ライフサイエンスコーパス: msec

1 error of the mean]) than at 0 degrees (10.0 msec +/- 0.7, P < .001) or 90 degrees (9.9 msec +/- 0.4,

2 .001) and 40% +/- 10 lower in canines (23.0 msec +/- 4.0 vs 39.3 msec +/- 2.5, P < .001).

3 scle tissue: T1, 1417 msec +/- 106; T2, 31.0 msec +/- 2.4; T2*, 11.3 msec +/- 1.7; and ADC, 1.40 x 10

4 uscle tissue: T1, 1386 msec +/- 88; T2, 32.0 msec +/- 4.3; T2*, 10.8 msec +/- 0.8; ADC, 1.39 x 10(-3)

5 MOLLI had similar precision to ShMOLLI (4.0 msec vs 5.6 msec; P = .07) but higher precision than SAP

6 f interest in an osteochondral lesion = 50.0 msec +/- 10.2) in comparison to adjacent intact cartilag

7 findings, mean T2 of both hemorrhagic (62.0 msec +/- 4.9) and nonhemorrhagic (71.7 msec +/- 7.3) inf

8 the 3D UTE technique (0.13-0.16 vs 1.32-3.03 msec) (P=.14, analysis of variance).

9 sotropic spatial resolution; echo time, 0.08 msec) were performed at 3 T.

10 rol subjects (1185.3 msec +/- 49.3 vs 1089.1 msec +/- 44.9, respectively; P < .001).

11 T1(rho) values were 14.7 msec +/- 5.5, 16.1 msec +/- 6.6, and 19.3 msec +/- 7.6 for the healthy, mil

12 s of recipient mice (mean, 21.7 msec vs 27.1 msec, respectively; P = .0444).

13 ts revealed a mean standard deviation of 4.1 msec (0.412%).

14 lative to mean T2 of remote myocardium (52.1 msec +/- 4.8) by 18% +/- 9 and 38% +/- 13, respectively

15 ximately 2 nm) and temporal (approximately 1 msec) localization accuracy.

16 red by BF electrical stimulation within 5-10 msec.

17 Sokolow-Lyon voltage (beta = 15.1 microV/10 msec, P = .004), lower QRS Cornell voltage (beta = 9.2 m

18 er QRS Cornell voltage (beta = 9.2 microV/10 msec, P = .031), and shorter QRS duration (beta = 0.16 m

19 nd shorter QRS duration (beta = 0.16 msec/10 msec, P = .049).

20 in both regions at each of 11 TEs spaced 10 msec apart were rated by seven neuroradiologists by usin

21 repetition time, 600-750 msec; echo time, 10 msec; in-plane resolution, 196 mm).

22 in the evoked potential ranged from 2 to 10 msec.

23 anscranial focused ultrasound (551.5 kHz, 10-msec bursts, 2-Hz pulse repetition frequency, 2 minute s

24 d in the bilateral hippocampus (1.68 MHz, 10-msec bursts, 1-Hz burst repetition frequency, 120-second

25 Most movements were initiated within 33-100 msec after stimulus onset.

26 right superior temporal gyrus [STG]) and 100 msec (N100 at Cz, M100 at left and right STG).

27 Ratio scores at 50 msec and 100 msec and S1 amplitude predicted variance in attention (p

28 illations that occurred within the first 100 msec after TMS, particularly in a cluster of electrodes

29 0 healthy control subjects to both long (100 msec) and short (50 msec) duration deviant sounds.

30 ion at 3.0 T and echo times of less than 100 msec, an average T2 value per metabolite should suffice

31 Abnormal 50- and 100-msec event-related brain activity derived from paired-cl

32 formance in patients support 50-msec and 100-msec ratio and amplitude scores as clinically significan

33 ardium across all examined subjects was 1031 msec +/- 33 (standard deviation).

34 ed from 1286 msec +/- 99 at baseline to 1077 msec +/- 50 at 6 months (P < .0001), whereas T2 decrease

35 d enabled temporal resolution as high as 108 msec.

36 arterial transit times of 750, 950, and 1100 msec to consecutive segments of the middle cerebral arte

37 92 and 60 msec +/- 19; skeletal muscle, 1100 msec +/- 59 and 44 msec +/- 9; and fat, 253 msec +/- 42

38 for native T1 relaxation times (cutoff, 1140 msec) were equivalent compared with those of the establi

39 th SASHA (13 msec; P < .05) and SAPPHIRE (12 msec; P < .05).

40 val of 240 msec or more, QRS duration of 120 msec or more, or second-degree or third-degree atriovent

41 pecified subgroup with a QRS interval of 120 msec or more, the peak oxygen consumption increased in t

42 include a prolonged QRS interval (> or =120 msec), in addition to other functional criteria.

43 uction block (IAB) (P wave duration, >or=120 msec), 46 had severe coronary artery disease (CAD) (>or=

44 ubgroup with a QRS interval of less than 120 msec (P=0.45).

45 lse inhibition was most prominent at the 120-msec interstimulus interval, which was not correlated to

46 msec +/- 77 and 47 msec +/- 10; spleen, 1232 msec +/- 92 and 60 msec +/- 19; skeletal muscle, 1100 ms

47 of infarcted myocardium decreased from 1286 msec +/- 99 at baseline to 1077 msec +/- 50 at 6 months

48 sec) and MOLLI (44 msec) than with SASHA (13 msec; P < .05) and SAPPHIRE (12 msec; P < .05).

49 action of 30% or less, a QRS duration of 130 msec or more, and New York Heart Association class I or

50 35% or less, a QRS duration of less than 130 msec, and echocardiographic evidence of left ventricular

51 failure and a QRS duration of less than 130 msec, CRT does not reduce the rate of death or hospitali

52 - 205 and 60 msec +/- 21; renal cortex, 1314 msec +/- 77 and 47 msec +/- 10; spleen, 1232 msec +/- 92

53 rsion preparations at inversion time 1 = 135 msec and inversion time 2 = 95 msec) sequences.

54 for mature skeletal muscle tissue: T1, 1386 msec +/- 88; T2, 32.0 msec +/- 4.3; T2*, 10.8 msec +/- 0

55 rence erector spinae muscle tissue: T1, 1417 msec +/- 106; T2, 31.0 msec +/- 2.4; T2*, 11.3 msec +/-

56 than in gray matter (GM; 221 msec +/- 7, 143 msec +/- 4, and 205 msec +/- 8, respectively) but not di

57 s of 174 msec +/- 11, 98 msec +/- 3, and 143 msec +/- 5, respectively.

58 ites were evident at 92 msec (C1) and at 146 msec (N1b).

59 group of patients with a QRS duration of 150 msec or more.

60 The mean arterial transit time was 1538 msec +/- 123 (standard deviation) in the pediatric cohor

61 .031), and shorter QRS duration (beta = 0.16 msec/10 msec, P = .049).

62 ues in white matter (WM; 298 msec +/- 6, 162 msec +/- 1, and 222 msec +/- 4 for NAA, Cr, and Cho, res

63 T1, T2, and ADC from cancer (mean, 1628 msec +/- 344, 73 msec +/- 27, and 0.773 x 10(-3) mm(2)/s

64 tively) or hypointense (296 msec +/- 27, 163 msec +/- 12, and 199 msec +/- 12, respectively) lesions,

65 th resulting temporal resolution of 82.5-165 msec.

66 ard error) for NAA (250 msec +/- 9), Cr (166 msec +/- 3), and Cho (221 msec +/- 6); shape was charact

67 nd T2 in metastatic adenocarcinoma were 1673 msec +/- 331 and 43 msec +/- 13, respectively, significa

68 those from NPZ (mean, 2247 msec +/- 450, 169 msec +/- 61, and 1.711 x 10(-3) mm(2)/sec +/- 0.269) (P

69 P < .001) and fetal brain (3.7 msec vs 7.17 msec; P = .02), whereas there was no significant differe

70 tion) and 31 msec +/- 6; renal medulla, 1702 msec +/- 205 and 60 msec +/- 21; renal cortex, 1314 msec

71 by full width at half maximum values of 174 msec +/- 11, 98 msec +/- 3, and 143 msec +/- 5, respecti

72 erence in the fetal liver (2.72 msec vs 3.18 msec; P = .47).

73 ferent from isointense (313 msec +/- 24, 188 msec +/- 12, and 238 msec +/- 17, respectively) or hypoi

74 e (296 msec +/- 27, 163 msec +/- 12, and 199 msec +/- 12, respectively) lesions, except for the Cho v

75 the same area before the transfer (T1, 137.2 msec +/- 39.3 and 239.5 msec +/- 17.6, respectively; P <

76 lower in patients (15.9 msec +/- 4.5 vs 35.2 msec +/- 2.1, P < .001) and 40% +/- 10 lower in canines

77 psychomotor speed (change in raw score, 5.2 msec and 0.9 msec, respectively).

78 ely; P = .006) and increased T2 values (64.2 msec +/- 10.9 vs 76.2 msec +/- 13.7, respectively; P = .

79 reased T2 values (64.2 msec +/- 10.9 vs 76.2 msec +/- 13.7, respectively; P = .01) in the proximal ul

80 had an ADC nadir of less than 0.83 microm(2)/msec (70.3% decrease from baseline), whereas 94% of cont

81 aseline] to 1.79 +/- 0.10 [hydration] mum(2)/msec, P = .0059; or 1.86 +/- 0.07 [furosemide] mum(2)/ms

82 .0059; or 1.86 +/- 0.07 [furosemide] mum(2)/msec, P = .0094).

83 iron overload (defined as midseptal T2* < 20 msec on any prior cardiac MR images).

84 ated by a T2* decay constant of less than 20 msec, was 5.0% +/- 4.9 (standard deviation) at the level

85 /cm, and 100 V/cm), and durations (10 and 20 msecs) of shocks delivered.

86 tal data, ranging from approximately 150-200 msec.

87 Inflow temporal resolution of 200 msec was demonstrated, revealing arterial transit times

88 (GM; 221 msec +/- 7, 143 msec +/- 4, and 205 msec +/- 8, respectively) but not different from isointe

89 2-KI mice (wildtype = 24.25 msec, KI = 25.22 msec; p = .011).

90 ec +/- 9), Cr (166 msec +/- 3), and Cho (221 msec +/- 6); shape was characterized by full width at ha

91 ficantly longer than in gray matter (GM; 221 msec +/- 7, 143 msec +/- 4, and 205 msec +/- 8, respecti

92 (WM; 298 msec +/- 6, 162 msec +/- 1, and 222 msec +/- 4 for NAA, Cr, and Cho, respectively) were all

93 ndard deviation, 2242 msec +/- 116), T2 (224 msec +/- 18), and T2* (33.3 msec +/- 3.6) values and ADC

94 higher T1 (mean +/- standard deviation, 2242 msec +/- 116), T2 (224 msec +/- 18), and T2* (33.3 msec

95 cantly lower than those from NPZ (mean, 2247 msec +/- 450, 169 msec +/- 61, and 1.711 x 10(-3) mm(2)/

96 e (313 msec +/- 24, 188 msec +/- 12, and 238 msec +/- 17, respectively) or hypointense (296 msec +/-

97 6 months (</=2.0 V at a pulse width of 0.24 msec and an increase of </=1.5 V from the time of implan

98 rhythm other than sinus, PR interval of 240 msec or more, QRS duration of 120 msec or more, or secon

99 nd controls for placenta (5.25 msec vs 11.25 msec; P < .001) and fetal brain (3.7 msec vs 7.17 msec;

100 s observed in Tph2-KI mice (wildtype = 24.25 msec, KI = 25.22 msec; p = .011).

101 n IUGR cases and controls for placenta (5.25 msec vs 11.25 msec; P < .001) and fetal brain (3.7 msec

102 values for pulse duration and amplitude, 25 msec and 1 muT, respectively.

103 e CEST parameters of saturation duration (25 msec) and amplitude (1 muT) were chosen on the basis of

104 by T2*-weighted gradient-echo (echo time, 25 msec; repetition time, 944 msec) imaging at 232-microm i

105 ean values (+/- standard error) for NAA (250 msec +/- 9), Cr (166 msec +/- 3), and Cho (221 msec +/-

106 y conditions at a temporal resolution of 252 msec.

107 msec +/- 59 and 44 msec +/- 9; and fat, 253 msec +/- 42 and 77 msec +/- 16, respectively.

108 combination of a gantry rotation time of 275 msec, wide volume coverage, iterative reconstruction, au

109 relaxation times of 840 msec +/- 113 and 28 msec +/- 3 (P < .0001 and P < .01) and those in hepatic

110 ge damage defined with a T2* threshold of 28 msec and less.

111 At ROC curve analysis, a T2* value of 28 msec was identified as the threshold for damaged cartila

112 A short echo-time (29 msec) single-voxel (1-cm(3)) proton (hydrogen 1 [(1)H])

113 ec +/- 17, respectively) or hypointense (296 msec +/- 27, 163 msec +/- 12, and 199 msec +/- 12, respe

114 Regional T2 values in white matter (WM; 298 msec +/- 6, 162 msec +/- 1, and 222 msec +/- 4 for NAA,

115 ec +/- 106; T2, 31.0 msec +/- 2.4; T2*, 11.3 msec +/- 1.7; and ADC, 1.40 x 10(-3) mm(2)/sec +/- 0.03)

116 myocarditis than in control subjects (1185.3 msec +/- 49.3 vs 1089.1 msec +/- 44.9, respectively; P <

117 .7 msec +/- 5.5, 16.1 msec +/- 6.6, and 19.3 msec +/- 7.6 for the healthy, mild OA, and severe OA gro

118 /- 116), T2 (224 msec +/- 18), and T2* (33.3 msec +/- 3.6) values and ADCs (1.53 x 10(-3) mm(2)/sec +

119 mes for normal cartilage (Beck score 1, 35.3 msec +/- 7.0) were significantly higher than those for c

120 lower in canines (23.0 msec +/- 4.0 vs 39.3 msec +/- 2.5, P < .001).

121 ) was associated with longer QTc (beta = 4.3 msec, P = .031).

122 - 2.9 [standard deviation]; range, 33.9-46.3 msec) were significantly lower than those of patients wh

123 ec +/- 26, P < .001) and mean PFR time (68.3 msec +/- 26.8 vs 103.7 msec +/- 41.8, P < .01).

124 ed mean PEC time (40.4 msec +/- 11.8 vs 91.3 msec +/- 26, P < .001) and mean PFR time (68.3 msec +/-

125 se in dispersion of repolarization (25 +/- 3 msecs to 37 +/- 7 msecs; p = .037).

126 -echo GRE spiral-out imaging (TE, 3.3 and 30 msec; flip angle, 72 degrees ; n = 5).

127 e and non-successive targets presented at 30 msec/item, suggesting that--regardless of whether they o

128 he AB and the success of target-cueing at 30 msec/item.

129 s were presented sequentially at rates of 30 msec/item or 90 msec/item.

130 Next, GRE echo-planar imaging (TE, 30 msec; flip angle, 90 degrees ; n = 22) was used to colle

131 GRE) echo-planar imaging (echo time [TE], 30 msec; flip angle, 90 degrees ; n = 10), small-flip-angle

132 a maximum exit velocity of 60 m/sec in a 30-msec burst.

133 , TS+ tones evoked a positive shift (200-300 msec) at bilateral frontal electrodes.

134 t (466 msec +/- 14, 406 msec +/- 59, and 303 msec +/- 53, respectively; P < .001).

135 n healthy volunteers (745 msec +/- 65 and 31 msec +/- 6, P < .0001 and P = .021, respectively).

136 745 msec +/- 65 (standard deviation) and 31 msec +/- 6; renal medulla, 1702 msec +/- 205 and 60 msec

137 on and temporal windows of 110, 210, and 310 msec further reduced volume CT dose index to 9.1-25.1 mG

138 times of lymphatic fluid at 3.0 T were 3100 msec +/- 160 (range, 2930-3210 msec; median, 3200 msec)

139 vely) but not different from isointense (313 msec +/- 24, 188 msec +/- 12, and 238 msec +/- 17, respe

140 +/- 160 (range, 2930-3210 msec; median, 3200 msec) and 610 msec +/- 12 (range, 598-618 msec; median,

141 0 T were 3100 msec +/- 160 (range, 2930-3210 msec; median, 3200 msec) and 610 msec +/- 12 (range, 598

142 oint-resolved spectroscopy (PRESS) with a 35-msec echo time.

143 C, TS+ tones evoked a negative shift (60-350 msec) at right temporal electrodes relative to Baseline.

144 volved contralateral brain were the same (36 msec+/-4 [standard deviation] vs 36 msec+/-5, respective

145 same (36 msec+/-4 [standard deviation] vs 36 msec+/-5, respectively), which might suggest similar oxy

146 peaks in the liver of three subjects was 36 msec+/-8.

147 greater than 5% was 0.84 at a cutoff of 383 msec.

148 acceptable pacing threshold (</=2.0 V at 0.4 msec) and an acceptable sensing amplitude (R wave >/=5.0

149 Mean T2 values were 11.4 msec +/- 3.9, 13.5 msec +/- 4.7, and 16.6 msec +/- 8.2 f

150 -T clinical MR system (repetition time, 11.4 msec; echo time [first echo], 3.7 msec; 18,000 projectio

151 eled control cells (T2 in vivo, 15.4 vs 24.4 msec; P < .05) and could be tracked in osteochondral def

152 F patients had prolonged mean PEC time (40.4 msec +/- 11.8 vs 91.3 msec +/- 26, P < .001) and mean PF

153 test attainable echo time of approximately 4 msec (T2* mapping).

154 on increased from 30 +/- 5 msecs to 57 +/- 4 msecs in the control group at 15 mins of ischemia.

155 heme B, the first IP echo (approximately 2.4-msec TE) and the third OP echo (approximately 5.8-msec T

156 th a temporal resolution of approximately 40 msec.

157 me was increased from approximately 20 to 40 msec, the measured cerebral microbleed volume increased

158 y increased (high-frequency power: 54 vs. 40 msec(2), p = 0.005; high-frequency normalized power: 23.

159 , TS+ tones evoked a positive shift (100-400 msec) at right frontotemporal electrodes.

160 gadolinium enhancement (466 msec +/- 14, 406 msec +/- 59, and 303 msec +/- 53, respectively; P < .001

161 adenocarcinoma were 1673 msec +/- 331 and 43 msec +/- 13, respectively, significantly different from

162 19; skeletal muscle, 1100 msec +/- 59 and 44 msec +/- 9; and fat, 253 msec +/- 42 and 77 msec +/- 16,

163 s lower with ShMOLLI (62 msec) and MOLLI (44 msec) than with SASHA (13 msec; P < .05) and SAPPHIRE (1

164 11.55 msec for male rat stem cells vs 15.45 msec for sex-matched rat stem cells; P = .02 and P = .04

165 mm(3); temporal resolution, approximately 45 msec) in 20 patients with hepatic cirrhosis, 20 healthy

166 20-kHz bandwidth, 0.81-kHz amplitude, and 45-msec echo time.

167 r QTc prolongation, defined as a QTc >/= 450 msec in men and >/= 460 msec in women, was 1.17 (95% CI:

168 ned as a QTc >/= 450 msec in men and >/= 460 msec in women, was 1.17 (95% CI: 1.01, 1.35) for a 1-SD

169 ith evident late gadolinium enhancement (466 msec +/- 14, 406 msec +/- 59, and 303 msec +/- 53, respe

170 on were 5.9%, 19.6%, 306.79 msec, and 162.47 msec, respectively.

171 /- 21; renal cortex, 1314 msec +/- 77 and 47 msec +/- 10; spleen, 1232 msec +/- 92 and 60 msec +/- 19

172 ve was stimulated hourly (30 pulses/min, 1.5 msec duration, 17.0 +/- 4.4 mA) during the surgery.

173 Mean T2 values were 11.4 msec +/- 3.9, 13.5 msec +/- 4.7, and 16.6 msec +/- 8.2 for the healthy, mil

174 gh-frequency normalized power: 23.5 vs. 20.5 msec, p = 0.001; root mean square successive differences

175 transfer (T1, 137.2 msec +/- 39.3 and 239.5 msec +/- 17.6, respectively; P < .001).

176 on of repolarization increased from 30 +/- 5 msecs to 57 +/- 4 msecs in the control group at 15 mins

177 heme A, the first OP echo (approximately 1.5-msec TE) and the second IP echo (approximately 4.9-msec

178 th truncation (GOIA-W[16,4]) pulses with 3.5-msec duration, 20-kHz bandwidth, 0.81-kHz amplitude, and

179 es with six widely spaced echo times (in 3.5-msec increments) were acquired to correlate R2* and musc

180 for native T1 mapping ( approximately 25-50 msec; P > .05) and ECV quantification ( approximately 0.

181 t-related potential maximal approximately 50 msec after the commission of errors.

182 aired-click ratio scores were obtained at 50 msec (P50 evoked potential at Cz, M50 at left and right

183 Ratio scores at 50 msec and 100 msec and S1 amplitude predicted variance in

184 bjects to both long (100 msec) and short (50 msec) duration deviant sounds.

185 CT at a temporal resolution of less than 50 msec.

186 cognitive performance in patients support 50-msec and 100-msec ratio and amplitude scores as clinical

187 to stimulate the brain noninvasively with 50-msec bursts at a 5% duty cycle, repetition frequency of

188 ith long stimulus trains ( approximately 500 msec).

189 ted to mechanical impact (40% strain for 500 msec).

190 g events with an approximate duration of 500 msec.

191 terstimulus intervals ranging from 30 to 500 msec.

192 A corrected QT interval >500 msecs was considered prolonged.

193 ex, multijoint movements revealed with a 500-msec duration intracortical stimulation in rat motor cor

194 was delivered to the eye at the end of a 500-msec tone; in the trace paradigm, the puff was delivered

195 At the 500-msec interstimulus interval, prepulse inhibition was sig

196 on was measured using paired clicks with 500-msec interstimulus intervals.

197 homotor speed (scores range from 100 to 5100 msec, with faster times representing better performance)

198 meniscus covered a range of values (247-515 msec) and patterns (homogeneous and focal variations).

199 ould be visualized with high temporal (21.54 msec per image) and sufficient spatial (</= 3 mm) resolu

200 l-flip-angle GRE echo-planar imaging (TE, 54 msec; flip angle, 35 degrees ; n = 7), or dual-echo GRE

201 n, 10.72 msec for human stem cells and 11.55 msec for male rat stem cells vs 15.45 msec for sex-match

202 an those of older MIs (925 msec+/-169 vs 551 msec+/-107, P<.001).

203 ereas T2 decreased from 84 msec +/- 10 to 58 msec +/- 4 (P < .0001).

204 .4 msec +/- 3.9, 13.5 msec +/- 4.7, and 16.6 msec +/- 8.2 for the healthy, mild OA, and severe OA gro

205 P = .021) in patients and by 8% +/- 5 (44.6 msec +/- 4.8, P = .012) in canines.

206 precession, with temporal resolution of 5.6 msec at 1.5 T).

207 imilar precision to ShMOLLI (4.0 msec vs 5.6 msec; P = .07) but higher precision than SAPPHIRE (6.8 m

208 bleaching light energy at a pulse width of 6 msec and a duty cycle of 50%.

209 msec +/- 10; spleen, 1232 msec +/- 92 and 60 msec +/- 19; skeletal muscle, 1100 msec +/- 59 and 44 ms

210 - 6; renal medulla, 1702 msec +/- 205 and 60 msec +/- 21; renal cortex, 1314 msec +/- 77 and 47 msec

211 ion time, 10-12 min; temporal resolution, 60 msec) or one cardiac cycle and time-of-flight (TOF) MR a

212 , 2930-3210 msec; median, 3200 msec) and 610 msec +/- 12 (range, 598-618 msec; median, 610 msec), res

213 sec +/- 12 (range, 598-618 msec; median, 610 msec), respectively.

214 al SSFSE imaging decreasing from 1358 to 613 msec for sagittal acquisitions and from 1494 to 621 msec

215 00 msec) and 610 msec +/- 12 (range, 598-618 msec; median, 610 msec), respectively.

216 uracy in phantoms was lower with ShMOLLI (62 msec) and MOLLI (44 msec) than with SASHA (13 msec; P <

217 r sagittal acquisitions and from 1494 to 621 msec for coronal oblique acquisitions.

218 Adding T2 information (98 msec+/-7 vs 68 msec+/-2, respectively) alone yields results that sugges

219 d mean PFR time (68.3 msec +/- 26.8 vs 103.7 msec +/- 41.8, P < .01).

220 ps were found: Mean T1(rho) values were 14.7 msec +/- 5.5, 16.1 msec +/- 6.6, and 19.3 msec +/- 7.6 f

221 ilage with early changes (Beck score 2, 20.7 msec +/- 6.0) and cartilage with more advanced degenerat

222 varial defects of recipient mice (mean, 21.7 msec vs 27.1 msec, respectively; P = .0444).

223 s 11.25 msec; P < .001) and fetal brain (3.7 msec vs 7.17 msec; P = .02), whereas there was no signif

224 time, 11.4 msec; echo time [first echo], 3.7 msec; 18,000 projection angles; imaging volume, 260 x 26

225 to adjacent intact cartilage (mean T2 = 32.7 msec +/- 4.2).

226 of abnormalities and higher T2 values (48.7 msec +/-4.35 vs 45.8 msec +/-3.93; P < .001) than did su

227 (62.0 msec +/- 4.9) and nonhemorrhagic (71.7 msec +/- 7.3) infarctions in canines was elevated relati

228 SAPPHIRE (6.8 msec; P = .002) and SASHA (8.7 msec; P < .001).

229 f repolarization (25 +/- 3 msecs to 37 +/- 7 msecs; p = .037).

230 paradigm, the puff was delivered after a 700-msec empty "trace" interval that followed the end of the

231 chondral defects of female rats (mean, 10.72 msec for human stem cells and 11.55 msec for male rat st

232 nificant difference in the fetal liver (2.72 msec vs 3.18 msec; P = .47).

233 ADC from cancer (mean, 1628 msec +/- 344, 73 msec +/- 27, and 0.773 x 10(-3) mm(2)/sec +/- 0.331, res

234 ve values included the following: liver, 745 msec +/- 65 (standard deviation) and 31 msec +/- 6; rena

235 epatic parenchyma in healthy volunteers (745 msec +/- 65 and 31 msec +/- 6, P < .0001 and P = .021, r

236 ing a GRE sequence (repetition time, 600-750 msec; echo time, 10 msec; in-plane resolution, 196 mm).

237 each target type were presented 300 and 750 msec after the prime.

238 msec +/- 9; and fat, 253 msec +/- 42 and 77 msec +/- 16, respectively.

239 in a typical three-echo protocol (with 0.78-msec increments).

240 ecruited population were 5.9%, 19.6%, 306.79 msec, and 162.47 msec, respectively.

241 sec +/- 88; T2, 32.0 msec +/- 4.3; T2*, 10.8 msec +/- 0.8; ADC, 1.39 x 10(-3) mm(2)/sec +/- 0.02 (ref

242 square successive differences: 16.7 vs. 14.8 msec, p = 0.007).

243 anced degeneration (Beck scores 3-6, </=19.8 msec +/- 5.6) (P < .001).

244 ured at an orientation of 54.7 degrees (21.8 msec +/- 2.8 [+/- standard error of the mean]) than at 0

245 was marginally elevated by 6% +/- 2.5 (37.8 msec +/- 2.5, P = .021) in patients and by 8% +/- 5 (44.

246 higher T2 values (48.7 msec +/-4.35 vs 45.8 msec +/-3.93; P < .001) than did subjects with low activ

247 .07) but higher precision than SAPPHIRE (6.8 msec; P = .002) and SASHA (8.7 msec; P < .001).

248 The T2 mapping showed a mean value of 89.8 msec+/-19.34.

249 TE) and the third OP echo (approximately 5.8-msec TE) were acquired.

250 ich the step rise-times can be as long as 80 msec.

251 ich temporal resolution was approximately 83 msec.

252 hs (P < .0001), whereas T2 decreased from 84 msec +/- 10 to 58 msec +/- 4 (P < .0001).

253 ing liver parenchyma relaxation times of 840 msec +/- 113 and 28 msec +/- 3 (P < .0001 and P < .01) a

254 speed (change in raw score, 5.2 msec and 0.9 msec, respectively).

255 (standard deviation) lower in patients (15.9 msec +/- 4.5 vs 35.2 msec +/- 2.1, P < .001) and 40% +/-

256 ation between groups (optimal threshold=22.9 msec, specificity=0.69 [11 of 16], sensitivity=0.60 [six

257 Mean T1rho values of volunteers (mean, 40.9 msec +/- 2.9 [standard deviation]; range, 33.9-46.3 msec

258 0 msec +/- 0.7, P < .001) or 90 degrees (9.9 msec +/- 0.4, P < .001).

259 E) and the second IP echo (approximately 4.9-msec TE) were acquired.

260 Successive targets presented at 90 msec improved performance compared with non-successive t

261 sequentially at rates of 30 msec/item or 90 msec/item.

262 -parietal electrode sites were evident at 92 msec (C1) and at 146 msec (N1b).

263 cts were longer than those of older MIs (925 msec+/-169 vs 551 msec+/-107, P<.001).

264 ho (echo time, 25 msec; repetition time, 944 msec) imaging at 232-microm in-plane resolution (0.05-mm

265 time 1 = 135 msec and inversion time 2 = 95 msec) sequences.

266 +/- 28 [standard deviation] in diastole, 959 msec +/- 21 in systole) and all segmental T1 values betw

267 t half maximum values of 174 msec +/- 11, 98 msec +/- 3, and 143 msec +/- 5, respectively.

268 Adding T2 information (98 msec+/-7 vs 68 msec+/-2, respectively) alone yields resu

269 Mean myocardial T1 (984 msec +/- 28 [standard deviation] in diastole, 959 msec +

270 on, with a 19% lower risk for every 817-mm . msec lower Cornell product treated as a continuous varia

271 density-weighted SE (repetition time msec/TE msec, 2000/15) and ultrashort TE (300/0.008, 6.6, echo-s

272 hted fat-suppressed (repetition time msec/TE msec, 2300/34), T1-weighted (700/10), and UTE (300/0.008

273 ing sequence (repetition time msec/echo time msec, 10 123/40; b=1200 sec/mm2).

274 (23)Na (repetition time msec/echo time msec, 160/0.35) and (35)Cl (40/0.6) MR imaging of both l

275 radient-echo (repetition time msec/echo time msec, 180/2.3; transmission angle, 55 degrees ) and T2-w

276 re generated (repetition time msec/echo time msec, 2000/67; section thickness, 4 mm; in-plane resolut

277 acquisitions (repetition time msec/echo time msec, 4/1.35; 20 degrees flip angle; 1 x 1 x 2-mm acquir

278 al MR images (repetition time msec/echo time msec, 8.86/4.51; flip angle, 25 degrees ) acquired with

279 cho sequence (repetition time msec/echo time msec, 800/1.8-49.8) was performed at embryonic day 19.

280 t and second repetition times msec/echo time msec: 72, 192/2.2; transmission angle: 60 degrees ) for

281 cho-planar imaging sequence (repetition time msec/echo time msec, 10 123/40; b=1200 sec/mm2).

282 (23)Na (repetition time msec/echo time msec, 160/0.35) and (35)Cl (40/0.6) MR im

283 l T1-weighted gradient-echo (repetition time msec/echo time msec, 180/2.3; transmission angle, 55 deg

284 and ADC maps were generated (repetition time msec/echo time msec, 2000/67; section thickness, 4 mm; i

285 Whole-heart acquisitions (repetition time msec/echo time msec, 4/1.35; 20 degrees flip angle; 1 x

286 three-dimensional MR images (repetition time msec/echo time msec, 8.86/4.51; flip angle, 25 degrees )

287 iple gradient-echo sequence (repetition time msec/echo time msec, 800/1.8-49.8) was performed at embr

288 ultiecho spin-echo sequence (repetition time msec/echo times msec, 1500/24, 36, 48, 60, 72, 84, 96, 1

289 ry short echo time sequence (repetition time msec/echo times msec, 30/0.075, 2, 5, 12, 18).

290 proton density-weighted SE (repetition time msec/TE msec, 2000/15) and ultrashort TE (300/0.008, 6.6

291 ity-weighted fat-suppressed (repetition time msec/TE msec, 2300/34), T1-weighted (700/10), and UTE (3

292 ho sequence (repetition time msec/echo times msec, 1500/24, 36, 48, 60, 72, 84, 96, 108, 120, 132, 14

293 me sequence (repetition time msec/echo times msec, 30/0.075, 2, 5, 12, 18).

294 e imaging (first and second repetition times msec/echo time msec: 72, 192/2.2; transmission angle: 60

295 er risk for heart failure for every 817-mm x msec (1 SD of the mean) lower Cornell product (hazard ra

296 patients with a reduction less than 236 mm x msec during treatment (6.8 per 1000 patient-years).

297 ts with an in-treatment decrease of 236 mm x msec or more (4.4 per 1000 patient-years) and 137 patien

298 ent reduction in Cornell product of 236 mm x msec or more (hazard ratio, 0.64 [CI, 0.47 to 0.89]; P <

299 ove or below the median decrease of 236 mm x msec) to predict heart failure hospitalization occurring

300 atment decrease in Cornell product (236 mm x msec) was associated with a 43% lower risk for heart fai