ライフサイエンスコーパス: mucous

1 has been measured in hypoxic cystic fibrosis mucous.

2 morphogenesis, and a lack of differentiated mucous acinar cells in submandibular and sublingual glan

3 f IRF6 in the developing ductal, serous, and mucous acinar cells of salivary glands.

4 n of prolonged diarrhea containing blood and mucous after recovery from an acute phase of myositis pr

5 cies underwent mass mortality, formed large, mucous aggregates, and sank out en masse (carbon sinkers

6 a serum-free medium that induces endocrine, mucous and absorptive lineage commitment by HRA-19 cells

7 ay 15.5-18.5), epithelial differentiation to mucous and chief cell lineages was rudimentary, with no

8 ed differentiation, with inhibition inducing mucous and endocrine cell differentiation while activati

9 ls and gland thread cells, which produce the mucous and fibrous components of the slime, respectively

10 copy to obtain force maps of human preocular mucous and purified ocular mucins by probing and locatin

11 es, mammary ducts, and glands such as sweat, mucous and sebaceous glands, are initiated in developmen

12 anism against pathogens that complements the mucous and secretory IgA Ab-mediated system in the prote

13 an normal (p = 0.031), but the proportion of mucous and serous cells in CF glands was normal.

14 echanisms involved in the deployment of both mucous and thread cells during the transition from thick

15 of mature chief cells, increased numbers of mucous and undifferentiated cells, and an increase in th

16 ent stem cells that give rise to absorptive, mucous, and endocrine lineages.

17 genesis and how Giardia's disruptions of the mucous barrier facilitate bacterial translocation that m

18 The mucous barrier of our digestive tract is the first line

19 om acidic reduction of nitrite and increases mucous barrier thickness and gastric blood flow (see the

20 fibrosis (CF), and the relationship between mucous cell abnormalities and neutrophilic inflammation

21 etinol metabolite retinoic acid (RA) induces mucous cell differentiation of normal human tracheobronc

22 utosomal recessive mutation, sld, attenuates mucous cell expression in murine sublingual glands with

23 Mucous cell hyperplasia and airway smooth muscle (ASM) h

24 educes allergen- and cigarette smoke-induced mucous cell hyperplasia in mice and in differentiated pr

25 l resolution process of inflammation-induced mucous cell hyperplasia may lead to sustained mucous hyp

26 , and during prolonged exposure to allergen, mucous cell hyperplasia remained elevated in STAT1(-/-)

27 d significantly less pulmonary inflammation, mucous cell hyperplasia, and eosinophilia compared with

28 osure of mice to allergen, IFN-gamma reduces mucous cell hyperplasia, but the signaling responsible f

29 Features of allergic airway disease such as mucous cell hyperplasia, infiltration of airway eosinoph

30 AECs and the resolution of allergen-induced mucous cell hyperplasia.

31 xyuridine, and for expression of the surface mucous cell marker Ulex europaeus.

32 endai virus triggered a switch to persistent mucous cell metaplasia and airway hyperreactivity after

33 ff and immunohistochemical staining revealed mucous cell metaplasia and muc5AC expression in RV1B- bu

34 or CLCA family members in the development of mucous cell metaplasia and possibly airway hyperreactivi

35 Indeed, mucous cell metaplasia is considered the critical preneo

36 uction on day 4 postinfection and subsequent mucous cell metaplasia on day 21 postinfection.

37 ute inflammatory response and the subsequent mucous cell metaplasia that accompanies the asthma pheno

38 Cell proliferation and mucous cell metaplasia were detected in newborn, but not

39 nificant increase in pulmonary inflammation, mucous cell metaplasia, airway hyperresponsiveness, and

40 3, dramatically blocked the allergen-induced mucous cell metaplasia, airway mucus formation, and the

41 tokines, C-C chemokines, IgE production, and mucous cell metaplasia, as compared with the adoptive tr

42 indicates that at least one of these traits, mucous cell metaplasia, depends on members of the calciu

43 onse that recruits CD4(+) T cells and drives mucous cell metaplasia, thus linking antiviral responses

44 Bcl-2 sustains mucous cell metaplasia, whereas p53(-/-) mice display ch

45 sensitization, TH2 cytokine production, and mucous cell metaplasia.

46 nd that FceRIa(-/-) mice no longer developed mucous cell metaplasia.

47 events, whereas blockade of CCL28 inhibited mucous cell metaplasia.

48 ng phagocyte recruitment, IgA secretion, and mucous cell metaplasia.

49 w enhanced expression of SPDEF and Bcl-2 and mucous cell metaplasia.

50 tokines, C-C chemokines, IgE production, and mucous cell metaplasia.

51 13, and alpha7-nAChRs are critical in airway mucous cell metaplasia/hyperplasia and mucus production

52 th loss of acid-secreting parietal cells and mucous cell metaplasias.

53 ealed edema within the submucosa and altered mucous cell morphology.

54 ated with Notch agonists displayed increased mucous cell numbers and decreased ciliated cell numbers.

55 ia, in which there are dramatic increases in mucous cell numbers.

56 tch misexpression resulted in an increase in mucous cells and a decrease in ciliated cells in the air

57 ecretory products from two cell types, gland mucous cells and gland thread cells, which produce the m

58 demonstrate MUC19 glycoproteins in glandular mucous cells and saliva.

59 nd-organ alterations (particularly in airway mucous cells and smooth muscle) as critical steps leadin

60 nd-organ alterations (particularly in airway mucous cells and smooth muscle) as critical steps leadin

61 Accordingly, more mucous cells are present in primary human airway culture

62 Normally, airway mucous cells are rare.

63 combination of no ciliated cells and excess mucous cells can account for the chronic rhinitis and in

64 ume, but serous cell transdifferentiation to mucous cells does not occur, nor are gland acini inflame

65 A significantly higher percentage of mucous cells expressed Bcl-2 in humans with cystic fibro

66 restricted localization to subpopulations of mucous cells in sld mice during postnatal gland developm

67 R, results in selective expansion of surface mucous cells in the body and fundus of the stomach.

68 Mucous cells of MECs were positive for CK7, CEA, as well

69 y investigated expression of Bcl-2 in airway mucous cells of persons with cystic fibrosis and tested

70 In addition, ER Bax was detected in mucous cells of STAT1(+/+) but not STAT1(-/-) airways of

71 Relatively few studies have characterized mucous cells or mucins in detail in cystic fibrosis (CF)

72 bleaching) probe, we have assessed in living mucous cells the relative importance of different protei

73 344/N rats, the percentage of Bcl-2-positive mucous cells was decreased to background levels before t

74 d 15 control subjects, and the morphology of mucous cells was measured.

75 sis of amplified cRNA from SPEM, and surface mucous cells were isolated by laser capture microdissect

76 Primary canine parietal or mucous cells were treated with IL-1beta.

77 ng LGR5-expressing cells, surface and antral mucous cells, and a diversity of gastric endocrine cells

78 thinner reticular basement membranes, fewer mucous cells, fewer eosinophils, and fewer mast cells th

79 were differentially expressed, in serous and mucous cells, respectively.

80 ent accumulation near the luminal borders of mucous cells.

81 on, displayed little or no redistribution in mucous cells.

82 t suppress Shh expression in primary gastric mucous cells.

83 rin cells and TFF2-expressing deep glandular mucous cells.

84 on is responsible for sustaining metaplastic mucous cells.

85 ehog (Ihh) and hyperproliferation of surface mucous cells.

86 p53(-/-) mice display chronically increased mucous cells.

87 ression patterns distinguished tumor goblet (mucous) cells from nontumorigenic airway and intestinal

88 intramuscular) contraceptives, high cervical mucous concentrations of interleukin (IL)-12, a positive

89 usea, vomiting, discomfort, and even gastric mucous damage.

90 n the disc consisted of cellular changes and mucous degeneration.

91 he cysteine protease inhibitor E64 prevented mucous degradation, mucin depletion, and the increase in

92 008 to April 2010, episodes of hematochezia, mucous diarrhea and epigastric pain intermittently manif

93 early biological effects of RA during normal mucous differentiation in NHTBE cells.

94 taining Ets-like factor (SPDEF), a driver of mucous differentiation.

95 igns of NLDO (epiphora, increased tear lake, mucous discharge) in both eyes on masked examination at

96 l surface that helps them evade predation by mucous filter feeders.

97 only partially exposed at the surface of the mucous gel.

98 to an exogenous probe in isolated mucins and mucous gels.

99 A and protein are expressed predominantly in mucous gland cells of the antrum and in mucous neck cell

100 s required for terminal maturation of antral mucous gland cells to protect animals from gastric infla

101 Spdef impaired terminal maturation of antral mucous gland cells, as reflected in reduced expression o

102 earch groups conclude that changes in airway mucous gland function in CFTR-deficient animals of these

103 have approximately the same number of airway mucous glands as humans.

104 and exocrine secretion of mucins by salivary mucous glands is regulated predominantly by parasympathe

105 LDA) was isolated from extracts derived from mucous glands that coat the dart before it is stabbed th

106 es abnormally viscous secretions from airway mucous glands that may be a major factor in CF pathology

107 Mouse airways have few mucous glands, and the mouse model of CF exhibits no sig

108 of ichthyotoxic peptides isolated from fish mucous glands.

109 erm ciliated cell hyperplasia coincides with mucous (goblet) cell metaplasia after respiratory viral

110 ng within mucin granules, we generated human mucous/goblet cells stably expressing a recombinant MUC5

111 ral components of egg coats, appendicularian mucous houses, and nematode dauer larvae, to serving as

112 Cases of cutaneous and mucous hyperpigmentations during interferon alfa treatme

113 triction leading to airflow obstruction, and mucous hypersecretion are clinical hallmarks of asthma.

114 Goblet cell hyperplasia and mucous hypersecretion contribute to the pathogenesis of

115 ucous cell hyperplasia may lead to sustained mucous hypersecretion in chronic diseases.

116 Mucous hypersecretion is a major cause of airway obstruc

117 Mucous hypersecretion is an important feature of obstruc

118 n lymphocytes, eosinophils, IL-13, IL-5, and mucous hypersecretion to wild-type levels, whereas eotax

119 ralizing antibody attenuated ILC2 expansion, mucous hypersecretion, and airways responsiveness.

120 ulmonary abnormalities of COPD/CF, including mucous hypersecretion, inflammatory and emphysematous ph

121 ner et al. show, in a mouse model of chronic mucous hypersecretion, that ciliated epithelial cell apo

122 neurogenic factors predominated in sinusitis mucous hypersecretion.

123 mice, as were airway hyperresponsiveness and mucous hypersecretion.

124 the PRD of p53 as a determinant for chronic mucous hypersecretion.

125 l in airway diseases associated with chronic mucous hypersecretion.Bcl-2 interacting killer (Bik) dec

126 GFR ligands and IL-13 are known to stimulate mucous induction, but the detailed mechanisms of epithel

127 ce infected with G. duodenalis had a thinner mucous layer and demonstrated differential Muc2 and Muc5

128 Most inhaled microbes are trapped on the mucous layer coating the nasal epithelium and upper resp

129 The intestinal mucous layer provides a critical host defense against pa

130 abitat for the commensal flora and the inner mucous layer that protects the epithelial cells by being

131 networks that in colon build the loose outer mucous layer that provides the habitat for the commensal

132 coadhesion of microparticles was tested on a mucous layer under shear stress, mimicking the human bli

133 ajor protein present in the small intestinal mucous layer, and that removal of this barrier in vitro

134 ls, between G. duodenalis and the intestinal mucous layer.

135 ella propel bacteria through urine and along mucous layers during ascension to the upper urinary trac

136 rmatologist when there were cutaneous and/or mucous lesions.

137 We show that endocrine and mucous lineage commitment is blocked in the presence of

138 9 cells significantly enhanced endocrine and mucous lineage commitment, while cells expressing the no

139 d negligible ability for either endocrine or mucous lineage commitment.

140 gnosis, most patients had other cutaneous or mucous localizations of cGVHD.

141 on, especially in case of other cutaneous or mucous localizations of cGVHD.

142 cal findings such as petechiae, bruising and mucous membrane bleeding, examination of peripheral bloo

143 ensitivity and UV radiation-induced skin and mucous membrane cancers.

144 fant through direct skin-to-skin and skin-to-mucous membrane contact while breastfeeding.

145 entification of Candida species from skin or mucous membrane cultures, and/or by culture or staining

146 actions characterized by widespread skin and mucous membrane detachments, including bronchial mucosa,

147 ugs have been reported to induce skin and/or mucous membrane discoloration.

148 servatively, but 2 eyes required free labial-mucous membrane grafting for persistent corneal exposure

149 ally alleviated with autologous serum drops, mucous membrane grafting to replace scarred tarsal conju

150 Free labial-mucous membrane grafting was performed in all cases of p

151 t secretion of components of the peritrophic/mucous membrane in the larval digestive tract.

152 Formaldehyde, a known human carcinogen and mucous membrane irritant, is emitted from a variety of b

153 There is also an increased risk of skin and mucous membrane malignancy.

154 fibrosis in the immunobullous disease ocular mucous membrane pemphigoid (MMP) causes blindness; the p

155 Mucous membrane pemphigoid (MMP) describes an autoimmune

156 dity of the First International Consensus on Mucous Membrane Pemphigoid (MMP) guidance, which recomme

157 Mucous membrane pemphigoid (MMP) has several subsets bas

158 Mucous membrane pemphigoid (MMP) is a heterogeneous grou

159 Mucous membrane pemphigoid (MMP) is a heterogeneous grou

160 A rare variant of mucous membrane pemphigoid (MMP) is characterized by cir

161 ancements in the diagnosis and management of mucous membrane pemphigoid (MMP) published in the Englis

162 A case of anti-laminin-332 mucous membrane pemphigoid (MMP) that developed shortly

163 ) in patients with clinically typical ocular mucous membrane pemphigoid (MMP).

164 ial pemphigoid (OCP), also labeled as ocular mucous membrane pemphigoid (MMP).

165 Ocular mucous membrane pemphigoid (OMMP) is an autoimmune disea

166 he immunosuppressive drugs used for treating mucous membrane pemphigoid and certain definitive catego

167 teers and 40 patients with other variants of mucous membrane pemphigoid and mAb GoH3 and BQ16 to inte

168 in we present a case of treatment-refractory mucous membrane pemphigoid and propose a mechanism to ex

169 Successful entropion repair in patients with mucous membrane pemphigoid can be achieved if control of

170 phigoid and certain definitive categories of mucous membrane pemphigoid continue to evolve.

171 Mucous membrane pemphigoid continues to be a potentially

172 A unique antigen in oral mucous membrane pemphigoid has not yet been identified.

173 ohnson syndrome in 41.7% (20 of 48 eyes) and mucous membrane pemphigoid in 41.7% (20 of 48 eyes).

174 Mucous membrane pemphigoid is an autoantibody-mediated d

175 Ocular mucous membrane pemphigoid is an immunobullous disease i

176 ocular disease among patients with oral only mucous membrane pemphigoid is estimated at 15-20% at 5 y

177 About 25% of mucous membrane pemphigoid patients generate autoantibod

178 aim of this article is to review articles on mucous membrane pemphigoid published in the English lite

179 However, not all cases of mucous membrane pemphigoid respond to rituximab.

180 Treatment for mucous membrane pemphigoid typically involves immunosupp

181 Circulating IgA against an antigen in mucous membrane pemphigoid was found in about 20% of pat

182 specific enough to confirm the diagnosis of mucous membrane pemphigoid with consistency.

183 indings and consisted of pemphigus vulgaris, mucous membrane pemphigoid, lichen planus, linear immuno

184 patients with lid malposition resulting from mucous membrane pemphigoid, particularly cicatricial ent

185 To review articles on mucous membrane pemphigoid, published between June 2004-

186 ts with an altered phenotype exist in ocular mucous membrane pemphigoid, we compared the functional c

187 t immunofluorescent testing in patients with mucous membrane pemphigoid.

188 dentified in the blood of some patients with mucous membrane pemphigoid.

189 on was found in skin of patients affected by mucous membrane pemphigoid.

190 advances have been achieved in the field of mucous membrane pemphigoid.

191 e two more common are oral lichen planus and mucous membrane pemphigoid.

192 e anti-mLAMalpha3 IgG-induced mouse model of mucous membrane pemphigoid.

193 olysis bullosa acquisita or anti-laminin-332 mucous membrane pemphigoid.

194 good efficacy in the treatment of refractory mucous membrane pemphigoid.

195 pemphigoid, epidermolysis bullosa acquisita, mucous membrane pemphigoid/cicatricial pemphigoid, or an

196 The conjunctiva is a mucous membrane that covers the sclera and lines the ins

197 Hyperpigmentation of the oral mucous membrane, acquired longitudinal melononychia, and

198 ften affects upper and lower gingiva, buccal mucous membrane, the retromolar triangle and the palate.

199 n of leptospirosis occurs through contact of mucous membranes and abraded skin with freshwater contam

200 Further research into contact allergies of mucous membranes and identification of its allergens is

201 ase that features blistering of the skin and mucous membranes caused by autoantibodies directed again

202 an autoimmune blistering disease of skin and mucous membranes caused by autoantibodies to the desmogl

203 eatening blistering disorder of the skin and mucous membranes caused by pathogenic autoantibodies to

204 y, by tense blisters and erosions on skin or mucous membranes close to the skin surface.

205 Skin and mucous membranes come in contact with external environme

206 all other shallow periodontal sites, and at mucous membranes followed by removal of calculus using c

207 In pemphigus, keratinocytes in epidermis and mucous membranes lose cell-cell adhesion, and in pemphig

208 ruses (HPVs) infect the epidermis as well as mucous membranes of humans.

209 red patchy depigmentation of skin, hair, and mucous membranes results from loss of melanocytes from i

210 d, natural exposure of the oral and/or nasal mucous membranes seems certain.

211 rvids commonly sustain minor lesions on oral mucous membranes that could have an impact on susceptibi

212 dermoscopic examination of skin, nails, and mucous membranes was performed, and skin biopsies were p

213 ired (e.g., orally or via lesions to skin or mucous membranes).

214 toxicities were as follows: esophagus, 4.7%; mucous membranes, 3.1%; and xerostomia, 3.1%.

215 neoplasia in sun-exposed areas of the skin, mucous membranes, and ocular surfaces.

216 utoimmune blistering disease of the skin and mucous membranes, characterized by autoantibodies agains

217 ), autoantibodies induce blisters on skin or mucous membranes, or both.

218 Nitrates are rapidly absorbed from mucous membranes, the gastrointestinal tract, and the sk

219 on can improve the integrity of the skin and mucous membranes, today's accomplishments are only the f

220 Damage is exclusive to the skin and mucous membranes, with no known direct compromise of the

221 ermatologic disorders involving the skin and mucous membranes.

222 onditions that initially affect the skin and mucous membranes.

223 ketosugars that are ubiquitous on mammalian mucous membranes.

224 pigmented spots of skin, overlying hair, and mucous membranes.

225 noglobulin G (IgG) and IgA antibodies across mucous membranes.

226 ng infections affecting the skin, nails, and mucous membranes.

227 s characterized by the fragility of skin and mucous membranes.

228 s characterized by the fragility of skin and mucous membranes.

229 potentially fatal blistering of the skin and mucous membranes.

230 ing in blisters and erosions of the skin and mucous membranes.

231 eding via particle capture occurs on a fine, mucous mesh (fiber diameter d approximately 0.1 microm)

232 o infection in lung diseases associated with mucous metaplasia and a mechanism by which patients with

233 of 6-d-old mice, but not mature mice, causes mucous metaplasia and airway hyperresponsiveness that ar

234 ven type 2 response, which causes persistent mucous metaplasia and airways hyperresponsiveness.

235 ablished role as an inflammatory mediator of mucous metaplasia and functions through Stat6-mediated g

236 d 11 mpi had less severe gastritis, atrophy, mucous metaplasia and hyperplasia (P < 0.01) and, additi

237 -IL-13 neutralizing Ab attenuated RV-induced mucous metaplasia and methacholine responses, and IL-4R

238 K19-kras transgenic mice uniquely displayed mucous metaplasia as early as 3 months and progressed to

239 The generation of mucous metaplasia in immature RV-infected mice involves

240 at Notch ligands, however, are able to cause mucous metaplasia in Stat6-null cultured trachea, thus i

241 mmation, hyperplasia, glandular atrophy, and mucous metaplasia in the corpus and for the level of hel

242 and negligible to mild glandular atrophy and mucous metaplasia in the fundic mucosa, while H. felis-i

243 During mucous metaplasia induced under inflammatory conditions,

244 nd a mechanism by which patients with florid mucous metaplasia may tolerate microbial burdens that ar

245 Mucous metaplasia was accompanied by induction of gob-5,

246 ation and marginally increased parietal cell mucous metaplasia with oxyntic atrophy.

247 opment of three cardinal features of asthma: mucous metaplasia, AHR, and the presence of inflammatory

248 rse agonist treatment, namely, reductions in mucous metaplasia, airway hyperresponsiveness (AHR), and

249 re sufficient for eosinophilic inflammation, mucous metaplasia, and airways hyperresponsiveness.

250 on leads to persistent airways inflammation, mucous metaplasia, and hyperresponsiveness, which are me

251 ed decreases in HDM-induced inflammation and mucous metaplasia, as well as reduced IL-4, IL-25, CD68,

252 Whether these twinned phenomena (mucous metaplasia, compromised host defenses) are causal

253 RV-induced mucous metaplasia, ILC2 expansion, airway hyperresponsiv

254 ses of the airway are often characterized by mucous metaplasia, in which there are dramatic increases

255 VAMP8 knock-out (KO) mice with IL-13-induced mucous metaplasia, mucin content in the bronchoalveolar

256 intranasal IL-33 and TSLP was sufficient for mucous metaplasia.

257 are required for maximum ILC2 expansion and mucous metaplasia.

258 nd IL-4R null mice failed to show RV-induced mucous metaplasia.

259 ably, Notch antagonists blocked IL13-induced mucous metaplasia.

260 identifying a novel pathway that stimulates mucous metaplasia.

261 airway epithelial cell cultures with induced mucous metaplasia/mucin hypersecretion.

262 XBP1 is also required for loss of mucous NC markers as ZCs form.

263 Compared with controls, numbers of total mucous neck and zymogen cells were significantly decreas

264 ronic gastritis followed by oxyntic atrophy, mucous neck cell hyperplasia, spasmolytic polypeptide-ex

265 cells (ZCs) as they differentiate from their mucous neck cell progenitors.

266 The transition of gastric epithelial mucous neck cells (NCs) to digestive enzyme-secreting zy

267 s study demonstrates that Agr2 expression by mucous neck cells in the stomach promotes the differenti

268 tiation of the zymogen cell lineage from the mucous neck cells in the stomachs of HKCre/Shh(KO) mice.

269 When Agr2 expression is absent, gastric mucous neck cells increased in number as does the number

270 y in mucous gland cells of the antrum and in mucous neck cells of the glandular corpus.

271 region of the gastric epithelium, namely in mucous neck cells.

272 und weak expression of FoxM1b protein in the mucous neck region of gastric mucosa, whereas we observe

273 and within 20 days, giving rise to parietal, mucous neck, and chief cells, but not to enterochromaffi

274 ISMCs express robust numbers of surface pit, mucous neck, chief, endocrine and parietal cells.

275 cells express robust numbers of surface pit, mucous neck, chief, endocrine and parietal cells.

276 ineages of the corpus including surface pit, mucous neck, zymogenic, and parietal cells expressed Shh

277 can evade filtration by slipping through the mucous nets of both pelagic and benthic tunicates.

278 uman tracheobronchial epithelial (NHTBE) and mucous NHTBE cells.

279 us metaplastic NHTBE cells but not in normal mucous NHTBE or normal bronchial epithelium.

280 h inflammation remains intact, heterogeneous mucous occlusion decreases by 74%.

281 moglobin degradation products 3. melanin, 4. mucous- or protein-containing lesions, 5. highly cellula

282 rt foreign particulate trapped in a layer of mucous out of the upper airway.

283 Existing dogma defines mucous phenotypes in asthma and chronic obstructive pulm

284 n cystic fibrosis) peribronchial thickening, mucous plugging and many other disorders that occur in t

285 l slices for the presence of bronchiectasis, mucous plugging, or other airway abnormalities (inspirat

286 ent revealed that ivacaftor decreased airway mucous plugging.

287 RSV rA2-line19F infection induces pulmonary mucous production and increased breathing effort in BALB

288 s without reducing goblet cell metaplasia or mucous production and may explain the lower risk of alle

289 f eosinophils, elevated Th2 cytokine levels, mucous production, and airway hyperresponsiveness.

290 of tissue loss, sclerite enlargement, excess mucous production, bleached commensal ophiuroids, and co

291 consider the therapeutic effects of blocking mucous production.

292 n, but the detailed mechanisms of epithelial mucous regulation have not been well defined.

293 tected in a total of 52 of 126 (41.3%) nasal mucous samples, showing the nasal cavity is a reservoir

294 Fasudil also reduced mucous secretion and MUC5AC expression in OVA-challenged

295 kotrienes stimulate conjunctival goblet cell mucous secretion with LTD(4) using the CysLT(1) receptor

296 ERK1/2 to stimulate conjunctival goblet cell mucous secretion, and that activation of all four histam

297 contributor to the viscoelastic property of mucous secretion, which is the major barrier to trapping

298 ses by permitting continuous accumulation of mucous secretions on the epithelial surface, establishin

299 Deletion of IL-4Ralpha abolished transient mucous secretory cell (MuSC) abundance and eosinophilia

300 t least monthly leakage of solid, liquid, or mucous stool), and pelvic organ prolapse (seeing/feeling