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1 tors in the injured retina are intrinsically multipotent.
2 actility (seeded on 2 mum gratings) remained multipotent.
3 ges, a large fraction of the progenitors are multipotent.
4  that individual neural crest precursors are multipotent.
5 hat at the population level Cux2(+) RGCs are multipotent.
6 ells: they are self-renewing, clonogenic and multipotent.
7 press the neural crest marker p75 and become multipotent.
8 ge/brown markers, suggesting the presence of multipotent adipogenic precursor cells.
9          There was no difference between the multipotent adult progenitor cell group and placebo grou
10 n the groups (64 [99%] of 65 patients in the multipotent adult progenitor cell group vs 59 [97%] of 6
11  score of 8-20 to treatment with intravenous multipotent adult progenitor cells (400 million or 1200
12                 In vitro, we show that human multipotent adult progenitor cells (MAPCs) have the abil
13 ations consisted of 65 patients who received multipotent adult progenitor cells and 61 patients who r
14 tients were randomly assigned (67 to receive multipotent adult progenitor cells and 62 to receive pla
15                                              Multipotent adult progenitor cells are a bone marrow-der
16 rolled, dose-escalation trial of intravenous multipotent adult progenitor cells in 33 centres in the
17 ved at 90 days in neurological outcomes with multipotent adult progenitor cells treatment, further cl
18            INTERPRETATION: Administration of multipotent adult progenitor cells was safe and well tol
19 t, well-tolerated, and safest single dose of multipotent adult progenitor cells, and if they were eff
20 potent murine cells spontaneously convert to multipotent adult spermatogonial-derived stem cells (MAS
21                              Basal cells are multipotent airway progenitors that generate distinct ep
22                      Berberine is an ancient multipotent alkaloid drug which derived from Coptis chin
23 tted cells are molecularly distinct, whereas multipotent and bipotent progenitors do not exhibit diff
24         While some progenitors are bona fide multipotent and contribute progeny to all major pancreat
25 ic stem cells and their differentiation into multipotent and downstream lymphoid-biased progenitors.
26                 Cardiac progenitor cells are multipotent and give rise to cardiac endothelium, smooth
27  post-embryonic retinal NSCs are exclusively multipotent and give rise to the complete spectrum of ce
28 ve also uncovered the presence of a range of multipotent and lineage-restricted progenitor cells in t
29  development and adulthood, is regarded as a multipotent and neural stem cell marker.
30 ude that pro-centrioles/pro-basal bodies are multipotent and not committed to form either a 9+2 or 9+
31          Endothelial markers colocalize with multipotent and osteogenic markers in calcified arteries
32                         Adult stem cells are multipotent and persist in small numbers in adult tissue
33                                              Multipotent and pluripotent stem cells are potential sou
34 ene silencing and variegation in cell lines, multipotent and pluripotent stem cells, and their differ
35 ng of viral and tissue-specific promoters in multipotent and pluripotent stem cells.
36 le for the efficient genetic modification of multipotent and pluripotent stem cells.
37 onal and population levels Fezf2(+) RGCs are multipotent and that at the population level Cux2(+) RGC
38 ad, only two progenitor classes predominate, multipotent and unipotent, with Er-Mk lineages emerging
39    Therefore, they are hereby termed induced multipotent cancer cells.
40 stem cell-like ability to self-renew and the multipotent capacity to reconstitute the entire spectrum
41           We have identified a population of multipotent cardiopulmonary mesoderm progenitors (CPPs)
42 hancers that become epigenetically primed in multipotent cardiovascular mesoderm, to regulate the div
43              The epicardium has emerged as a multipotent cardiovascular progenitor source with therap
44 ese findings support ex vivo fucosylation of multipotent CD34(+) CB cells as a clinically feasible me
45   In humans, mast cells can be cultured from multipotent CD34(+) progenitor cells.
46 ethod that generated neural derivatives with multipotent cell fate potential and normal karyotype.
47 e, thus resulting in a rapid decrease in the multipotent cell population and compromising their regen
48  The neural crest (NC) is a highly migratory multipotent cell population that forms at the interface
49          The neural crest is a migratory and multipotent cell population that plays a crucial role in
50 evelopment, the glycoprotein 2 (GP2) marks a multipotent cell population that will differentiate into
51 ine lineage-specification program within the multipotent cell population.
52 eneration through paracrine effects and as a multipotent cell source, and has received recent attenti
53 duced cell plasticity occurs via a transient multipotent cell state and that concomitant exposure to
54 eals adult pancreatic duct cells as a latent multipotent cell type.
55 ur cytokine priming, increased the number of multipotent cells (CD34+CD90+) generated; however, the d
56 both the sequence of lineage choices made by multipotent cells and to identify the genes influencing
57              Perturbation of this balance in multipotent cells of the dermomyotome influences cell fa
58 enitors, but its ability to act on upstream, multipotent cells remains to be established.
59             The neural crest (NC) represents multipotent cells that arise at the interphase between e
60        Solid malignancies contain subsets of multipotent cells that grow as spheres and efficiently p
61 lexity of gene regulatory networks that lead multipotent cells to acquire different cell fates makes
62                        Neural stem cells are multipotent cells with the ability to differentiate into
63 s genes that cause them to become migratory, multipotent cells, distinguishing them from adjacent sta
64 ocrine-selective transcription in epithelial multipotent cells, nascent endocrine progenitors, and di
65            Mesenchymal stem cells (MSCs) are multipotent cells, which can give rise to variety of cel
66 e observed a biphasic distribution of Yan in multipotent cells, with a rapid inductive phase and slow
67 lonal organoid cultures derived from primary multipotent cells.
68 unipotent, with Er-Mk lineages emerging from multipotent cells.
69 on of specific lung epithelial lineages from multipotent cells.
70 that are characteristic of undifferentiated, multipotent cells.
71 product of its target gene HMGA1, encoding a multipotent chromatin modifier.
72                                     Further, multipotent compounds were discovered, characterized, an
73 ntiate hESC and hiPSC into neural-restricted multipotent derivatives or specialized cell types under
74 om exfoliated deciduous teeth (SHED) possess multipotent differentiation and immunomodulatory propert
75 ions due to their long-term self-renewal and multipotent differentiation capacities.
76 e postnatal stem cell population, possessing multipotent differentiation capacity and immunomodulator
77                  These cells were capable of multipotent differentiation in vitro, generating both ci
78 ing of the epigenetic programs necessary for multipotent differentiation of MSCs that may prove benef
79 ages and yet capable of self-replication and multipotent differentiation, being able to differentiate
80 ncer stem cells, capable of self-renewal and multipotent differentiation, influence tumor behavior th
81 ion and the balance between self-renewal and multipotent differentiation.
82 ives and their pharmacological evaluation as multipotent drugs for the treatment of Alzheimer's disea
83             The neural crest is a transient, multipotent embryonic cell population in vertebrates giv
84 dels can be developed in situ from different multipotent embryonic cerebellar progenitor cells via co
85              Here we show that mouse Sox9(+) multipotent embryonic lung progenitors can be isolated a
86          Hepatocyte growth factor (HGF) is a multipotent endogenous repair factor secreted primarily
87                                              Multipotent epithelial cells with high Aldehyde dehydrog
88 ose tissue originates from the epicardium, a multipotent epithelium that until now is only establishe
89 eveal the births of larval astrocytes from a multipotent glial lineage, their allocation to reproduci
90 is driven by the action of a small number of multipotent haematopoietic stem cells.
91                                          The multipotent hematopoietic cell line EML has emerged as a
92 EML cell line suggests that interconvertible multipotent hematopoietic cell subsets coexist in a home
93 the heterogeneity and interconvertibility of multipotent hematopoietic cells.
94 x/flox):Scl-Cre-ER(T)) and demonstrated that multipotent hematopoietic colonies form despite the abse
95 ne receptor CCR9 controls the immigration of multipotent hematopoietic progenitor cells into the thym
96 ents diagnosed with BCR-ABL1-positive ALL, a multipotent hematopoietic progenitor is affected by the
97                  Ddb1 is highly expressed in multipotent hematopoietic progenitors and its deletion l
98 on of different lineages of blood cells from multipotent hematopoietic stem cells.
99        We show that intrathymic injection of multipotent hematopoietic stem/progenitor cells into irr
100 , hepatoblasts (rHBs), two lineage stages of multipotent, hepatic precursors with overlapping but als
101  engraftment potential but failed to produce multipotent HPCs and lineage-defined blood cells.
102 ll population that shares many features with multipotent HSCs and serves as a lineage-restricted emer
103                 The concept of self-renewing multipotent HSCs was born, but accompanied by perplexing
104 le scaffolds capable of supporting growth of multipotent human bone marrow mesenchymal stem cells (hM
105                 Recently, a subpopulation of multipotent human luminal cells defined by CD26 expressi
106                                    Isolating multipotent human NC has proven challenging, limiting ou
107 eloid lineages suggesting the involvement of multipotent, immature hematopoietic cells in the pathoph
108 scribe a protocol to generate expandable and multipotent induced cardiac progenitor cells (iCPCs) fro
109  After in vitro enzymatic inhibition assays, multipotent inhibitors showing potencies in the nanomola
110                         We documented stable multipotent long-term hematopoietic clonal output of mon
111                                              Multipotent long-term repopulating hematopoietic stem ce
112 onal pioneering during B cell programming of multipotent lymphoid progenitors by restricting chromati
113           Our findings provide evidence of a multipotent lymphomyeloid HSC origin of SF3B1 mutations
114                        The identification of multipotent mammary stem cells (MaSCs) has provided an e
115 es (MDS), we establish the existence of rare multipotent MDS stem cells (MDS-SCs), and their hierarch
116  memory formation depends on elucidating how multipotent memory precursor (MP) cells maintain develop
117  to H19 in the cytoplasm of undifferentiated multipotent mesenchymal C2C12 cells, and this interactio
118 ess and application of mechanical stretch to multipotent mesenchymal cells stimulated the nuclear tra
119                        During nephrogenesis, multipotent mesenchymal nephron progenitors develop into
120 yoblasts in embryos but is also expressed in multipotent mesenchymal progenitors.
121                                              Multipotent mesenchymal stem (stromal) cells (MSCs) have
122                                              Multipotent mesenchymal stem cells (MSCs) promise a ther
123 omplexes to direct the fate determination of multipotent mesenchymal stem cells (MSCs).
124                         Glial cells generate multipotent mesenchymal stem cells that produce pulp cel
125 ation consisting of mature white adipocytes, multipotent mesenchymal stem cells, committed progenitor
126 ly responsible for the beneficial effects of multipotent mesenchymal stem cells, for example in the t
127 llicular thyroid cancers and the presence of multipotent mesenchymal stem/stromal cells (MSCs) in non
128                     Advances in the field of Multipotent Mesenchymal Stromal cell (MSC) biology have
129 imum (estimate+/-SE, 0.01+/-0.002; P<0.001), multipotent mesenchymal stromal cell colony maximum (est
130 differentiation were assessed in bone marrow multipotent mesenchymal stromal cell models.
131                                        Human multipotent mesenchymal stromal cells (hMSCs) produce tu
132 ce, we demonstrate expression exclusively in multipotent mesenchymal stromal cells (MSCs) in the bone
133                                              Multipotent mesenchymal stromal cells (MSCs) possess rep
134 oduce therapeutically relevant quantities of multipotent mesenchymal stromal cells (MSCs) via in vitr
135                        Presentations covered multipotent (mesenchymal and hematopoietic) and pluripot
136 opriate cell fate decisions to occur in this multipotent mesoderm lineage.
137 omprise the head and face are derived from a multipotent migratory progenitor cell population called
138                                        Human multipotent muscle derived stem cells (MDSC) can be a po
139               In this study, we surveyed the multipotent myogenic B7.5 lineage in Molgula spp.
140 (ESC)-based model yields high proportions of multipotent NC cells (expressing SOX10, PAX7 and TFAP2A)
141 vival, differentiation, and migration of the multipotent NCCs critical for mammalian cardiovascular d
142  with the appearance of highly patterned and multipotent NCCs in stem vertebrates.
143 sion that both Fezf2(+) and Cux2(+) RGCs are multipotent neocortical progenitors.
144                During embryonic development, multipotent neural crest cells are specified at the late
145 ipheral nervous system (PNS), originate from multipotent neural crest cells that also give rise to ot
146 ved skilled forelimb function after grafting multipotent neural progenitor cells into sites of SCI.
147                    In the developing retina, multipotent neural progenitors undergo unidirectional di
148 involving permanently labeled cells revealed multipotent neural stem cells (NSCs) of embryonic origin
149 epithelial anterior PS cells are Sox2(+)T(+) multipotent NMPs and form the bulk of neural progenitors
150 or the existence of a sortable population of multipotent non-epithelial cells in the adult pancreas t
151 Herein, we studied the effects of ETH on rat multipotent NSC proliferation and neuronal differentiati
152 oma function in vivo, which is mediated by a multipotent NT5E(+) (CD73)(+) ENG(-) (CD105)(-) LY6A(+)
153 monstrates that BMI1 is expressed in vivo by multipotent OE progenitors, validating our culture model
154 s do not require transit through a requisite multipotent or bipotent megakaryocyte-erythrocyte progen
155 ells produce all cells of an organism, while multipotent or unipotent stem cells regenerate only spec
156 c exocrine and endocrine lineages arise from multipotent pancreatic progenitor cells (MPCs).
157 ping pancreata, indicating that it marks the multipotent pancreatic progenitors in vivo.
158  cell fate, before migration to the limb, in multipotent Pax3(+) cells in the somite of the mouse emb
159                                              Multipotent Pax3-positive (Pax3(+)) cells in the somites
160          SMC-derived AdvSca1 cells exhibit a multipotent phenotype capable of differentiating in vivo
161             The neural crest is a transient, multipotent population of cells that arises at the borde
162  Omental adipose stromal cells (O-ASC) are a multipotent population of mesenchymal stem cells contain
163 e vertebrate body and differentiation of the multipotent posterior progenitor cells to the muscle cel
164 ther types of ILC2, MPP(type2) cells exhibit multipotent potential and do not express T1/ST2 or IL-7R
165 hich have been proposed to arise either from multipotent precursor cells or pools of heterogeneous, r
166 ng interneuron allocation from telencephalic multipotent precursors are poorly understood.
167 l checkpoints that dictate the commitment of multipotent precursors to the T cell lineage and their s
168                                 In prethymic multipotent precursors, however, titration of GATA-3 act
169 transition from naive/primed pluripotency to multipotent primary neural progenitor cells (NPCs).
170 ing of changes with age in the heterogeneous multipotent progenitor (MPP) cell compartment, which is
171 rease in hematopoietic stem cells (HSCs) and multipotent progenitor (MPP) cells in conditional Runx1-
172 CL12, but whether a separate niche instructs multipotent progenitor (MPP) differentiation remains unc
173 al progenitors and PMPs derive from a common multipotent progenitor called the neuromesodermal progen
174 mming to pluripotency to generate an induced multipotent progenitor cell (iMPC) state from which endo
175                     Within the epithelium, a multipotent progenitor cell (MPC) population is specifie
176 thelium, for the subsequent formation of the multipotent progenitor cell (MPC) population that gives
177 s the effects of the loss of Dok proteins on multipotent progenitor cell cycle.
178               The stem cell factor-dependent multipotent progenitor cell line HPC-7 represents a well
179  of JMML using mice that express KrasG12D in multipotent progenitor cells (Flt3Cre+ KrasG12D mice).
180 letion leads to a reduced pool of pancreatic multipotent progenitor cells (MPCs) due to decreased pro
181 raftment of human PSC-derived (hPSC-derived) multipotent progenitor cells (MPP) has hampered the clin
182  functional and immunophenotypic features of multipotent progenitor cells (MPPs).
183  roles in the migration and specification of multipotent progenitor cells at the onset of cardiogenes
184 capacity to predict and control when and how multipotent progenitor cells commit to the desired cell
185 e of differentiated tissue-specific stem and multipotent progenitor cells for regenerative medicine a
186 ) mice, and the frequency of lymphoid-primed multipotent progenitor cells in bone marrow was reduced.
187 ke receptor stimulation, short-term HSCs and multipotent progenitor cells produce copious amounts of
188                                     Stem and multipotent progenitor cells show scant H3K4me2 marking
189           Cell fate choice and commitment of multipotent progenitor cells to a differentiated lineage
190 , the biological secretome of Amnion-derived Multipotent Progenitor cells, contains multiple anti-inf
191 monstrates the persistence of stem cells and multipotent progenitor cells, their nature in the OE rem
192 tion and asymmetric cell division to produce multipotent progenitor cells.
193 but that the marrow contained 3-5 times more multipotent progenitor cells.
194  their order; the final mutations occur in a multipotent progenitor derived from the preleukemic HSC
195 tiation and that inhibition of Ezh2 promotes multipotent progenitor expansion.
196                           The existence of a multipotent progenitor for all anatomic and cellular com
197  epithelium of the branching mouse lung is a multipotent progenitor pool that self-renews and produce
198  the expansion had shifted to the phenotypic multipotent progenitor population by 1 year.
199   The emergence of horizontal basal cells, a multipotent progenitor population in the adult epitheliu
200 ferentiation of globose basal cells, another multipotent progenitor population in the adult OE, is al
201 l crest evolved through the acquisition of a multipotent progenitor regulatory state upstream of mult
202 ey transit into short-term self-renewing and multipotent progenitor states, with the first major line
203 multipotent progenitor-2 and lymphoid-primed multipotent progenitor, respectively, found in cord bloo
204 A(hi)]) that were functionally equivalent to multipotent progenitor-2 and lymphoid-primed multipotent
205                                 However, how multipotent progenitors (MPP) switch into common lymphoi
206 l analysis, was used to evaluate and compare multipotent progenitors (MPPs) and common lymphoid proge
207 ction, lineage specification at the level of multipotent progenitors (MPPs) remains poorly understood
208 s of the composition of HSCs and HSC-derived multipotent progenitors (MPPs) revealed a significantly
209 cells (HSCs), short-term HSCs (ST-HSCs), and multipotent progenitors (MPPs) were all significantly re
210                        Endogenous pancreatic multipotent progenitors (PMPs) are ideal candidates for
211 ed depletion of hematopoietic stem cells and multipotent progenitors across all subtypes.
212 tion, we find that homing of lymphoid-primed multipotent progenitors and common lymphoid progenitors
213  finding that short-term HSCs and downstream multipotent progenitors are potent and biologically rele
214 opoietic precursors with IL-1, we found that multipotent progenitors are targets of IL-1.
215 ensure precision in lineage specification as multipotent progenitors become restricted in cell fate.
216 of the early B cell factor 1 (Ebf1) locus in multipotent progenitors determines this lineage choice.
217 ferentiation recapitulates the generation of multipotent progenitors during embryonic development, wh
218 driving the expansion and maintenance of the multipotent progenitors during nephrogenesis.
219                                        These multipotent progenitors have a high proliferation abilit
220     We further show that adenosine increased multipotent progenitors in a mouse embryonic stem cell c
221 ecific progenitors, we identify bipotent and multipotent progenitors in ducts and TDLUs, respectively
222            An increase in the cellularity of multipotent progenitors is observed in young Dok1/Dok2-d
223              How these cell types arise from multipotent progenitors is poorly understood.
224           Apoptosis was markedly elevated in multipotent progenitors lacking RECQL4 compared with WT
225                                        These multipotent progenitors of the spleen (MPPS) develop fro
226 of AML LSC resembling either lymphoid-primed multipotent progenitors or granulocyte/macrophage progen
227                                        These multipotent progenitors originate from Eya1-expressing o
228                   During T cell development, multipotent progenitors relinquish competence for other
229 ferentiation of lineage-committed cells from multipotent progenitors requires the establishment of ac
230 forming haematopoietic stem cells (HSCs) and multipotent progenitors results in more aggressive AML t
231 ort-term hematopoietic stem cells (HSCs) and multipotent progenitors than controls.
232 gest that canine hemangiosarcomas arise from multipotent progenitors that differentiate into distinct
233  layer was damaged, MG respond by generating multipotent progenitors that migrate to all nuclear laye
234 nation of key transcription factors (TFs) in multipotent progenitors that transition them away from o
235 6 deficiency increased the ratio of Flt3(hi) multipotent progenitors to CD150(+) stem cells in the mo
236 he mechanisms underlying the transition from multipotent progenitors to distinct muscle precursors re
237  of the trunk domain in the pancreas causing multipotent progenitors to lose acinar, while gaining en
238                      T cell development from multipotent progenitors to specialized effector subsets
239                    Two important TFs for the multipotent progenitors to T lineage transition are RUNX
240 es, implying that cells in the CE domain are multipotent progenitors, and suggesting that an asymmetr
241 lts identified CLPs, and not lymphoid-primed multipotent progenitors, as the requisite CD11a-dependen
242  present with development of lymphoid-primed multipotent progenitors, common lymphoid progenitors and
243  first major lineage commitment occurring in multipotent progenitors, thus giving rise to progenitors
244 e for the directed homing mechanism of these multipotent progenitors.
245 phoid progenitors (CLPs) and lymphoid-primed multipotent progenitors.
246 -erythrocyte progenitors and lymphoid-primed multipotent progenitors.
247  roles in inducing cell differentiation from multipotent progenitors.
248 endent on Runx1, a factor already present in multipotent progenitors.
249  sea star larvae begins with soxc-expressing multipotent progenitors.
250 differentiated cells to dedifferentiate into multipotent proliferative cells with the capacity to reg
251  adult sheath tissues possess clonogenic and multipotent properties comparable to those of stem/proge
252 clear whether mesoderm induction generates a multipotent PS progenitor or several distinct ones with
253 diate progenitors (IPs) are derived from the multipotent radial glia (RGs) and serve as the direct pr
254 teraction in the regulation of Six2-positive multipotent renal progenitor cells and formation of the
255 es, retinal ganglion cells (RGCs) arise from multipotent retinal progenitor cells (RPCs), and their f
256                                          How multipotent retinal progenitors know when to switch from
257 ows that, whereas the prostate develops from multipotent SCs, only unipotent SCs mediate mammary glan
258 ed stem cell-like cells (iMuSCs) displayed a multipotent state with sensitiveness and strong migratio
259 le for maintaining Drosophila eye cells in a multipotent state.
260  maintains eye field neural progenitors in a multipotent state; then, in combination with Pax6, Tbx3
261 imary somatic cells into tissue-regenerative multipotent stem (iMS) cells.
262 ression was restricted to the most immature, multipotent stem and progenitor populations.
263 er gene expression, and differentiation of a multipotent stem cell line.
264             The vertebrate body forms from a multipotent stem cell-like progenitor population that pr
265 ferentiation of white adipose tissue-derived multipotent stem cells (ADMSCs) into lipid-accumulating,
266               A challenge has been to obtain multipotent stem cells and/or progenitors that can gener
267 e of soluble cues directs differentiation of multipotent stem cells into discrete populations of spec
268  directed differentiation of pluripotent and multipotent stem cells into mesodiencephalic dopaminergi
269 uring the development and differentiation of multipotent stem cells into specialised cell types remai
270                  Terminal differentiation of multipotent stem cells is achieved through a coordinated
271                        T-IPs did not include multipotent stem cells or molecular evidence of T cell-r
272 rise via a hierarchical scheme starting with multipotent stem cells that become increasingly restrict
273 e neural crest is an embryonic population of multipotent stem cells that form numerous defining featu
274 opoietic stem cells (HSCs) are self-renewing multipotent stem cells that generate mature blood lineag
275 ne neural progenitor cells (NPCs), which are multipotent stem cells that give rise to cells in the ce
276 C-MSCs), originating in Wharton's jelly, are multipotent stem cells that home to damaged tissues and
277                                              Multipotent stem cells with neural crest-like properties
278 vital for the differentiation of ES cells to multipotent stem cells, little is known regarding the ro
279  DGCR8 to reprogram adult somatic cells into multipotent stem cells.
280 em cells and may play similar roles in other multipotent stem cells.
281 s robust identification and isolation of the multipotent stem cells.
282  broadly permissive chromatin established in multipotent stem cells.
283 y gland evokes cell dedifferentiation into a multipotent stem-like state, suggesting this to be a mec
284  mouse of a previously unknown population of multipotent stem/progenitor cells that are capable of no
285  neuronal precursors to dedifferentiate into multipotent stem/progenitor cells that contribute signif
286  prevent neurons from dedifferentiating to a multipotent, stem cell-like state.
287                                              Multipotent stromal cells (MSCs) derived from bone marro
288 ast cancer cell fusion with mesenchymal stem/multipotent stromal cells (MSCs) involves apoptosis.
289            Mesenchymal stem cells (MSCs) are multipotent stromal cells that exist in many tissues and
290            Mesenchymal stem cells (MSCs) are multipotent stromal cells within the bone marrow.
291 arrow stromal cells (BMSCs), a major type of multipotent stromal cells, produces pain relief (antihyp
292 ical markers that together are predictive of multipotent subpopulations, in vitro and in vivo.
293                   Long-lived, self-renewing, multipotent T memory stem cells (TSCM) can trigger profo
294                                              Multipotent Tet2(-/-);Flt3(ITD) progenitors (LSK CD48(+)
295  the current view of endogenous pericytes as multipotent tissue-resident progenitors and suggest that
296 o function as mesenchymal stem cells (MSCs), multipotent tissue-resident progenitors with great poten
297 ults indicate that HSCs are not sufficiently multipotent to produce hepatocytes, cholangiocytes, or o
298 m-like cells represent poorly differentiated multipotent tumor-propagating cells that contribute disp
299  animal stem cell niches, maintain a pool of multipotent, undifferentiated cells that divide and diff
300                         These HSCs are fully multipotent, yet they display both higher lymphoid cell

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