ライフサイエンスコーパス: multipolar

1 , unipolar, bipolar, inverted pyramidal, and multipolar.

2 monopolar morphology of the P cell becoming multipolar.

3 ng the phenotypic conversion from bipolar to multipolar.

4 halic trigeminal neurons, some of which were multipolar.

5 ied, though the spindles may be short and/or multipolar [3, 4] and the fidelity of chromosome distrib

6 ted the efficacy of a novel linear irrigated multipolar ablation catheter capable of creating linear

7 g CDK2 inhibition, lung cancer cells develop multipolar anaphase and undergo multipolar cell division

8 extra centrosomes generate CIN by promoting multipolar anaphase, a highly abnormal division that pro

9 amplify during papillar endocycles, causing multipolar anaphase.

10 evere HDR disruption additionally results in multipolar anaphases and loss of clonogenic survival.

11 They displayed multipolar and complex dendritic arbors in ipsilateral L

12 rafish embryos leads to mitotic defects with multipolar and disorganized mitotic spindles.

13 ECs causes increases in centrosome numbers, multipolar and disorganized spindles, and unaligned chro

14 rcollicular nucleus were large and generally multipolar and had extensive, sparsely branching central

15 ogically, these cells varied from bipolar to multipolar and included basket-like and chandelier cells

16 overall movement is radial, but they become multipolar and move nonradially in the intermediate zone

17 itional dependence of molecular orientation (multipolar) and orbital (quadrupolar) order in the perov

18 they were generally larger, were more often multipolar, and (in cervical enlargement) had stronger N

19 le spindle aberrations, including elongated, multipolar, and fractured spindles.

20 e classified morphologically into pyramidal, multipolar, and fusiform types.

21 The overall densities of unipolar, bipolar, multipolar, and inverted pyramidal neurons did not diffe

22 ant (e.g., flattened pyramidal, nonpyramidal multipolar, and inverted pyramidal neurons).

23 gets is very diverse and includes pyramidal, multipolar, and neurogliaform cells.

24 e rest of the NK1R-ir neurons of the VRG are multipolar, are larger (somatic cross section: 252 +/- 1

25 cal types of neurons--unipolar, bipolar, and multipolar--are important for information processing and

26 ficantly fewer processes than non-neurogenic multipolar astrocytes.

27 have a more heterogeneous representation of multipolar, bipolar, and unipolar interneurons.

28 Neurons typically assume multipolar, bipolar, or unipolar morphologies.

29 in the rat developing neocortex affects the multipolar-bipolar transition of neurons leading to dela

30 AC precursors are initially multipolar but lose neurites as they migrate through the

31 roanatomical mapping system was used in 70%, multipolar catheter in 51%, and real-time image integrat

32 In 21 patients, a multipolar catheter placed within scar visualized spatia

33 in 45 patients with AF were identified using multipolar catheter recordings.

34 geting the earliest of LPs visualized on the multipolar catheter, and the impact on later LPs was rec

35 With the use of a multipolar catheter, pacing was performed from electrode

36 risk ratio=0.49 [0.33-0.74]), and the use of multipolar catheters (R(2)=0.08; P=0.05; risk ratio=0.75

37 All clinical high-resolution multipolar catheters are of sufficient resolution to acc

38 Contact mapping was performed using biatrial multipolar catheters in 36 AF subjects (29 persistent).

39 and use of scar integration from imaging and multipolar catheters to focus high-density mapping are i

40 by AFM interactions between the dipolar and multipolar Ce moments .

41 Terminals of auditory nerve fibers in the multipolar cell area included both large and small endin

42 selves ensheathed by a layer of processes of multipolar cell body glia.

43 ls with more than two centrosomes to undergo multipolar cell division leading to apoptosis, defined a

44 ells develop multipolar anaphase and undergo multipolar cell division with the resulting progeny apop

45 lls with multiple centrosomes rarely undergo multipolar cell divisions, and the progeny of these divi

46 ed aberrant mitosis including asymmetric and multipolar cell divisions.

47 is required for cells to transit out of the multipolar cell phase and to enter into the cortical pla

48 nregulation of FoxG1 at the beginning of the multipolar cell phase induces Unc5D expression, the timi

49 The projections of one type of labeled multipolar cell, planar neurons, were traced into the ve

50 A small group of large, multipolar cells along the ventral edge projects to DP/P

51 First, the number of labeled VCN planar multipolar cells and the amount of labeling in the LSO w

52 rade tracing data revealed that medium-sized multipolar cells from the magnocellular part of the LRN

53 tly generated by soma-targeting fast-spiking multipolar cells in layer III, which in turn were driven

54 premotor neurons within the OPt, as well as multipolar cells in the nucleus of the posterior commiss

55 Time-lapse imaging of multipolar cells in the subventricular zone revealed tha

56 Ventrotubercular cells are multipolar cells in the ventral cochlear nucleus (VCN) t

57 ells, thick-smooth dendrite cells, and large multipolar cells of the intermediate layer were present

58 We investigated whether multipolar cells of the ventral cochlear nucleus have pr

59 These multipolar cells showed altered localization of a leadin

60 The other type comprised multipolar cells that were located at the origin and int

61 onsists of short pyramidal neurons and large multipolar cells with "polarized" dendritic trees; group

62 Multipolar cells with onset chopper (O(C)) responses inn

63 We suggest that multipolar cells within the PVCN have the distribution a

64 We infer that planar multipolar cells, in addition to bushy cells, are a sour

65 d terminals, as well as retrogradely labeled multipolar cells, were present in the contralateral OPt,

66 Labeled cells in DCN were either fusiform or multipolar cells, whereas those in Sp5 varied in size an

67 n, and had morphological features of radiate multipolar cells.

68 stem is composed of a considerable number of multipolar centrifugal neurons, resembling the "ectopic"

69 imple point charge models and more elaborate multipolar charge models for the CO-ligand are smaller b

70 er from monkeys in having a preponderance of multipolar ChAT-ir interneurons in the caudate nucleus a

71 f disorganized spindle microtubules owing to multipolar configurations and defects in chromosome segr

72 us identifies the K-fiber meshwork of linked multipolar connectors as a key integrator and determinan

73 The K-fiber mesh is made of linked multipolar connectors.

74 l evaluation of electrograms recorded on the multipolar coronary sinus and ablation catheters was und

75 ently recorded interneuron subtype had short multipolar dendrites and a dense local axonal arborizati

76 Thus, multipolar divisions cannot explain observed rates of CI

77 Although most multipolar divisions failed and cell fusion occurred, so

78 oo low to cause mitotic arrest and result in multipolar divisions instead.

79 ase, whereas, severe HDR deficiency leads to multipolar divisions that are prohibitive for cell proli

80 omes in tumor cells create the potential for multipolar divisions that can lead to aneuploidy and cel

81 tosis through a signaling cascade leading to multipolar divisions, and its knockout promotes clusteri

82 nesin-14 HSET, which likely accounts for the multipolar divisions, and overexpression of HSET reduced

83 This aneuploidy resulted from multipolar divisions, chromosome missegregation, and fai

84 mitosis, avoiding the detrimental effects of multipolar divisions.

85 f live NPCs revealed lagging chromosomes and multipolar divisions.

86 cancer cells with extra centrosomes to avoid multipolar divisions.

87 e injected with neurobiotin were found to be multipolar Dogiel type II neurons.

88 e >250 s) in 10 procedures (group 1), with a multipolar duty-cycled radiofrequency pulmonary group 2)

89 ed radiofrequency, argon plasma coagulation, multipolar electrocoagulation, and photodynamic therapy.

90 ctivation pattern recorded from conventional multipolar electrode catheters was characteristic of clo

91 , resulting in e.g. dipolar, quadrupolar, or multipolar emission on demand.

92 In order to facilitate the design of multipolar fluorine-backbone interactions in protein-lig

93 sed at protein interfaces, we postulate that multipolar fluorine-backbone interactions may represent

94 acking the IGF-1 receptor remain arrested as multipolar forming a highly disorganized tissue.

95 na I projection neurons have been described: multipolar, fusiform, and pyramidal.

96 ynapses on somata and dendritic processes of multipolar GABAergic interneurons, recognized sites for

97 The resulting multipolar geometries resemble electrostatic charge conf

98 Individual activated projection neurons were multipolar, globular, or fusiform in shape.

99 well with experimental data for bipolar and multipolar HT-29 colorectal cancer cells.

100 Multipolar interactions involving fluorine and the prote

101 We show that nonlinear multipolar interference allows achieving not only unidir

102 Multipolar interneurones with narrow spikes generated th

103 of Cajal (ICC)-like cells (AIL cells) with a multipolar, irregular, elongated shape and with numerous

104 f the left atrium (LA) was performed using a multipolar Lasso catheter guided by intracardiac echocar

105 y ablation in a linear pattern, an irrigated multipolar linear ablation catheter safely delivers cont

106 te the excitation of 16 distinct high-order, multipolar, localized surface phonon polariton resonance

107 even assuming beamed emission, but a strong multipolar magnetic field can describe its properties.

108 e areas of slow conduction with simultaneous multipolar mapping.

109 gthened mitosis, centrosome overduplication, multipolar metaphase spindles, and misaligned chromosome

110 with somal translocation induces a switch to multipolar migration.

111 intact centrioles whose presence results in multipolar mitoses and aneuploidy.

112 -phase entry, giant nuclei, multinucleation, multipolar mitoses and centrosome hyperamplification.

113 leted cells showed a significant increase of multipolar mitoses and other spindle pole defects.

114 ls with supernumerary centrosomes to undergo multipolar mitoses resulting in apoptotic cell death.

115 HPV-induced centrosome abnormalities, multipolar mitoses, and aneuploidy often occur at early

116 acquisition of > or =3 centrosomes-generates multipolar mitoses, aneuploidy, and chromosome instabili

117 to significant increases in multinucleation, multipolar mitoses, failed abscission, asymmetric segreg

118 47 and low-dose paclitaxel induces aberrant, multipolar mitoses, mitotic slippage and multinucleation

119 cells, thereby increasing the propensity for multipolar mitoses, which can lead to chromosome missegr

120 in abolishes centrosome clustering, yielding multipolar mitoses.

121 ancer cells defined mechanisms that suppress multipolar mitoses.

122 ary nor sufficient to cause the formation of multipolar mitoses.

123 y divide because of mechanisms that suppress multipolar mitoses.

124 Importantly, multipolar mitosis results in global unbalanced chromoso

125 y phosphatase knockdown, multinucleation and multipolar mitosis were markedly reduced, resulting in e

126 n by shRNA caused focal cytokinesis defects, multipolar mitosis, and multinuclearity as observed in t

127 gression and centrosome number, resulting in multipolar mitosis.

128 The deletion of MdmX induces multipolar mitotic spindle formation and the loss of chr

129 overduplication and has been shown to cause multipolar mitotic spindle formation, a diagnostic hallm

130 Here we show that multipolar mitotic spindles form frequently in mouse pol

131 tinucleated cells, centrosome amplification, multipolar mitotic spindles, aneuploidy, and ability for

132 of microtubule dynamics by MAP2 resulted in multipolar mitotic spindles, defects in cytokinesis and

133 genomic instability through the formation of multipolar mitotic spindles.

134 by inducing abnormal centrosome numbers and multipolar mitotic spindles.

135 This multipolar mode is less efficient but still leads to suc

136 e unambiguously associated with any specific multipolar mode.

137 may be associated with interference between multipolar modes.

138 number of cells exhibiting distinct round or multipolar morphologies, corresponding to cells expressi

139 ar and bipolar neurons causes them to assume multipolar morphologies.

140 ajority of migratory new neurons exhibited a multipolar morphology and moved in a nonlinear manner fo

141 ike transcription factor Dar1 determines the multipolar morphology of postmitotic neurons in Drosophi

142 one to the overlaying cortical plate, assume multipolar morphology while passing through the transien

143 in the lower intermediate zone either showed multipolar morphology with short neurites or possessed n

144 yed TH-IR (9 with bipolar morphology, 6 with multipolar morphology) while the remaining 28 neurons di

145 y TH-IR (18 with bipolar morphology, 10 with multipolar morphology).

146 cal phenotypes caused by Merlin loss, namely multipolar morphology, enhanced cell-matrix adhesion, fo

147 ons by transiently adopting a characteristic multipolar morphology.

148 morphology; the remainder had Dogiel type II multipolar morphology.

149 motor activity was necessary to maintain the multipolar morphology.

150 By examining the critical behavior of the multipolar nematic order parameter, we show that it driv

151 We uncovered a multipolar nematic phase of matter in the metallic pyroc

152 hat the transcription factor Dar1 determines multipolar neuron morphology in postmitotic neurons by r

153 at these genes are required for Dar1-induced multipolar neuron morphology.

154 st common morphological type of CR+ neurons, multipolar neuronal morphology was typical among PV+ neu

155 Dar1 is specifically expressed in multipolar neurons and loss of dar1 gradually converts m

156 se data suggest that cochlear nucleus planar multipolar neurons drive the MOC neuron's response to so

157 rence between the two classes is that type I multipolar neurons have fewer than 35% of their somata a

158 were also found on globular bushy cells and multipolar neurons in the PVCN and AVCN.

159 kely due to selective preservation of large, multipolar neurons in the subcoeruleus.

160 ological transformation and migration of the multipolar neurons in the SVZ/IZ.

161 neurons and loss of dar1 gradually converts multipolar neurons into the bipolar or unipolar morpholo

162 ial nucleus of the trapezoid body, and small multipolar neurons of the contralateral ventral nucleus

163 uron types: spherical bushy cells and planar multipolar neurons of the ipsilateral ventral cochlear n

164 Depletion of kinesin-6 results in multipolar neurons that either are stationary or continu

165 Pase and N-cadherin (NCad) are important for multipolar neurons to polarize their migration toward th

166 and vertically oriented bipolar neurons and multipolar neurons were approximately equally frequent a

167 Multipolar neurons were found caudally, dorsal and ventr

168 Nonpyramidal cells ranged from large multipolar neurons with radiating dendrites, to Martinot

169 oneurons, Rohde nucleus cells, small ventral multipolar neurons, and Edinger cells.

170 argest cells, which were pyramidal and spiny multipolar neurons, appeared earliest.

171 orrelated with the number of labeled radiate multipolar neurons, suggesting radiate neurons as the pr

172 rescues the effect of Cx43 knockdown in the multipolar neurons.

173 possesses a dense core region consisting of multipolar neurons.

174 rom posteroventral cochlear nucleus stellate/multipolar neurons.

175 keletal compartmentalization is conserved in multipolar neurons.

176 n (FRA) and occurred preferentially in large multipolar neurons.

177 tecture with a random distribution of simple multipolar neurons.

178 Many nonpyramidal cells were multipolar, of which three subtypes (large, medium, and

179 onnected pyramidal neurons (presynaptic) and multipolar or bitufted interneurons (postsynaptic).

180 These cells were multipolar or bitufted, had a widely extending axon, and

181 ation that was 30-40 micrometer in diameter, multipolar or pyramidal in shape, and immunoreactive for

182 ns, we observed spindles that were unipolar, multipolar, or frayed with no defined poles.

183 Recent discoveries of pseudogap, magnetic multipolar ordered and possible d-wave superconducting p

184 ransition to a less directionally persistent multipolar phase of migration.

185 y discovered electronic nematic phases, this multipolar phase spontaneously breaks rotational symmetr

186 in the absence of UNC-6 and SLT-1 induces a multipolar phenotype with undirected outgrowths.

187 nd loss of its function caused a distinctive multipolar phenotype.

188 Multipolar plasmon resonances up to fourth order were me

189 at exhibits optical properties influenced by multipolar plasmonic coupling.

190 ctin cDNAs caused a dramatic accumulation of multipolar progenitor cells within the subventricular zo

191 Rac activation, leading to the formation of multipolar protrusions and increased cell circularity, a

192 romote actin polymerization and formation of multipolar protrusions, thereby retarding cell migration

193 Multipolar Purkinje cells are located in the central reg

194 spond to parallel fiber activation, but only multipolar Purkinje cells showed characteristic all-or-n

195 e an orbital disorder/order transition and a multipolar reorientation transition, each occurring at d

196 Due to their multipolar resonant response and low intrinsic losses th

197 allows for the manipulation of the nonlinear multipolar response of a meta-atom, resulting in e.g. di

198 rates leads to new features appearing in the multipolar response.

199 rates of MT formation and were predominantly multipolar, revealing a function of augmin in stabilizin

200 resent new opportunities for engineering the multipolar scattering response of dielectric optical ant

201 nged metaphase arrest, anaphase bridges, and multipolar segregations.

202 lls (41 out of 131 neurons, i.e., 31%) had a multipolar soma, while the other group (87 out of 131 ne

203 ar, horizontal; and perpendicular bipolar or multipolar somata.

204 , as predicted by quantum theories involving multipolar spin interactions.

205 centrosomes can result in the assembly of a multipolar spindle and lead to chromosome mis-segregatio

206 rosomes from spindle poles, the formation of multipolar spindle arrays and cytokinetic defects.

207 The model results explain why multipolar spindle could inhibit SAC silencing and spind

208 ion of beta-tubulin isotypes and resulted in multipolar spindle formation and apoptosis.

209 Inhibition of centrosome clustering triggers multipolar spindle formation and mitotic catastrophe, of

210 en correlated with an increased incidence of multipolar spindle formation in some cancer cells that c

211 ndicating that Eg5 function is necessary for multipolar spindle formation in the absence of hTPX2.

212 cancer cells, inhibited mitosis and induced multipolar spindle formation in these cells.

213 auses multiple centrosomal defects, aberrant multipolar spindle formation, and chromatin missegregati

214 Loss of control over mitotic progression, multipolar spindle formation, and cytokinesis defects ar

215 erary centrosomes, multinucleated cells, and multipolar spindle formation.

216 rosome amplification, chromosome lagging and multipolar spindle formation.

217 ient to produce centrosome amplification and multipolar spindle formation.

218 blastic leukemia cells resulted in increased multipolar spindle frequency that correlated with centro

219 As a result, GRP94 knockdown cells form a multipolar spindle instead of bipolar morphology and con

220 quence of cells passing through a transient 'multipolar spindle intermediate' in which merotelic kine

221 ntrosome and spindle pole numbers, producing multipolar spindles (most ESCRT-III/VPS4 proteins) or mo

222 ntrosomal and mitotic abnormalities, such as multipolar spindles and asymmetric, bipolar spindles wit

223 Multipolar spindles and bipolar spindles with misaligned

224 rturbation of IKK2 promotes the formation of multipolar spindles and chromosome missegregation.

225 Moreover, imaging studies revealed multipolar spindles and chromosome segregation defects i

226 osome replication, resulting in tripolar and multipolar spindles and defective cytokinesis.

227 ell division, resulting in the generation of multipolar spindles and free microtubule-organizing cent

228 ent before anaphase onset, which can lead to multipolar spindles and genomic instability.

229 ed loss of the shRNA screening hits leads to multipolar spindles and heterogeneous chromosome content

230 eated cells, with supernumerary centrosomes, multipolar spindles and lagging chromosomes during anaph

231 cells show an aberrant number of centrosome, multipolar spindles and micronuclei formation.

232 Mitotically arrested cells display multipolar spindles and misalignment of chromosomes, ind

233 tions and accumulation of mitotic cells with multipolar spindles and unaligned chromosomes.

234 uploid cells result from aberrant mitosis as multipolar spindles are induced by the stabilizing drugs

235 reas HBXIP overexpression causes tripolar or multipolar spindles due to excessive centrosome replicat

236 ci of microtubule organization, formation of multipolar spindles from adjacent centrosome pairs, and

237 y in mitosis, amplified centrosomes assemble multipolar spindles in CDK5RAP2 mutant cells.

238 evelopment, as we observe both monopolar and multipolar spindles in Eg5 transgenic mice.

239 also induced the formation of monopolar and multipolar spindles in mitotic cells.

240 l clustering that prevented the formation of multipolar spindles in noncancer cells.

241 Depletion of Kif2a generated multipolar spindles in stage 12 embryos.

242 crosslinks sufficient to stabilize aberrant multipolar spindles may form.

243 pecifically associated with the formation of multipolar spindles only when centrosomes were present.

244 overcome this clustering mechanism to allow multipolar spindles to form at a high frequency.

245 The formation of multipolar spindles via centrosome splitting after LIC d

246 ormation of acentrosomal poles, meaning that multipolar spindles were observable only in cells with a

247 mation of multiple numbers of centrioles and multipolar spindles with abnormal chromosome arrangement

248 Multipolar spindles with poles that contain single centr

249 terfering RNA (siRNA) resulted in cells with multipolar spindles, aberrant mitosis, formation of mult

250 letion resulted in distorted, elongated, and multipolar spindles, accompanied by aberrant chromosomal

251 RD9-depleted cells have fragmented PCM, form multipolar spindles, activate the spindle assembly check

252 ncluding formation of unstructured spindles, multipolar spindles, and chromosome missegregation, lead

253 l cells, including centrosome amplification, multipolar spindles, and chromosome missegregation.

254 d abnormal cytokinesis with delayed mitosis, multipolar spindles, and increased apoptosis, rescued by

255 ncluding misaligned and lagging chromosomes, multipolar spindles, and increased tetraploidy.

256 cells with extra centrosomes initially form multipolar spindles, but these spindles ultimately becom

257 Cells lacking TOGp assembled multipolar spindles, confirming results of Gergely et al

258 accumulated high centrosome numbers, formed multipolar spindles, displayed micronuclei formation, an

259 ividual depletion enhances the generation of multipolar spindles, increases mitotic transit time, and

260 cancer cells, including multinucleation and multipolar spindles, indicating that these changes are s

261 These AMtOCs assemble multipolar spindles, leading to chromosome missegregatio

262 ng lamin B caused formation of elongated and multipolar spindles, which could be reversed by partiall

263 cts, including chromosome missegregation and multipolar spindles, which eventually lead to the activa

264 soform, induced centrosome amplification and multipolar spindles.

265 g that results in formation of elongated and multipolar spindles.

266 these assembly pathways caused formation of multipolar spindles.

267 romatid separation, lagging chromosomes, and multipolar spindles.

268 ation of the centrosome and the formation of multipolar spindles.

269 a pronounced attenuation at prometaphase and multipolar spindles.

270 at HBx induced supernumerary centrosomes and multipolar spindles.

271 arrested by 5-Br-nosc and Rd 5-Br-nosc form multipolar spindles.

272 t cells lacking hTPX2 arrest in mitosis with multipolar spindles.

273 myt1 mutant spermatocytes enter meiosis with multipolar spindles.

274 increases the number of anaphase bridges and multipolar spindles.

275 the central region of the lobe, with large, multipolar, spiny dendrites and locally ending axons.

276 migration was independently disrupted at the multipolar stage and accompanied by premature ectopic ex

277 tion and microtubule organization during the multipolar stage as important determinants of axon forma

278 NAi have revealed that transition out of the multipolar stage depends on the function of filamin A, L

279 The multipolar stage occurs as bipolar progenitor cells beco

280 The multipolar stage therefore seems to be a critical point

281 e passage of migrating neurons through their multipolar stage via p27 signaling and that interference

282 rphological stage of neuronal migration, the multipolar stage, is vulnerable and is disrupted in seve

283 ontinuously from nearly zero at remnant in a multipolar state, to a large value under the maximum ele

284 implications of cooperative quadrupolar and multipolar states for the design of relaxor-like hybrid

285 Rather, at the pyramidal-to-multipolar synapse, raised P(r) increases the transient

286 At the pyramidal-to-multipolar synapse, which shows paired-pulse depression,

287 radial glial cells and are distinct from the multipolar Tbr2((+)) intermediate progenitors, which div

288 up-regulation disrupts the transition from a multipolar to a bipolar neuronal morphology in the inter

289 The change of the morphology from multipolar to bipolar cells was also attenuated.

290 otin1 or Kif20b hindered the transition from multipolar to bipolar cells.

291 e-born neurons fail to correctly switch from multipolar to bipolar morphology, resulting in impaired

292 nsition of newly born migrating neurons from multipolar to bipolar morphology.

293 rol distinct steps of the migratory process, multipolar to bipolar transition in the intermediate zon

294 m a complete failure in progression from the multipolar to the migratory bipolar state, as revealed b

295 suggest that SARA regulates the orientation, multipolar-to-bipolar transition and the positioning of

296 r interactions--such as extremely high-order multipolar transitions, two-plasmon spontaneous emission

297 ten considered "weak", including "orthogonal multipolar" types represented by, for example, F-CO, sul

298 The explicit mobile moieties are the ion, multipolar waters, and the carbonyls and amides of the p

299 s with locally branching axons fall into the multipolar (with ventrally protruding dendrites) and fla

300 ed, they cannot be inactivated, resulting in multipolar zygotic spindles.