1 eviously identified arf2/hss, arf3/ett, arf5/mp, and arf7/nph4 mutants, suggesting that there are fun

2  a second C terminus CaM-binding domain (CB) mp (CBmp), in conjunction with IQmp, on single L-type Ca

3 ation in a glycopeptidolipid synthesis gene (mps) that results in hyperaggregation of cells and fully

4 us (SON), and in the anterior (ap-), medial (mp-), and lateral (lp-) parvocellular, and posterior mag

5 asing factor (CRF)-expressing dorsal-medial (mpd) neurons of the neonatal mouse hypothalamus.

6 ell types, bundle sheath (bs) and mesophyll (mp), which provide the foundation for this highly effici

7 ormed1 (pin1), pinoid (pid), and monopteros (mp), which are defective in either auxin transport or au

8 lings that closely phenocopy the monopteros (mp) mutant phenotype, with an absence of roots and defec

9 E540A) and bred it onto the MPS VII (gus(mps/mps)) background.

10            The murine model MPS VII (gus(mps/mps)) has been very well characterized and used extensiv

11 acteristics of the original MPS VII (gus(mps/mps)) mouse.

12 I mice carrying a deletion mutation (gus(mps/mps)).

13 the highest densities in the medial pallium (mp; homolog of the mammalian hippocampus), accumbens, an

14 cretory neurons in the medial parvicellular (mp) part of the paraventricular hypothalamic nucleus (PV

15 also significantly reduced GR-ir in the POA, mp, MeA and BNST, but not in the rostral pars distalis.

16 ys significantly decreased GR-ir in the POA, mp, medial amygdala (MeA), BNST, and rostral pars distal

17  coat protein (cp) and the movement protein (mp), as well as inside Rep and cp and in the short inter