ライフサイエンスコーパス: multiple organ dysfunction syndrome

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1 n is a key element in the development of the multiple organ dysfunction syndrome.

2 nfiltrates, or occurrence of septic shock or multiple organ dysfunction syndrome.

3 t of systemic inflammatory response syndrome/multiple organ dysfunction syndrome.

4 sregulation of the inflammatory response and multiple organ dysfunction syndrome.

5 for systemic inflammatory response syndrome/multiple organ dysfunction syndrome.

6 o systemic inflammatory response syndrome or multiple organ dysfunction syndrome.

7 ology of endothelial cell dysfunction in the multiple organ dysfunction syndrome.

8 of tissue injuries appears to be involved in multiple organ dysfunction syndrome.

9 t has been implicated in the pathogenesis of multiple organ dysfunction syndrome.

10 Complications in ICU were multiple-organ dysfunction syndrome (34%), shock (28%),

11 ical complications, including progression to multiple organ dysfunction syndrome and even death.

12 n endothelium-targeted therapy to ameliorate multiple organ dysfunction syndrome and improve outcome

13 d liver, correlates with several features of multiple organ dysfunction syndrome and independently pr

14 ma, and burn injury, is often complicated by multiple organ dysfunction syndrome and is accompanied b

15 stablished criteria for sepsis syndrome with multiple organ dysfunction syndrome, and ten noninfected

16 is frequently associated with inflammation, multiple-organ dysfunction syndrome, and the accumulatio

17 dentified as a target for acute pancreatitis multiple organ dysfunction syndrome (AP-MODS); a devasta

18 reatening illness characterized by shock and multiple organ dysfunction syndrome, are discussed.

19 anges in SLPI correlate with the severity of multiple organ dysfunction syndrome as measured by the m

20 itation, therefore, may help protect against multiple-organ dysfunction syndrome as a result of this

21 Persistent organ failure and multiple organ dysfunction syndrome but not necessarily

22 ial translocation and the development of the multiple organ dysfunction syndrome during sepsis.

23 Patients with low and medium multiple organ dysfunction syndrome experienced greater

24 ients with injuries severe enough to develop multiple organ dysfunction syndrome have multiple immune

25 hat their pathogenesis is similar to that of multiple organ dysfunction syndrome in other populations

26 the acute respiratory distress syndrome and multiple organ dysfunction syndrome in patients with sep

27 provide unprecedented opportunities to treat multiple organ dysfunction syndrome in the 21st century.

28 llows assessment of the severity of cases of multiple organ dysfunction syndrome in the PICU with a c

29 ococcus aureus, synergize to cause shock and multiple organ dysfunction syndrome in the rat.

30 systemic inflammatory response syndrome and multiple organ dysfunction syndrome is poorly understood

31 Multiple organ dysfunction syndrome is the main cause of

32 -1-one (ODQ), on the circulatory failure and multiple organ dysfunction syndrome (kidney, liver, lung

33 nterruption was compared and analyzed across multiple organ dysfunction syndrome levels and sedative

34 Multiple organ dysfunction syndrome levels were defined

35 s for 28 days to evaluate outcomes including multiple organ dysfunction syndrome (MODS) and death.

36 of sterile inflammation leading to systemic multiple organ dysfunction syndrome (MODS) and death.

37 condary outcome measures were improvement in multiple organ dysfunction syndrome (MODS) and developme

38 f reactive oxygen species contributes to the multiple organ dysfunction syndrome (MODS) caused by end

39 te lung injury (ALI) is a major component of multiple organ dysfunction syndrome (MODS) following pul

40 y showed rapid improvement in their modified Multiple Organ Dysfunction Syndrome (MODS) score (which

41 inflammatory response syndrome (SIRS score), multiple organ dysfunction syndrome (MODS) score, admiss

42 c inflammatory response, which culminates in multiple organ dysfunction syndrome (MODS) that is norma

43 Multiple organ dysfunction syndrome (MODS) was present i

44 improve lung injury score (LIS), progressive multiple organ dysfunction syndrome (MODS), and an unfav

45 Severe bacterial infection can cause sepsis, multiple organ dysfunction syndrome (MODS), and death.

46 temic inflammatory response syndrome (SIRS), multiple organ dysfunction syndrome (MODS), and mortalit

47 on inflammatory infiltration and subsequent multiple organ dysfunction syndrome (MODS), associated w

48 gressive, fulminant illness resulting in the multiple organ dysfunction syndrome (MODS), with or with

49 storm" can lead to poor outcomes, including Multiple Organ Dysfunction Syndrome (MODS).

50 of its relationship with the development of multiple organ dysfunction syndrome (MODS).

51 an important role in the pathogenesis of the multiple organ dysfunction syndrome (MODS).

52 creased respiratory rate variability for low multiple organ dysfunction syndrome patients (p < 0.05 f

53 ratory rate variability occurred in the high multiple organ dysfunction syndrome patients.

54 nd further suggest that progression into the multiple organ dysfunction syndrome reflects progressive

55 g, severe head injury, or the development of multiple organ dysfunction syndrome remains all too comm

56 ty of organ dysfunction was assessed through multiple organ dysfunction syndrome scores, and sedative

57 for new treatment approaches for those with multiple organ dysfunction syndrome; several examples ar

58 limination of sedation in patients with high multiple organ dysfunction syndrome suggests a different

59 t of systemic inflammatory response syndrome/multiple organ dysfunction syndrome that is causal to cr

60 a variety of human diseases like sepsis and multiple organ dysfunction syndrome, the leading cause o

61 f variation), compared to patients with high multiple organ dysfunction syndrome, who failed to mount

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