ライフサイエンスコーパス: multi-drug resistance

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1 ii is an important human pathogen due to its multi-drug resistance.

2 metabolism, stress responses, virulence and multi-drug resistance.

3 major contributor to the spread of bacterial multi-drug resistance.

4 tions in topoII have been linked to atypical multi-drug resistance.

5 tant diseases, including cystic fibrosis and multi-drug resistance.

6 report that expression of an exogenous human multi-drug resistance 1 (MDR1) gene enables dramatic ex

7 genes such as CYP (cytochrome)3A4 and MDR-1 (multi-drug resistance-1), that are involved in this proc

8 bacteria increased colon inflammation in the multi-drug resistance 1a-deficient (Mdr1a (-/-) ) mouse

9 of DHM, which induces apoptosis and reverses multi-drug resistance against ovarian cancer cells throu

10 ydrophobic compounds have important roles in multi-drug resistance and can cause a number of diseases

11 Horizontal transfer of mobile DNA conferring multi-drug resistance and expression of a new superantig

12 formulate and analyse a stochastic model for multi-drug resistance and investigate the dependence of

13 Lowering multi-drug resistance and involving influx transportatio

14 rculosis Beijing genotype is associated with multi-drug resistance and is emerging.

15 . cruzi, demonstrate that this can result in multi-drug resistance, and indicate that vigilance will

16 xplaining the elevated rates of triazole and multi-drug resistance associated with C. glabrata.

17 ths due to bacteria and reduce the spread of multi-drug-resistance, but cannot be regularly performed

18 de the development of asymptomatic shedding, multi-drug resistance during prolonged antiviral therapy

19 and we found evidence of the persistence of multi-drug resistance following export from East Asia.

20 ression of drug resistance proteins, such as multi-drug resistance gene-1 P-glycoprotein (MDR1) and m

21 n of the message of MDR1, a gene involved in multi-drug resistance in cancer cells.

22 a major role in both intrinsic and acquired multi-drug resistance in Gram-negative bacteria.

23 also applies to antibiotics that may lead to multi-drug resistance in pathogenic bacteria, aptamer-ba

24 In a previous study, we discovered that multi-drug resistance is common in bacteria isolated fro

25 High prevalence of multi-drug-resistance makes it dangerous and difficult t

26 tibiotic resistance remarkably rapidly, with multi drug-resistance (MDR) rates exceeding 60%.

27 1) protease to develop mutations that confer multi-drug resistance (MDR) has been a major obstacle in

28 s of world, the extremely high prevalence of multi-drug resistance (MDR) in nematodes of sheep and go

29 Multi-drug resistance (MDR) is a phenomenon by which tum

30 es due to the lack of tumor-selectivity, the multi-drug resistance (MDR) to free chemotherapeutic dru

31 In contrast, multi-drug resistance (MDR), resistance to traditional f

32 motherapy because of its tendency to develop multi-drug resistance (MDR), whose various underlying me

33 essful chemotherapy is intrinsic or acquired multi-drug resistance (MDR).

34 The human multi-drug resistance membrane transporter, P-glycoprote

35 s like WhiB7, which account for the observed multi-drug resistance phenotypes.

36 l pfcrt alleles that could contribute to new multi-drug resistance phenotypes.

37 oli O157:H7, as well as the broad host range multi-drug resistance plasmid pB10 from an unknown host.

38 e expression of the ABC transporter proteins multi-drug resistance protein-1 (MDR1, ABCB1, P-glycopro

39 otic polyspecific metabolite transporter and multi-drug resistance pumps.

40 the processes of metastasis, development of multi-drug resistance, the 'Warburg effect', angiogenesi

41 Kit and stably express P-glycoprotein (P-gp)/multi-drug resistance type 1 (MDR1).

42 target-specific ARGs to ARGs associated with multi-drug resistance was seen across influents and effl

43 Polyamine transport alterations and multi-drug resistance were eliminated as causes of the r

44 onstrate that the Bcl-2 oncoprotein produces multi-drug resistance when assessed in primary lymphomas

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