ライフサイエンスコーパス: mouse genetics

1 for this apparent noncanonical signal using mouse genetics.

2 e xenografts, patient-derived xenografts and mouse genetics.

3 g molecular and chemical expression aided by mouse genetics.

4 this apparent discrepancy between human and mouse genetics.

5 bral cortex using loss- and gain-of-function mouse genetics.

6 nship between Zic2 and the Shh pathway using mouse genetics.

7 nimals including lesion studies, imaging and mouse genetics.

8 mline capacity of ES cells, and the power of mouse genetics.

9 red mouse strains provide the foundation for mouse genetics.

10 e electronic publication of classic books in mouse genetics.

11 remains one of the longstanding mysteries of mouse genetics.

12 Here we employ mouse genetics, 2-photon microscopy, electrophysiology a

13 Using mouse genetics and a BCR-ABL model of CML, we observed c

14 ue of Neuron, Cho et al. (2017) use advanced mouse genetics and biochemical experiments to unravel th

15 Perhaps, the power of mouse genetics and biology can be harnessed to identify

16 hich have taken advantage of improvements to mouse genetics and circuit-mapping tools.

17 Integrating rat physiology with mouse genetics and clinical results from human by using

18 We have used mouse genetics and genome-wide analyses to determine how

19 Mouse genetics and genomics will help to identify all th

20 cladistic analysis, which is well suited for mouse genetics and has increased flexibility over existi

21 scaffolding protein families and explore how mouse genetics and human genetics have intersected to ad

22 By using mouse genetics and in utero electroporation, we provide

23 Using the tools of mouse genetics and multielectrode recording, we demonstr

24 in contributes to AML using a combination of mouse genetics and primary patient samples.

25 ns of SL and SP cells using a combination of mouse genetics and retrograde labeling.

26 Here, we use mouse genetics and structure function analysis to invest

27 We combined mouse genetics and viral strategies to determine how the

28 Inbred strains are a distinctive feature of mouse genetics and we discuss their history, advantages

29 ree complementary disciplines: cell biology, mouse genetics, and dermatology.

30 Using an in vivo reporter of Shh signaling, mouse genetics, and systems modeling, we show that a spa

31 To test this, we devised a mouse genetics approach to silence glutamatergic signall

32 A mouse genetics approach was used to separate the matted

33 With the mouse genetics approach, we show that ablation of ACF7 i

34 With genome-editing and mouse genetics approach, we show that loss of function o

35 ew focuses on the last year's progress using mouse genetics as a tool to study intrinsic mechanisms o

36 cluded in this account are the beginnings of mouse genetics at the Bussey Institute, Columbia Univers

37 Recent advances in mouse genetics combined with neuroanatomical and electro

38 Mouse genetics combined with pharmacological probing in

39 s work will become a useful resource for the mouse genetics community to understand gene functions.

40 Mouse genetics confirmed the importance of canonical Wnt

41 d using high-resolution approaches combining mouse genetics, designer receptor exclusively activated

42 Here we combined mouse genetics, electrophysiology, and circuit mapping w

43 Here we use mouse genetics, electrophysiology, imaging and optogenet

44 Described are biochemical and mouse genetics experiments demonstrating that membrane-a

45 Mouse genetics experiments have defined crucial roles fo

46 xP technology is widely used in the field of mouse genetics for spatial and/or temporal regulation of

47 st anticipated and less heralded outcomes of mouse genetics has been to rediscover whole organism phy

48 The advent of modern mouse genetics has benefited many fields of diseased-bas

49 The use of mouse genetics has changed this approach and has rejuven

50 by genome scans of multi-case families, and mouse genetics has contributed to mapping and identifica

51 Mouse genetics has made crucial contributions to the und

52 Mouse genetics has revived this holistic view of physiol

53 Discoveries from human and mouse genetics have identified cytoskeletal and signalin

54 Recent progress in human and mouse genetics have led to the identification of novel c

55 Using mouse genetics, histology, liver damage assays and trans

56 Mouse genetics is a frequently used tool that allows dis

57 ystem that has been profoundly influenced by mouse genetics is skeletal physiology.

58 Using mouse genetics, our data reveal that in the complete abs

59 cale phenotyping projects such as the Sanger Mouse Genetics project are ongoing efforts to help ident

60 We describe a program, the Sanger Institute Mouse Genetics Project, that provides a step toward the

61 set from the Wellcome Trust Sanger Institute Mouse Genetics Project, where the treatment is a gene ab

62 se mutants generated by the Sanger Institute Mouse Genetics Project.

63 Strategies using reverse mouse genetics provide excellent methods for validating

64 erve as a valuable practical resource in the mouse genetics research community, and also accelerate t

65 In this in vivo study, we used complex mouse genetics, single-cell dye tracing, and behavioral

66 devise genomics, in situ hybridization, and mouse genetics strategies to uncover diverse allelic eff

67 We show using mouse genetics that zolpidem only needs to work on speci

68 Combining this technique with mouse genetics, the authors demonstrate that vesicle pri

69 Based on mouse genetics, the current model of melanophilin functi

70 o not negate the invaluable contributions of mouse genetics to biomedical science, but they do show t

71 to mutant phenotypes and unlocks the use of mouse genetics to determine functions of orthologous hum

72 We establish a precedent for applying mouse genetics to dissect viral entry and validate the r

73 Here, we use mouse genetics to exploit mosaicism to study cell segreg

74 a combination of high-content screening and mouse genetics to identify the miR-34/449 family as key

75 To enable the use of mouse genetics to investigate mechanisms underlying gang

76 e of Blood, Yan et al and Walz et al exploit mouse genetics to investigate the contribution of signal

77 We used mouse genetics to model how polygenic thresholds for the

78 use Vibrio cholerae neuraminidase (VCN) and mouse genetics to probe the molecular composition of the

79 eel, who have exploited the use of human and mouse genetics to revolutionize our understanding of the

80 Here we employ mouse genetics to show that RA derived from the paraxial

81 We used conditional mouse genetics to test the hypothesis that Purkinje cell

82 eir epithelial counterparts, we used in vivo mouse genetics tools to characterize four prostate strom

83 Using mouse genetics we demonstrate that female cells lacking

84 Using mouse genetics, we dissected the influence of IPC-derive

85 Using mouse genetics, we find that Pten and Tsc2 act synergist

86 ing a combination of functional genomics and mouse genetics, we identified a role for the transcripti

87 Using mouse genetics, we show increasing Hedgehog signaling vi

88 Here, using in vivo mouse genetics, we show that increasing UBE3A in the nuc

89 Here, using NGS and human and mouse genetics, we show that influenza A virus (IAV) rib

90 Using mouse genetics, we show that the pleuroperitoneal folds

91 Using mouse genetics, we show that the status of Nkx6.1 expres

92 Furthermore, using mouse genetics, we showed that CR cell density severely

93 13 issue of the JCI, Cutando et al. combined mouse genetics with classic mouse behavioral analysis to

94 ww.genes2cognition.org) that links human and mouse genetics with proteomic studies.

95 in various biological contexts, e.g. cancer, mouse genetics, yeast genetics, and analysis of brain im