ライフサイエンスコーパス: mucus secretion

1 recently been reported to promote excessive mucus secretion.

2 nflammation, microvascular permeability, and mucus secretion.

3 f autophagosomes were required for efficient mucus secretion.

4 leukin-13 (IL-13) is a mediator of pulmonary mucus secretion.

5 d several mechanisms that inhibit neurogenic mucus secretion.

6 es were assessed for airway inflammation and mucus secretion.

7 sal glands do not appear to be involved with mucus secretion.

8 ad to particles that rapidly penetrate human mucus secretions.

9 a disease characterized by hyperviscoelastic mucus secretions.

10 les are similar to those in many other human mucus secretions.

11 eosinophilia, type 2 cytokine production and mucus secretion after allergen inhalation.

12 ole in murine asthma, mediating both AHR and mucus secretion after HDM exposure.

13 h2 cytokine production, airway inflammation, mucus secretion, airway hyperresponsiveness, and serum o

14 bronchoconstriction, airway plasma leakage, mucus secretion and cough.

15 matory, and infectious insults induce airway mucus secretion and goblet cell metaplasia to preserve a

16 tered secretory state can lead to changes in mucus secretion and luminal pH.

17 utonomic and trigeminal innervation controls mucus secretion and may release neurotransmitters into n

18 healthy, intact epithelium results in rapid mucus secretion and movement of Ly6C(+)7/4(+) monocytes

19 negative regulatory role in allergen-induced mucus secretion and MUC5AC expression by regulating STAT

20 l asthma, such as smooth muscle contraction, mucus secretion and recruitment of inflammatory cells, a

21 ammatory responses, and direct activation of mucus secretion and smooth muscle cell constriction.

22 creased IgE production, eosinophil activity, mucus secretion and smooth muscle reactivity, effected t

23 erentiated epithelium with functional cilia, mucus secretion and subepithelial fibroblasts within typ

24 We measured mucus secretion and the expression of Rho-kinase in the

25 loped epithelial cell hyperplasia, increased mucus secretion, and airway hyperreactivity.

26 here it regulates eosinophilic inflammation, mucus secretion, and airway hyperresponsiveness.

27 ncluding gastric acid/bicarbonate secretion, mucus secretion, and cell migration.

28 d mediators involved in bronchoconstriction, mucus secretion, and cell trafficking in asthmatic patie

29 ce showed increased Th2 cytokine production, mucus secretion, and lung infiltration of eosinophils an

30 issue eosinophilia, goblet cell hyperplasia, mucus secretion, and peribronchial edema and also inhibi

31 vented eosinophilia, airway hyperreactivity, mucus secretion, and Th2 cyto-kine production.

32 y shown to regulate airway cell cytokine and mucus secretion, and transepithelial Cl(-) current.

33 of the nanoscale barrier properties of human mucus secretions, and to achieve more uniform and longer

34 d IL-5, IL-13, and TNF-alpha levels; reduced mucus secretion; and improved airway function.

35 lation of human epithelial cells resulted in mucus secretion as measured by MUC5AC mRNA and protein.

36 Here, we show that mucus secretion by goblet cells is altered in the colon

37 clude reflex stimulation of submucosal gland mucus secretion by sensory neurons that release substanc

38 dition, DF of BR increased significantly the mucus secretion compared to control group.

39 nfected IL-27rKO mice showed exacerbation of mucus secretion compared with wild type, as well as enha

40 crease microvascular permeability, stimulate mucus secretion, decrease mucociliary clearance, and app

41 rrhea, coughing, bronchoconstriction, airway mucus secretion, dysphagia, altered gastrointestinal mot

42 n as the most likely mechanism for defective mucus secretion from CF glands.

43 ncrease the rate of cholinergically mediated mucus secretion from CF glands.

44 nor amiloride increased forskolin-stimulated mucus secretion from porcine submucosal glands (75 gland

45 ing regulation of airway smooth-muscle tone, mucus secretion from submucosal glands and surface epith

46 Mucus secretions from X. laevis previously exposed to B.

47 eatment of disorders of epithelial fluid and mucus secretion, hypertension, asthma, and possibly canc

48 at nanoparticles can rapidly penetrate human mucus secretions if they are densely coated with low MW

49 Although clarithromycin reduced mucus secretion in both rhinitis patients and normal sub

50 olinergic pathways still elicit strong gland mucus secretion in CF subjects, it is unclear whether VI

51 Loss of "housekeeping" gland mucus secretion in CF, in combination with demonstrated

52 rinic receptor subtypes mediating neurogenic mucus secretion in ferret trachea were characterized in

53 w levels of VIP and ACh produced significant mucus secretion in human glands via strong synergistic i

54 rine lactone (C4-HSL), on cell viability and mucus secretion in LS174T cells.

55 bition of TMEM16A-CaCC significantly impairs mucus secretion in primary human airway surface epitheli

56 mucin gene expression, mucus composition, or mucus secretion in response to intestinal microbes or ho

57 T-I CD8 T cells, attenuated eosinophilia and mucus secretion in the lungs of sensitized mice in an an

58 Thus, altered mucus secretion in TMF(-/-) mouse colons is accompanied

59 inistration to the lung resulted in enhanced mucus secretion, inflammatory cell recruitment, and cyto

60 tive autophagy in goblet cells and abrogated mucus secretion into the large intestinal lumen.

61 Increased mucus secretion is an important clinical symptom and con

62 Mucus secretion is an important protective mechanism for

63 The dominant neural control of human airway mucus secretion is cholinergic.

64 tic nanoparticles must rapidly penetrate the mucus secretions lining the surfaces of the respiratory,

65 une regulatory pathway governing goblet cell mucus secretion, linking nonhematopoietic inflammasome s

66 cant depletion of goblet cell metaplasia and mucus secretion markers after HDM exposure.

67 vasculature, inflammatory cell recruitment, mucus secretion, mucociliary clearance and airway surfac

68 TMEM16B regulate diverse processes including mucus secretion, neuronal excitability, smooth muscle co

69 ts that may be functionally analogous to the mucus secretions of higher eukaryotes.

70 ese two hypotheses, we measured single gland mucus secretion optically and applied ENaC inhibitors to

71 hohexital nor propofol significantly affects mucus secretion or clearance in healthy dogs.

72 rocess begins when the bacteria aggregate in mucus secretions outside the light organ.

73 d tracheas, using optical methods to monitor mucus secretion rates from multiple glands in parallel.

74 ild infection induces a strong activation of mucus secretion-related genes in young immunocompetent r

75 ory pathways involved in goblet cell-induced mucus secretion remain largely unknown.

76 hma is characterized by airway inflammation, mucus secretion, remodeling and hyperresponsiveness (AHR

77 It appears that increased mucus secretion results from increased mucin gene expres

78 ng of the airways with significantly reduced mucus secretion, subepithelial fibrosis, smooth muscle t

79 Cholinergic stimulation, which elicits mucus secretion, substantially reduced microdisk movemen

80 al range, indicating a rapid recovery in the mucus secretion system.

81 ormally active and stimulate low-level gland mucus secretion that is a component of innate mucosal de

82 therefore investigated the role of HCO3- in mucus secretion using mouse small intestine segments ex

83 PGE(2) is known to induce mucus secretion, vasodilation, and edema, and acts as an

84 hea, cholinergic nerve stimulation increases mucus secretion via muscarinic M3 receptors on the submu

85 increases in airway hyperresponsiveness and mucus secretion were similar to those observed in wild-t