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1  66.4 +/- 12.2 y) for uHCC (36 uninodular, 5 multinodular, 36 diffuse) with (90)Y TARE (41 resin, 36
2 acteristic CT findings of massively enlarged multinodular adrenal glands.
3                 Eleven patients had enlarged multinodular adrenal glands: Nodules were 0.1-5.5 cm.
4 ing syndrome and with bilateral nonpigmented multinodular adrenal hyperplasia.
5  applies to tumors presenting with a diffuse/multinodular as well as solitary nodule pattern.
6  of 56 cases (27%) in which the HCC was in a multinodular, bilobar form (T4), sufficient discordance
7 iltrative), focality (solitary oligonodular, multinodular), disease distribution (unilobar, bilobar),
8  patients referred for a diffuse goiter or a multinodular gland, ultrasonography detected discrete no
9 d higher D2 to D1 mRNA ratios than normal or multinodular glands suggesting differential regulation o
10 ten display thyroid abnormalities, including multinodular goiter and follicular adenomas, and are at
11  of 123] vs 24.3% [70 of 288], P < .001) and multinodular goiter or thyroid nodule (40.7% [50 of 123]
12 land disease (OR, 7.63; 95% CI, 3.49-16.69), multinodular goiter or thyroid nodule (OR, 1.82; 95% CI,
13  clinical practice in nontoxic uninodular or multinodular goiter.
14 ed by any type of hyperthyroidism--eg, toxic multinodular goitre or solitary autonomously functioning
15 ndrome is Graves' disease, followed by toxic multinodular goitre, and solitary hyperfunctioning nodul
16  = 1.81; 95% CI = 1.21-2.71; P = 0.004), and multinodular (&gt;3) HCC (HR = 1.79; 95% CI = 1.21-2.63; P
17 mice, with N-nitrosodiethylamine resulted in multinodular HCC with steatotic features and associated
18 lar adenomas, 10 papillary carcinomas, or 10 multinodular hyperplasias.
19  practice widely applied among patients with multinodular, large, and macrovascular invasive HCC, pro
20 umber: uninodular tumors 2 cm or smaller and multinodular or uninodular tumors 2 cm or larger.
21 n contrast, patients in the study group with multinodular or uninodular tumors larger than 2 cm had w
22 ), nodular (OR = 0.76, p = 3.1 x 10(-5)) and multinodular (OR = 0.69, p = 3.9 x 10(-5)) goiters, and
23 r, clinical decompensation of cirrhosis, and multinodular tumor are independent prognostic predictors
24  a transient viral hepatitis and resulted in multinodular tumorigenesis within 5 to 8 weeks.
25 e for patients with asymptomatic noninvasive multinodular tumors in intermediate-stage disease.
26  total beta-catenin protein were observed in multinodular tumors independent of beta-catenin mutation

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