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1 4+ T cells, bone marrow dendritic cells, and mouse embryonic stem cells.
2 ising features of nucleosome organization in mouse embryonic stem cells.
3 omotes an elevated transcriptional output in mouse embryonic stem cells.
4 thod using gene expression measurements from mouse Embryonic Stem Cells.
5 regulation of the pluripotency gene Nanog in mouse embryonic stem cells.
6 ed protein 1 (KAP1; also known as TRIM28) in mouse embryonic stem cells.
7 in of any of the three Tet proteins and from mouse embryonic stem cells.
8 etic changes within the homeotic clusters of mouse embryonic stem cells.
9 ariants inserted at the same genomic site in mouse embryonic stem cells.
10 cept experiments in a human cell line and in mouse embryonic stem cells.
11 sms with methylated DNA, as we illustrate in mouse embryonic stem cells.
12 s to the ChIP-Seq data of 91 pairs of TFs in mouse embryonic stem cells.
13 nd gene expression during differentiation of mouse embryonic stem cells.
14 mic map of 5-methylcytosine, 5hmC and 5fC in mouse embryonic stem cells.
15 ble for the decay of uridylated pre-let-7 in mouse embryonic stem cells.
16 s demonstrated by culturing and transfecting mouse embryonic stem cells.
17 nce protein interaction partners of NANOG in mouse embryonic stem cells.
18 sing MNs from both wild-type and mutant SOD1 mouse embryonic stem cells.
19 zes haploidy in human HAP1 cells and haploid mouse embryonic stem cells.
20 nog gene expression was reported recently in mouse embryonic stem cells.
21 separate genomic loci and a plasmid donor in mouse embryonic stem cells.
22 unctional when integrated into the genome of mouse embryonic stem cells.
23 ximately 50 kb from the Hoxa gene cluster in mouse embryonic stem cells.
24 n identity when expressed in differentiating mouse embryonic stem cells.
25 ften in the context of bivalent chromatin in mouse embryonic stem cells.
26 chromosome transfer of Hsa21 into recipient mouse embryonic stem cells.
27 CR function in T cells derived in vitro from mouse embryonic stem cells.
28 rease mesoderm at the expense of endoderm in mouse embryonic stem cells.
29 -containing polycomb repressive complexes in mouse embryonic stem cells.
30 to relate these to gene expression levels in mouse embryonic stem cells.
31 it has a major effect on PRC2 recruitment in mouse embryonic stem cells.
32 lomere dynamics of respective chromosomes in mouse embryonic stem cells.
33 activation of bivalent gene transcription in mouse embryonic stem cells.
34 duce the expression of inflammatory genes in mouse embryonic stem cells.
35 f PRC2 to CpG island-containing promoters in mouse embryonic stem cells.
36 -binding, and regulatory element function in mouse embryonic stem cells.
37 otherwise microRNA-deficient Dgcr8 knockout mouse embryonic stem cells.
38 to the global chromatin affinity of BRG1 in mouse embryonic stem cells.
39 that is required for the differentiation of mouse embryonic stem cells.
40 itive DNA elements during differentiation of mouse embryonic stem cells.
41 emonstrate our approach on Oct4 and Nanog in mouse embryonic stem cells.
42 enine as another form of DNA modification in mouse embryonic stem cells.
43 constitutive heterochromatin organization in mouse embryonic stem cells.
44 that polycomb repressive complex 2-deficient mouse embryonic stem cells also release Bmp4 but retain
45 in repression at many developmental genes in mouse embryonic stem cells and are required for early de
46 lements at a defined chromosomal position in mouse embryonic stem cells and assessed their ability to
47 w approach for two typical qPCR datasets (of mouse embryonic stem cells and blood stem/progenitor cel
49 lied MCORE to map the chromatin landscape in mouse embryonic stem cells and differentiated neural cel
50 We also applied SingleSplice to data from mouse embryonic stem cells and discovered a set of genes
51 tate transition following differentiation of mouse embryonic stem cells and during reprogramming of s
53 o generate myelinating oligodendrocytes from mouse embryonic stem cells and established a myelin form
54 amily members in both human HCT116 cells and mouse embryonic stem cells and find that MLL4 is prefere
55 iation capacity of heparan sulfate-deficient mouse embryonic stem cells and functioning in concert wi
56 es, including Zscan4-expressing cells within mouse embryonic stem cells and hemoglobin-expressing cel
57 ssue fundamental to kidney development, from mouse embryonic stem cells and human induced pluripotent
58 evious approaches with the epigenome data in mouse embryonic stem cells and human lung fibroblast cel
59 he algorithm we present 3D architectures for mouse embryonic stem cells and human lymphoblastoid cell
61 g the transient G1 phase of rapidly dividing mouse embryonic stem cells and identifying a window for
62 , inhibited hematopoietic differentiation of mouse embryonic stem cells and impaired the formation of
63 ell blastomeres, and by direct conversion of mouse embryonic stem cells and induced pluripotent stem
64 on promoted precardiac mesoderm formation in mouse embryonic stem cells and involved endodermal produ
65 nvestigation to the inflammatory response in mouse embryonic stem cells and mouse embryonic stem cell
68 ast adenocarcinoma cell line MCF7 as well as mouse embryonic stem cells and observed similarly high c
70 ral resolution (6-hourly) in differentiating mouse embryonic stem cells and report new insight into m
72 duction and suppression of antiviral RNAi in mouse embryonic stem cells and suckling mice(10,11).
73 ogether on individual chromatin fragments in mouse embryonic stem cells and that half of the H3K9me3
74 H3K4 methylation genome-wide in human cells, mouse embryonic stem cells, and Drosophila Biochemical a
75 lling network that maintains pluripotency in mouse embryonic stem cells, and find an incoherent feedf
76 ammatory response mechanism is not active in mouse embryonic stem cells, and in vitro differentiation
77 vents TET2 downregulation in differentiating mouse embryonic stem cells, and short hairpin RNA agains
78 ny TFs remain associated with chromosomes in mouse embryonic stem cells, and that the exclusion previ
80 direct differentiation of motor neurons from mouse embryonic stem cells as a tool to identify genes t
82 2-, or 3-bp substitutions in MMR-proficient mouse embryonic stem cells as effectively as in MMR-defi
83 ity, we established synchronized cultures of mouse embryonic stem cells as they exit the ground state
85 rise Myst2/Kat7/Hbo1 protein interactions in mouse embryonic stem cells by affinity purification coup
87 osine increased multipotent progenitors in a mouse embryonic stem cell colony-forming assay and in em
89 nerating organized germ layers from a single mouse embryonic stem cell cultured in a soft fibrin matr
91 centromeric chromatin-associated proteins in mouse embryonic stem cells deficient for either the meth
93 nction of engineered cardiac tissues made of mouse embryonic stem cell-derived cardiomyocytes (mESC-C
95 cess BMP2 signaling in zebrafish embryos and mouse embryonic stem cell-derived embryoid bodies substa
99 l infection, but not to LPS, was observed in mouse embryonic stem cell-differentiated fibroblasts.
100 e applied PIQ to analyze DNase-seq data from mouse embryonic stem cells differentiating into prepancr
101 amming towards HC fate, both during in vitro mouse embryonic stem cell differentiation and following
103 an important role for DNA replication during mouse embryonic stem cell differentiation, which could s
108 ions genome-wide across an isogenic panel of mouse embryonic stem cell (ESC) and neuronal progenitor
109 ion and early neurogenesis using an in vitro mouse embryonic stem cell (ESC) clonal assay system.
110 n single cells during the earliest stages of mouse embryonic stem cell (ESC) differentiation and duri
112 To model X-SCID in vitro, we generated a mouse embryonic stem cell (ESC) line in which a disease-
114 wever, hPSCs are unstable in classical naive mouse embryonic stem cell (ESC) WNT and MEK/ERK signal i
115 ere, using the self-organizing properties of mouse embryonic stem cell (ESC), we report that ESC-deri
117 y using an RNA interference (RNAi) screen in mouse embryonic stem cells (ESC) with a Nanog reporter.
121 , we performed comparative RNA sequencing of mouse embryonic stem cells (ESCs) and defined a pluripot
122 (3) The binding sites for SOX2 and POU5F1 in mouse embryonic stem cells (ESCs) and EpiSCs are diverge
123 ave generated Tet1/2/3 triple-knockout (TKO) mouse embryonic stem cells (ESCs) and examined their dev
125 repression, we carried out an RNAi screen in mouse embryonic stem cells (ESCs) and identified a list
126 encing across multiple clonal populations of mouse embryonic stem cells (ESCs) and neural progenitor
127 ncing screen in clonal populations of hybrid mouse embryonic stem cells (ESCs) and neural progenitor
128 T1-(MOF)-containing MSL and NSL complexes in mouse embryonic stem cells (ESCs) and neuronal progenito
131 hibition of Erk1/2 and Gsk3beta signaling in mouse embryonic stem cells (ESCs) by small-molecule inhi
134 Using CRISPR/Cas9 technology, we generated mouse embryonic stem cells (ESCs) deficient for all thre
135 ons that maintain an undifferentiated state, mouse embryonic stem cells (ESCs) differentiate into var
137 stream of LIF, WNT and MAPK-ERK to stabilize mouse embryonic stem cells (ESCs) in their naive state h
138 ntiation of inner ear sensory epithelia from mouse embryonic stem cells (ESCs) in three-dimensional c
139 e found that 30% of the genome in interphase mouse embryonic stem cells (ESCs) is marked with H3S10ph
142 ression from the naive status represented by mouse embryonic stem cells (ESCs) to a state capacitated
143 rformed a large-scale pooled shRNA screen in mouse embryonic stem cells (ESCs) to discover genes asso
145 and GSK3beta can be selectively inhibited in mouse embryonic stem cells (ESCs) using a chemical-genet
150 -range chromatin interactions genome-wide in mouse embryonic stem cells (ESCs), iPSCs, and fibroblast
152 s at four developmental stages in the mouse [mouse embryonic stem cells (ESCs), mesoderm (MES), cardi
154 tion of DNA methylation at imprinted loci in mouse embryonic stem cells (ESCs), suggesting that epige
155 he human hematopoietic stem cells (HSCs) and mouse embryonic stem cells (ESCs), we demonstrated that
156 rtoire of GSK-3-dependent phosphorylation in mouse embryonic stem cells (ESCs), we found that approxi
157 n-coding transcripts (lincRNAs) expressed in mouse embryonic stem cells (ESCs), which might be incomp
174 kle-like and filament structures occurred in mouse embryonic stem cells expressing little or no NEAT1
175 ilobases (kb) of mouse and human sequence in mouse embryonic stem cells for enhancer activity we iden
176 f RUNX1 in the transition of differentiating mouse embryonic stem cells from hemogenic to hematopoiet
178 that hundreds of (lowly expressed) genes in mouse embryonic stem cells have reduced noise due to sub
179 bly in the formation of induced neurons from mouse embryonic stem cells, human fibroblasts, and norma
180 1p levels and L1 mobilization in pluripotent mouse embryonic stem cells, implying that Tex19.1 preven
181 to generate mechanosensitive hair cells from mouse embryonic stem cells in a three-dimensional (3D) c
183 asure the dynamics of the chromatin fiber in mouse embryonic stem cells, in combination with dynamica
184 out single-molecule imaging of CHD4 in live mouse embryonic stem cells, in the presence and absence
185 ly, we show that in vitro differentiation of mouse embryonic stem cells into alpha motor neurons reca
187 A to restore Galpha expression in Ric8A(-/-) mouse embryonic stem cells, involved two helical domains
188 e deficiency in the inflammatory response in mouse embryonic stem cells is also attributed to the lac
189 ng present initially in both male and female mouse embryonic stem cells is lost from the active X dur
190 in A2-deletion-induced cellular apoptosis in mouse embryonic stem cells is partly rescued by S477D/T4
193 ring at the euchromatic ROSA26 locus between mouse embryonic stem cells lacking either ATM, H2AX, or
194 ration of an Rsx transgene on an autosome in mouse embryonic stem cells leads to gene silencing in ci
195 ctivity in vitro, and knockdown of Dis3l2 in mouse embryonic stem cells leads to the stabilization of
197 Furthermore, genetically modified Dnmt2-only mouse embryonic stem cells lost the DNA methylation patt
198 that CTCF binds thousands of transcripts in mouse embryonic stem cells, many in close proximity to C
199 c retinoic acid (RA) signaling and modulates mouse embryonic stem cell (mESC) differentiation in part
201 more than 3700 compounds in three syngeneic mouse embryonic stem cell (mESC) lines: htt(-/-), extend
203 acetyltransferase (HAT) Mof is essential for mouse embryonic stem cell (mESC) pluripotency and early
204 ere, we clarify the molecular foundations of mouse embryonic stem cell (mESC) self-renewal by applyin
207 whether ERbeta influences differentiation of mouse embryonic stem cells (mESC) into neural lineages,
211 formed an unbiased loss-of-function study in mouse embryonic stem cells (mESCs) and identified 20 lin
213 metabolism, thereby impacting maintenance of mouse embryonic stem cells (mESCs) and subsequent embryo
217 that amongst different developmental stages, mouse embryonic stem cells (mESCs) are resistant to cell
219 rin regulates the naive pluripotent state of mouse embryonic stem cells (mESCs) by enabling LIF-depen
220 Here, we show that depletion of Nup153 in mouse embryonic stem cells (mESCs) causes the derepressi
225 ist in a state of primed pluripotency, while mouse embryonic stem cells (mESCs) display a naive or pr
226 he catalytic SET domains of MLL3 and MLL4 in mouse embryonic stem cells (mESCs) does not disrupt self
227 g during adherent culture differentiation of mouse embryonic stem cells (mESCs) generates a populatio
228 on heterogeneity in the pluripotent state of mouse embryonic stem cells (mESCs) has been increasingly
229 c cells derived from murine embryos and from mouse embryonic stem cells (mESCs) have primarily been s
230 is both necessary and sufficient to convert mouse embryonic stem cells (mESCs) into 2-cell-embryo-li
232 to widespread erasure of DNA methylation in mouse embryonic stem cells (mESCs) lacking the de novo D
233 anscriptional activity of Oct3/4 (Pou5f1) in mouse embryonic stem cells (mESCs) maintained under stan
239 or nuclease (TALEN)-mediated gene editing in mouse embryonic stem cells (mESCs) to produce mice with
241 romosomal translocations can be generated in mouse embryonic stem cells (mESCs) via CRISPR/Cas9.
242 onsible for self-renewal and pluripotency of mouse embryonic stem cells (mESCs) was extracted from se
243 increases globally during differentiation of mouse embryonic stem cells (mESCs), and is particularly
244 the temporal dynamics of NANOG expression in mouse embryonic stem cells (mESCs), and to dissect the l
245 critically required for the pluripotency of mouse embryonic stem cells (mESCs), but the detailed mec
246 own or Parp1 heterozygosity of Brca2(cko/ko) mouse embryonic stem cells (mESCs), carrying a null (ko)
248 types considered hard to transduce, such as mouse embryonic stem cells (mESCs), human ESCs (hESCs),
249 ogen synthase kinase-3 (GSK-3) substrates in mouse embryonic stem cells (mESCs), providing a broad pr
252 e we describe a transcriptional regulator of mouse embryonic stem cells (mESCs), Yin-yang 2 (YY2), th
261 Using Sm-ChIPi, we demonstrated that within mouse embryonic stem cells, one polycomb repressive comp
262 ated using 2D methods or MLOs generated from mouse embryonic stem cells, our human MLOs produced neur
263 ntal route of hemangiogenic progenitors from mouse embryonic stem cells, perform genome-wide CRISPR s
264 of RXR for these functions was validated in mouse embryonic stem cells, primary neurons, and APOE3 a
265 lysis of published genome-wide datasets from mouse embryonic stem cells revealed that the Ring1b subu
268 m the GAPDH housekeeping gene (5.45 ng total mouse embryonic stem cell RNA) and measured associated s
272 ificial chromosome (BAC) transgenic HeLa and mouse embryonic stem cells stably expressing enhanced gr
273 motor neurons in both chick neural tube and mouse embryonic stem cells, suggesting that miR-218 play
274 ly assess Ago-bound small RNAs, we adapted a mouse embryonic stem cell system to express a single epi
278 rovide genomic evidence in budding yeast and mouse embryonic stem cells that the efficiency-accuracy
279 The LIF network was used to encapsulate mouse embryonic stem cells; the encapsulated cells remai
281 y the method to an experiment on pluripotent mouse embryonic stem cells to classify a set of previous
282 EMOIR as a proof of principle, we engineered mouse embryonic stem cells to contain multiple scratchpa
283 nd Hi-Cap data as well as ChIA-PET data from mouse embryonic stem cells to investigate promoter-cente
286 Here, using a de novo targeting assay in mouse embryonic stem cells we unexpectedly discover that
289 high resolution Hi-C dataset generated from mouse embryonic stem cells, we found that most local gen
293 Using the auxin-inducible degron system in mouse embryonic stem cells, we show that CTCF is absolut
297 h1 as regulating FLK1+ mesoderm formation in mouse embryonic stem cells, which in turn specifies hema
298 roduce a targeted autosome loss in aneuploid mouse embryonic stem cells with an extra human chromosom
299 tiated motile fibroblast cells from isogenic mouse embryonic stem cells with or without disruption of
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