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1 leukemia)-2H3 mast cells (a tumor analog of mucosal mast cells).
2 antibody dissociation from its receptors on mucosal mast cells.
3 are not essential for exocytosis in RBL-2H3 mucosal mast cells.
4 blood eosinophils, and increased intestinal mucosal mast cells.
5 pus of mice appear to be bone marrow-derived mucosal mast cells.
6 involved in colonic goblet cell release and mucosal mast cell activation after immobilization stress
8 expulsion is correlated with high numbers of mucosal mast cells and an increase in IL-13 and IL-10 se
9 accompanied by accelerated degranulation of mucosal mast cells and increased Ag-specific production
12 vity reflects the permanent sensitization of mucosal mast cells by allergen-specific IgE antibodies b
16 nstrate a critical role for IL-10 in driving mucosal mast cell expansion and activation, suggesting t
18 ifferentiation of intraepithelial intestinal mucosal mast cells expressing mouse mast cell protease 1
19 r (SCF), whereas helminth-induced intestinal mucosal mast cell hyperplasia also requires T cell-deriv
21 r results reveal a potential function of gut mucosal mast cells in HIV-1 dissemination in tissues.
22 d beta(6)(-/-) mice, we show accumulation of mucosal mast cells in the lamina propria of the small in
26 finity receptor for IgE (FcepsilonRI) on the mucosal mast cell line, RBL-2H3, results in the rapid an
28 leukemia cells (RBL-2H3 m1), an immortalized mucosal mast cell line, was studied at the single-channe
30 These results provide the mechanism whereby mucosal mast cells mediate parasite expulsion from the i
31 inal nematodes is associated with pronounced mucosal mast cell (MMC) hyperplasia, differentiation, an
34 tification of multifunctional IL-9-producing mucosal mast cells (MMC9s) that can secrete prodigious a
37 toplasm of rat basophilic leukemia (RBL-2H3) mucosal mast cells or anchored (via LynB protein) to the
38 ntributing factor to the observed changes in mucosal mast cell protease and epithelial cytokine expre
39 tocytosis was absent, the protease unique to mucosal mast cells, rat mast cell protease II (RMCPII),
41 usly compromised in their ability to mount a mucosal mast cell response after infection, and there is
44 Our aim was to elucidate the contribution of mucosal mast cells to the effector phase of a secondary
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