ライフサイエンスコーパス: mule deer

1 n analysis to identify resources selected by mule deer.

2 ulated with brain tissue from a CWD-infected mule deer.

3 repared from the brainstem of a CWD-affected mule deer.

4 of the PRNP gene in susceptibility to CWD in mule deer.

5 Comparable data have not been derived for mule deer, a species susceptible to the TSE chronic wast

6 Mule deer abundance declined by 36% during the developme

7 and-use change with the demographic rates of mule deer, an iconic species in the western United State

8 ttle in the United Kingdom and Europe and in mule deer and elk in parts of the United States has emph

9 same prion strain caused CWD in the analyzed mule deer and elk.

10 rt-term studies of 2-3 years have shown that mule deer and other ungulates avoid energy infrastructur

11 prion protein in tissues from sheep, cattle, mule deer, and elk with naturally occurring transmissibl

12 l lymph node samples from white-tailed deer, mule deer, and moose, collected in the field from areas

13 e findings suggest that CWD prions from elk, mule deer, and white-tailed deer can be readily transmit

14 behavioral effects of energy development on mule deer are long term and may affect population abunda

15 Impacts to mule deer are of particular concern because some of the

16 on levels appeared to influence selection by mule deer because of variability in crop rotation and su

17 uantified antler size of 11,000 male elk and mule deer born throughout the intermountain western US (

18 Primary cultures derived from uninfected mule deer brain tissue were transformed by transfection

19 Mule deer consistently avoided energy infrastructure thr

20 ntain proviruses that are closely related to mule deer CrERVgamma in a conserved region of pol; more

21 e show that prairie voles are susceptible to mule deer CWD prions in vivo and that sPMCA amplificatio

22 We captured 205 adult female mule deer, equipped them with GPS collars, and observed

23 re, on average, 100 CrERVgamma copies in the mule deer genome based on quantitative PCR analysis.

24 ars during development, to determine whether mule deer habituated to natural gas development and if t

25 bal Positioning System collars to monitor 14 mule deer in an agricultural area near public lands in s

26 n exists to understand resource selection of mule deer in response to annual variation in crop rotati

27 arious levels of insertional polymorphism in mule deer individuals.

28 e hypothesized that prion transmission among mule deer might also be enhanced in ranges with relative

29 arrival on birthing areas, especially where mule deer migrate over longer distances or for greater d

30 Here we show that asymptomatic CWD-infected mule deer (Odocoileus hemionus) excrete CWD prions in th

31 en isolated from North American free-ranging mule deer (Odocoileus hemionus) exhibiting mucocutaneous

32 video footage taken from systems deployed on mule deer (Odocoileus hemionus) in north-central Washing

33 range and arrival to summer range of female mule deer (Odocoileus hemionus) in northwestern Colorado

34 fat-free body mass; IFFFBMass) of 136 female mule deer (Odocoileus hemionus) over 8 years.

35 Mule deer (Odocoileus hemionus) populations in the weste

36 of chronic wasting disease (CWD) in captive mule deer (Odocoileus hemionus) that is attributable to

37 or cervid endogenous gammaretrovirus) in the mule deer (Odocoileus hemionus) that is insertionally po

38 In a population of migratory mule deer (Odocoileus hemionus), 31% surfed plant phenol

39 ite-tailed deer (Odocoileus virginianus) and mule deer (Odocoileus hemionus), denoted Tg(DePrP).

40 development on habitat selection patterns of mule deer on their winter range in Colorado.

41 ance of expanding residential development on mule deer populations, a factor that has received little

42 he open reading frame (ORF) in exon 3 of the mule deer PRNP gene revealed polymorphisms in all 145 sa

43 Analysis of BAC clones containing mule deer PRNP genes revealed a full length functional g

44 rom CWD-positive elk, white-tailed deer, and mule deer produced disease in Tg(ElkPrP) mice between 18

45 production and reclamation efforts underway, mule deer remained >1 km away from well pads.

46 One CrERVgamma provirus was detected in all mule deer sampled but was absent from white-tailed deer,

47 ing changes in exon 3 were identified in the mule deer samples examined.

48 We tested the hypothesis that mule deer select certain crops, and in particular sunflo

49 Mule deer selected areas closer to forest and alfalfa fo

50 functional gene alleles from 47 CWD-positive mule deer showed the predominant allele encoded 20D225S

51 Concurrently, we measured abundance of mule deer to indirectly link behavior with demography.

52 on disease) of North American cervids, i.e., mule deer, white-tailed deer, and elk (wapiti).

53 Therefore, CWD isolates from mule deer, white-tailed deer, and elk were inoculated in

54 first to correlate a demographic response in mule deer with residential and energy development at lar