mouse embryos, we observed randomly distributed spontane

286 Using mouse embryos, we show that cell protrusions emanating f

287 neurons obtained from Klotho-overexpressing mouse embryos were more resistant to both cytotoxic insu

288 -in mice, and found that homozygous knock-in mouse embryos were typically small in size and had a hig

289 T1 at an early postimplantation stage of the mouse embryo, when its paralogs Tet2 and Tet3 are not de

290 Usp36 depletion is lethal in preimplantation mouse embryos, where it blocks the transition from morul

291 urbation of BMP signaling in preimplantation mouse embryos, whether by treatment with a small molecul

292 ction of RPGRIP1L, we analyzed Rpgrip1l(-/-) mouse embryos, which display a ciliopathy phenotype and

293 chromatin remodeling ATPase LSH (HELLS)-null mouse embryos, which lack DNA methylation from centromer

294 ith special focus on palate formation, using mouse embryos with a complete knockout of Tmem107.

295 Co-microinjection of mouse embryos with Cas9 mRNA and single guide RNAs induc

296 g-term (24 h) time-lapse imaging of E6.5-8.5 mouse embryos with light-sheet microscopy, we developed

297 malformations observed in human patients and mouse embryos with mutations in Foxc1.

298 Mouse embryos with systemic or endothelium-selective Grk

299 siological conditions, including how to hold mouse embryos without agarose embedding, how to transfer

300 replace wild-type cells in vitro and in the mouse embryo--without perturbing normal development.