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1 than to those of oncoretroviral RTs, such as murine leukemia virus.
2 or with the amphotropic envelope protein of murine leukemia virus.
3 (HIV-1), simian immunodeficiency virus, and murine leukemia virus.
4 Hrs had no effect on the release of Moloney murine leukemia virus.
5 nter in response to infection by the Abelson murine leukemia virus.
6 rted infection of one cell line (DEL) with a murine leukemia virus.
7 of the RNAs packaged by a model retrovirus, murine leukemia virus.
8 dimerization and packaging domain of Moloney murine leukemia virus.
9 ived growth factor (PDGF)-containing Moloney murine leukemia viruses.
11 PIM1 (Proviral Integration site for Moloney murine leukemia virus 1) has emerged as a key regulator
12 kinase proviral integration site for Moloney murine leukemia virus-1 (PIM-1), which in turn regulates
16 l protein tyrosine kinase encoded by Abelson murine leukemia virus (Ab-MLV) transforms pre-B cells.
17 ginal pipeline, can identify integrations of murine leukemia virus, adeno-associated virus, Tol2 tran
19 rupted restriction activity against N-tropic murine leukemia virus and equine infectious anemia virus
21 fective HIV-1 or Vpu(-) retroviruses such as murine leukemia virus and simian immunodeficiency virus.
23 on site pattern of XMRV with those found for murine leukemia virus and two human retroviruses, human
24 gene delivery by wild-type ecotropic Moloney murine leukemia virus and vesicular stomatitis virus (VS
25 some glycoproteins, such as those encoded by murine leukemia virus and vesicular stomatitis virus, we
26 OBEC3F (A3F), which are potent inhibitors of murine leukemia virus and Vif-deficient human immunodefi
27 ene whose overexpression blocks infection by murine leukemia viruses and human immunodeficiency virus
28 stigate to what extent virions of HERV-Kcon, murine leukemia virus, and HIV-1 have the ability to tra
29 ents derived from three retroviruses (HIV-1, murine leukemia virus, and Mason-Pfizer monkey virus), t
30 ell lines blocked cytokine induction by HIV, murine leukemia virus, and simian immunodeficiency virus
32 een XMRV and the intensively studied Moloney murine leukemia virus, architectures of the regulatory d
33 tiviruses and distinct from those of Moloney murine leukemia virus, avian sarcoma leukosis virus, and
34 mbined immunodeficiency (SCID-X1), a Moloney murine leukemia virus-based gamma-retrovirus vector expr
37 r more than 1 year after transduction with a murine leukemia virus-based vector encoding the T-cell g
39 us, MoFe2, or with the parent virus, Moloney murine leukemia virus, caused significant reduction in B
41 so suggest that the leader region of Moloney murine leukemia virus contains inhibitory/regulatory seq
42 ollowed by reverse transcription via Moloney murine leukemia virus, degradation of chromosomal DNA wi
44 se APOBEC3 does not catalyze base changes in murine leukemia virus DNA, it can be recovered from thes
48 rt of this hypothesis, infection of HIV-1 or murine leukemia virus Env (MLV-Env)-pseudotyped HIV-1 pa
49 lays a defect in proteolytic cleavage of the murine leukemia virus Env cytoplasmic tail in pseudotype
52 urface (SU) or transmembrane (TM) protein of murine leukemia virus Env, along with point mutations th
54 native viral glycoproteins, including Friend murine leukemia virus envelope (F-MLV Env) are actively
57 or-binding sequence of the ecotropic Moloney murine leukemia virus envelope glycoprotein with the pep
58 studied how the protomeric units of Moloney murine leukemia virus envelope protein (Env) are activat
59 udotyped with vesicular stomatitis virus and murine leukemia virus envelopes, indicating that defensi
60 ammaretrovirus glycoproteins, such as Friend murine leukemia virus (F-MLV) Env, but not with the rela
61 ) in the envelope glycoprotein of the Friend murine leukemia virus (F-MLV) ISD has been reported to a
62 ith defined mixtures of the ecotropic Friend murine leukemia virus (F-MuLV) and different polytropic
63 kely the functionally equivalent sequence in murine leukemia virus Gag has been inferred by mutationa
64 velopment of antitumor immunity using Friend murine leukemia virus gag-expressing mouse EL-4 (EL-4 ga
65 main inserted at random positions throughout murine leukemia virus Gag-Pol, then selecting for varian
66 ells are shown to restrict the expression of murine leukemia virus genomes but not retroviral genomes
67 ith the parental wild-type ecotropic Moloney murine leukemia virus glycoprotein through the ecotropic
69 on by retroviruses that are derived from the murine leukemia virus, human immunodeficiency virus type
70 magnetic resonance structure of the Moloney murine leukemia virus IN (M-MLV) C-terminal domain (CTD)
71 estricts N-tropic (N-MLV), but not B-tropic, murine leukemia virus in a manner dependent upon residue
73 hronically infected with the gammaretrovirus murine leukemia virus in which receptor has been downreg
76 crossover rate similar to that of HIV-1 and murine leukemia virus, indicating that the extremely hig
78 mic lymphocytes by a mink cell focus-forming murine leukemia virus induces apoptosis during the prele
79 se, encoded by the v-Abl oncogene of Abelson murine leukemia virus induces transformation of progenit
82 -5-positive (Lgr5(+)) and B lymphoma moloney murine leukemia virus insertion region homolog-1-positiv
84 is (Arabidopsis thaliana) B lymphoma Moloney murine leukemia virus insertion region1 homolog (BMI1) P
86 e the role of Bmi-1 (B-cell-specific Moloney murine leukemia virus integration site 1) as a regulator
89 profile of raltegravir on the replication of murine leukemia virus is similar to that for HIV, and th
91 ession of proviral insertion site of Moloney murine leukemia virus kinases (Pim-1, -2, and -3) in can
95 mal recessive mutation (Lamc2(jeb)) due to a murine leukemia virus long terminal repeat insertion in
96 LTR) was found to be higher than the Moloney murine leukemia virus LTRs in both LNCaP and WPMY-1 (sim
99 that gPr80gag facilitates release of Moloney murine leukemia virus (M-MuLV) from cells along an IFN-s
100 erated within the 3' terminus of the Moloney murine leukemia virus (M-MuLV) pol gene encoding the con
102 deficiency virus, type 1 (HIV-1) and Moloney murine leukemia virus (M-MuLV), we determined the effect
103 n immunodeficiency virus type 1) and Moloney murine leukemia virus (M-MuLV), we evaluated how individ
105 ock the release of HIV-1, as well as that of murine leukemia virus (MLV) and Ebola virus (EBOV); knoc
107 tion by mouse mammary tumor virus (MMTV) and murine leukemia virus (MLV) and that there are polymorph
108 the pol gene of gammaretroviruses, including murine leukemia virus (MLV) and xenotropic murine leukem
111 The envelope protein (Env) from the CasBrE murine leukemia virus (MLV) can cause acute spongiform n
113 IM proteins previously identified to inhibit murine leukemia virus (MLV) demonstrated an ability to i
115 tionship between two recent additions to the murine leukemia virus (MLV) ecotropic subgroup: Mus cerv
117 y, we found that the cytoplasmic tail of the murine leukemia virus (MLV) Env could functionally subst
118 The infectivity of HIV-1 pseudotyped with murine leukemia virus (MLV) Env was not affected by Vpu.
119 ty, we generated chimeric constructs between murine leukemia virus (MLV) Gag and HBV Core to determin
123 and Sleeping Beauty (SB) transposons and the murine leukemia virus (MLV) in mouse embryonic stem cell
125 The roles of cellular proteases in Moloney murine leukemia virus (MLV) infection were investigated
127 n 3 protein under conditions whereby Moloney murine leukemia virus (MLV) integrase failed to do so, s
128 of understanding the molecular mechanisms of murine leukemia virus (MLV) integration into host chroma
129 We found that the preintegration complex of murine leukemia virus (MLV) interacts with the dynein co
130 n A3G but is sensitive to murine A3, whereas murine leukemia virus (MLV) is relatively resistant to m
132 interferon-alpha-responsive manner, captures murine leukemia virus (MLV) particles and mediates their
133 the efficiencies of restriction of HIV-1 and murine leukemia virus (MLV) particles containing various
134 w analyzed the mRNA content of Psi- and Psi+ murine leukemia virus (MLV) particles using both microar
135 infectious anemia virus (EIAV), or N-tropic murine leukemia virus (MLV) postentry and supported late
136 rs to integrate within active genes, whereas murine leukemia virus (MLV) prefers to integrate near tr
138 binding site (PBS)-dependent restriction of murine leukemia virus (MLV) replication in embryonic ste
140 ted glycosylated Gag (glycoGag) protein of a murine leukemia virus (MLV) similarly enhance the infect
141 viruses, we engineered a fluorescent Moloney murine leukemia virus (MLV) system consisting of MLV-int
146 enhancement of the photonic inactivation of Murine Leukemia Virus (MLV) via 805 nm femtosecond pulse
149 also by vectors bearing the envelope of 10A1 murine leukemia virus (MLV), a murine retrovirus that ca
151 well as simian immunodeficiency virus (SIV), murine leukemia virus (MLV), and the retrotransposon Mus
153 oviral vectors, including those derived from murine leukemia virus (MLV), bearing heterologous Env pr
155 imers in the case of gammaretroviruses, like murine leukemia virus (MLV), led to the underestimation
156 eported to be contaminated with an ecotropic murine leukemia virus (MLV), raising questions about the
157 four different retroviruses: HIV-1, Moloney murine leukemia virus (MLV), Rous sarcoma virus (RSV), a
159 se, we used a shuttle vector system in which murine leukemia virus (MLV)-based proviral targets prese
160 ng primary human CD4(+) T lymphocytes with a murine leukemia virus (MLV)-based retroviral vector or x
161 ments in the long terminal repeats (LTRs) of murine leukemia virus (MLV)-based vectors and the vector
162 an gene therapy approaches utilize HIV-1- or murine leukemia virus (MLV)-based vectors, which prefere
163 t may be safer than that of standard Moloney murine leukemia virus (MLV)-derived retroviral vectors.
164 n immunodeficiency virus type 1 (HIV-1)- and murine leukemia virus (MLV)-derived viral vectors, respe
165 ing, are a newly appreciated viral target in murine leukemia virus (MLV)-induced neurodegeneration.
167 four subsequent reports failed to detect any murine leukemia virus (MLV)-related virus gene sequences
168 virus-related virus (XMRV) as well as other murine leukemia virus (MLV)-related viruses, though not
174 lated to the hypothetical common ancestor of murine leukemia viruses (MLVs) and other gammaretroviral
180 The generation of cytopathic effects by murine leukemia viruses (MLVs) in different cell types c
183 Members of the gammaretroviruses--such as murine leukemia viruses (MLVs), most notably XMRV [xenot
186 he 3' end processing site within the Moloney murine leukemia virus (MMLV) LTR d(TCTTTCATT), a host-gu
187 elity, we measured the error rate of Moloney murine leukemia virus (MMLV) RT in the presence of sever
188 markedly inhibits the replication of Moloney murine leukemia virus (Mo-MLV) and is required for the a
189 A crystallographic study of the Moloney murine leukemia virus (Mo-MLV) RNase H domain was perfor
191 a novel domain in the Gag protein of Moloney murine leukemia virus (MoLV) that is important for the f
192 eas the prototypical gammaretrovirus Moloney murine leukemia virus (MoMLV) favors strong enhancers an
194 oviral reverse transcriptase (RT) of Moloney murine leukemia virus (MoMLV) is expressed in the form o
195 o examine the role of this domain in Moloney murine leukemia virus (MoMLV) replication, we analyzed 1
197 ase, but not the RNase H function of Moloney Murine Leukemia Virus (MoMLV) RT and also inhibited Esch
198 h show that tetherin does not affect Moloney murine leukemia virus (MoMLV) spread, and only minimally
199 cle ultrastructure highly similar to Moloney murine leukemia virus (MoMLV), another gammaretrovirus.
201 studies with small fragments of the Moloney murine leukemia virus (MoMuLV) genome suggested that sel
203 enic temperature-sensitive mutant of Moloney murine leukemia virus (MoMuLV-ts1), results in motor neu
205 odies against clade C HIV-1 gp140, gp120, or murine leukemia virus (MuLV) gp70-scaffolded V1/V2 and t
210 from scarce, femtomole quantities of Moloney murine leukemia virus (MuLV) RNA inside authentic virion
214 on with mouse mammary tumor virus (MMTV) and murine leukemia virus (MuLV) via an adaptive immune mech
216 n the gammaretroviruses, typified by Moloney murine leukemia virus (MuLV), gag and pol are in the sam
217 opic virus 1 (Bxv1), a xenotropic endogenous murine leukemia virus (MuLV), is present in these 2 rece
220 wever, the mechanism of APOBEC inhibition of murine leukemia viruses (MuLVs) does not appear to be G-
223 nfected with mixtures of mouse retroviruses (murine leukemia viruses [MuLVs]) exhibit dramatically al
225 rs TRIM5alpha(hu) and Fv-1 restrict N-tropic murine leukemia virus (N-MLV) infection at an early post
226 capsid, we had previously selected N-tropic murine leukemia virus (N-MLV) mutants escaping from rhes
227 iction of the TRIM5alpha-sensitive, N-tropic murine leukemia virus (N-MLV) rendered HIV-1 transductio
229 The temperature-sensitive form of Abelson murine leukemia virus offers a reversible model to study
231 The infectivity of HIV-1 virions bearing murine leukemia virus or vesicular stomatitis virus glyc
232 The provirus integration site for Moloney murine leukemia virus (Pim) 1 kinase is an oncogenic ser
234 ecific proviral integration site for Moloney murine leukemia virus (PIM) kinases PIM1 and PIM2 have b
236 ous, replication-competent MMTV or into MMTV/murine leukemia virus pseudotypes had no effect on incor
237 cture of the double hairpin from the Moloney murine leukemia virus ([Psi(CD)](2), 132 nt, 42.8 kDa) u
238 neonatal infection of rats with the PVC-211 murine leukemia virus (PVC-211 MuLV) and its underlying
240 We investigated the prevalence of xenotropic murine leukemia virus-related virus (XMRV) among 293 par
241 tly, CFS has been associated with xenotropic murine leukemia virus-related virus (XMRV) as well as ot
246 tions regarding the prevalence of xenotropic murine leukemia virus-related virus (XMRV) in patients w
247 ecent study identified DNA from a xenotropic murine leukemia virus-related virus (XMRV) in peripheral
259 iency virus type-1 (HIV-1) and of xenotropic murine leukemia virus-related virus (XMRV), a gammaretro
261 (MLV)-based retroviral vector or xenotropic murine leukemia virus-related virus (XMRV), and isolated
263 DNA from a human gammaretrovirus, xenotropic murine leukemia virus-related virus (XMRV), in 68 of 101
264 described gammaretrovirus genome, xenotropic murine leukemia virus-related virus (XMRV), in prostate
265 e newly identified retrovirus-the xenotropic murine leukemia virus-related virus (XMRV)-has recently
267 d with the human retrovirus XMRV (xenotropic murine leukemia virus-related virus) can induce rare foc
269 a gammaretrovirus, termed "XMRV" (xenotropic murine leukemia virus-related virus) in prostate cancers
272 ow that a retrovirus called XMRV (xenotropic murine leukemia virus-related virus) was present in the
274 inding to Fc; the same hot-spots control HIV/murine leukemia virus restriction by TRIM5alpha and medi
275 eonatal infection of these mice with Moloney murine leukemia virus resulted in accelerated tumor onse
276 tor resistance of several commercial Moloney murine leukemia virus reverse transcriptase (MMLV RT) en
277 t to increase the thermostability of Moloney Murine Leukemia Virus reverse transcriptase (MMLV RT), w
281 Tan-1 RT enzymatically mimics oncoretroviral murine leukemia virus RT which is characterized by its l
283 l replication in vivo, I constructed a novel murine leukemia virus strain (FMLV-IL-1beta) that encode
284 , feline leukemia virus subgroup T, and 10A1 murine leukemia virus, this receptor is the human type I
286 1 with a replaced envelope gene from Moloney murine leukemia virus to allow HTLV-1 to fuse with murin
289 neurovirulent viruses FrCas(NC) and Moloney murine leukemia virus ts1 indicate that the nascent enve
290 be increased by 3- to 5-fold by placing the murine leukemia virus UAG read-through element upstream
291 icrobe, Stavrou et al. (2015) reveal how the murine leukemia virus uses a sugar-protein shield to pro
294 ffect was specific to HIV in that release of murine leukemia virus was minimally affected by the pres
295 stingly, our fusion protein did not restrict murine leukemia virus, which does not incorporate Vpr.
297 r, mouse APOBEC3 protein blocks infection by murine leukemia viruses without catalyzing this base cha
298 variant nonpermissive to XMRV and xenotropic murine leukemia virus (X-MLV) infection, suggesting that
299 iruses (MLVs), most notably XMRV [xenotropic murine leukemia virus (X-MLV)-related virus--have been r
300 g murine leukemia virus (MLV) and xenotropic murine leukemia virus (XMRV), named the CAE (cytoplasmic
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