コーパス検索結果 (1語後でソート)
通し番号をクリックするとPubMedの該当ページを表示します
1 ranscription state of a GR-induced promoter (mouse mammary tumor virus).
2 uses efficient -1 ribosomal frameshifting in mouse mammary tumor virus.
3 against infections by viruses like HIV-1 and mouse mammary tumor virus.
4 simplex viruses, human papillomaviruses, and mouse mammary tumor viruses.
5 nt-6 (eIF3e), a frequent integration site of mouse mammary tumor viruses.
6 is one of the frequent integration sites for mouse mammary tumor viruses.
8 nce to an endogenous superantigen encoded by mouse mammary tumor virus 8 (Mtv-8) by either deletion o
9 mphoid periphery by superantigens encoded by mouse mammary tumor viruses-8 and -9 in an MHC class II-
10 e binding sites for two of these viruses-the mouse mammary tumor virus (a retrovirus) and Machupo vir
11 lex class I (MHC-I), C-type lectins, and the mouse mammary tumor virus and herpesvirus saimiri supera
12 lucocorticoid-inducible enhancer-promoter of mouse mammary tumor virus and introduced it into the lef
13 nous retrovirus K (HERV-K) is related to the mouse mammary tumor virus and is present in the genomes
14 ukemogenic virus (TBLV) is highly related to mouse mammary tumor virus but induces rapidly appearing
15 n primary mammary adenocarcinomas arising in mouse mammary tumor virus-c-erbB2 transgenic mice harbor
16 ed Akt activation and survival in transgenic mouse mammary tumor virus-c-Myc mouse mammary carcinoma
17 sion of tumors initiated by an overexpressed mouse mammary tumor virus-c-myc transgene, which on its
18 ng a T47D cell line stably integrated with a mouse mammary tumor virus-chloramphenicol acetyltransfer
19 y dexamethasone occurs via the disruption of mouse mammary tumor virus chromatin structure and the re
21 ithelial cells isolated from Cav-1 null(-/-)/mouse mammary tumor virus-CR-1 transgenic animals showed
22 receptor type I (TGFbetaRI), either by using mouse mammary tumor virus-Cre mice or by delivering aden
26 l-type breast adenomas with lung metastases [mouse mammary tumor virus-driven polyoma virus middle T
28 ction by a process that depends upon neither mouse mammary tumor virus-encoded superantigens nor MHC
29 virus middle T antigen, under control of the mouse mammary tumor virus enhancer/promoter, was used to
32 mmary tumor virus-ErbB2 Ink4a/Arf(+/-) mice, mouse mammary tumor virus-ErbB2 Ink4a/Arf(wt) mammary tu
34 suppresses ER-negative tumor development in mouse mammary tumor virus-erbB2 transgenic mice with min
38 ivity equivalent to the entire 3'-end of the mouse mammary tumor virus genome, but further deletions
39 nized dexamethasone-induced activation of an mouse mammary tumor virus-glucocorticoid-response elemen
41 -MS) to directly analyze protein profiles in mouse mammary tumor virus/HER2 transgenic mouse frozen t
44 expression of cyclin D1 and wingless-related mouse mammary tumor virus integration site 10b (Wnt10b)
45 where they become inducible by wingless-type mouse mammary tumor virus integration site family member
46 he balance between mesenchymal Wingless-type Mouse Mammary Tumor Virus integration site family, membe
47 he timed secretion of Wingless-related MMTV (mouse mammary tumor virus) integration site 3 (WNT3) by
48 secrete the morphogen Wingless-related MMTV (mouse mammary tumor virus) integration site 3 (WNT3).
49 e roles of the canonical wingless-type MMTV (mouse mammary tumor virus) integration site family (WNT)
50 e that express Msx1 under the control of the mouse mammary tumor virus long terminal repeat (MMTV LTR
51 el cDNA was driven by the hormone-responsive mouse mammary tumor virus long terminal repeat (MMTV-LTR
52 on, whereas the sex-limited protein gene and mouse mammary tumor virus long terminal repeat do not.
53 ell-cell junctions, under the control of the mouse mammary tumor virus long terminal repeat promoter,
55 e promoter (NFI-Ad), and 2) the more complex mouse mammary tumor virus long terminal repeat promoter.
59 using a neutralizing CSF-1R antibody in the mouse mammary tumor virus long-terminal region-driven po
60 ilizing a Cre transgene under control of the mouse mammary tumor virus-long terminal repeat (MMTV-LTR
61 ey acidic protein (WAP) gene promoter or the mouse mammary tumor virus-long terminal repeat (MMTV-LTR
62 ing the KI-GSK3beta under the control of the mouse mammary tumor virus-long terminal repeat develop m
63 control of the whey acidic protein (WAP) or mouse mammary tumor virus-long terminal repeat promoters
64 d copies of the steroid responsive MMTV-LTR (mouse mammary tumor virus-long terminal repeat), we show
65 nesis, transgenic mice were generated with a mouse mammary tumor virus-long terminal repeat-driven, c
66 Transgenic expression of this gene using a mouse mammary tumor virus LTR enhancer causes extensive
67 ng the known positions of nucleosomes on the mouse mammary tumor virus LTR, and additionally, we char
72 ant I/LnJ mice, which control infection with mouse mammary tumor virus (MMTV) and murine leukemia vir
73 wn that mouse APOBEC3 restricts infection by mouse mammary tumor virus (MMTV) and murine leukemia vir
74 ivation function on two different reporters, mouse mammary tumor virus (MMTV) and prostate-specific a
75 transactivate the glucocorticoid-responsive mouse mammary tumor virus (MMTV) and TAT3 promoters in a
76 to have 95% to 97% nucleotide homology with mouse mammary tumor virus (MMTV) and with retrovirus seq
77 /N x WAP-TGF-beta1 mating were injected with mouse mammary tumor virus (MMTV) at 8-10 weeks of age.
79 clinical trial, have suggested that a human mouse mammary tumor virus (MMTV) causes primary biliary
80 east cancer cells stably transfected with an mouse mammary tumor virus (MMTV) chloramphenicol acetylt
86 he HIV TM-CT with sequences derived from the mouse mammary tumor virus (MMTV) envelope glycoprotein,
93 breast cancer with sequences similar to the mouse mammary tumor virus (MMTV) has been shown, but con
97 e mouse APOBEC3 (mA3) restricts infection by mouse mammary tumor virus (MMTV) in its natural host.
100 iously, we showed that IFN-gamma elicited by mouse mammary tumor virus (MMTV) infection in I/LnJ mice
112 liparous mice expressing Sim2s driven by the mouse mammary tumor virus (MMTV) long terminal repeat (L
113 he Cre recombinase gene under control of the mouse mammary tumor virus (MMTV) long terminal repeat or
114 rrepressor (SR) protein under control of the mouse mammary tumor virus (MMTV) long terminal repeat pr
115 to epithelial cells under the control of the mouse mammary tumor virus (MMTV) long terminal repeat pr
116 GFalpha), have been cloned downstream of the mouse mammary tumor virus (MMTV) long terminal repeat pr
117 iddle T (PyMT) oncogene under control of the mouse mammary tumor virus (MMTV) long-terminal repeat (M
118 biting 42% of the maximal effect of DEX in a mouse mammary tumor virus (MMTV) luciferase reporter tra
120 tein for the activation of B cells by either mouse mammary tumor virus (MMTV) or murine leukemia viru
121 The Gag protein of the murine retrovirus mouse mammary tumor virus (MMTV) orchestrates the assemb
122 ong been known for shedding large amounts of mouse mammary tumor virus (MMTV) particles in milk and f
123 ssion of the glucocorticoid receptor-induced mouse mammary tumor virus (MMTV) promoter and of the Tat
126 WI/SNF chromatin-remodeling complex, and the mouse mammary tumor virus (MMTV) promoter assembled in a
128 omatin structure using a tandem array of the mouse mammary tumor virus (MMTV) promoter driving a ras
131 RANK overexpression under the control of the mouse mammary tumor virus (MMTV) promoter in a transgeni
133 otic stress response was investigated on the mouse mammary tumor virus (MMTV) promoter in different c
134 ion of activated human SMO (SmoM2) under the mouse mammary tumor virus (MMTV) promoter in transgenic
135 those domains required for activation of the mouse mammary tumor virus (MMTV) promoter in two distinc
138 is study examines the mechanism by which the mouse mammary tumor virus (MMTV) promoter is repressed b
140 e dynamics of the interaction of PR with the mouse mammary tumor virus (MMTV) promoter reconstituted
142 Cdk2 fusion protein under the control of the mouse mammary tumor virus (MMTV) promoter results in mam
144 nsformation of the mammary gland we used the mouse mammary tumor virus (MMTV) promoter to target over
145 rticoid receptor-mediated stimulation of the mouse mammary tumor virus (MMTV) promoter, suggesting th
146 bition on steroid receptor activation of the mouse mammary tumor virus (MMTV) promoter, we found that
148 GR)-mediated transcriptional activation of a mouse mammary tumor virus (MMTV) promoter-driven lucifer
149 In vivo, deletion of Ppm1d in mice bearing mouse mammary tumor virus (MMTV) promoter-driven oncogen
158 ne-dependent activation of stably integrated mouse mammary tumor virus (MMTV) promoters, and this coa
160 WAP-p53(172H) in tumorigenesis, we performed mouse mammary tumor virus (MMTV) proviral mutagenesis.
162 es Moloney murine leukemia virus (MMuLV) and mouse mammary tumor virus (MMTV) showed cell surface exp
165 B leukemogenic virus (TBLV) is a variant of mouse mammary tumor virus (MMTV) that causes T-cell lymp
166 Type B leukemogenic virus is a variant of mouse mammary tumor virus (MMTV) that causes thymic lymp
167 We previously have shown that CDP represses mouse mammary tumor virus (MMTV) transcription in tissue
171 A novel common integration site for the mouse mammary tumor virus (MMTV) was identified (designa
172 tein (CDP) is a transcriptional repressor of mouse mammary tumor virus (MMTV), a betaretrovirus that
173 receptor alpha, c-myc, and those encoded by mouse mammary tumor virus (MMTV), a glucocorticoid-respo
174 This report shows for the first time that mouse mammary tumor virus (MMTV), a mammalian retrovirus
177 ludes Jaagsiekte sheep retrovirus (JSRV) and mouse mammary tumor virus (MMTV), as well as many endoge
178 MR structure of one such pseudoknot, that of mouse mammary tumor virus (MMTV), has revealed that it i
179 iency or mammary-specific deletion inhibited mouse mammary tumor virus (MMTV)- PyMT- and MMTV- Wnt1-d
181 n tumorigenesis as judged by findings with a mouse mammary tumor virus (MMTV)-c-rel transgenic mouse
183 gth Brca1 in mammary epithelial cells of the mouse mammary tumor virus (MMTV)-Cre Brca1 conditional e
184 of the first coding exon in the germ line of mouse mammary tumor virus (MMTV)-Cre transgenic mice.
188 nase (PI3-K)-Akt kinase signaling pathway in mouse mammary tumor virus (MMTV)-Her-2/neu NF639 mouse b
189 ering the role of HER2/Neu in breast cancer, mouse mammary tumor virus (MMTV)-Her2/neu transgenic mic
190 ontext of HER-2 overexpression, we generated mouse mammary tumor virus (MMTV)-Hoxb7 transgenic mice,
192 wth of mammary carcinomas arising in situ in mouse mammary tumor virus (MMTV)-infected female C3H/HeN
193 splants and decreases mammary cancer risk in mouse mammary tumor virus (MMTV)-infected females even a
198 ng growth factor beta (TGF-beta), we crossed mouse mammary tumor virus (MMTV)-Neu mice with MMTV-TGF-
200 ited breast tumor formation in xenograft and mouse mammary tumor virus (MMTV)-neu mouse models in a m
201 increased angiogenic signaling in the NeuYD [mouse mammary tumor virus (MMTV)-Neu(ndl)-YD5] mammary t
203 R2-evoked breast tumorigenesis, we generated mouse mammary tumor virus (MMTV)-NeuNT transgenic mice l
204 nesis, Apc(Min/+) mice were crossed with the mouse mammary tumor virus (MMTV)-Polyoma virus middle T
205 versely, generation of mammary tumors in the mouse mammary tumor virus (MMTV)-polyoma virus middle T
206 thology after crossing the mutation into the mouse mammary tumor virus (MMTV)-polyoma virus middle T
207 e activator E2F transcription factors in the mouse mammary tumor virus (MMTV)-polyomavirus middle T o
209 r (FTI) L-744,832 causes tumor regression in mouse mammary tumor virus (MMTV)-v-Ha-ras transgenic mic
210 p53 cancer mutant knock-in (R175H) mice and mouse mammary tumor virus (MMTV)-Wnt-1 transgenic (mWnt-
212 berrantly overexpressed in mammary tumors of mouse mammary tumor virus (MMTV)-Wnt1-transgenic mice an
218 78 markedly delays breast tumor formation in mouse mammary tumor virus (MMTV)/Neu + MMTV/transforming
221 ing the activated c-neu oncogene driven by a mouse mammary tumor virus (MMTV-neu) in vivo, we show th
222 using multiparous, ErbB2/Neu-overexpressing mouse mammary tumor virus (MMTV-Neu) mice have shown tha
224 terminal repeats (LTRs) of acquired somatic mouse mammary tumor viruses (MMTV) can activate juxtapos
229 olution and accelerated tumor progression in mouse mammary tumor virus/neu transgenic mice by inhibit
230 the carboxyl-terminal (Cys(2)HisCys) ZBD of Mouse Mammary Tumor Virus nucleocapsid protein (MMTV NCp
231 or transcriptional activation from chromatin mouse mammary tumor virus or endogenous promoters in viv
233 aling once tumors have developed, we use the mouse mammary tumor virus-Polyoma Middle T (MMTV-PyMT),
235 ancer development, we established cohorts of mouse mammary tumor virus-polyoma middle T (PyMT) PAR1(-
236 nic mouse model of breast cancer, MMTV-PyMT (mouse mammary tumor virus-polyoma middle T antigen), wit
237 lac altered breast cancer development in the mouse mammary tumor virus-polyoma middle T-antigen model
238 /-) mice were generated and crossed with the Mouse Mammary Tumor Virus-Polyoma Middle T-Antigen mouse
239 g for this antitumor response, we engineered mouse mammary tumor virus-polyoma virus middle T antigen
240 c resulted in increased pAkt levels in total mouse mammary tumor virus-polyoma virus middle T antigen
241 sed mouse models of Kiss1r gene knockout and mouse mammary tumor virus-polyoma virus middle T antigen
242 ammary tumorigenesis, we generated wild-type mouse mammary tumor virus/polyoma middle-T (WT/PyMT) and
243 Collagen(-/-) mice in the background of the mouse mammary tumor virus/polyoma virus middle T oncogen
244 rom spontaneous mammary tumors that arose in mouse mammary tumor virus-polyomavirus (MMTV-PyV) Middle
245 d-type (WT) and AIB1(-/-) mice harboring the mouse mammary tumor virus-polyomavirus middle T (PyMT) t
246 om both implantable (4T1) and autochthonous (mouse mammary tumor virus-polyomavirus middle T Ag (MMTV
248 et promoter, repressing transactivation from mouse mammary tumor virus, probasin, and prostate-specif
249 ssed phorbol-12-myristate-13-acetate-induced mouse mammary tumor virus promoter activity and phorbol
250 iption from an integrated steroid-responsive mouse mammary tumor virus promoter and also from an inte
251 moter and diminished activity on the complex mouse mammary tumor virus promoter compared with human G
252 ed progesterone-dependent PR activation of a mouse mammary tumor virus promoter in both prostate (PC-
253 Cortisol-dependent transactivation from the mouse mammary tumor virus promoter in cells lacking plas
254 fered in their ability to associate with the mouse mammary tumor virus promoter in organized chromati
255 us middle T antigen under the control of the mouse mammary tumor virus promoter in transgenic mice re
256 ice expressing MTA1 under the control of the mouse mammary tumor virus promoter long terminal repeat
257 n used to locate hSwi-Snf complexes bound to mouse mammary tumor virus promoter nucleosomal arrays, a
258 emodeling complex on individual, single-copy mouse mammary tumor virus promoter nucleosomal arrays.
259 (GR) and nuclear factor 1 (NF1) to bind the mouse mammary tumor virus promoter organized as regular
260 nd that LeTx repressed the activation of the mouse mammary tumor virus promoter related to overexpres
261 lucocorticoid receptor, to a tandem array of mouse mammary tumor virus promoter sites in live cells,
262 DAC inhibitors (KDACis) potently repress the mouse mammary tumor virus promoter through transcription
263 DeltaTR)-human MUC1 under the control of the mouse mammary tumor virus promoter to further examine th
264 G transgenic mice model under the control of mouse mammary tumor virus promoter was established and t
265 xpressing Polyoma middle T antigen under the mouse mammary tumor virus promoter were combined with mi
266 ining a unique C-->U substitution within the mouse mammary tumor virus promoter, one located within e
267 consisting of about 200 tandem repeats of a mouse mammary tumor virus promoter-driven ha-v-ras gene.
268 ous mammary cancers in mice transgenic for a mouse mammary tumor virus promoter-driven polyomavirus m
269 xpress ERalpha, were stably transfected with mouse mammary tumor virus promoter-luciferase (MMTV-LUC)
270 ction, as tested from a GR-responsive 330-bp mouse mammary tumor virus promoter-luciferase reporter c
283 ol experiments were performed on a mutant of mouse mammary tumor virus pseudoknot (VPK), for which an
284 ess the stability of the folded structure of mouse mammary tumor virus pseudoknot in the presence of
285 tisense oligonucleotides (ASOs) in the MMTV (mouse mammary tumor virus)-PyMT mouse mammary carcinoma
287 in these mice is conferred by an endogenous mouse mammary tumor virus superantigen (Mtv-7 sag) that
289 a5(+) T cells are tolerized to an endogenous mouse mammary tumor virus superantigen either by deletio
291 d a group of replication-competent exogenous mouse mammary tumor viruses that failed to induce mammar
292 dogenous nucleic acid sequence homology with mouse mammary tumor virus; therefore all viral insertion
294 AIB1(+/-), and AIB1(-/-) mice harboring the mouse mammary tumor virus/v-Ha-ras (ras) transgene that
296 DKK1), an antagonist of the wingless-related mouse mammary tumor virus (WNT) signaling pathway, is on
297 S-15 and MDA-MB-231 breast cancer cells, and mouse mammary tumor virus-Wnt-1 mammary tumor-derived ce
300 otent antitumor efficacy was shown using the mouse mammary tumor virus-Wnt1 tumor model under dosing
WebLSDに未収録の専門用語(用法)は "新規対訳" から投稿できます。