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1 ions in the event of an outbreak of foot-and-mouth disease.
2 pment as effective vaccines against foot-and-mouth disease.
3 h CNS involvement and 29 with skin, eye, and mouth disease.
4 spreading the much more contagious foot and mouth disease.
5 ant animal pathogen responsible for foot-and-mouth disease.
6 o now of the epidemiology of hand, foot, and mouth disease.
7 ction are usually limited, such as hand-foot-mouth disease.
8 es to cause large outbreaks of hand foot and mouth disease across Asia, associated with neurological
9 DSLINE database was searched using the term "mouth diseases." Additional references were identified f
10 ential route of vaccination against foot-and-mouth disease and may be useful for eliciting protection
12 ng pathogens associated with hand, foot, and mouth disease and pediatric respiratory disease worldwid
14 is the major cause of severe hand, foot, and mouth disease and viral encephalitis in children across
15 sease), two datasets of serotype A (Foot-and-Mouth disease) and two datasets of influenza when the sc
16 7,200,092 probable cases of hand, foot, and mouth disease (annual incidence, 1.2 per 1000 person-yea
17 cts children, manifesting as hand, foot, and mouth disease, aseptic meningitis, poliomyelitis-like ac
18 ve samples) from suspected cases of foot-and-mouth disease collected from 65 countries between 1965 a
19 present an analysis of the current foot-and-mouth disease epidemic in Great Britain over the first 2
22 tainty as management proceeds, with foot-and-mouth disease (FMD) culling and measles vaccination as c
26 fox-hunting during the outbreak of foot-and-mouth disease (FMD) in 2001 to examine this issue and fo
27 e show that, during the outbreak of foot and mouth disease (FMD) in 2001, there was a significant red
28 essing IFNs can effectively control foot-and-mouth disease (FMD) in cattle and swine during experimen
30 In 1997, a devastating outbreak of foot-and-mouth disease (FMD) in Taiwan was caused by a serotype O
38 table vaccine candidates.IMPORTANCE Foot-and-mouth disease (FMD) is the most devastating disease affe
44 spongiform encephalopathy (BSE) and foot-and-mouth disease (FMD), and the advent of new technologies,
45 ith FMDV developed typical signs of foot-and-mouth disease (FMD), including fever, vesicular lesions,
46 ase of notifiable diseases, such as foot-and-mouth disease (FMD), these analyses provide important in
48 e major etiological agents of hand, foot and mouth disease (HFMD) and are often associated with neuro
49 an emerging pathogen causing hand, foot, and mouth disease (HFMD) and fatal neurological diseases in
50 (EV-A71) is a major cause of hand, foot, and mouth disease (HFMD) and is particularly prevalent in pa
51 mily and are major causes of hand, foot, and mouth disease (HFMD) and pediatric respiratory disease w
52 rus 71 (EV71) and associated hand, foot, and mouth disease (HFMD) are recognized as emerging public h
53 Epidemiology and etiology of hand, foot, and mouth disease (HFMD) based on large sample size or evalu
54 million/11.3 million) of all hand, foot, and mouth disease (HFMD) cases reported to WHO during 2010-2
57 To monitor search trends on Hand, Foot and Mouth Disease (HFMD) in Guangdong Province, China, we te
63 ary causes of the epidemics of hand-foot-and-mouth disease (HFMD) that affect more than a million chi
64 aviridae), a common cause of hand, foot, and mouth disease (HFMD), may also cause severe neurological
65 rus that causes outbreaks of hand, foot, and mouth disease (HFMD), primarily in the Asia-Pacific area
66 1) causes large outbreaks of hand, foot, and mouth disease (HFMD), with severe neurological complicat
68 y emerged as a major cause of hand, foot and mouth disease in children worldwide but no vaccine is av
70 cterised the epidemiology of hand, foot, and mouth disease in China on the basis of enhanced surveill
73 k is used to analyse an outbreak of foot-and-mouth disease in the UK, enhancing current understanding
75 rus infection and in the control of foot-and-mouth disease infection highlight the problems caused by
76 and animal (bovine brucellosis and foot-and-mouth disease) infections clearly differentiating infect
80 irus (FMDV), the causative agent of foot-and-mouth disease, is an Aphthovirus within the Picornavirid
81 irus (FMDV), the causative agent of foot-and-mouth disease, is an Apthovirus within the Picornavirida
82 irus), jump dispersal on a network (foot-and-mouth disease), or a combination of these (Sudden oak de
83 del run for the 2001 United Kingdom foot and mouth disease outbreak and compare the efficacy of diffe
85 tio-temporal model of the spread of foot-and-mouth disease, parameterized to match the 2001 UK outbre
87 aboratory data from cases of hand, foot, and mouth disease reported to the Chinese Center for Disease
89 problem using the specific case of foot-and-mouth disease spreading between farms using the formulat
90 he severe, atypical cases of hand, foot, and mouth disease that have been reported worldwide since 20
91 71 is a picornavirus causing hand, foot, and mouth disease that may progress to fatal encephalitis in
92 e performed experimental studies of foot-and-mouth disease transmission in cattle and estimated this
93 results on three datasets of SAT2 (Foot-and-Mouth disease), two datasets of serotype A (Foot-and-Mou
98 retion are induced by expression of foot-and-mouth disease virus (FMDV) 3C(pro) and that this require
101 a from transmission experiments for foot-and-mouth disease virus (FMDV) and African swine fever virus
102 sted in this study: one recognizing foot-and-mouth disease virus (FMDV) and another recognizing the 1
103 encephalomyocarditis virus (EMCV), foot-and-mouth disease virus (FMDV) and other picornaviruses comp
105 t to which the genetic diversity of foot-and-mouth disease virus (FMDV) arising over the course of in
107 t the leader proteinase (L(pro)) of foot-and-mouth disease virus (FMDV) blocks cap-dependent mRNA tra
110 tabilizing SAT2 vaccines.IMPORTANCE Foot-and-mouth disease virus (FMDV) causes a highly contagious ac
117 ifferential laboratory detection of foot-and-mouth disease virus (FMDV) from viruses that cause clini
124 ith clearance versus persistence of foot-and-mouth disease virus (FMDV) in micro-dissected compartmen
128 Here, we show that the picornavirus foot-and-mouth disease virus (FMDV) induces the formation of auto
129 lymphocyte subsets in recovery from foot-and-mouth disease virus (FMDV) infection in calves was inves
130 The pathogenesis of persistent foot-and-mouth disease virus (FMDV) infection was investigated in
132 investigation, the manner in which foot-and-mouth disease virus (FMDV) interacts with the innate and
133 t the leader proteinase (L(pro)) of foot-and-mouth disease virus (FMDV) interferes with the innate im
134 slation initiation dependent on the foot-and-mouth disease virus (FMDV) internal ribosome entry site
135 ors were constructed containing the foot-and-mouth disease virus (FMDV) internal ribosome entry site
139 he final steps in the maturation of foot-and-mouth disease virus (FMDV) is cleavage of the VP0 protei
140 reviously shown that replication of foot-and-mouth disease virus (FMDV) is highly sensitive to alpha/
142 The leader proteinase (L(pro)) of foot-and-mouth disease virus (FMDV) is involved in antagonizing t
144 animals with chemically inactivated foot-and-mouth disease virus (FMDV) is widely practiced to contro
148 feron [IFN-alpha/beta]) can inhibit foot-and-mouth disease virus (FMDV) replication in cell culture,
149 Ns have proven effective to inhibit foot-and-mouth disease virus (FMDV) replication in swine, a simil
150 rative analysis, of 103 isolates of foot-and-mouth disease virus (FMDV) representing all seven seroty
153 Adsorption and plaque formation of foot-and-mouth disease virus (FMDV) serotype A12 are inhibited by
154 human rhinovirus type 2 (HRV2) and foot-and-mouth disease virus (FMDV) to control the translation of
155 ly demonstrated that the ability of foot-and-mouth disease virus (FMDV) to form plaques in cell cultu
156 Adaptation of field isolates of foot-and-mouth disease virus (FMDV) to grow in cells in culture c
158 netic and epidemiological data in a Foot and Mouth Disease Virus (FMDV) veterinary outbreak in Englan
160 velopment of a serological test for foot-and-mouth disease virus (FMDV) which is quick and easy to us
163 rin receptors on cultured cells for foot-and-mouth disease virus (FMDV), and high-efficiency utilizat
165 for several diverse species such as foot-and-mouth disease virus (FMDV), hemagglutinin (HA) and neura
170 that the key replication enzyme of foot-and-mouth disease virus (FMDV), the RNA-dependent RNA polyme
171 esentatives of several serotypes of foot-and-mouth disease virus (FMDV), we discovered a putative cre
172 We have previously reported that Foot-and-mouth disease virus (FMDV), which is virulent for cattle
173 were used to map antigenic sites on foot-and-mouth disease virus (FMDV), which resulted in the identi
183 y the method to two UK epidemics of Foot-and-Mouth Disease Virus (FMDV): the 2007 outbreak, and a sub
185 ntibiotic resistance gene, with the foot and mouth disease virus 2A self-cleaving sequence placed bet
186 binding and explains the ability of foot-and-mouth disease virus 3C(pro) to cleave sequences containi
187 The X-ray crystal structure of the foot-and-mouth disease virus 3C(pro), mutated to replace the cata
189 he gene encoding the 2A protease of foot-and-mouth disease virus and then inserted in frame between t
193 nction derived from domain 3 of the foot-and-mouth disease virus internal ribosome entry site (IRES);
194 x 10(7) c.f.u./ml, indicating that foot-and-mouth disease virus IRES provides high-titer bicistronic
196 with a multiple cloning site 3' to foot-and-mouth disease virus IRES, was used to construct vectors
197 1) British field strain serotype of foot-and-mouth disease virus is a high-affinity ligand for alpha
200 se of core catalytic domains of the foot-and-mouth disease virus leader protease and coronavirus PLPs
201 hese methods to analyze data from a foot-and-mouth disease virus outbreak in the United Kingdom in 20
203 predict the antigenic similarity in foot-and-mouth disease virus strains and in influenza strains, wh
204 e genome-linked protein, VPg wheras foot-and-mouth disease virus uniquely encodes three copies of VPg
205 te responses against infection with foot-and-mouth disease virus was analyzed on consecutive 5 d foll
207 ed a salient genome segmentation of foot-and-mouth disease virus, an important animal pathogen whose
208 has been documented for poliovirus, foot-and-mouth disease virus, and coxsackievirus B3 and can lead
210 us family, including poliovirus and foot-and-mouth disease virus, are widespread pathogens of humans
211 QVLAQKVART (A20FMDV2), derived from foot-and-mouth disease virus, as a potent inhibitor of alphavbeta
213 nent G-H loop of the VP1 protein of foot-and-mouth disease virus, raised substantial levels of antipe
214 larger picornavirus IRESs (those of foot-and-mouth disease virus, rhinovirus, encephalomyocarditis vi
215 s, including all seven serotypes of foot-and-mouth disease virus, two serotypes of vesicular stomatit
223 ogens: classical swine fever virus; foot-and-mouth disease virus; vesicular stomatitis virus, New Jer
225 affinity ligands of alpha v beta6 (foot-and-mouth-disease virus, latency associated peptide), have a
228 ols during a nationwide epidemic of foot and mouth disease, which substantially delayed removal of TB
229 ent large-scale outbreaks of hand, foot, and mouth disease worldwide and represent a major etiologica
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