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1 tors in the injured retina are intrinsically multipotent.
2 actility (seeded on 2 mum gratings) remained multipotent.
3 ges, a large fraction of the progenitors are multipotent.
4 that individual neural crest precursors are multipotent.
5 hat at the population level Cux2(+) RGCs are multipotent.
6 ells: they are self-renewing, clonogenic and multipotent.
7 press the neural crest marker p75 and become multipotent.
10 n the groups (64 [99%] of 65 patients in the multipotent adult progenitor cell group vs 59 [97%] of 6
11 score of 8-20 to treatment with intravenous multipotent adult progenitor cells (400 million or 1200
13 ations consisted of 65 patients who received multipotent adult progenitor cells and 61 patients who r
14 tients were randomly assigned (67 to receive multipotent adult progenitor cells and 62 to receive pla
16 rolled, dose-escalation trial of intravenous multipotent adult progenitor cells in 33 centres in the
17 ved at 90 days in neurological outcomes with multipotent adult progenitor cells treatment, further cl
19 t, well-tolerated, and safest single dose of multipotent adult progenitor cells, and if they were eff
20 potent murine cells spontaneously convert to multipotent adult spermatogonial-derived stem cells (MAS
23 tted cells are molecularly distinct, whereas multipotent and bipotent progenitors do not exhibit diff
25 ic stem cells and their differentiation into multipotent and downstream lymphoid-biased progenitors.
27 post-embryonic retinal NSCs are exclusively multipotent and give rise to the complete spectrum of ce
28 ve also uncovered the presence of a range of multipotent and lineage-restricted progenitor cells in t
30 ude that pro-centrioles/pro-basal bodies are multipotent and not committed to form either a 9+2 or 9+
34 ene silencing and variegation in cell lines, multipotent and pluripotent stem cells, and their differ
37 onal and population levels Fezf2(+) RGCs are multipotent and that at the population level Cux2(+) RGC
38 ad, only two progenitor classes predominate, multipotent and unipotent, with Er-Mk lineages emerging
40 stem cell-like ability to self-renew and the multipotent capacity to reconstitute the entire spectrum
42 hancers that become epigenetically primed in multipotent cardiovascular mesoderm, to regulate the div
44 ese findings support ex vivo fucosylation of multipotent CD34(+) CB cells as a clinically feasible me
46 ethod that generated neural derivatives with multipotent cell fate potential and normal karyotype.
47 e, thus resulting in a rapid decrease in the multipotent cell population and compromising their regen
48 The neural crest (NC) is a highly migratory multipotent cell population that forms at the interface
50 evelopment, the glycoprotein 2 (GP2) marks a multipotent cell population that will differentiate into
52 eneration through paracrine effects and as a multipotent cell source, and has received recent attenti
53 duced cell plasticity occurs via a transient multipotent cell state and that concomitant exposure to
55 ur cytokine priming, increased the number of multipotent cells (CD34+CD90+) generated; however, the d
56 both the sequence of lineage choices made by multipotent cells and to identify the genes influencing
61 lexity of gene regulatory networks that lead multipotent cells to acquire different cell fates makes
63 s genes that cause them to become migratory, multipotent cells, distinguishing them from adjacent sta
64 ocrine-selective transcription in epithelial multipotent cells, nascent endocrine progenitors, and di
66 e observed a biphasic distribution of Yan in multipotent cells, with a rapid inductive phase and slow
73 ntiate hESC and hiPSC into neural-restricted multipotent derivatives or specialized cell types under
74 om exfoliated deciduous teeth (SHED) possess multipotent differentiation and immunomodulatory propert
76 e postnatal stem cell population, possessing multipotent differentiation capacity and immunomodulator
78 ing of the epigenetic programs necessary for multipotent differentiation of MSCs that may prove benef
79 ages and yet capable of self-replication and multipotent differentiation, being able to differentiate
80 ncer stem cells, capable of self-renewal and multipotent differentiation, influence tumor behavior th
82 ives and their pharmacological evaluation as multipotent drugs for the treatment of Alzheimer's disea
84 dels can be developed in situ from different multipotent embryonic cerebellar progenitor cells via co
88 ose tissue originates from the epicardium, a multipotent epithelium that until now is only establishe
89 eveal the births of larval astrocytes from a multipotent glial lineage, their allocation to reproduci
92 EML cell line suggests that interconvertible multipotent hematopoietic cell subsets coexist in a home
94 x/flox):Scl-Cre-ER(T)) and demonstrated that multipotent hematopoietic colonies form despite the abse
95 ne receptor CCR9 controls the immigration of multipotent hematopoietic progenitor cells into the thym
96 ents diagnosed with BCR-ABL1-positive ALL, a multipotent hematopoietic progenitor is affected by the
100 , hepatoblasts (rHBs), two lineage stages of multipotent, hepatic precursors with overlapping but als
102 ll population that shares many features with multipotent HSCs and serves as a lineage-restricted emer
104 le scaffolds capable of supporting growth of multipotent human bone marrow mesenchymal stem cells (hM
107 eloid lineages suggesting the involvement of multipotent, immature hematopoietic cells in the pathoph
108 scribe a protocol to generate expandable and multipotent induced cardiac progenitor cells (iCPCs) fro
109 After in vitro enzymatic inhibition assays, multipotent inhibitors showing potencies in the nanomola
112 onal pioneering during B cell programming of multipotent lymphoid progenitors by restricting chromati
115 es (MDS), we establish the existence of rare multipotent MDS stem cells (MDS-SCs), and their hierarch
116 memory formation depends on elucidating how multipotent memory precursor (MP) cells maintain develop
117 to H19 in the cytoplasm of undifferentiated multipotent mesenchymal C2C12 cells, and this interactio
118 ess and application of mechanical stretch to multipotent mesenchymal cells stimulated the nuclear tra
125 ation consisting of mature white adipocytes, multipotent mesenchymal stem cells, committed progenitor
126 ly responsible for the beneficial effects of multipotent mesenchymal stem cells, for example in the t
127 llicular thyroid cancers and the presence of multipotent mesenchymal stem/stromal cells (MSCs) in non
129 imum (estimate+/-SE, 0.01+/-0.002; P<0.001), multipotent mesenchymal stromal cell colony maximum (est
132 ce, we demonstrate expression exclusively in multipotent mesenchymal stromal cells (MSCs) in the bone
134 oduce therapeutically relevant quantities of multipotent mesenchymal stromal cells (MSCs) via in vitr
137 omprise the head and face are derived from a multipotent migratory progenitor cell population called
140 (ESC)-based model yields high proportions of multipotent NC cells (expressing SOX10, PAX7 and TFAP2A)
141 vival, differentiation, and migration of the multipotent NCCs critical for mammalian cardiovascular d
145 ipheral nervous system (PNS), originate from multipotent neural crest cells that also give rise to ot
146 ved skilled forelimb function after grafting multipotent neural progenitor cells into sites of SCI.
148 involving permanently labeled cells revealed multipotent neural stem cells (NSCs) of embryonic origin
149 epithelial anterior PS cells are Sox2(+)T(+) multipotent NMPs and form the bulk of neural progenitors
150 or the existence of a sortable population of multipotent non-epithelial cells in the adult pancreas t
151 Herein, we studied the effects of ETH on rat multipotent NSC proliferation and neuronal differentiati
152 oma function in vivo, which is mediated by a multipotent NT5E(+) (CD73)(+) ENG(-) (CD105)(-) LY6A(+)
153 monstrates that BMI1 is expressed in vivo by multipotent OE progenitors, validating our culture model
154 s do not require transit through a requisite multipotent or bipotent megakaryocyte-erythrocyte progen
155 ells produce all cells of an organism, while multipotent or unipotent stem cells regenerate only spec
158 cell fate, before migration to the limb, in multipotent Pax3(+) cells in the somite of the mouse emb
162 Omental adipose stromal cells (O-ASC) are a multipotent population of mesenchymal stem cells contain
163 e vertebrate body and differentiation of the multipotent posterior progenitor cells to the muscle cel
164 ther types of ILC2, MPP(type2) cells exhibit multipotent potential and do not express T1/ST2 or IL-7R
165 hich have been proposed to arise either from multipotent precursor cells or pools of heterogeneous, r
167 l checkpoints that dictate the commitment of multipotent precursors to the T cell lineage and their s
169 transition from naive/primed pluripotency to multipotent primary neural progenitor cells (NPCs).
170 ing of changes with age in the heterogeneous multipotent progenitor (MPP) cell compartment, which is
171 rease in hematopoietic stem cells (HSCs) and multipotent progenitor (MPP) cells in conditional Runx1-
172 CL12, but whether a separate niche instructs multipotent progenitor (MPP) differentiation remains unc
173 al progenitors and PMPs derive from a common multipotent progenitor called the neuromesodermal progen
174 mming to pluripotency to generate an induced multipotent progenitor cell (iMPC) state from which endo
176 thelium, for the subsequent formation of the multipotent progenitor cell (MPC) population that gives
179 of JMML using mice that express KrasG12D in multipotent progenitor cells (Flt3Cre+ KrasG12D mice).
180 letion leads to a reduced pool of pancreatic multipotent progenitor cells (MPCs) due to decreased pro
181 raftment of human PSC-derived (hPSC-derived) multipotent progenitor cells (MPP) has hampered the clin
183 roles in the migration and specification of multipotent progenitor cells at the onset of cardiogenes
184 capacity to predict and control when and how multipotent progenitor cells commit to the desired cell
185 e of differentiated tissue-specific stem and multipotent progenitor cells for regenerative medicine a
186 ) mice, and the frequency of lymphoid-primed multipotent progenitor cells in bone marrow was reduced.
187 ke receptor stimulation, short-term HSCs and multipotent progenitor cells produce copious amounts of
190 , the biological secretome of Amnion-derived Multipotent Progenitor cells, contains multiple anti-inf
191 monstrates the persistence of stem cells and multipotent progenitor cells, their nature in the OE rem
194 their order; the final mutations occur in a multipotent progenitor derived from the preleukemic HSC
197 epithelium of the branching mouse lung is a multipotent progenitor pool that self-renews and produce
199 The emergence of horizontal basal cells, a multipotent progenitor population in the adult epitheliu
200 ferentiation of globose basal cells, another multipotent progenitor population in the adult OE, is al
201 l crest evolved through the acquisition of a multipotent progenitor regulatory state upstream of mult
202 ey transit into short-term self-renewing and multipotent progenitor states, with the first major line
203 multipotent progenitor-2 and lymphoid-primed multipotent progenitor, respectively, found in cord bloo
204 A(hi)]) that were functionally equivalent to multipotent progenitor-2 and lymphoid-primed multipotent
206 l analysis, was used to evaluate and compare multipotent progenitors (MPPs) and common lymphoid proge
207 ction, lineage specification at the level of multipotent progenitors (MPPs) remains poorly understood
208 s of the composition of HSCs and HSC-derived multipotent progenitors (MPPs) revealed a significantly
209 cells (HSCs), short-term HSCs (ST-HSCs), and multipotent progenitors (MPPs) were all significantly re
212 tion, we find that homing of lymphoid-primed multipotent progenitors and common lymphoid progenitors
213 finding that short-term HSCs and downstream multipotent progenitors are potent and biologically rele
215 ensure precision in lineage specification as multipotent progenitors become restricted in cell fate.
216 of the early B cell factor 1 (Ebf1) locus in multipotent progenitors determines this lineage choice.
217 ferentiation recapitulates the generation of multipotent progenitors during embryonic development, wh
220 We further show that adenosine increased multipotent progenitors in a mouse embryonic stem cell c
221 ecific progenitors, we identify bipotent and multipotent progenitors in ducts and TDLUs, respectively
226 of AML LSC resembling either lymphoid-primed multipotent progenitors or granulocyte/macrophage progen
229 ferentiation of lineage-committed cells from multipotent progenitors requires the establishment of ac
230 forming haematopoietic stem cells (HSCs) and multipotent progenitors results in more aggressive AML t
232 gest that canine hemangiosarcomas arise from multipotent progenitors that differentiate into distinct
233 layer was damaged, MG respond by generating multipotent progenitors that migrate to all nuclear laye
234 nation of key transcription factors (TFs) in multipotent progenitors that transition them away from o
235 6 deficiency increased the ratio of Flt3(hi) multipotent progenitors to CD150(+) stem cells in the mo
236 he mechanisms underlying the transition from multipotent progenitors to distinct muscle precursors re
237 of the trunk domain in the pancreas causing multipotent progenitors to lose acinar, while gaining en
240 es, implying that cells in the CE domain are multipotent progenitors, and suggesting that an asymmetr
241 lts identified CLPs, and not lymphoid-primed multipotent progenitors, as the requisite CD11a-dependen
242 present with development of lymphoid-primed multipotent progenitors, common lymphoid progenitors and
243 first major lineage commitment occurring in multipotent progenitors, thus giving rise to progenitors
250 differentiated cells to dedifferentiate into multipotent proliferative cells with the capacity to reg
251 adult sheath tissues possess clonogenic and multipotent properties comparable to those of stem/proge
252 clear whether mesoderm induction generates a multipotent PS progenitor or several distinct ones with
253 diate progenitors (IPs) are derived from the multipotent radial glia (RGs) and serve as the direct pr
254 teraction in the regulation of Six2-positive multipotent renal progenitor cells and formation of the
255 es, retinal ganglion cells (RGCs) arise from multipotent retinal progenitor cells (RPCs), and their f
257 ows that, whereas the prostate develops from multipotent SCs, only unipotent SCs mediate mammary glan
258 ed stem cell-like cells (iMuSCs) displayed a multipotent state with sensitiveness and strong migratio
260 maintains eye field neural progenitors in a multipotent state; then, in combination with Pax6, Tbx3
265 ferentiation of white adipose tissue-derived multipotent stem cells (ADMSCs) into lipid-accumulating,
267 e of soluble cues directs differentiation of multipotent stem cells into discrete populations of spec
268 directed differentiation of pluripotent and multipotent stem cells into mesodiencephalic dopaminergi
269 uring the development and differentiation of multipotent stem cells into specialised cell types remai
272 rise via a hierarchical scheme starting with multipotent stem cells that become increasingly restrict
273 e neural crest is an embryonic population of multipotent stem cells that form numerous defining featu
274 opoietic stem cells (HSCs) are self-renewing multipotent stem cells that generate mature blood lineag
275 ne neural progenitor cells (NPCs), which are multipotent stem cells that give rise to cells in the ce
276 C-MSCs), originating in Wharton's jelly, are multipotent stem cells that home to damaged tissues and
278 vital for the differentiation of ES cells to multipotent stem cells, little is known regarding the ro
283 y gland evokes cell dedifferentiation into a multipotent stem-like state, suggesting this to be a mec
284 mouse of a previously unknown population of multipotent stem/progenitor cells that are capable of no
285 neuronal precursors to dedifferentiate into multipotent stem/progenitor cells that contribute signif
288 ast cancer cell fusion with mesenchymal stem/multipotent stromal cells (MSCs) involves apoptosis.
291 arrow stromal cells (BMSCs), a major type of multipotent stromal cells, produces pain relief (antihyp
295 the current view of endogenous pericytes as multipotent tissue-resident progenitors and suggest that
296 o function as mesenchymal stem cells (MSCs), multipotent tissue-resident progenitors with great poten
297 ults indicate that HSCs are not sufficiently multipotent to produce hepatocytes, cholangiocytes, or o
298 m-like cells represent poorly differentiated multipotent tumor-propagating cells that contribute disp
299 animal stem cell niches, maintain a pool of multipotent, undifferentiated cells that divide and diff
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