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1 ired for subsequent sorting of Arn1 into the multivesicular body.
2 radation of Smf1p by trafficking through the multivesicular body.
3 , or not required for protein sorting at the multivesicular body.
4 ing into intraluminal vesicles (ILVs) of the multivesicular body.
5 ng, including defects in sorting through the multivesicular body.
6 the accumulation of the uroplakin-degrading multivesicular bodies.
7 such as microdomain-dependent biogenesis of multivesicular bodies.
8 port (ESCRT) complexes 0, I, II and III into multivesicular bodies.
9 both retrogradely transported to the soma in multivesicular bodies.
10 Golgi phosphoprotein of 130 kDa (GPP130) to multivesicular bodies.
11 xosomes and could originate from cytoplasmic multivesicular bodies.
12 posed of 50- to 80-nm exosomes released from multivesicular bodies.
13 g and scission of intralumenal vesicles into multivesicular bodies.
14 d, sciatic nerve and brain, but no excess of multivesicular bodies.
15 omes, including late endosomes/lysosomes and multivesicular bodies.
16 rnae, accumulation of abnormal lysosomes and multivesicular bodies.
17 ation of cargo into intraluminal vesicles of multivesicular bodies.
18 helial cells, infected AmEpCs made dispersed multivesicular bodies.
19 te and help generate the luminal vesicles of multivesicular bodies.
20 e phagosome through the endocytic network to multivesicular bodies.
21 uman immunodeficiency virus (HIV) virions in multivesicular bodies.
22 of host proteins into endosomal vesicles and multivesicular bodies.
23 ittle internalized PAM-1/OSX was detected in multivesicular bodies.
24 which normally function in the formation of multivesicular bodies.
25 mbrane ruffles and subsequently processed to multivesicular bodies.
26 required to incorporate late endosomes into multivesicular bodies.
27 ontrolling the morphogenesis and function of multivesicular bodies.
28 al compartments and for protein sorting into multivesicular bodies.
29 smembrane proteins into internal vesicles of multivesicular bodies.
30 n sorting ubiquitylated protein cargoes into multivesicular bodies.
31 ease of endosomal intraluminal vesicles into multivesicular bodies.
32 IL-1beta was detected within LAMP-1-positive multivesicular bodies.
33 olving endosomal budding of HAV capsids into multivesicular bodies.
34 ne at the ciliary base and not via fusion of multivesicular bodies.
35 ker of exosomes and intralumenal vesicles of multivesicular bodies.
36 ed into internal vesicles of late endosomes (multivesicular bodies), a ubiquitin-dependent event that
39 delayed sequestering of p75NTR-bound NGF in multivesicular bodies and delayed degradation in lysosom
40 is interaction eliminated Gag trafficking to multivesicular bodies and diminished HIV particle format
41 tor-interacting protein LIP5 targets AQP2 to multivesicular bodies and facilitates lysosomal degradat
43 uces a marked accumulation of cholesterol in multivesicular bodies and late endosomes, which inhibits
44 res, likely to be endosomes, and with sparse multivesicular bodies and lysosomes found in our reconst
45 tion is required for the trafficking of both multivesicular bodies and lysosomes to the LD surface du
47 NGF accumulates with a significant delay in multivesicular bodies and organelles of the degradation
48 V) are cell membranous sacs originating from multivesicular bodies and plasma membranes that facilita
49 ACA-containing vesicles, likely secreted as multivesicular bodies and presumably involved in the for
51 at the limiting membranes of late endosomes/multivesicular bodies and that Anx2 depletion is associa
52 his complex is required for the formation of multivesicular bodies and the degradation of internalize
53 ed a novel interaction between CD63-positive multivesicular bodies and the intracellular chlamydiae,
54 e vesicles produced through the formation of multivesicular bodies and their subsequent fusion with t
55 2 is involved in sorting at both the TGN and multivesicular body and that the first step can occur wi
56 sicles in late endosomal compartments called multivesicular bodies, and for the sorting of ubiquitina
57 res endosome trafficking, is associated with multivesicular bodies, and is regulated by Wnt5a through
58 arly endosomes, Rab7-positive late endosomes/multivesicular bodies, and LAMP1-positive lysosomes and
59 er of plasma membranes, recycling endosomes, multivesicular bodies, and released exosomes; 2) a commo
60 oscopy and found to be present in endosomes, multivesicular bodies, and vacuoles, all known to be end
61 oteins that are deposited into the lumens of multivesicular bodies are either sorted for lysosomal-me
63 eceptors to intra-lumenal vesicles (ILVs) of multivesicular bodies, are thought to terminate signalin
64 5, known to be required for the formation of multivesicular bodies, as a key sensor of thresholds for
65 pivotal role in receptor downregulation and multivesicular body biogenesis and is conserved from yea
66 AAA+ ATPase VPS4 plays an essential role in multivesicular body biogenesis and is thought to act by
69 udding is a key step in vesicular transport, multivesicular body biogenesis, and enveloped virus rele
70 ery functions in HIV-1 budding, cytokinesis, multivesicular body biogenesis, and other pathways, in t
71 emodeling events that accompany cytokinesis, multivesicular body biogenesis, and retrovirus budding,
72 n a number of biological processes including multivesicular body biogenesis, cytokinesis, and envelop
73 or transport (ESCRT) system is essential for multivesicular body biogenesis, in which cargo sorting i
74 ed for transport (ESCRT) are responsible for multivesicular body biogenesis, membrane abscission duri
75 le cellular pathways, including cytokinesis, multivesicular body biogenesis, repair of the plasma mem
76 rt (ESCRT) pathway remodels membranes during multivesicular body biogenesis, the abscission stage of
78 MITF stabilization caused an increase in multivesicular body biosynthesis, which in turn increase
79 ndependent of GRASP proteins, autophagy, and multivesicular bodies but involves enclosure within endo
80 calized with markers for early endosomes and multivesicular bodies but not the trans-Golgi network.
81 re initially thought to be late endosomes or multivesicular bodies, but it has since been shown that
82 fecting vacuolar trafficking to and from the multivesicular body bypassed the requirement for the ess
84 the cell, leading to particle assembly in a multivesicular body compartment and defective release of
87 ting of ubiquitinated membrane proteins into multivesicular bodies en route to lysosomes for degradat
91 ulated with aging in, the outer membranes of multivesicular bodies, especially in neuronal processes
93 is transported via the ESCRT pathway through multivesicular bodies for degradation, can also target t
94 omes, where it facilitates the generation of multivesicular bodies for TCR degradation and signal ter
96 Y trafficking, whereas VipD interferes with multivesicular body formation at the late endosome and e
101 s that originate as the internal vesicles in multivesicular bodies from every renal epithelial cell t
102 in a robust inhibition of APP transport into multivesicular bodies, further demonstrating that Gas1 n
104 e their discovery in 2001: the biogenesis of multivesicular bodies in endolysosomal sorting; the budd
105 f the protein degradation pathway, including multivesicular bodies in the axons and lysosomes within
106 drive membrane scission for trafficking into multivesicular bodies in the endocytic pathway and for t
108 re elucidated, including transport involving multivesicular body inner vesicles/tubules and exocytosi
109 ed Wnt signaling by increasing the number of multivesicular bodies into which the Wnt signalosome/des
110 rade signaling through Pincher-generated Trk-multivesicular bodies is distinctively refractory to sig
111 d membrane proteins into lumenal vesicles of multivesicular bodies is mediated by the Endosomal Sorti
112 it: 1) storage of exosomal FasL and TRAIL in multivesicular bodies is protected from proteolytic clea
114 rafficking of internalized Ag to specialized multivesicular bodies known as MHC class II compartments
115 ough the early endosome to the late endosome/multivesicular body (LE/MVB) does not change, but exitin
116 eration of peptide-MHC class II complexes in multivesicular body-like MIIC compartments of B cells.
117 se other organelles such as mitochondria and multivesicular bodies located in close proximity to the
119 Rab7 leads to gross morphological changes of multivesicular bodies, lysosomes, and autophagosomes, co
120 ial urothelial cells, including increases in multivesicular bodies, lysosomes, and expression of the
123 tein Shrub has a central role in endosome-to-multivesicular body membrane trafficking, with synaptic
124 mice revealed a reduction in mature type-II multivesicular bodies (MVB II) and an accumulation of la
125 AA-ATPase, Vps4 is important for function of multivesicular bodies (MVB) sorting pathway, which invol
126 rk, plasma membrane, apoplast, late endosome/multivesicular bodies (MVB), transitory late endosome/ t
129 h likely takes place on the membranes of the multivesicular bodies (MVB)/late endosomal compartments.
130 s involving membrane invagination, including multivesicular body (MVB) biogenesis, viral budding, and
134 te membrane exvagination processes including multivesicular body (MVB) formation, enveloped virus bud
137 rocess appeared to be blocked at the step of multivesicular body (MVB) fusion with the vacuolar membr
138 e sorting of transmembrane proteins into the multivesicular body (MVB) internal vesicles requires the
141 additional function of targeting CD4 to the multivesicular body (MVB) pathway for eventual delivery
144 of ubiquitinated cargo transport through the multivesicular body (MVB) pathway using a dominant negat
145 ns is a signal for active inclusion into the Multivesicular Body (MVB) pathway, resulting in lysosoma
152 27 is a component of ESCRT-0 involved in the multivesicular body (MVB) sorting pathway during endocyt
158 mplex, which is required for function of the multivesicular body (MVB), an endosomal structure that f
159 HIV budding and in vesicle formation at the multivesicular body (MVB), where they interact with othe
160 hologous endosomal NHE Nhx1 is important for multivesicular body (MVB)-vacuolar lysosome fusion, the
165 resides within the intraluminal vesicles of multivesicular bodies (MVBs) and inside exosomes, which
166 T-2 co-localized with HBV surface protein at multivesicular bodies (MVBs) and physically interacted w
167 e necessary for the sorting of proteins into multivesicular bodies (MVBs) and the budding of several
168 es and is required for both the formation of multivesicular bodies (MVBs) and the endocytic host cell
171 radation are sorted into lumenal vesicles of multivesicular bodies (MVBs) by the endosomal sorting co
173 cargo proteins into the lumenal vesicles of multivesicular bodies (MVBs) depends on the recruitment
174 mulates in a subset of LBPA-rich perinuclear multivesicular bodies (MVBs) distinct from those carryin
175 rich regions near/at the basolateral LIS and multivesicular bodies (MVBs) expressing early endosomal
176 pathway and targeting of internalized CD4 to multivesicular bodies (MVBs) for eventual degradation in
177 estration of the ubiquitinated receptor into multivesicular bodies (MVBs) for subsequent degradation.
178 sembly to intracellular compartments such as multivesicular bodies (MVBs) generally leads to a signif
181 idase S (Cps1p) into the luminal vesicles of multivesicular bodies (MVBs) in Saccharomyces cerevisiae
182 found that overexpressed PARK9 localized to multivesicular bodies (MVBs) in the human H4 cell line.
183 mbrane proteins into the lumenal vesicles of multivesicular bodies (MVBs) is dependent on the attachm
185 acuolar lumen, sorting endosomes mature into multivesicular bodies (MVBs) through the action of ENDOS
186 the endocytic delivery of cell surface Hh to multivesicular bodies (MVBs) via an endosomal sorting co
187 secreted vesicles arising from the fusion of multivesicular bodies (MVBs) with the plasma membrane.
188 light-sensitive rhabdomeres and localized to multivesicular bodies (MVBs) within the photoreceptor cy
189 ane, leading to the extracellular release of multivesicular bodies (MVBs), initially contained within
190 sequestration of GSK3 from the cytosol into multivesicular bodies (MVBs), so that this enzyme become
192 bset of late-endosomal compartments known as multivesicular bodies (MVBs), whose formation is control
221 suggest that nucleotide exchange of Ypt7p on multivesicular bodies (MVBs)/late endosomes must take pl
223 ng to late endosomal compartments, including multivesicular bodies, occurs prior to viral particle bu
224 with Abeta42 being prominently localized to multivesicular bodies of neurons, as shown in Alzheimer'
225 d fourth-stage larvae, the body channels and multivesicular bodies of third-stage larvae and the proc
228 ynaptic boutons demonstrated the presence of multivesicular bodies, organelles involved in the produc
229 ith the idea that HIV uses the late endosome/multivesicular body pathway during virion budding from m
230 Ubiquitinated Fpn is trafficked through the multivesicular body pathway en route to degradation in t
231 genetic interactions with components of the multivesicular body pathway in fission yeast and budding
232 independently of the function of Vps4 in the multivesicular body pathway, as dominant-negative Vps4 p
233 etion of Hrs and Tsg101, acting early in the multivesicular body pathway, retained APP in early endos
238 -8 functions in vesicle trafficking from the multivesicular body/pre-vacuolar compartment to the lyti
239 anelles indicates that, differently from the multivesicular bodies present in dendritic cells, in mon
240 from the tubulovesicular organization of the multivesicular bodies previously reported in dendritic c
241 the degradation of the ESCRT protein-charged multivesicular body protein (CHMP2B), thus generating a
244 different human ESCRT-III subunits, charged multivesicular body protein 1B (CHMP1B) and increased so
245 nd identify strong interactions with charged multivesicular body protein 1B (CHMP1B), CHMP2A, and inc
248 and cellular analyses, we identified charged multivesicular body protein 2B (CHMP2B), which is part o
249 valosin-containing protein (n = 5), charged multivesicular body protein 2B (n = 4), and linked to ch
251 1 (TSG101) and the ESCRT-III subunit charged multivesicular body protein 4b (CHMP4B) are sequentially
252 ed for transport (ESCRT)-III subunit charged multivesicular body protein 4B (CHMP4B) colocalizes and
255 e Arabidopsis thaliana ESCRT-related CHARGED MULTIVESICULAR BODY PROTEIN/CHROMATIN MODIFYING PROTEIN1
257 that the ESCRT-III subunit paralogs CHARGED MULTIVESICULAR BODY PROTEIN1 (CHMP1A) and CHMP1B are req
260 proteins are AAA(+) ATPases required to form multivesicular bodies, release viral particles, and comp
262 r (EGFR) to the intralumenal vesicles of the multivesicular body requires the coordinated action of s
263 Rab7) are minivacuoles that are competent in multivesicular body sorting and cargo degradation but re
264 tify a new component of the ESCRT-I complex, multivesicular body sorting factor of 12 kD (Mvb12), and
265 bscission and retroviral budding, but not in multivesicular body sorting of activated epidermal growt
266 y of membrane remodeling processes including multivesicular body sorting, abscission during cytokines
267 impact multiple cellular processes including multivesicular body sorting, abscission, and viral buddi
269 mes are lipid vesicles derived from cellular multivesicular bodies that are enriched in specific miRN
270 t may involve the P2X7R-induced formation of multivesicular bodies that contain exosomes with entrapp
272 nthesizing enzymes localize to intracellular multivesicular bodies that, upon stimulation, release th
273 athway functions in vesicle formation at the multivesicular body, the budding of enveloped RNA viruse
274 required for the sequestration of MHC II in multivesicular bodies, this modification is essential fo
275 and then fuse with lysosomes, endosomes and multivesicular bodies through mechanisms that involve ac
278 f resident protein and lipid constituents of multivesicular bodies to the intracellular chlamydiae.
279 is trafficked through either early endosome/multivesicular bodies to the late endosome-Golgi for sur
281 ese studies confirm CD63 as a constituent in multivesicular body-to-inclusion transport; however, oth
282 of Golgi cisternae, plasma membrane, select multivesicular bodies, tonoplast, dense intravacuolar bo
284 his trafficking pathway with an inhibitor of multivesicular body transport and the delivery of exogen
288 omparison of the morphology of intracellular multivesicular bodies, we detect changes in their distri
289 or the formation of intralumenal vesicles in multivesicular bodies, were also found to be required fo
290 IL are expressed on the limiting membrane of multivesicular bodies where, by membrane invagination, i
291 herin and HBV L protein at the intracellular multivesicular body, where the budding of HBV virions ta
292 ling was absent from the Golgi apparatus and multivesicular bodies, which are associated with protein
293 r degradation are internalized and sorted to multivesicular bodies, which fuse with lysosomes, where
294 BiP is transported to the lytic vacuole via multivesicular bodies, which represent the plant prevacu
295 tetherin colocalizes with HBV virions on the multivesicular body, which is the HBV virion budding sit
296 reen fluorescent protein-SKD1 colocalizes on multivesicular bodies with fluorescent fusion protein en
297 olgi-derived exocytic vesicles and endosomal multivesicular bodies with the bacteria-containing paras
299 into multivesicular endosomes (also known as multivesicular bodies) with subsequent fusion of the mul
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