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1 nd Sox2(COND) hypomorphic alleles, result in multiple abnormalities.
2 However, the cells had multiple abnormalities.
3 normal in LEA rats, whereas LEC rats showed multiple abnormalities.
4 with 0 to 1 abnormality versus patients with multiple abnormalities.
5 often develops in HLVH patients and involves multiple abnormalities.
6 nexpected layers of genetic complexity, with multiple abnormalities associated with disease progressi
14 lts indicate that it is possible to identify multiple abnormalities in some offspring of type II diab
15 ice is lethal in the embryonic period due to multiple abnormalities in the liver, heart, and lung, th
16 e is lethal in the embryonic period, causing multiple abnormalities in the liver, heart, lung, and bl
18 HR) model of genetic hypertension, there are multiple abnormalities in VSMC function, including incre
19 embryos die at embryonic day 9.5, displaying multiple abnormalities including lack of somites, forebr
20 ed by generating Ace -/- mice, which exhibit multiple abnormalities including renal structural defect
21 otic entry, RINT-1-deficient cells exhibited multiple abnormalities, including aberrant Golgi dynamic
22 s are present on most tumor vessels but have multiple abnormalities, including altered expression of
24 hat Bmal1(lox/lox)/Ren1(d)Cre mice exhibited multiple abnormalities, including increased urine volume
25 r targeted disruption of the Alx-4 gene have multiple abnormalities, including preaxial polydactyly.
26 e structures in these adult mutants unmasked multiple abnormalities, including supernumerary spermath
28 lonic carcinoma tissue mRNA and confirm that multiple abnormalities of CD44 mRNA processing occur in
35 The CR rates for patients with 0 to 1 and multiple abnormalities were similar (69% and 54%, respec
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