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1 = 71) of the 76 episodes of candidiasis were mucocutaneous.
2 failure syndrome characterized by a triad of mucocutaneous abnormalities and a predisposition to canc
3 failure syndrome characterized by a triad of mucocutaneous abnormalities and an increased predisposit
4 congenita, a rare condition characterized by mucocutaneous abnormalities and bone marrow failure, is
5 characterized by multiple features including mucocutaneous abnormalities, bone marrow failure and an
6 lopecia in addition to the dramatic skin and mucocutaneous acantholysis observed in pemphigus patient
7 tivity, and oral ulcers, and 3 (23%) met the mucocutaneous ACR criteria plus positive antinuclear and
8 uring our study did so mostly by meeting the mucocutaneous ACR criteria, and the majority developed n
9 and pathologic description of the lichenoid mucocutaneous adverse effects seen in patients receiving
10 Of the 13 patients, 1 (8%) solely met the mucocutaneous American College of Rheumatology (ACR) cri
11 13-cRA reports and included mild to moderate mucocutaneous and flu-like symptoms; occasional signific
15 s: (1) Langerhans-related, (2) cutaneous and mucocutaneous, and (3) malignant histiocytoses as well a
16 bone marrow failure syndrome associated with mucocutaneous anomalies, pulmonary fibrosis, and cirrhos
19 ly exclusive pathways underlying immunity to mucocutaneous as opposed to invasive fungal infections.
21 ly tested, however, acute major bleeding and mucocutaneous bleeding during chronic administration wer
23 udied a 16-year-old white male with lifelong mucocutaneous bleeding manifestations and abnormal plate
27 molysis bullosa is an incurable, often fatal mucocutaneous blistering disease caused by mutations in
28 ne pemphigoid or cicatricial pemphigoid is a mucocutaneous blistering disease characterized by autoan
29 phigus vulgaris (PV) is a potentially lethal mucocutaneous blistering disease characterized by cell-c
33 s study, we present a patient with extensive mucocutaneous blisters, epidermolytic palmoplantar kerat
35 alpha/beta that causes platelet dysfunction, mucocutaneous blood loss and suppression of erythropoies
37 cells are also critical in host immunity to mucocutaneous candida infections and Staphylococcus aure
40 have recently been shown to underlie chronic mucocutaneous candidiasis (CMC), while inborn errors of
44 ial disease (hypomorphic alleles) to chronic mucocutaneous candidiasis (CMC; hypermorphic alleles).
45 rge family in which a combination of chronic mucocutaneous candidiasis (fungal infections of the skin
47 ator of transcription 1 (STAT1) with chronic mucocutaneous candidiasis and PML was reported previousl
48 linkage region on chromosome 2p for chronic mucocutaneous candidiasis and thyroid disease, previousl
49 cells; meanwhile, some patients with chronic mucocutaneous candidiasis disease might also have viral
51 nti-IL-17F, or anti-IL-22 autoantibodies and mucocutaneous candidiasis in the setting of either APECE
52 ciency (AI), hypoparathyroidism, and chronic mucocutaneous candidiasis plus autoantibodies neutralizi
53 ients with unusual susceptibility to chronic mucocutaneous candidiasis resulting from T(H)17 deficien
56 d clinical manifestations, including chronic mucocutaneous candidiasis, AI, and asplenia, respectivel
57 matoceles, eczema, staphylococcal abscesses, mucocutaneous candidiasis, and abnormalities of bone and
58 ponses, improved TH17 differentiation, cured mucocutaneous candidiasis, and maintained remission of i
59 rized by multiple endocrine failure, chronic mucocutaneous candidiasis, and various ectodermal defect
60 in IL17F and IL17R in patients with chronic mucocutaneous candidiasis, as well as neutralizing autoa
61 mutations have been associated with chronic mucocutaneous candidiasis, but the role of CARD9 in inte
62 eficiency in mice or humans leads to chronic mucocutaneous candidiasis, but the specific downstream m
63 rent bacterial infections, viral infections, mucocutaneous candidiasis, cutaneous warts, and skin abs
66 autoimmune disorder characterized by chronic mucocutaneous candidiasis, hypoparathyroidism, and adren
67 pecies infection, Norwegian scabies, chronic mucocutaneous candidiasis, hypothyroidism, and esophagea
69 nt, both subjects with mosaicism had chronic mucocutaneous candidiasis, suggesting that candidiasis i
70 risingly, it is also associated with chronic mucocutaneous candidiasis, through as yet undetermined m
71 -17A and interleukin-17F (IL-17A/F) underlie mucocutaneous candidiasis, whereas inborn errors of inte
72 utations, including recurrent infections and mucocutaneous candidiasis, which are suggestive of TH17
81 ive stomatitis (CUS) is a recently described mucocutaneous condition in which patients experience chr
84 ratin 14 (K14) promoter, developed GVHD-like mucocutaneous disease and weight loss following transfer
86 nts with advanced disease such as widespread mucocutaneous disease, lymphedema, and visceral disease
87 disease, such as in patients with widespread mucocutaneous disease, lymphedema, and visceral disease,
92 significantly less frequent nonopportunistic mucocutaneous disorders and respiratory infections (P<.0
93 Cicatricial pemphigoid is one of a number of mucocutaneous disorders that can present in the oral cav
94 frequency of minor opportunistic infections, mucocutaneous disorders, and respiratory infections and
96 luded generally mild to moderate fatigue and mucocutaneous dryness, moderate to severe neutropenia (3
100 ratosis congenita, typically with associated mucocutaneous features, and others (TERC and TERT) for m
103 d with severe disseminated mycobacterial and mucocutaneous fungal infections and was ultimately cured
104 ly from that of Crohn disease (CD); however, mucocutaneous granulomatous lesions have not been consid
106 hrombasthenia is clinically characterized by mucocutaneous hemorrhage with episodes of intracranial a
107 ms involved in host clearance of symptomatic mucocutaneous herpes simplex virus (HSV) infection are u
109 famciclovir, and valacyclovir, treatment of mucocutaneous HSV is a practice of everyday medical care
111 srupt various cytokine pathways that control mucocutaneous immunity against Candida species, especial
112 indicate that human IL-17RC is essential for mucocutaneous immunity to C. albicans but is otherwise l
114 tations in either STAT1 or STAT3 that affect mucocutaneous immunity to Candida and Staphylococcus spe
118 a range of diseases from mild uncomplicated mucocutaneous infection to those that are life threateni
120 es a wide spectrum of diseases, ranging from mucocutaneous infections like oral thrush to disseminate
121 d fertile and only rarely develop the severe mucocutaneous infections or pulmonary inflammation chara
122 ssing all three selectins (ELP(-/-)) develop mucocutaneous infections that eventually lead to death.
123 amily with 2 siblings who have had recurrent mucocutaneous infections with Candida albicans and Staph
124 ukocyte adhesion deficiency characterized by mucocutaneous infections, plasma cell proliferation, hyp
130 re was a focus of PCNA-positive cells in the mucocutaneous junction and a few scattered PCNA-positive
131 gland volume and a forward migration of the mucocutaneous junction anterior to the gland orifice; si
132 tival cells remained within 1 to 2 mm of the mucocutaneous junction at all postinjection intervals.
133 at progressively greater distances from the mucocutaneous junction in the animals killed at 1, 3, an
137 counted in a series of 0.4-mm zones from the mucocutaneous junction of the eyelid, through the fornix
142 ithout IRIS (+158 vs +53 cells/muL, P = .04, mucocutaneous KS; +261 vs +113, P = .04, tuberculosis).
143 ght a distinct biosignature in patients with mucocutaneous leishmaniasis (MCL) or localized cutaneous
145 nnia) braziliensis is the causative agent of mucocutaneous leishmaniasis (ML) in South America, and M
148 sera from human patients with cutaneous and mucocutaneous leishmaniasis indicated that these individ
152 characterized by the development of multiple mucocutaneous lesions and benign tumors, and enhanced ca
153 ons in the genital tract are responsible for mucocutaneous lesions and transmission and manifest as d
154 Laboratory diagnosis of HSV in cutaneous or mucocutaneous lesions has historically been performed wi
157 ge of diseases in humans, from uncomplicated mucocutaneous lesions to life-threatening infections.
159 Twenty-one patients (47%) had more than 50 mucocutaneous lesions, 14 (32%) had lymphedema, and none
163 ually associated with a slightly more severe mucocutaneous lipoid proteinosis phenotype, but neurolog
164 and marrow transplantation can attenuate the mucocutaneous manifestations of the disease and improve
165 cterized by intestinal hamartomatous polyps, mucocutaneous melanin deposition, and increased risk of
168 the normal flora of the alimentary tract and mucocutaneous membranes, is the leading cause of invasiv
169 tment, remission was induced, notably in the mucocutaneous, musculoskeletal, cardiovascular/respirato
170 Protocol-defined "minor" bleeding, usually mucocutaneous, occurred in 0% to 32% of patients in the
171 f the patients developed fever, respiratory, mucocutaneous, or central nervous system manifestations.
172 cal disease that can present with cutaneous, mucocutaneous, or visceral manifestation and affects mil
174 drome associated with benign tumours, spotty mucocutaneous pigmentation, and endocrine overactivity (
175 hers syndrome (PJS), which includes aberrant mucocutaneous pigmentation, and somatic LKB1 mutations o
176 eutz-Jeghers syndrome (PJS) characterized by mucocutaneous pigmentation, predisposition to benign ham
179 patients who developed widespread blistering mucocutaneous reactions without any suspected drug causa
184 f the epithelial lineage, causing lesions on mucocutaneous surfaces, HSVs also establish latent infec
185 with biallelic RASGRP2 variants had abnormal mucocutaneous, surgical, and dental bleeding from childh
187 ity of clinical features, ranging from small mucocutaneous telangiectases to life-threatening viscera
188 ascular disorder characterized by epistaxis, mucocutaneous telangiectases, and arteriovenous malforma
189 ch is characterized by recurrent nosebleeds, mucocutaneous telangiectases, and visceral AVMs and caus
190 , numerous secondary lesions on the skin and mucocutaneous tissues, severe respiratory disease, death
191 Both regimens were quite toxic, with more mucocutaneous toxicity in the FAP arm and more myelosupp
193 Dyskeratosis congenita is characterized by a mucocutaneous triad, bone marrow failure (BMF), and pres
194 (n=13) and FS (n=15) patients, and in 79% of mucocutaneous-type PV patients (n=33), but in none of th
197 stigated the etiology of autosomal-dominant, mucocutaneous ulceration in a family whose proband was d
199 pes simplex virus 1 (HSV-1) causes recurrent mucocutaneous ulcers and is the leading cause of infecti
201 in IL-12Rbeta1 may have candidiasis, usually mucocutaneous, which is frequently recurrent or persiste
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