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1 hotspot driver mutations specifically in the mucinous tumor.
2 e a distinct subset of genes was specific to mucinous tumors.
3 ociation of NKX2-1 with the genes induced in mucinous tumors.
4 between ductal type of intraductal papillary mucinous tumors.
5 ely related to nonmucinous tumors but not to mucinous tumors.
6 l/Met variant of COMT decreases the risk for mucinous tumors.
7 y mucinous ascites and multifocal peritoneal mucinous tumors.
8 cer of all principal histologic types except mucinous tumors.
9 erapy, whereas no association was found with mucinous tumors.
11 ations for serous tumors, KRAS mutations for mucinous tumors, and beta-catenin and PTEN mutations and
12 tones, localization of intraductal papillary mucinous tumors, and localization and treatment of hemob
16 d with an increased risk in endometrioid and mucinous tumors [e.g., testosterone, endometrioid tumors
17 inous tumors and 1.03 (95% CI 0.88-1.21) for mucinous tumors; for each year of oral contraceptive use
18 erefore, we present the first mouse model of mucinous tumor formation from ovarian cells and supporti
20 s 67%, 72%, and 67%, respectively, with most mucinous tumors having predominantly high SI and a perip
21 s and some included a heterogeneous group of mucinous tumors, including both benign and malignant app
23 indings strongly support the conclusion that mucinous tumors involving the appendix and ovaries in wo
25 creas of patients with intraductal papillary mucinous tumors (IPMT) and to analyze their relation to
26 rofiles in a series of intraductal papillary-mucinous tumors (IPMTs) of the pancreas, which represent
27 ce interval: 1.0, 2.4) but decreased risk of mucinous tumors (IRR = 0.3, 95% confidence interval: 0.1
32 endiceal mucinous adenomas (MAs) and ovarian mucinous tumors of low malignant potential (MLMPs) unass
34 iant was not significant overall, it was for mucinous tumors of the ovary, with an adjusted RR of 0.2
36 inous tumors and 0.98 (95% CI 0.93-1.04) for mucinous tumors (p = 0.00051 and p = 0.0040, respectivel
39 an be differentiated with MR imaging because mucinous tumors show high SI on T2-weighted fast SE imag
41 ordinate variants between paired Brenner and mucinous tumors supports a shared origin or progression.
42 convey greater increased risk for women with mucinous tumors than for women with neoplasms of other h
44 demonstrated that a set of genes induced in mucinous tumors was shared with genes induced in a nontu
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