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1 infection with ECTV, the causative agent of mousepox.
2 nant type I IFN bp protects mice from lethal mousepox.
3 ovided significant protection against lethal mousepox.
4 capable of protecting susceptible mice from mousepox.
5 by long-lived memory CD8(+) T cells prevent mousepox, a disease caused by ectromelia virus, a close
6 ential for the resistance of C57BL/6 mice to mousepox, a disease caused by the Orthopoxvirus ectromel
10 rus infection of humans (smallpox) and mice (mousepox [ectromelia virus {ECTV}]) despite the lack of
11 ss II-restricted TCD4+ repertoire induced by mousepox (ECTV) infection and the functional profile of
12 uivalent ectromelia virus (ECTV; an agent of mousepox), encode immune response modifiers (IRMs) that
13 virus, which causes a smallpox-like disease (mousepox) in mice, was determined and allows for classif
14 EVM1 is abundantly expressed early during mousepox infection and is able to selectively bind CC ch
17 d NKT cells are not important in the C57BL/6 mousepox model and CD4+ and CD8+ T cells do not exhibit
18 We show that mice genetically resistant to mousepox (the mouse parallel of human smallpox) lose res
20 T(m)s, 61.3 to 63.7 degrees C), 8 strains of mousepox virus (T(m), 61.9 degrees C), and 8 strains of
22 ts are immunogenic in cowpox and ectromelia (mousepox) virus infections, and immunization with the im
24 ural resistance of nonimmune C57BL/6 (B6) to mousepox, we show that memory CD8(+) T cells of single s
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