ライフサイエンスコーパス: mucus plug

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1 al ascension across a dysfunctional cervical mucus plug.

2 which is filled with a dense and protective mucus plug.

3 t intercrypt crevices, and absence of apical mucus plugs.

4 from airway inflammation, bronchospasm, and mucus plugging.

5 c target for airway diseases associated with mucus plugging.

6 nce in mice with AAD, resulting in increased mucus plugging.

7 e of barotrauma, hemodynamic instability, or mucus plugging.

8 e from uninfected mice with AAD demonstrated mucus plugging after 14 and 21 days of ovalbumin-aerosol

9 aluation of lung sections revealed extensive mucus plugging and epithelial cell hypertrophy/hyperplas

10 of airway remodeling and contributes to the mucus plugs and airflow obstruction associated with seve

11 ll as MRI-defined airway wall abnormalities, mucus plugging, and abnormal lung perfusion in infants a

12 ed bronchial wall thickening/bronchiectasis, mucus plugging, and perfusion deficits from the first ye

13 goblet cell hyperplasia; hyper IgE syndrome; mucus plugging; and extensive inducible BALT.

14 flecting decreased formation of asphyxiating mucus plugs; and 3) in Scnn1b-Tg mice, neutrophilia, muc

15 of disease heterogeneity, including regional mucus plugging associated with abnormal lung perfusion i

16 ace enlargement, but had no effect on airway mucus plugging, bacterial infection, or pulmonary mortal

17 ailure, ventilation failure, barotraumas, or mucus plugging between treatment groups.

18 disease was believed to arise from abnormal mucus plugging exocrine ducts.

19 pneumothorax development in one patient and mucus plug formation in one patient.

20 Here, we demonstrated that mucus plugs from individuals with fatal asthma are heter

21 tasis and likely represents a major cause of mucus plugging in asthma.

22 may serve to identify occult central airway mucus plugging in the ventilated asthmatic patient.

23 ever, some features are different, including mucus plugging, mucus "tethering" to goblet cells, plasm

24 In in vivo models of airway mucus plugs, neutrophil migration was inhibited by thick

25 lbumin-aerosol challenge, with resolution of mucus plugging occurring by 42 days.

26 act infection can result in inflammation and mucus plugging of airways.

27 t represent a protective strategy to prevent mucus plugging of distal airways and thus impaired venti

28 f partially dispersed Paneth granules in the mucus plugs of CF mouse intestinal crypts, and this mucu

29 n alone is not sufficient to trigger luminal mucus plugging or airways inflammation/goblet cell hyper

30 n adult Scnn1b-Tg mice, but did not decrease mucus plugging or neutrophilia.

31 ter the overall permeability of the cervical mucus plug, our findings suggest that the latter mechani

32 secretion, which formed a thick, protective mucus plug overlying the surface epithelium, entrapping

33 radiographic infiltrates, coughing up thick mucus plugs, peripheral and pulmonary eosinophilia, and

34 reduced the airway eosinophil infiltration, mucus plugging, smooth muscle hyperplasia, and subepithe

35 In MCMV/AAD mice, mucus plugging was observed after 7 days of ovalbumin-ae

36 from airway epithelial cells and subsequent mucus plugging, which serves as the focus for infections

37 from airway epithelial cells and subsequent mucus plugging, which serves as the focus for infections

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