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1 an factors (n = 348, approximately 1% of the mouse genome).
2 ed deletions at 17 sites in four loci of the mouse genome.
3 ll as introduction of polymorphisms into the mouse genome.
4 recombination, we totally deleted it in the mouse genome.
5 erminal repeats [sLTRs]) within the C57BL/6J mouse genome.
6 le-specific DNA methylation (ASM) map in the mouse genome.
7 and then mapping these sequences back to the mouse genome.
8 2 sgRNAs for human and 40,860 sgRNAs for the mouse genome.
9 iral superantigen (sag) genes encoded in the mouse genome.
10 ion of recombination initiation sites in the mouse genome.
11 he breaks are nonrandomly distributed in the mouse genome.
12 acid sequence, using targeted editing of the mouse genome.
13 s, but a SHOX ortholog does not exist in the mouse genome.
14 etic nephropathy since Nox5 is absent in the mouse genome.
15 l CpG clusters that were integrated into the mouse genome.
16 p to 50% of CpG dinucleotides throughout the mouse genome.
17 g sites, are under negative selection in the mouse genome.
18 d in the rhesus monkey genome but not in the mouse genome.
19 ted and partially redundant MMP genes in the mouse genome.
20 map the orthologous promoter regions in the mouse genome.
21 functionally diverse set of genes across the mouse genome.
22 at the desired genomic locations within the mouse genome.
23 e extent of molecular scars inflicted on the mouse genome.
24 have applied it to the analysis of the whole mouse genome.
25 lotypes at a targeted location (Hprt) in the mouse genome.
26 ites are stronger for the human than for the mouse genome.
27 f heterozygous congenic strains spanning the mouse genome.
28 disease-causing I130T mutant allele into the mouse genome.
29 edite the functional characterization of the mouse genome.
30 d-like CVC (Prd-L:CVC) homeobox genes in the mouse genome.
31 stigating the recombination landscape of the mouse genome.
32 of DNA microarrays representing the complete mouse genome.
33 t are located adjacent to one another in the mouse genome.
34 ith cis-regulatory motifs and modules in the mouse genome.
35 0 probes randomly distributed throughout the mouse genome.
36 utionarily conserved TF binding sites on the mouse genome.
37 we have inactivated this binding site in the mouse genome.
38 ing genomic region at insertion sites in the mouse genome.
39 ing evidence for periodicity in the human or mouse genome.
40 f antisense transcripts in the human and the mouse genome.
41 fficiency for transgene integration into the mouse genome.
42 otropic murine leukemia virus content of the mouse genome.
43 had only been partially characterized in the mouse genome.
44 s it enabled the routine manipulation of the mouse genome.
45 the hotspots lie in noncoding regions of the mouse genome.
46 igh-throughput, targeted manipulation of the mouse genome.
47 largest public collection of SNPs across the mouse genome.
48 n genomes, but appears to be absent from the mouse genome.
49 e brought in cis-regulatory modules into the mouse genome.
50 er unrepresented or totally absent, from the mouse genome.
51 e most recently active ERVs in the human and mouse genome.
52 ied, representing over 60% of the TFs in the mouse genome.
53 of 41 imprinting gene clusters known in the mouse genome.
54 ssed Pol II-transcribed genes throughout the mouse genome.
55 and neurodegeneration when knocked into the mouse genome.
56 elerate tumorigenesis when engineered in the mouse genome.
57 iated from endogenous L1 elements across the mouse genome.
58 maintain silencing of a large portion of the mouse genome.
59 the method to comprehensively map pAs in the mouse genome.
60 gth XP-MLV ERVs found in the sequenced C57BL mouse genome.
61 lite markers that are located throughout the mouse genome.
62 obacter sphaeroides and chromosome 16 of the mouse genome.
63 and its third year of investigations in the mouse genome.
64 sposase to insert a large transgene into the mouse genome.
65 at are expressed across the entire human and mouse genomes.
66 -responsive elements (EREs) in the human and mouse genomes.
67 annotation of bimodal genes in the human and mouse genomes.
68 ranscripts have been identified in human and mouse genomes.
69 annel subunits encoded by the human, rat and mouse genomes.
70 comparing the 129X1/SvJ and C57BL/6J inbred mouse genomes.
71 s comprise a large fraction of the human and mouse genomes.
72 d its targets-are prevalent in the human and mouse genomes.
73 Ri and CRISPRa libraries targeting human and mouse genomes.
74 l known and predicted genes in the human and mouse genomes.
75 the RE1 and used it to search the human and mouse genomes.
76 istribution of retroposed genes in human and mouse genomes.
77 tion of all predicted genes in the human and mouse genomes.
78 ults of this method applied to the human and mouse genomes.
79 port a novel DNA sequence motif in human and mouse genomes.
80 n evolution of GGI networks in the human and mouse genomes.
81 rtebrates, with an emphasis on the human and mouse genomes.
82 caspase that is found in the human, and not mouse, genome.
83 f their target genes in mammalian (human and mouse) genomes.
84 tforms: the short oligonucleotide Affymetrix Mouse Genome 430 2.0 GeneChip and a spotted cDNA array u
89 of this Mef2c neural crest enhancer from the mouse genome abolishes Endothelin induction of Mef2c exp
90 erformed genomic DNase I footprinting of the mouse genome across 25 cell and tissue types, collective
92 ely 1,200 odorant receptor (OR) genes in the mouse genome, an olfactory sensory neuron is thought to
95 th K-ras and p53 missense mutations into the mouse genome and established a more faithful genetic mod
96 human mutant huntingtin allele in the native mouse genome and gradually manifest symptoms late in lif
97 or site within intron 1, is conserved in the mouse genome and is widely expressed in mouse tissues.
98 ional Nna1-like genes were identified in the mouse genome and named cytosolic carboxypeptidase (CCP)
99 alysis to identify similar structures in the mouse genome and validated that these host structures al
100 inding sites for RBP motifs across human and mouse genomes and allows large scale querying of predict
101 enes have alternative promoters in human and mouse genomes and approximately 40% of promoters are tis
102 earch in the intergenic regions of human and mouse genomes and between the Burkholderia cenocepacia J
103 We tested GRAPeFoot using the human and mouse genomes and compared its performance to a set of p
105 -length DNA sequence motifs in the human and mouse genomes and sub-selected those with multiple recur
106 s approaching that of the finished human and mouse genomes and suggests an affordable roadmap to high
107 roximately 5000 processed pseudogenes in the mouse genome, and estimated that approximately 60% are l
108 The annotated sequences add up to 11% of the mouse genome, and include more than 70% of conserved non
109 ci of both proviruses were determined in the mouse genome, and integration site information was used
111 lf of the fragments showed homology with the mouse genome, and several sequences could be mapped to o
112 cisely mapped, and their distribution in the mouse genome appears random relative to transcription un
116 50 kb and map to over 95.6% of the reference mouse genome assembly (NCBIm37), covering 98.8% of Ensem
117 of 205 kb and map to 93.9% of the reference mouse genome assembly, covering 95.7% of Ensembl genes.
118 new interfaces for exploring regions of the mouse genome associated with cancer phenotypes and incor
121 e with mouse cDNA sequence (BC006598), using mouse genome browser, defines that PNRC2 gene, located o
122 provements include integration of the latest mouse genome build (GRCm38), improved access to comparat
124 ir genetic blueprint is still present in the mouse genome but is normally modified to the needs of th
127 extend our initial observation to the entire mouse genome by enriching for methylated DNA with the Me
128 e report a cloning-free method to target the mouse genome by pronuclear injection of a commercial Cas
129 sults suggest that SB Tn insertions into the mouse genome can be discriminated by DNA methylation mac
131 oadmap of hepatic PXR bindings in the entire mouse genome [chromatin immunoprecipitation (ChIP)-Seq].
135 mutagenesis projects, we calculate that the mouse genome contains approximately 3479-4825 embryonic
138 mutation into the homologous position in the mouse genome, creating mice that were heterozygous and h
146 currence, we surveyed the Jackson Laboratory Mouse Genome Database for knockout mouse strains and the
155 P-MLV ERVs map to the 95% of the laboratory mouse genome derived from P-MLV-infected M. m. domesticu
156 and are embedded in the 5% of the laboratory mouse genome derived from the Asian Mus musculus musculu
157 Individual deletion of these sites from the mouse genome did not alter expression of any of the gene
158 se divergent intervals span 19% of the house mouse genome, disproportionately aggregating on the X ch
160 d probes, optical microscopy techniques, and mouse genome editing hold great potential over the next
162 sceptible to elimination, as occurred in the mouse genome, either by chance or selection on one of th
163 nd molecular functions in both the human and mouse genomes even though the underlying sequences are n
164 ed survey of asynchronous replication in the mouse genome, excluding known asynchronously replicated
165 rehensive, unified, non-redundant catalog of mouse genome features generated by distilling gene predi
166 high-density BAC arrays covering >95% of the mouse genome for analysis of genomic patterns of aberrat
167 The binding motif was used to search the mouse genome for candidate 2.102 reactive allopeptides t
168 ces and/or the vast noncoding regions of the mouse genome for large-scale functional genomic analysis
173 of variation found in the inbred laboratory mouse genome has increased to 71 M SNPs and 12 M indels.
175 e immense challenge of annotating the entire mouse genome has stimulated the development of cutting-e
177 tes located in the intergenic regions of the mouse genome have been identified and molecularly charac
178 far, most of the functional sequences in the mouse genome have yet to be found, and the cis-regulator
179 the transcriptional output of the human and mouse genomes have revealed that there are many more tra
181 ee-dimensional organization of the human and mouse genomes in embryonic stem cells and terminally dif
182 the structure and dynamics of the human and mouse genomes in space and time with the goal of gaining
183 of parameters, our designs cover 78% of the mouse genome including many regions previously considere
184 tially in putative regulatory regions of the mouse genome, including CpG islands and non-exonic ultra
185 ave constructed a revised genetic map of the mouse genome, incorporating 10,195 single nucleotide pol
188 combined with genetic and phenotypic data in Mouse Genome Informatics (MGI) and made readily accessib
189 e Database (MGD) is a major component of the Mouse Genome Informatics (MGI) database resource and ser
193 gical context through integration with other Mouse Genome Informatics (MGI) resources and interconnec
194 type Ontology, EBI's Ontology Working Group, Mouse Genome Informatics and the Monarch Initiative amon
197 to generate mutations for every gene in the mouse genome is a first and essential step in this endea
199 implies that a near-complete coverage of the mouse genome is obtainable with this approach using 20 g
202 al collaborative efforts, a large portion of mouse genome is still poorly characterized for cellular
204 in a region conserved between the human and mouse genomes, it is possible to reduce the number of va
205 n variable-gene repertoire (2.7 Mb) into the mouse genome, leaving the mouse constant regions intact.
206 t1 transcripts or deletion of Mist1 from the mouse genome led to increased cell proliferation and a c
207 (Hsa21) are syntenic to three regions in the mouse genome, located on mouse chromosome 10 (Mmu10), Mm
211 ntly contains binding sites across the whole-mouse genome of 8 NRs identified in 40 chromatin immunop
213 coincided with the site of insertion in the mouse genome of the 335 bp of human DNA containing the H
214 principal technology for manipulation of the mouse genome, offering unrivalled accuracy in allele des
215 s concluded that paralogous genes within the mouse genome or the human genome are more functionally s
216 vidence of PXR DNA-binding signatures in the mouse genome, paving the path for predicting and further
219 xed models using worked examples from Sanger Mouse Genome Project focusing on Dual-Energy X-Ray Absor
220 etions (indels) present in SPRET/Ei from the Mouse Genomes Project (Wellcome Trust Sanger Institute)
224 te-specific integration are conserved in the mouse genome proximal to the recently identified Mbs85 g
225 g human FcgammaRIIA gene, not present in the mouse genome, recapitulated the human situation and resc
229 m line, occupy ~8% and ~10% of the human and mouse genomes, respectively, and affect their structure,
230 We identified 1301 and 997 RE1s inhuman and mouse genomes, respectively, of which >40% are novel.
232 d as transgenes, have been inserted into the mouse genome resulting in recapitulation of the unique p
239 pping the BAC end-sequences to the reference mouse genome sequence, was 7.1-fold across the autosomes
240 an histone acetylation island sequences with mouse genome sequences, we find that despite the conserv
241 ject was launched a decade ago to complement mouse genome sequencing efforts by promoting new phenoty
246 leases of the human, chimp, dog, macaque and mouse genomes, so that the results can be compared direc
247 than 50 class I MHC (MHCI) molecules in the mouse genome, some of which are now known to be expresse
248 2 PDZ domains and 93 peptides encoded in the mouse genome, successfully predicts interactions involvi
249 ne targeting allows precise tailoring of the mouse genome such that desired modifications can be intr
250 t the same respective loci in both human and mouse genomes, suggesting functional relevance and indic
251 SNP/genome ratio of CpG dinucleotides in the mouse genome support the "loss of CpG islands" model in
252 efficiency between two L1 variants from the mouse genome, T(FC) and T(Fspa), to a single amino acid
254 s and fewer losses of duplicate genes in the mouse genome than in the human genome, although the popu
255 troducing an X chromosome duplication in the mouse genome that contains Plp1 and five neighboring gen
257 million primer pairs targeting the human and mouse genomes that is readily usable for rapid and flexi
262 combining chromatin immunoprecipitation with mouse genome tiling array profiling, we identify Foxp3 b
263 used CRISPR-Cas9 gene editing to modify the mouse genome to encode two amino acids (positions 288 an
266 length Emu region (core plus MARsEmu) in the mouse genome to study VH gene repertoire and IgH express
268 oding regulatory sequences between human and mouse genomes to enable identification of conserved TFBS
269 annotated conserved islands in the human and mouse genomes to transcribed regions and to RFBSs, relyi
270 y (which allows for rapid engineering of the mouse genome) to quickly develop and evaluate antibodies
271 nnotated bimodal or switch-like genes in the mouse genome using a large collection of microarray data
272 sis of recent duplication content within the mouse genome using a whole-genome assembly comparison me
273 (TDMRs) were identified and localized in the mouse genome using second generation virtual RLGS (vRLGS
277 ion of cytosine methylation in 6.2 Mb of the mouse genome was tested using cohybridization of genomic
280 fying thousands of TERRA target sites in the mouse genome, we demonstrate that TERRA can bind both in
281 roughput sequencing of a 2.5Mb region of the mouse genome, we discovered a mutation in the grainyhead
282 robable transcription factors encoded in the mouse genome, we identify 12 novel transcription factors
283 ity candidate genes in a small region of the mouse genome, we noticed that many genes when knocked ou
284 istribute loxP sites randomly throughout the mouse genome, we present a simple but comprehensive meth
285 ated CpG islands and promoter regions in the mouse genome, we report the genome-wide DNA methylation
287 Using markers distributed throughout the mouse genome, we use a hybrid zone between two recently
289 affinities for core histone proteins in the mouse genome were not altered by removal of core histone
292 used a glioma model to identify loci in the mouse genome, which were repeatedly targeted by platelet
293 allelic variants of one or more genes in the mouse genome whose human orthologues should be important
294 vised sgRNA design rules to create human and mouse genome-wide libraries, perform positive and negati
295 In contrast to human population studies, our mouse genome-wide searches find loci that individually e
297 uman genome, so a thorough annotation of the mouse genome will be of significant value to understandi
299 e map of genetic variation in the laboratory mouse genome will provide insights into the evolutionary
300 positions of practically all hotspots in the mouse genome, with the exception of the pseudo-autosomal
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